コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 n of the overlying epidermis is an effect of epiregulin.
2 clin D2, aromatase, inhibin-alpha, SF-1, and epiregulin.
3 -1 (SF-1), cholesterol side chain (SCC), and epiregulin.
4 D2, inhibin-alpha, aromatase, SCC, SF-1, and epiregulin.
5 t Y1173 phosphorylation, but has no bias for epiregulin.
6 y, we found that the addition of recombinant epiregulin, amphiregulin, CX3CL1, and interleukin-11 sig
7 75% amino acid sequence identity with mouse epiregulin, an epidermal growth factor (EGF)-related gro
9 factor receptor (HER) ligands neuregulin and epiregulin and activation of the HER2 and HER3 receptors
10 ors that have high gene expression levels of epiregulin and amphiregulin and patients with wild-type
11 that express high levels of the EGFR ligands epiregulin and amphiregulin are more likely to have dise
12 ate-induced but not constitutive shedding of epiregulin and had no effect on betacellulin (BTC) proce
13 epidermal growth factor (EGF)-family ligands epiregulin and HB-EGF, the chemokine CX3CL1, and the tra
17 nd ANGPTL-2), as well as ErbB (amphiregulin, epiregulin, and neuregulins) and TGF-ss, which activate
19 wo additions to the family, betacellulin and epiregulin, are exquisitely restricted to the mouse uter
20 inding EGF-like growth factor, amphiregulin, epiregulin, betacellulin, or heregulin beta1 that bind t
23 jury-upregulated, low-affinity EGFR ligands (epiregulin, epigen, TGFalpha), which also require solubl
25 ied with a variety of EGF-like ligands--EGF, epiregulin (EPR), heparin binding EGF (HB-EGF), and here
26 ith amphiregulin (AR), betacellulin (BTC) or epiregulin (EPR), increased fetal type II cell different
27 sforming growth factor-alpha (TGF-alpha) and epiregulin (EPRG) were expressed as FimH fusion proteins
29 her approaches, we show how the EGFR ligands epiregulin (EREG) and epigen (EPGN) stabilize different
30 idermal growth factor receptor (EGFR) ligand epiregulin (EREG) from the basolateral to the apical cel
31 ling, we observed differential expression of epiregulin (EREG) in mouse models of colitis-associated,
36 GFR) pathway, including amphiregulin (AREG), epiregulin (EREG), and ectodomain cleavage protease MMP1
37 h factor C (VEGFC), betacellulin (BTC), EGF, epiregulin (EREG), and other members of the transforming
38 eptor (EGFR) ligands amphiregulin (AREG) and epiregulin (EREG), and systemic lipopolysaccharide admin
47 several EGFR family ligands (EGF, HB-EGF and epiregulin), however only increases in HB-EGF were detec
48 evidence that expression of betacellulin and epiregulin in the uterus requires the presence of an act
49 Taken together, these results indicate that epiregulin is a potent VSMC-secreted mitogen, induced in
51 released by mesenchymal cells, revealed more epiregulin mRNA in fibroblast-like angiofibroma and peri
54 al staining of wild type mice confirmed that epiregulin, one of the limbal epithelium-enriched genes,
55 also demonstrate that amphiregulin, but not epiregulin, partially compensates for the loss of HB-EGF
56 and beta) receptor 2, decorin, osteoglycin, epiregulin, proliferins 2 and 3, stromal cell-derived fa
58 with real-time PCR, and increased amounts of epiregulin protein were demonstrated with immunoprecipit
59 r progression and metastasis, including EREG/epiregulin, PTGS2/COX2, MMP1, MMP2, and CD44, along with
60 (epidermal growth factor), amphiregulin and epiregulin, resulting in autocrine activation of the EGF
61 ectively, our data indicate that Yap-induced Epiregulin signaling promotes the identity of SMG ductal
63 the epidermal growth factor receptor ligand epiregulin, the cyclooxygenase COX2, and the matrix meta
65 ET-1, or alpha-Thr for 1 h, induction of two epiregulin transcripts was observed, including a 4.8-kb
66 lulin, and ADAM17 as the major convertase of epiregulin, transforming growth factor alpha, amphiregul
67 Mice deficient in either amphiregulin or epiregulin, two EGFR ligands, display delayed or reduced
70 regulin, follistatin, and inhibin-beta-A and epiregulin were activated by an autocrine loop involving
71 eptor tyrosine phosphorylation stimulated by epiregulin were less than those induced by EGF or betace
72 oduction of a potent epidermal growth factor epiregulin, which may serve as a paracrine survival and
73 reduced expression of the EGF family member Epiregulin, which we show is required for the expansion