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1 f CDH1 and CDH2 expression does not indicate epithelial-mesenchymal transformation.
2 of valves and septa, is a classic example of epithelial-mesenchymal transformation.
3 arrangements that characterize renal tubular epithelial-mesenchymal transformation.
4 ptive myocytes at the time they initiate the epithelial-mesenchymal transformation.
5  the cardiac valves and form by a process of epithelial-mesenchymal transformation.
6 such as TGFbeta3, which are known to promote epithelial-mesenchymal transformation.
7  phenotypic plasticity resembling aspects of epithelial-mesenchymal transformation.
8 ium, although these cells did not undergo an epithelial/mesenchymal transformation.
9        We propose that loss of Sox9 inhibits epithelial-mesenchymal transformation after delamination
10 inase A were co-localized to sites of active epithelial-mesenchymal transformation and basal lamina d
11 ymal cells in endocardial cushions following epithelial-mesenchymal transformation and in mature valv
12 7 phosphorylation, and activates programs of epithelial-mesenchymal transformation and metastasis.
13 that neurofibromin normally acts to modulate epithelial-mesenchymal transformation and proliferation
14 on AV cushion endocardial cells to stimulate epithelial-mesenchymal transformation and that TGFbeta m
15 ation, cells in the primitive streak undergo epithelial-mesenchymal transformation and the resulting
16                               No concomitant epithelial-mesenchymal transformation could, however, be
17                    Our results indicate that epithelial-mesenchymal transformation does not occur in
18 cription factor Slug/Snai2 is a regulator of epithelial-mesenchymal transformation during development
19 differentiation of cells that have undergone epithelial-mesenchymal transformation during embryogenes
20 3258 promoted PCa cell migration by inducing epithelial mesenchymal transformation (EMT) in vitro as
21                                              Epithelial mesenchymal transformation (EMT) of the media
22             MMP-3 is known to be involved in epithelial mesenchymal transformation (EMT), and its per
23         We have previously demonstrated that epithelial-mesenchymal transformation (EMT) brings about
24                     In the developing heart, epithelial-mesenchymal transformation (EMT) in a subpopu
25 oglin and Alk5 were not directly involved in epithelial-mesenchymal transformation (EMT) in the heart
26                                Regulation of epithelial-mesenchymal transformation (EMT) is of centra
27                                              Epithelial-mesenchymal transformation (EMT) occurs durin
28                                          The epithelial-mesenchymal transformation (EMT) process invo
29 KO hearts that suggest defects in epicardial epithelial-mesenchymal transformation (EMT), a process t
30                   EC formation occurs via an epithelial-mesenchymal transformation (EMT), involving t
31 programmed process consisting of endocardial epithelial-mesenchymal transformation (EMT), mesenchymal
32 y the SNAI2 gene, has been shown to modulate epithelial-mesenchymal transformation (EMT), the convers
33                                              Epithelial-mesenchymal transformation (EMT), the process
34  of the valves and membranous septa, through epithelial-mesenchymal transformation (EMT).
35  due to decreased apoptosis and dysregulated epithelial-mesenchymal transformation (EMT).
36  and cell differentiation, which is known as epithelial-mesenchymal transformation (EMT).
37 or cells are derived from the epicardium via epithelial-mesenchymal transformation (EMT).
38 t described in developing heart valves as an epithelial mesenchymal transformation, EndMT begins in r
39       Cardiac endothelial cells that undergo epithelial-mesenchymal transformation express TBRIII, an
40 type that does not require ERK activation or epithelial-mesenchymal transformation for progression.
41 role for MMPs during a specific stage of the epithelial mesenchymal transformation in the embryonic h
42 matrix glycoprotein ES/ 130 is necessary for epithelial--mesenchymal transformation in the developing
43                Gelatinase A may also mediate epithelial-mesenchymal transformation in a paracrine man
44  treatment degraded beta-catenin and induced epithelial-mesenchymal transformation in cultured mammar
45 ocardial, are required to start and complete epithelial-mesenchymal transformation in cushion-forming
46 e gelatinase A alone is sufficient to induce epithelial-mesenchymal transformation in the absence of
47                                              Epithelial-mesenchymal transformation in the atrioventri
48 tive gelatinase A is absolutely required for epithelial-mesenchymal transformation induced by TGF-bet
49                                              Epithelial-mesenchymal transformation is a critical deve
50                                              Epithelial-mesenchymal transformation is a critical even
51              Expression of SNAI1 protein and epithelial-mesenchymal transformation markers was then a
52                                              Epithelial-mesenchymal transformation of the embryonic e
53  other causes are explored, including direct epithelial-mesenchymal transformations of the lens epith
54                                              Epithelial-mesenchymal transformation plays an important
55 ignaling, completely abolished the excessive epithelial-mesenchymal transformation seen in the absenc
56 mation of the midline seam are necessary for epithelial-mesenchymal transformation to be triggered.
57   Here we show that epicardial cells undergo epithelial-mesenchymal transformation to become coronary
58  maintained squamous differentiation whereas epithelial-mesenchymal transformation was frequent in no
59 isappear and palates become confluent due to epithelial-mesenchymal transformation, while seams remai