ライフサイエンスコーパス: epitopic

1 The epitopic and extraepitopic compensatory mutations introd

2 +) T cells were expanded by using autologous epitopic and variant peptides.

3 Both proteins were recognized by a mono-epitopic antibody raised against a peptide of GMEB-2.

4 irs were also predicted by netMHCpan to have epitopic binding.

5 ency virus replication, the relevance of the epitopic breadth of those CTL responses remains unexplor

6 Although no epitopic changes were detected under the conditions of t

7 o the NH(2)-terminal region can modulate the epitopic conformation and troponin I and tropomyosin bin

8 adaptive immunity balances specificity with epitopic coverage is a key challenge for the field, and

9 Epitopic cross-reactivity does not explain the increased

10 of full length proteins (containing multiple epitopic determinants) for presentation to T cells.

11 Allele families showed extreme epitopic differences, underscoring genetic variability o

12 ent expansion of the magnitude, breadth, and epitopic diversity of Env-specific binding antibody resp

13 is achieved through convergence on a common epitopic focus by utilizing various complementarity-dete

14 ine candidates containing IFN-gamma Inducing epitopic fragments from Leishmania major (L. major) glyc

15 se cardiomyocyte phosphoprotein with limited epitopic homology to p53.

16 Z12 elicited an integrated and multi-epitopic immune response with persistent effector T cell

17 We identified epitopic-like regions in 206 parasite proteins and prese

18 d of the selected sites were associated with epitopic mutational escapes from CTLs.

19 vumeran was well tolerated and elicited poly-epitopic neoantigen-specific responses, encompassing CD4

20 Recognition of epitopic peptide antigens presented on class I major his

21 ween TRBV7 and non-TRBV7 clonotypes and this epitopic peptide.

22 Epitopic peptides are identified from a direct compariso

23 r histocompatibility complex (MHC-I) display epitopic peptides derived from endogenous proteins on th

24 cell receptors (TCRs) or antibodies against epitopic peptides presented by class I major histocompat

25 Moreover, the HPV-6 epitopic peptides recognized by WIL differed to some ext

26 n to afford reproducible spectra that enable epitopic peptides to be identified in complex mixtures a

27 otal role in adaptive immunity by displaying epitopic peptides to CD8+ T cells.

28 i Shiga toxin subtypes viz., stx1 & stx2 via epitopic peptides.

29 ascribed to both the optimized design of its epitopic region and the superior surface interacting pro

30 alescent serum antibodies targeting the same epitopic region as these three mAbs may function coopera

31 led a unique P376S mutation within the F-4-2 epitopic region of the F gene of one Pakistani virus, wh

32 to bind to each of 2 sites within the Lym-1 epitopic region, have been linked to generate small (<2

33 The nine epitopic regions analyzed were also preserved so that, w

34 udy subjects, with a median of 14 individual epitopic regions targeted per person (range, 2 to 42), a

35 In these subjects, the median number of epitopic regions targeted was 13 (range, 2 to 39) and th

36 However, the number of epitopic regions targeted, the protein subunits recogniz

37 of significant similarity, inclusive of the epitopic regions, between allergens and helminth protein

38 in alcoholic hepatitis focused on individual epitopic regions.

39 ntibody-S complexes identified corresponding epitopic regions.

40 he A(k)/p46-61 complex, which is hindered by epitopic residue(s) within p46-61.

41 suggests that T cell receptor (TCR) contact (epitopic) residue(s) flanking the minimal 51-59 determin

42 The identification of the epitopic sequence of a monoclonal antibody raised agains

43 ition to the nucleotide changes defining the epitopic sequence of He, a single C-to-G nucleotide tran

44 These modifications created an epitopic site that can be readily distinguished from the

45 ency, suggesting potential reversions of CTL epitopic sites recognized by the immune system of the tr

46 To augment our understanding of epitopic sites seen by a set of anti-MHC-I mAb, we deter

47 Nevertheless, the NKTcrs recognised distinct epitopic sites within these antigens, including alpha-ga

48 ssible significance of these antibodies, the epitopic specificity of the anti-rhodopsin antibodies wa

49 antibodies examined exhibited the identical epitopic specificity, it is likely that a common mechani

50 e of the time course of the response and the epitopic specificity, using peptides derived from the cy

51 ose of D10/B7 was combined with an IgG1 anti-epitopic tag monoclonal antibody, possibly because decor

52 tion occurs because the Ab binds to a common epitopic tag present at two sites on each of the two VHH

53 e produced and characterized a collection of epitopic tagged, heavy chain-only antibody VH domains (V