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1 es in macrophages degrading red blood cells (erythrophagocytosis).
2 in over 75% of phagocytic cups during amebic erythrophagocytosis.
3 moved at an accelerated rate in the liver by erythrophagocytosis.
4 ase, EhC2PK is involved in the initiation of erythrophagocytosis.
5 ophages died of exposure to heme released on erythrophagocytosis.
6 gnificantly reduced the efflux of 59Fe after erythrophagocytosis.
7 erythrocytes, nor did it enhance the rate of erythrophagocytosis.
8 -British (FAB) M4/5 morphology and prominent erythrophagocytosis.
9 d therapeutics against alloantibody-mediated erythrophagocytosis.
10 d by splenic red pulp macrophages (RPMs) via erythrophagocytosis.
11 ity and protects red blood cells (RBCs) from erythrophagocytosis.
12 roglial HO-1 and reduced injury by enhancing erythrophagocytosis.
13 CR3) on activated monocytes, thus leading to erythrophagocytosis.
14                                              Erythrophagocytosis (70%), leukemia cutis (58%), and dis
15 sociated with host parasitemia; mediators of erythrophagocytosis and cellular stress were notable com
16 rythroid-specific deletion of TLR9 abrogated erythrophagocytosis and decreased local and systemic cyt
17  at 9 and 14 days after inoculation revealed erythrophagocytosis and deposition of an iron-negative p
18 ion of a PH domain in trophozoites inhibited erythrophagocytosis and enhanced motility, providing gen
19                                              Erythrophagocytosis and inflammation from activated macr
20 In vivo, CpG-carrying RBCs drove accelerated erythrophagocytosis and innate immune activation charact
21 2(-/-) liver macrophages exhibited defective erythrophagocytosis and lysosome maturation.
22 ld-type animals exhibit comparable levels of erythrophagocytosis and platelet clearance in response t
23 pared with HM-1:IMSS, Rahman has a defect in erythrophagocytosis and the ability to cause amoebic col
24 d infiltrates, Kupffer cell hyperplasia with erythrophagocytosis, and an inconstant presence of eosin
25 formed granulomas, Kupffer cell hyperplasia, erythrophagocytosis, and microvesicular fatty metamorpho
26  establish phosphatidylserine involvement in erythrophagocytosis by amebae and suggest the existence
27        We also demonstrated an impairment of erythrophagocytosis by Hri-/- macrophages both in vitro
28 e of EhRab35 in the early and late phases of erythrophagocytosis by the amoeba.
29 erican-British M4/5 morphology and prominent erythrophagocytosis by the blast cells.
30 ation of maternal blood sinuses, the massive erythrophagocytosis by trophoblasts, the alteration of t
31  increased the phagocytosis of erythrocytes (erythrophagocytosis) by macrophages in the spleen, which
32 exporter ferroportin, which diminishes their erythrophagocytosis capacity and lysosomal activity.
33                                              Erythrophagocytosis caused the death of HO-1(-/-) macrop
34                                        After erythrophagocytosis, FPN1 mRNA levels were also up-regul
35 t FPN1 functions in the export of iron after erythrophagocytosis, FPN1 was stably expressed in J774 m
36 se activity and regulating the initiation of erythrophagocytosis in E. histolytica.
37                  The degree of C3dg-mediated erythrophagocytosis in samples from different PNH patien
38 populations, enhancing alloantibody-mediated erythrophagocytosis in SCD both in vivo in mice and in i
39 escribe a novel pathophysiologic pathway for erythrophagocytosis in the context of tissue macrophage
40  complement via complement receptor-mediated erythrophagocytosis in the spleen; and (3) when opsonize
41    In rat peritoneal macrophages, engaged in erythrophagocytosis in vitro, endotoxin stimulated heme
42 thy, fever, liver failure, pancytopenia, and erythrophagocytosis indicative of a hemophagocytic syndr
43                                       Amebic erythrophagocytosis is characteristic of invasive amebia
44                                   Therefore, erythrophagocytosis is traditionally considered as one o
45                     In murine cultures, avid erythrophagocytosis is triggered by transduction of marr
46 ad mutant of EhC2PK displayed a reduction in erythrophagocytosis, it appears that kinase activity is
47 as splenic histiocytes engage in more robust erythrophagocytosis, leading to pathological mechanical
48 e-dependent increase in (59)Fe release after erythrophagocytosis of labeled red blood cells.
49 m, we examined the effect of iron status and erythrophagocytosis on FPN1 expression in J774 macrophag
50 orthern analyses of iron-related genes after erythrophagocytosis revealed a 16-fold increase in FPN1
51 nd single-cell RNA sequencing, we found that erythrophagocytosis skewed liver macrophages into an ant
52 ressed FPN1 mRNA and protein induction after erythrophagocytosis, suggesting that FPN1 induction resu
53 nd strong induction of FPN1 expression after erythrophagocytosis suggests that FPN1 plays a role in i
54 expressing FPN1 released 70% more 59Fe after erythrophagocytosis than control cells, consistent with
55 anslocation in acute monocytic leukemia with erythrophagocytosis that fuses MOZ with CBP.
56 nt viral and nonviral proteins do not induce erythrophagocytosis to any marked degree.
57                     A reduction and delay in erythrophagocytosis was observed in E. histolytica cells
58                                       Marked erythrophagocytosis was present.
59 ntial YM1(+) tissue macrophage accumulation, erythrophagocytosis within the liver, spleen, and bone m