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1 ecimens (seven Barrett's metaplasias and six esophageal carcinomas).
2 r, in development of Barrett's esophagus and esophageal carcinoma.
3 atients undergoing nonoperative treatment of esophageal carcinoma.
4 ion does not alter the TNM classification of esophageal carcinoma.
5 of 171 patients identified, 2 had died from esophageal carcinoma.
6 reasingly used in treatment of patients with esophageal carcinoma.
7 ) for intramucosal (T1a) or submucosal (T1b) esophageal carcinoma.
8 ificantly improved survival in patients with esophageal carcinoma.
9 ging and impacts on therapy of patients with esophageal carcinoma.
10 mor types, including breast, pancreatic, and esophageal carcinoma.
11 diation followed by surgery in patients with esophageal carcinoma.
12 nd p53 mutations in patients with late-stage esophageal carcinoma.
13 y influence further follow-up guidelines for esophageal carcinoma.
14 atocellular carcinoma, and one SF complex in esophageal carcinoma.
15 ions were examined in two main histotypes of esophageal carcinomas.
16 and confers treatment resistance in lung and esophageal carcinomas.
17 eal cancer-derived cell lines and in primary esophageal carcinomas.
19 4 Barrett's esophagus (BA) metaplasias and 8 esophageal carcinomas (3 squamous cell carcinomas and 5
21 ients undergoing elective surgery for either esophageal carcinoma (adenocarcinoma or squamous cell ca
22 o-species (human and canine) and two-cancer (esophageal carcinoma and B-cell lymphoma) analysis of sp
24 tecan had significant activity in metastatic esophageal carcinoma and resulted in significant relief
25 nd EUS FNA for preoperative nodal staging of esophageal carcinoma and to measure the impact of each s
26 ate that 15-LOX-1 is down-regulated in human esophageal carcinomas and that NSAIDs induce apoptosis i
27 s an important new agent in the treatment of esophageal carcinoma, and further evaluation of this age
28 rative chemoradiation, long-term outcomes of esophageal carcinoma are best predicted utilizing histol
29 ithelium, including lung, head and neck, and esophageal carcinomas, are the leading causes of cancer-
30 of a 5.4 cM region at 7q31-q35 in 43 primary esophageal carcinomas, as well as mutational analyses of
31 at: (a) 15-LOX-1 was down-regulated in human esophageal carcinomas; (b) NSAIDs induced 15-LOX-1 expre
32 rigin or progression of at least a subset of esophageal carcinomas, but that ST7 is not the target ge
35 Squamous carcinoma of the head and neck and esophageal carcinoma demonstrate avid (18)F-FDG uptake.
36 one for patients with potentially resectable esophageal carcinoma did not demonstrate a statistically
37 ng thresholds in patients with biopsy-proven esophageal carcinoma (differentiation of stage T0 diseas
43 s for the treatment of partially obstructive esophageal carcinoma, high-grade dysplasia associated wi
44 leading to its overexpression in breast and esophageal carcinomas; however, the contribution of stab
46 rformance of MRI in determining the stage of esophageal carcinoma in patients before esophagectomy an
47 11, patients with a resectable intrathoracic esophageal carcinoma, including the gastroesophageal jun
50 , the prognosis for patients with inoperable esophageal carcinoma is still poor and the reliability o
52 cinomas and dysplasias, gastric cancers, and esophageal carcinomas, manifest microsatellite instabili
54 e development or progression of most primary esophageal carcinomas or UCANs, although lack of express
57 ith medically inoperable and/or irresectable esophageal carcinoma, referred for dCRT, were randomly a
58 ave sequenced exon 5 of cyclin D1 in primary esophageal carcinoma samples and in cell lines derived f
59 cember 1999 to March 2001, all patients with esophageal carcinoma seen at the Mayo Clinic Rochester w
60 in determining the appropriate management of esophageal carcinoma (squamous cell carcinoma and adenoc
61 gnosed with stage pT3-4Nx-0M0 or pT1-4N1-3M0 esophageal carcinoma (squamous cell or adenocarcinoma) f
62 was diminished in stomach adenocarcinoma and esophageal carcinoma (STAD-ESCA) and completely disappea
63 ican Joint Committee on Cancer, 7th Edition, esophageal carcinoma staging system is derived from pati
64 cer Audit who underwent potentially curative esophageal carcinoma surgery in 2011 to 2018, were inclu
66 complex setting of multimodal treatments of esophageal carcinoma; this leads to rather undifferentia
67 schizophrenia and one for the MMP-2 gene and esophageal carcinoma, to evaluate the performance of the
68 cancer, including liver metastasectomy, and esophageal carcinoma treated primarily with chemoradiati
69 y of 43 patients with potentially resectable esophageal carcinoma treated with an intensive regimen o
70 ot for LRC in patients with locally advanced esophageal carcinoma treated with definitive radiochemot
73 ity but low specificity for the detection of esophageal carcinoma, which shows promise for determinin
74 nd multifocal neoplasia in patients with pT1 esophageal carcinoma who underwent esophagectomy without
75 substantial antitumor activity in metastatic esophageal carcinoma, with a remarkable complete respons