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1 cted to superior endoscopy (with biopsies of esophageal mucosa).
2             No eosinophils were found in the esophageal mucosa.
3 oE), an allergic inflammatory disease of the esophageal mucosa.
4 sin B activity across normal and tumor human esophageal mucosa.
5 normal, inflamed, metaplastic, and malignant esophageal mucosa.
6 le 1, 29 patients (81%) had no cancer in the esophageal mucosa.
7 tly increased ERK2 phosphorylation levels in esophageal mucosa.
8 without acid is not sufficient to damage the esophageal mucosa.
9 extensive expansion involving up to 17 cm of esophageal mucosa.
10 s that cause an eosinophilic infiltration of esophageal mucosa.
11 ly compatible system for manufacturing human esophageal mucosa.
12 may cause irritation and inflammation of the esophageal mucosa.
13 esponse to food antigens in contact with the esophageal mucosa.
14 ach, or the infrequent methylation of normal esophageal mucosa accompanied by methylation in all norm
15 with the aim to achieve a barrier to protect esophageal mucosa against reflux damages and neutralizat
16 e and protein expression of ASIC1 and 3 from esophageal mucosa and dorsal root ganglia (DRG) neurons
17 rade IIa display internal enhancement of the esophageal mucosa and enhancement of the outer wall conf
18 y significant cis-eQTLs in FAM120AOS in both esophageal mucosa and esophageal muscularis tissue.
19 ) was found in the basal layer of the normal esophageal mucosa and in IEUs and cytomegalovirus ulcera
20  system, we show comprehensive microscopy of esophageal mucosa and of coronary arteries in vivo.
21 A, MGMT, and TIMP3) of methylation in normal esophageal mucosa and stomach, or the infrequent methyla
22 led to esophagitis, marked thickening of the esophageal mucosa, and enhanced expression of COX-2.
23 s of RE and NERD were similarly increased in esophageal mucosa as well as T3-T5 DRG neurons compared
24 ncreatic enzymes in gastric contents damages esophageal mucosa, causing inflammation and increased pa
25 the evaluation of the macroscopic aspects of esophageal mucosa during EGD.
26 geal involvement and several biopsies of the esophageal mucosa for histopathologic confirmation of Eo
27          We performed bulk RNA-sequencing on esophageal mucosa from 14 achalasia and 8 healthy subjec
28                                              Esophageal mucosa from 3 patients whose ulcers healed or
29               We used electron microscopy on esophageal mucosa from an affected family member carryin
30 issues and in 96% (96/100) of distant normal esophageal mucosa from cancer specimens.
31  xeno-free, scalable strategy for generating esophageal mucosa from human pluripotent stem cells (hPS
32 e level of TNF-alpha messenger RNA (mRNA) in esophageal mucosa from patients with cytomegalovirus-ass
33              Electron microscopy of squamous esophageal mucosa harboring the S631G variant revealed d
34 gus (BE) and malignant transformation of the esophageal mucosa in mice.
35        Long-term systematic follow-up of the esophageal mucosa including multistaged biopsies is requ
36 ether an esophageal prick test, in which the esophageal mucosa is challenged by local injection of al
37 he biofilm microorganisms suggested that the esophageal mucosa is more permissive to invasion than th
38 We profile 421,312 individual cells from the esophageal mucosa of 7 healthy and 15 EoE participants,
39 ressed genes (DEGs) were found in the distal esophageal mucosa of achalasia subjects and 120 DEGs wer
40 stological changes have been reported in the esophageal mucosa of achalasia, suggesting its involveme
41  not differ significantly between the distal esophageal mucosa of controls (median, 25.5 cell layers
42 oglobulin G4 (IgG4) and food proteins in the esophageal mucosa of eosinophilic esophagitis (EoE) pati
43 pression of mediators of inflammation in the esophageal mucosa of patients with different GERD endosc
44 ucosa of patients with NERD, from the distal esophageal mucosa of patients with ERD, and the distal-m
45 tions as dilated intercellular spaces in the esophageal mucosa of patients with GERD compared to cont
46                          Proximal and distal esophageal mucosa of patients with NERD have more superf
47 s were obtained from the proximal and distal esophageal mucosa of patients with NERD, from the distal
48 pithelium lining the upper two-thirds of the esophageal mucosa possess a non-neuronal cholinergic sys
49 ith extensive alterations of the lingual and esophageal mucosa that were absent in mice given the hwp
50                              Exposure of the esophageal mucosa to food allergens can cause acute muco
51 ial and the sensitivity or resistance of the esophageal mucosa to the reflux material are important f
52  The epithelial turnover time of the healthy esophageal mucosa was approximately 11 days (twice that
53                                  Biopsies of esophageal mucosa were taken from the (1) proximal esoph
54 to be a poor substitute for tissue biopsy of esophageal mucosa when evaluating microflora patterns.
55                We then treated ex vivo human esophageal mucosa with a cytokine cocktail to closely mi
56 lammatory mediators S100a8 and S100a9 in the esophageal mucosa with accompanying esophageal epithelia
57                                   Contact of esophageal mucosa with acid, before inducing heartburn,