ライフサイエンスコーパス: ethanolamine

1 histamine, glutamine, xanthurenic acid, and ethanolamine).

2 phosphatidylcholine or dioleoyl-phosphatidyl-ethanolamine.

3 hionate), which supports anaerobic growth on ethanolamine.

4 , filamentous growth, and is auxotrophic for ethanolamine.

5 egulator gene was induced by the presence of ethanolamine.

6 anolamine, and the satiating factor N-oleoyl ethanolamine.

7 ctivase EutA protein under aerobic growth on ethanolamine.

8 (12) are necessary and sufficient to grow on ethanolamine.

9 nia lyase (EA-lyase), a catabolic enzyme for ethanolamine.

10 ay forming phosphatidylethanolamine from CDP-ethanolamine.

11 gomyelin and lysophosphatidylcholine, versus ethanolamine.

12 nhibit phosphatidylcholine biosynthesis from ethanolamine.

13 t of an aptamer-based assay for detection of ethanolamine.

14 mately 56%), which was partially restored by ethanolamine.

15 feedback regulation of FAAH activity by free ethanolamine.

16 zes these bioactive fatty acid conjugates of ethanolamine.

17 es, phosphatidyl glycerols, and phosphatidyl ethanolamines.

18 ived lipids, and both n-3PUFA lowered N-acyl ethanolamines.

19 AcsC together are able to condense citrate, ethanolamine, 2,4-diaminobutyrate, and alpha-ketoglutara

20 rmediates formed with the substrates, [1-13C]ethanolamine, [2-13C]ethanolamine, and unlabeled ethanol

21 he type 1 [choline: 3.4 +/- 1.5% (P < 0.01); ethanolamine: 5.9 +/- 2.5% (P < 0.05)] and type 2 [choli

22 in the control group (choline: 5.5 +/- 2.2%; ethanolamine: 7.5 +/- 2.5%).

23 Microcompartment-mediated catabolism of ethanolamine, a host cell breakdown product, fuels the c

24 Ethanolamine, a product of the breakdown of phosphatidyl

25 e bisretinoid compound diretinoid-pyridinium-ethanolamine (A2E) were increased in Rdh12-deficient mic

26 d with accumulation of diretinoid-pyridinium-ethanolamine (A2E), a condensation product of all-trans-

27 n products, including di-retinoid-pyridinium-ethanolamine (A2E), are thought to be transferred to RPE

28 dihydro-N-retinylidene-N-retinylphosphatidyl-ethanolamine (A2PE-H(2)), also accumulates in retinas of

29 s, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenols, fragrances, glycol ethers,

30 Furthermore, in addition to PME synthesis, ethanolamine also contributes to the production of phosp

31 d tannic acid (TA), low-toxic cationic PGEA (ethanolamine-aminated poly(glycidyl methacrylate)) and t

32 ata obtained support the conclusion that the ethanolamine ammnonia-lyase (EAL) enzyme (encoded by the

33 Ado-B12) is both the cofactor and inducer of ethanolamine ammonia lyase (EA-lyase), a catabolic enzym

34 netic field effect (MFE) investigations with ethanolamine ammonia lyase (EAL).

35 (9 microM) of the first degradative enzyme, ethanolamine ammonia lyase.

36 in the adenosylcobalamin (AdoCbl)-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

37 trate radical pair catalytic intermediate in ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

38 n coenzyme B12 (adenosylcobalamin)-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

39 dulation (ESEEM) spectroscopic properties of ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

40 (AdoCbl; coenzyme B(12)) in AdoCbl-dependent ethanolamine ammonia-lyase (EAL) from Salmonella typhimu

41 yzed by adenosylcobalamin (AdoCbl)-dependent ethanolamine ammonia-lyase (EAL).

42 bstrate, to study radical pair generation in ethanolamine ammonia-lyase from Salmonella typhimurium a

43 trate radical pair catalytic intermediate in ethanolamine ammonia-lyase from Salmonella typhimurium h

44 rrinoid adenosyltransferase and for the EutA ethanolamine ammonia-lyase reactivase.

45 The model predicts that EutBC (ethanolamine-ammonia lyase) lies outside the compartment

46 The ethanolamine ammonialyase microcompartment of Escherichi

47 The endocannabinoid arachidonoyl ethanolamine (anandamide) is a lipid transmitter synthes

48 For example, N-arachidonoyl-ethanolamine and 2-arachidonoyl-glycerol can be metaboli

49 ith an MIC(50) of 22.5 and 15 mug/ml without ethanolamine and an MIC(50) of 75 and 60 mug/ml with eth

50 roarray assay relying on competition between ethanolamine and an oligonucleotide complementary to the

51 ditional interactions involving phosphatidyl ethanolamine and cholesterol.

52 oxyl groups are chemically tagged with (15)N-ethanolamine and detected using a 2D heteronuclear corre

53 n the endoplasmic reticulum by fusion of CDP-ethanolamine and diacylglycerol.

54 rasites exhibited severe growth defects when ethanolamine and exogenous lipids became limited or when

55 Competitive binding of ethanolamine and fluorescently labeled complementary oli

56 cid amide hydrolase (FAAH) degrades NAE into ethanolamine and free fatty acid to terminate its signal

57 inner membrane [palmitoyloleoylphosphatidyl ethanolamine and palmitoyloleoylphosphatidylglycerol (PO

58 referential cleavage of the C-O bond between ethanolamine and phosphate, enabling the selective ident

59 ncluding increased abundance of phosphatidyl ethanolamine and phosphatidyl choline species, corrobora

60 itamin B(12) biosynthesis and degradation of ethanolamine and propanediol was apparently acquired by

61 uired during anoxic growth of S. enterica on ethanolamine and tetrathionate.

62 EPR spectra of samples prepared with [1-13C]ethanolamine and the absence of such splitting in spectr

63 e hydrolase (FAAH), which hydrolyzes NAEs to ethanolamine and their corresponding free fatty acids.

64 he related pigments, all-trans-retinal dimer-ethanolamine and unconjugated all-trans-retinal dimer, i

65 tic MWFs, as well as duration of exposure to ethanolamines and nitrosamines.

66 sed surface (PFP/protein G'/whole antibodies/ethanolamine) and one optimized Fab' fragment-based surf

67 hatidylethanolamine, all-trans-retinal dimer-ethanolamine, and all-trans-retinal dimer) increased wit

68 ay intermediates phosphoethanolamine and CDP-ethanolamine, and an increase in the methylated derivati

69 ite can acquire the lipid precursors serine, ethanolamine, and choline from its environment and use t

70 hanolamine generated all-trans-retinal dimer-ethanolamine, and protonation/deprotonation of the Schif

71 pyrolysis products include alanine, glycine, ethanolamine, and small dipeptides, and many of these, t

72 ide, the anti-inflammatory lipid N-palmitoyl ethanolamine, and the satiating factor N-oleoyl ethanola

73 the substrates, [1-13C]ethanolamine, [2-13C]ethanolamine, and unlabeled ethanolamine were acquired u

74 cans in the absence or presence of exogenous ethanolamine, and YU253467 and YU254403 were identified

75 of the Kennedy pathways (CDP-choline and CDP-ethanolamine) are the predominant pathways responsible f

76 growth of an S. enterica cobA eutT strain on ethanolamine as a carbon and energy or nitrogen source.

77 Effective utilization of ethanolamine as a carbon and nitrogen source may provide

78 We evaluated the role of the metabolism of ethanolamine as a potential nitrogen and carbon source f

79 The role of ethanolamine as a potential nutrient source during UTIs

80 sing Salmonella enterica by providing [13C2]-ethanolamine as a sole carbon source.

81 peron that allows Salmonella enterica to use ethanolamine as a sole source of nitrogen, carbon, and e

82 al functions needed by this bacterium to use ethanolamine as a source of carbon and energy.

83 sferase (SDPM) pathway using host serine and ethanolamine as precursors.

84 Here, the ability of E. faecalis to utilize ethanolamine as the sole carbon source is shown to be de

85 o survive on small organic molecules such as ethanolamine as the sole source for carbon and nitrogen.

86 respectively, methoxyethanol (solvent), and ethanolamine (base).

87 fied 5 members (PhFT1-5) of the phosphatidyl ethanolamine-binding proteins (PEBP) family from moso ba

88 resent distinct binding modes to the choline/ethanolamine-binding site of P. falciparum choline kinas

89 human disorder arising due to defective CDP-ethanolamine biosynthesis and provide new insight into t

90 a mixture of ethanolaminium bicarbonate and ethanolamine bisulphide is also produced.

91 phosphoethanolamine, an intermediate in the ethanolamine branch of the Kennedy pathway of phosphatid

92 d the synthesis of phospholipids via the CDP-ethanolamine branch of the Kennedy pathway were controll

93 esis of phosphatidylethanolamine via the CDP-ethanolamine branch of the Kennedy pathway.

94 Regulation involves specific sensing of ethanolamine by a sensor histidine kinase (EutW), result

95 nvolving conjugation of arachidonic acid and ethanolamine by fatty-acid amide hydrolase.

96 tion in the synthesis of PC from choline and ethanolamine by the compound.

97 n of the resulting glycerophospho-N-modified ethanolamines by liquid chromatography-tandem mass spect

98 Ethanolamine capping was applied to avoid unspecific int

99 osome is needed to concentrate low levels of ethanolamine catabolic enzymes, to keep the level of tox

100 particular genes involved in propanediol and ethanolamine catabolism and cobalamin biosynthesis.

101 The role of GSH in ethanolamine catabolism is complex and requires further

102 We confirmed that mutants deficient in ethanolamine catabolism or in the type VII secretion sys

103 ica, a microcompartment encloses enzymes for ethanolamine catabolism.

104 osome is not involved in the biochemistry of ethanolamine catabolism.

105 e steady-state radical in the deamination of ethanolamine catalyzed by adenosylcobalamin (AdoCbl)-dep

106 l cloning strategy, we here identified a CDP-ethanolamine:ceramide ethanolamine phosphotransferase as

107 At pH 7.0, the standard ethanolamine concentration (41 mM) provides enough Eth0

108 The ethanolamine concentration in urine was comparable to th

109 ted in fluorescence intensities dependent on ethanolamine concentration with a limit of detection of

110 e show here that use of a specific nutrient (ethanolamine) confers a marked growth advantage on Salmo

111 yltransferase with prominent activity toward ethanolamine-containing lysophospholipids, which we term

112 Ethanolamine-containing molecules are essential in host

113 4), Ethanolamine-containing phosphoglycerides are generally

114 -adenosylmethionine-dependent methylation of ethanolamine-containing phospholipids to produce the abu

115 s an important enzyme in the biosynthesis of ethanolamine-containing phospholipids, especially in bra

116 ant for sicariid predatory behavior, because ethanolamine-containing sphingolipids are common in inse

117 enzyme B(12)- (adenosylcobalamin-) dependent ethanolamine deaminase from Salmonella typhimurium have

118 le mediator of radical pair recombination in ethanolamine deaminase.

119 d to phospholipid by the cytidinediphosphate-ethanolamine-dependent Kennedy pathway.

120 arboamination reaction between a substituted ethanolamine derivative and an aryl or alkenyl bromide.

121 exhibited much greater activity with N-acyl ethanolamines (e.g. anandamide) and N-acyl taurines.

122 The ability of bacteria to utilize ethanolamine (EA) as a carbon and nitrogen source may co

123 Salmonellae can use ethanolamine (EA) as a sole source of carbon and nitroge

124 Ethanolamine (EA) is an important metabolite for EHEC in

125 Ethanolamine (EA) metabolism is a trait associated with

126 following the pyrolysis of citric acid (CA)-ethanolamine (EA) precursor at different temperatures.

127 In2O3 film precursor solutions consisting of ethanolamine (EAA) and InCl3 in methoxyethanol.

128 In2O3 film precursor solutions consisting of ethanolamine (EAA) and InCl3 in methoxyethanol.

129 However ethanolamine (ETA) content was lower.

130 Supplemental ethanolamine (ETA), which can be converted to PE via the

131 scued by supplementing the growth media with ethanolamine (ETA).

132 (Eth0) does not enter cells, while uncharged ethanolamine (Eth0) diffuses freely across the membrane.

133 Evidence is presented that protonated ethanolamine (Eth0) does not enter cells, while uncharge

134 with the phosphorylation of choline (Cho) or ethanolamine (Etn) catalyzed by either choline or ethano

135 athway has evolved to be the major route for ethanolamine (EtN) synthesis, as EtN supplementation com

136 Blocking ceramide degradation with N-oleoyl-ethanolamine exacerbated Abeta cytotoxicity; and additio

137 Amides of fatty acids with ethanolamine (FAE) are biologically active lipids that p

138 were sensitive to corticosterone, selenium, ethanolamine, fatty acids and/or antioxidants.

139 umbelliferone), an amine (propargylamine and ethanolamine), fluoride, or a nucleoside monophosphate (

140 olic labeling revealed an increased usage of ethanolamine for PtdEtn synthesis by the mutant.

141 The second pigment, A2-dihydropyridine-ethanolamine, forms from phosphate hydrolysis of A2-DHP-

142 Alanine, choline, ethanolamine, glucose, lactate, myoinositol, phosphochol

143 ospholipid and phosphatidylinositol than for ethanolamine glycerophospholipid and phosphatidylserine

144 EGF receptor-containing rafts contained more ethanolamine glycerophospholipids and less sphingomyelin

145 Ethanolamine glycerophospholipids are ubiquitous cell me

146 Leishmania synthesize the majority of their ethanolamine glycerophospholipids as 1-O-alk-1'-enyl-2-a

147 lysis of arachidonoyl-containing choline and ethanolamine glycerophospholipids by other phospholipase

148 species; 2) alterations in both choline and ethanolamine glycerophospholipids, including a decreased

149 re [1-(14)C]16:0 was targeted to choline and ethanolamine glycerophospholipids, whereas more [1-(14)C

150 is well-defined in the electron density, the ethanolamine group is poorly defined, suggesting structu

151 OPA reacts with the ethanolamine head group of PE in human cells to form pyr

152 sphate, indicating that either a glycerol or ethanolamine headgroup is the chemical determinant for s

153 e recently, nonenzymatic modification of the ethanolamine headgroup of phosphatidylethanolamine (PE)

154 utions correlated with the capability of the ethanolamine headgroups to engage in hydrogen bonding wi

155 that contain phospholipids with phosphatidyl-ethanolamine headgroups.

156 forms the corrinoid-dependent degradation of ethanolamine if given vitamin B12, but it can make B12 f

157 The metabolism of ethanolamine in artificial urine medium (AUM) induced me

158 tment is used to sequester the metabolism of ethanolamine in bacteria such as Escherichia coli and Sa

159 Original data on the habitual presence of ethanolamine in beers are presented.

160 the rate of PtdEtn synthesis from exogenous ethanolamine in hepatocytes.

161 esis of phosphatidylcholine from choline and ethanolamine in P. falciparum, and provide evidence for

162 es, such as propionate, 1,2-propanediol, and ethanolamine, in addition to melibiose and ascorbate, th

163 Ethanolamine induced a 15-fold increase in the rate of a

164 y the absence of phospholipids with choline, ethanolamine, inositol, and serine head groups.

165 Salmonella enterica catabolizes ethanolamine inside a compartment known as the metabolos

166 the enzymatic machinery needed to metabolize ethanolamine into acetyl coenzyme A (acetyl-CoA).

167 ve C3-alkylation of indoles with N-protected ethanolamines involving the "borrowing hydrogen" strateg

168 These data suggest that urinary ethanolamine is a significant additional carbon and nitr

169 n a growth advantage in AUM, suggesting that ethanolamine is also utilized as a carbon source.

170 Ethanolamine is an abundant compound in the human intest

171 The choline/ethanolamine kinase (CEK) family catalyzes the initial s

172 olamine (Etn) catalyzed by either choline or ethanolamine kinase (CEK).

173 The level of ethanolamine kinase activity increased when zinc was dep

174 ingly, these compounds primarily inhibit the ethanolamine kinase activity of the P. falciparum cholin

175 leted cells, indicating that the increase in ethanolamine kinase activity was attributed to a transcr

176 strated that the zinc-mediated regulation of ethanolamine kinase and the synthesis of phospholipids v

177 Regulation of the EKI1-encoded ethanolamine kinase by inositol and choline was examined

178 Regulation of the EKI1-encoded ethanolamine kinase by the essential nutrient zinc was e

179 Ethanolamine kinase catalyzes the committed step in the

180 Ethanolamine kinase catalyzes the first step in the CDP-

181 ne was confirmed by corresponding changes in ethanolamine kinase mRNA, protein, and activity levels.

182 The eas(+) gene encodes for the protein Ethanolamine Kinase, involved in phospholipid biosynthes

183 Delta mutant defective in choline kinase and ethanolamine kinase, we examined the consequences of a b

184 hosphatidylation of eggPC in the presence of ethanolamine), lyso-phosphatidylcholine (LPC), and lyso-

185 in the IL-4-treated macrophages suggest that ethanolamine lysophospholipid (LPE) is an sPLA2-V-derive

186 only tested for PCCC applications, including ethanolamine (MEA), methyldiethanolamine (MDEA), and pip

187 s 2-arachidonoyl-glycerol and N-arachidonoyl-ethanolamine mediate an array of pro- and anti-inflammat

188 Our data show that the ethanolamine metabolosome is not involved in the biochem

189 like structure (hereafter referred to as the ethanolamine metabolosome) is thought to contain the enz

190 ith the protein, independent of the state of ethanolamine methylation, with introduction of polyunsat

191 t drug moieties (an aromatic ring and a beta-ethanolamine moiety) were further screened for aerobic b

192 inked to the A2E molecule via its pyridinium ethanolamine moiety.

193 s along with 56-60% loss of C1 and C2 phenyl ethanolamine-N-methyltransferase (PNMT)-ir neurones.

194 H characterized to date belong to the N-acyl ethanolamine (NAE) class of fatty acid amides, including

195 Termination of the activity of the N-acyl ethanolamine (NAE) class of lipid-signaling molecules, i

196 tic pathway(s) for anandamide and its N-acyl ethanolamine (NAE) congeners remain enigmatic.

197 ntative fatty acid amides include the N-acyl ethanolamines (NAEs) anandamide, which serves as an endo

198 N-Acyl ethanolamines (NAEs) are a large class of signaling lipi

199 N-Acyl ethanolamines (NAEs) constitute a large and diverse clas

200 ith the mitochondrial uncoupler, niclosamide ethanolamine (NEN), to determine the effects of mitochon

201 le cross-linking monomer N,O-bismethacryloyl ethanolamine (NOBE) along with template, initiator, and

202 (f) the effect of the AC inhibitor N-oleoyl-ethanolamine (NOE) on cytotoxicity and ceramide species.

203 ggests a role in transport, eutH mutants use ethanolamine normally under standard conditions (pH 7.0)

204 have previously selected ssDNA aptamers for ethanolamine, one of the smallest aptamer targets so far

205 its deficiency could not be complemented by ethanolamine or choline supplementation for the synthesi

206 ges in the ratio of phospholipids containing ethanolamine, or glycerol as phospholipid head group and

207 rius terrosus showed a strong preference for ethanolamine over choline, whereas two paralogous enzyme

208 rence for positively charged (choline and/or ethanolamine) over neutral (glycerol and serine) headgro

209 e kinase catalyzes the first step in the CDP-ethanolamine pathway for the formation of the major memb

210 ulation correlated with increases in the CDP-ethanolamine pathway intermediates phosphoethanolamine a

211 ion of diacylglycerol utilization by the CDP-ethanolamine pathway led to a 10-fold increase in triacy

212 hesis of phosphatidylethanolamine by the CDP-ethanolamine pathway.

213 osphatidylethanolamine synthesis via the CDP-ethanolamine pathway.

214 Arabidopsis cannot form PC from phosphatidyl ethanolamine (PE), and demonstrates that methylation of

215 ps of phosphatidylcholine (PC), phosphatidyl-ethanolamine (PE), phosphatidylinositol (PI), phosphatid

216 psulated in polyethylene glycol-phosphatidyl ethanolamine (PEG-PE) conjugated micelles.

217 classes, viz., phosphatidyl-cholines (PCs), -ethanolamines (PEs), -serines (PSs), -inositoles (PIs),

218 DC1, acts as a phosphodiesterase removing an ethanolamine phosphate (EtN-P) from mannose 2 of the gly

219 competent for transfer to protein, (ii) the ethanolamine phosphate group on the third mannose residu

220 tical for GPI recognition by GPIT, (iii) the ethanolamine phosphate residue linked to the first manno

221 enzyme that modifies the second mannose with ethanolamine phosphate, which is removed soon after GPI

222 Results of in vivo studies also show that ethanolamine-phosphate (EA-P) is a substrate of CbiB, bu

223 enzyme of the Kennedy pathway, the cytosolic ethanolamine-phosphate cytidylyltransferase (TbECT).

224 trates, initiated by the methylation of free ethanolamine-phosphate.

225 cerides and red blood cell (RBC) choline and ethanolamine phosphoglyceride FAs were assessed.

226 s than of the control subjects, and cord RBC ethanolamine phosphoglycerides were lower in DHA (P < 0.

227 ion promoted uptake of exogenous choline and ethanolamine phospholipids.

228 nd TbSLS4, a bifunctional sphingomyelin (SM)/ethanolamine phosphorylceramide (EPC) synthase, were ina

229 am-stage parasites contain sphingomyelin and ethanolamine phosphorylceramide (EPC), but no detectable

230 reviously to be a bifunctional sphingomyelin/ethanolamine phosphorylceramide synthase, whereas functi

231 inositol phosphorylceramide, TbSLS2 produces ethanolamine phosphorylceramide, and TbSLS3 is bifunctio

232 nia major through the characterization of an ethanolamine phosphotransferase (EPT) mutant.

233 novo phospholipogenesis, mediated by choline-ethanolamine phosphotransferase 1 (CEPT1), is essential

234 mic glucose metabolism, we perturbed choline/ethanolamine phosphotransferase 1 (CEPT1), the terminal

235 here identified a CDP-ethanolamine:ceramide ethanolamine phosphotransferase as the enzyme responsibl

236 sferase, diacylglycerol acyltransferase, and ethanolamine phosphotransferase were not affected by Scd

237 ificant decrease in the level of the choline/ethanolamine-phosphotransferase (PfCEPT), a key enzyme i

238 of A2E (adduct of two vitamin A aldehyde and ethanolamine) photodegradation products, and in a zymogr

239 reported effect of FABP on plasmalogen mass, ethanolamine plasmalogen mass was reduced 30% in gene-ab

240 subtype of ether phospholipids also known as ethanolamine plasmalogen whose functions are not well ch

241 A and treated with di-palmitoyl-phosphatidyl-ethanolamine polyethylene glycol (DPPE-PEG), a CD1d-bind

242 yl)-1,2-dihexadecanoyl-sn-glycero-3 -phospho-ethanolamine present in bilayer vesicles.

243 el reactions examining adduct formation with ethanolamine produced stable products of exact masses (m

244 these isoquinolinediones with methylamine or ethanolamine produced the isoquinolinedione alkaloids ca

245 Together, these findings suggest that ethanolamine production is likely required for Leishmani

246 The deuterium-labeled ethanolamine reaction product ([(2)H(4)]EA) was analyzed

247 turally occurring amide of palmitic acid and ethanolamine, reduces pain and inflammation through an a

248 Breakdown of ethanolamine requires adenosylcobalamin (AdoCbl) as a co

249 with large and small headgroups (choline and ethanolamine, respectively), and of the removal of a lip

250 mine and an MIC(50) of 75 and 60 mug/ml with ethanolamine, respectively.

251 To grow aerobically on ethanolamine, Salmonella enterica must be provided with

252 During growth on ethanolamine, Salmonella enterica synthesizes a multimol

253 Here we show that niclosamide ethanolamine salt (NEN) uncouples mammalian mitochondria

254 ve, direct acting S1P1 agonists utilizing an ethanolamine scaffold containing a terminal carboxylic a

255 sporter, EutH contributed to the toxicity of ethanolamine seen under some conditions; furthermore, fu

256 Our findings demonstrate that ethanolamine sensing and EutR-dependent regulation of th

257 n the Enterobacteriaceae and is required for ethanolamine sensing and metabolism.

258 g in spectra of samples prepared with [2-13C]ethanolamine show that the unpaired electron is localize

259 Of the five different head groups, only ethanolamine showed appreciable activity.

260 In(4)Sn(4)O(15), grown from 2-methoxyethanol/ethanolamine solutions, were used to fabricate thin-film

261 NA was established as a soluble protein with ethanolamine-specific kinase activity that was most high

262 residues are "masked" by positively charged ethanolamine substituents, leading to an overall zero ne

263 the mutant's growth defect in the absence of ethanolamine supplementation.

264 In the anaerobic environment of the gut, ethanolamine supports little or no growth by fermentatio

265 osome membranes, is synthesized de novo from ethanolamine through the Kennedy pathway.

266 Two central enzymes convert ethanolamine to acetaldehyde (EutBC) and then to acetyl-

267 richia coli (EHEC) O157:H7, EutR responds to ethanolamine to activate expression of traits required f

268 se) was required for UPEC strains to utilize ethanolamine to gain a growth advantage in AUM, suggesti

269 sion to imine functionality by reaction with ethanolamine to give ZIF-91 and ZIF-92, respectively.

270 We used MALDI-MS to verify eptA as an ethanolamine transferase for the lipid-A portion of V. f

271 her GSH nor EutA was needed during growth on ethanolamine under reduced-oxygen conditions.

272 found that the N-terminal sequences from the ethanolamine utilization (Eut) and glycyl radical-genera

273 The ethanolamine utilization (eut) locus of Enterococcus fae

274 shell constituents of a functionally complex ethanolamine utilization (Eut) microcompartment.

275 ng specific functions encoded by the 17-gene ethanolamine utilization (eut) operon established the mi

276 The ethanolamine utilization (eut) operon of this bacterium

277 EHEC encodes the ethanolamine utilization (eut) operon that allows EHEC t

278 rase (EutT) is encoded within the operon for ethanolamine utilization (eut).

279 nt expression of the Salmonella enterica LT2 ethanolamine utilization bacterial microcompartment shel

280 tory strategies that influence expression of ethanolamine utilization genes (eut) in Enterococcus, Cl

281 sing EA and regulating the expression of the ethanolamine utilization genes.

282 ultiple regulatory strategies for control of ethanolamine utilization genes.

283 c metabolic process (e.g. 1,2-propanediol or ethanolamine utilization).

284 serovar Typhimurium include those coding for ethanolamine utilization, a universal stress protein, a

285 by the eut operon proved to be essential for ethanolamine utilization, when subjected to sufficiently

286 somes for CO2-fixation, and propanediol- and ethanolamine-utilizing microcompartments that contain B1

287 th of E. faecalis in a synthetic medium with ethanolamine was abolished in the response regulator RR1

288 In vitro ethanolamine was found to be utilized as a sole source o

289 iff base nitrogen of all-trans-retinal dimer-ethanolamine was pH-dependent.

290 nolamine, [2-13C]ethanolamine, and unlabeled ethanolamine were acquired using RMFQ trapping methods f

291 in, as N-arachidoyl glycine and N-arachidoyl ethanolamine, which did not inhibit the Ca(v)3.3 current

292 ability to use an abundant simple substrate, ethanolamine, which is provided by the host.

293 med gut is because of its ability to respire ethanolamine, which is released from host tissue, but is

294 Supplementation with ethanolamine, which led to increased PE synthesis, or wi

295 ) synthase/decarboxylase are auxotrophic for ethanolamine, which must be transported into the cell an

296 noid receptors, and N-oleoyl and N-palmitoyl ethanolamine, which produce satiety and anti-inflammator

297 ublimation produced DL-alanine, glycine, and ethanolamine, while in the presence of hydrogen sulfide,

298 Replacement of ethanolamine with ethylene diamine or 1,3-diaminopropane

299 For the series of ethanolamines with different levels of headgroup methyla

300 utants isolated for their ability to degrade ethanolamine without added DMB converted Cbi to pseudo-B