ライフサイエンスコーパス: euthyroid

1 based asymptomatic individuals classified as euthyroid (0.25< thyroid-stimulating hormone [TSH] 4.0 u

2 e was 60 years, 65% were female, 21,025 were euthyroid, 1342 had subclinical hypothyroidism and 671 s

3 of TED diagnosis, 611 patients (14.9%) were euthyroid, 477 (11.6%) were hypothyroid, and 3018 (73.5%

4 thyroidism (52%), hypothyroidism (17.6%) and euthyroid (9.7%).

5 ulated thyroglobulin less than 2 ng/ml (95%, euthyroid; 96%, hypothyroid).

6 f thyroxine therapy, and all patients became euthyroid after treatment with bexarotene was stopped.

7 riteria of negative whole body scans (84% of euthyroid and 94% of hypothyroid patients) and stimulate

8 Values for thyroid uptake of 123I in euthyroid and hyperthyroid patients in Boston have remai

9 ganded TR, we deleted NCoR1 in the livers of euthyroid and hypothyroid mice and examined the effects

10 ed to the baseline TSH strata (classified as euthyroid and mild and marked SCH; all P<0.05).

11 experimental platform that exploited paired euthyroid and severe hypothyroid serum samples from huma

12 The association of TSH levels in the euthyroid and subclinical hypothyroid range with inciden

13 hyroidism who were clinically and chemically euthyroid and were receiving levothyroxine sodium, 0.1 o

14 At the end of treatment, hypothyroid, euthyroid, and hyperthyroid status was confirmed.

15 BAT)-specific hypothyroidism in an otherwise euthyroid animal.

16 rked increase in message level in T3-treated euthyroid animals depends primarily on post-transcriptio

17 ificant, increase in HCN2 mRNA occurred when euthyroid animals were made hyperthyroid.

18 ABR thresholds develop rapidly in normal, euthyroid animals, decreasing as much as 80 dB between P

19 e in that they were obtained in systemically euthyroid animals, revealing that brain D2 plays a domin

20 nes the magnitude of the response to T(3) in euthyroid animals.

21 er (1.8-fold increase) but not in T3-treated euthyroid animals.

22 formation markers in a sequential manner in euthyroid but not hypothyroid mice, thus providing evide

23 ce had normal serum T3 and were systemically euthyroid, but exhibited an approximately 3-fold elevati

24 is study shows that hyperthyroid compared to euthyroid cats have higher serum concentrations for some

25 -99 ratios are 0.47 and 0.32 for healthy and euthyroid cats, respectively, which differs significantl

26 ysis of thyroid tissue from hyperthyroid and euthyroid cats, we identified differentially expressed g

27 other areas showing high TR expression under euthyroid conditions.

28 ignificantly inhibited (> 80%) compared with euthyroid control animals.

29 ished urinary concentration as compared with euthyroid controls (CTL) and hypothyroid rats replaced w

30 ion of CYP4A2 mRNA was decreased relative to euthyroid controls, but DHEA was as effective an inducer

31 een reported to have a leaner phenotype than euthyroid controls.

32 d rats and hypothyroid rats were compared to euthyroid controls.

33 we used FloxD2 mice to generate systemically euthyroid fat-specific (FAT), astrocyte-specific (ASTRO)

34 All subjects were euthyroid for at least six months.

35 One standard deviation (SD) increase in euthyroid FT4 regulated by genetic variants in deiodinas

36 om the carotid artery and thyroid vein of 10 euthyroid goiter patients and one patient with a toxic s

37 AF was also longer, compared with 11% in the euthyroid group (all P<0.05).

38 Relative to euthyroid hamsters (EH), HH exhibited decreased D1 recep

39 t vary with states of photoresponsiveness in euthyroid hamsters, but blockade of thyroid function acc

40 women with normal functioning thyroids (ie, euthyroid) have been associated with several complicatio

41 f the hypothalamus-pituitary-thyroid axis to euthyroid hormone levels.

42 nd aged rats (26 months old) were studied at euthyroid, hyperthyroid and hypothyroid conditions.

43 er rats at 4, 12 and 24 months of age during euthyroid, hypothyroid and hyperthyroid states.

44 unoassays can be successfully eliminated for euthyroid individuals as well as for patients with thyro

45 Higher fT4 levels at baseline in euthyroid individuals were associated with increased AF

46 or hypothyroidism or during off-label use in euthyroid individuals, has not proven to be an effective

47 In euthyroid individuals, higher circulating fT4 levels, bu

48 Additionally, in euthyroid individuals, local regulation of thyroid hormo

49 ly(A) signal in hypothyroid hearts were 0.26 euthyroid levels (P < 0.05).

50 total mRNA beta 1 content decreased to 0.18 euthyroid levels, which was associated with a disproport

51 The prerequisites for an euthyroid metabolic state include a normally developed t

52 hyroid WT mice exposed to VILI compared with euthyroid mice, indicating that the lungs were functiona

53 In addition to being leaner than euthyroid mice, owing in part to reduced food intake, th

54 and ghrelin(+) cells compared to grafts from euthyroid mice.

55 ivity of (131)I to treat hyperthyroidism and euthyroid multinodular goiter.

56 thin the study group, 60 of these cats had a euthyroid (n = 23) or hyperthyroid (n = 37) status, all

57 gery were randomized into hypothyroid (N=9), euthyroid (N=9), and hyperthyroid (N=9) groups.

58 Euthyroid, nondiabetic, healthy, adult Pima Indians (n =

59 ent in the liver, deletion of SMRT in either euthyroid or hypothyroid animals had little effect on TH

60 s and also after restricting the analysis to euthyroid participants (defined by thyroid-stimulating h

61 he absolute 10-year risk of SCD increased in euthyroid participants from 1% to 4% with increasing FT4

62 significantly associated with incident AF in euthyroid participants or those with subclinical hypothy

63 In euthyroid participants, higher thyroid function was asso

64 In euthyroid participants, we additionally examined the ass

65 ts remained similar or became stronger among euthyroid participants.

66 iated with an increased risk of SCD, even in euthyroid participants.

67 ith US guidance and conscious sedation in 20 euthyroid patients (mean age, 44.5 years) with a benign

68 measurements using 123I were obtained in 671 euthyroid patients and 274 hyperthyroid patients, of whi

69 lasma T(4) concentrations in critically ill, euthyroid patients and suggest that targeting the imbala

70 thyroid function test results that occur in euthyroid patients who are receiving amiodarone.

71 er end of the normal range, as often seen in euthyroid patients who are receiving amiodarone.

72 62 euthyroid patients with HT and 58 healthy individuals we

73 For euthyroid patients, there were no significant difference

74 In euthyroid Pima Indians, lower free T(3) but not free T(4

75 urons in each nucleus were identical in both euthyroid rats and hypothyroid rats induced by 6-n-propy

76 ted effects were robust in the current work, euthyroid rats retain thyroid hormone sensitivity which

77 approximately 80% alpha-MHC/20% beta-MHC in euthyroid rats to 100% beta-MHC, without altering the ex

78 Compared to euthyroid rats, hyperthyroidism in 4-month-old rats was

79 As seen in euthyroid rats, T3 administration potently suppressed DH

80 ed the effects of similar microinjections in euthyroid rats.

81 ly 12-fold, respectively, in hypothyroid and euthyroid rats.

82 ffective an inducer of this mRNA as it is in euthyroid rats.

83 In euthyroid rodent kidney, which only expresses CYP4A2 und

84 ss; however, the mechanisms involved in the "euthyroid sick syndrome" remain poorly understood.

85 ent with this observation and reminiscent of euthyroid sick syndrome, a stress-associated clinical co

86 Originally termed the euthyroid sick syndrome, this phenomenon is now more com

87 The euthyroid state was defined as thyroid-stimulating hormo

88 e of excess radioiodine from the body in the euthyroid state with Thyrogen stimulation has significan

89 Remarkably, in the euthyroid state, expression of many T(3)-targets is also

90 large changes in weight can occur despite a euthyroid state, there are notable changes in weight, ap

91 lated in L-NCoRDeltaID mice in the hypo- and euthyroid state, there was little effect seen on negativ

92 hormone levels, thus creating a biochemical euthyroid state.

93 odalities necessary to restore patients to a euthyroid state.

94 nagement with hormone substitution to secure euthyroid status in both groups.

95 ely on serum TSH measurements to confirm the euthyroid status of these patients.

96 he goal of radioiodine therapy is to achieve euthyroid status.

97 n January 30, 2004, and June 20, 2007, of 50 euthyroid study participants aged 18 to 65 years who wer

98 HB mRNA was consistently detected in AT from euthyroid subjects, and positively associated with serum

99 al stiffness (all P<0.05) when compared with euthyroid subjects.

100 nonthyroidal illness, also known as the sick euthyroid syndrome, is characterized by a low plasma T3

101 on, contributing to the etiology of the sick euthyroid syndrome.

102 In patients with TED who were euthyroid, the 4-year cumulative incidence was 41% for a

103 Neither euthyroid thyroid stimulating hormone (TSH) nor free thy

104 Data from 368 euthyroid volunteers who provided blood samples at basel

105 ter stopping CBZ treatment and were rendered euthyroid with either long-term CBZ (n=3) or radioiodine

106 f a middle-aged clinically and biochemically euthyroid woman with a stable HT, who developed a severe

107 atment would increase live-birth rates among euthyroid women who had thyroid peroxidase antibodies an

108 The use of levothyroxine in euthyroid women with thyroid peroxidase antibodies did n