ライフサイエンスコーパス: excitatory synaptic transmission

1 and are recognized as regulatory elements of excitatory synaptic transmission.

2 ribution of the glutamate-glutamine cycle to excitatory synaptic transmission.

3 in the brain where its activation depresses excitatory synaptic transmission.

4 ne's actions on depression-like behavior and excitatory synaptic transmission.

5 mediate a slow Ca(2+)-permeable component of excitatory synaptic transmission.

6 d by ambient glutamate to regulate levels of excitatory synaptic transmission.

7 y is usually correlated with the strength of excitatory synaptic transmission.

8 tral nervous system and play a major role in excitatory synaptic transmission.

9 synaptic states to maintain the strength of excitatory synaptic transmission.

10 , whereas HDAC2 overexpression (OE) reduced, excitatory synaptic transmission.

11 rm potentiation and long-term depression, of excitatory synaptic transmission.

12 endritic spines and functional plasticity of excitatory synaptic transmission.

13 t, neuronal or synaptic structures, or basal excitatory synaptic transmission.

14 ransporters have additional roles in shaping excitatory synaptic transmission.

15 al gene expression, and deficits in cortical excitatory synaptic transmission.

16 utamate receptors that mediate a majority of excitatory synaptic transmission.

17 eptors (AMPARs) at synapses is essential for excitatory synaptic transmission.

18 ent predominantly increased postsynaptically excitatory synaptic transmission.

19 E STATEMENT Synaptic AMPARs are critical for excitatory synaptic transmission.

20 the shift is expressed by a modification of excitatory synaptic transmission.

21 y been implicated in long-term plasticity of excitatory synaptic transmission.

22 om a shift of balance between inhibitory and excitatory synaptic transmission.

23 receptors mediate the majority of vertebrate excitatory synaptic transmission.

24 sease by removing dendritic spines, sites of excitatory synaptic transmission.

25 eased the spine density to markedly increase excitatory synaptic transmission.

26 ty in hippocampal circuits, thereby reducing excitatory synaptic transmission.

27 d neuropathic pain facilitation by enhancing excitatory synaptic transmission.

28 tamate receptors (GluRs) play major roles in excitatory synaptic transmission.

29 K signaling, AMPAR membrane trafficking, and excitatory synaptic transmission.

30 e of dendritic spines--postsynaptic sites of excitatory synaptic transmission.

31 o and from synapses controls the strength of excitatory synaptic transmission.

32 ssical mushroom-head morphology, and impairs excitatory synaptic transmission.

33 t neurons show normal morphologies and basal excitatory synaptic transmission.

34 A2 is a synaptic adaptor protein involved in excitatory synaptic transmission.

35 enous neuromodulator adenosine and inhibited excitatory synaptic transmission.

36 , we report a role for opioids in modulating excitatory synaptic transmission.

37 tally with difficulty breathing and impaired excitatory synaptic transmission.

38 proteins or their binding partners regulates excitatory synaptic transmission.

39 itical for the regulation of the efficacy of excitatory synaptic transmission.

40 ion leads to short- and long-term changes in excitatory synaptic transmission.

41 d decreases both dendritic spine density and excitatory synaptic transmission.

42 ve channels that mediate a slow component of excitatory synaptic transmission.

43 ontaining, native receptors involved in fast excitatory synaptic transmission.

44 uced dendritic spine density, and diminished excitatory synaptic transmission.

45 sting that this SNARE complex is involved in excitatory synaptic transmission.

46 nc-sensitive signaling system that regulates excitatory synaptic transmission.

47 ly, the light stimuli transiently facilitate excitatory synaptic transmission.

48 tic increase in inhibitory and a decrease in excitatory synaptic transmission.

49 NMDA receptors play crucial roles in excitatory synaptic transmission.

50 Rs and their auxiliary proteins control fast excitatory synaptic transmission.

51 ptors are responsible for fast inhibitory or excitatory synaptic transmission.

52 re ligand-gated cation channels that mediate excitatory synaptic transmission.

53 nged the electrical properties and increased excitatory synaptic transmission.

54 ns in dendritic spine density and diminished excitatory synaptic transmission.

55 PA receptor levels, and thus the strength of excitatory synaptic transmission.

56 injection of JWH133 into mice also increased excitatory synaptic transmission.

57 ted ion channels that play a crucial role in excitatory synaptic transmission.

58 uronal activity to fine-tune the strength of excitatory synaptic transmission.

59 s through FSI and not via a direct effect on excitatory synaptic transmission.

60 ate uptake in astrocytes and how this shapes excitatory synaptic transmission among neurons.

61 physiological concentrations enhances basal excitatory synaptic transmission and ameliorates deficit

62 utamate receptors (AMPARs) mediate most fast excitatory synaptic transmission and are crucial for man

63 soxazole-propionate (AMPA) receptors mediate excitatory synaptic transmission and are dynamically reg

64 AMPA receptors mediate fast excitatory synaptic transmission and are essential for s

65 receptors mediate the majority of vertebrate excitatory synaptic transmission and are therapeutic tar

66 Postsynaptic expression of parkin dampens excitatory synaptic transmission and causes a marked los

67 t this early life stressor leads to enhanced excitatory synaptic transmission and decreased levels of

68 rotein phosphatase-1 and actin and modulates excitatory synaptic transmission and dendritic spine mor

69 prevailing view, AMPA receptors mediate fast excitatory synaptic transmission and effect short-term p

70 exes of NR1 and NR2A-D subunits that mediate excitatory synaptic transmission and have a role in neur

71 ionotropic glutamate receptors that mediate excitatory synaptic transmission and have been implicate

72 Both excitatory synaptic transmission and network firing acti

73 Excitatory synaptic transmission and plasticity are crit

74 These results suggest that IgSF11 regulates excitatory synaptic transmission and plasticity through

75 l recordings show that Aph1b is required for excitatory synaptic transmission and plasticity.

76 (NMDAR) to dendritic spines is essential for excitatory synaptic transmission and plasticity.

77 euroadaptive changes in hippocampal area CA1 excitatory synaptic transmission and plasticity.

78 x 10(-7)), which is implicated in regulating excitatory synaptic transmission and plasticity.

79 rtate (NMDA) receptors (NMDARs) mediate fast excitatory synaptic transmission and play a critical rol

80 e DRN through both presynaptic inhibition of excitatory synaptic transmission and postsynaptic activa

81 NMDA receptors mediate excitatory synaptic transmission and regulate synaptic p

82 ynchronous burst onset mediated by recurrent excitatory synaptic transmission and similar intrinsic s

83 150 (AKAP79/150) signaling complex regulates excitatory synaptic transmission and strength through te

84 d the impact of NPAS2 disruption on accumbal excitatory synaptic transmission and strength, along wit

85 enhancement of paired-pulse facilitation in excitatory synaptic transmission and stronger paired-pul

86 , these mediators have been shown to enhance excitatory synaptic transmission and suppress inhibitory

87 S-R1a activation in the hippocampus enhances excitatory synaptic transmission and synaptic plasticity

88 the AMPA receptor plays an important role in excitatory synaptic transmission and synaptic plasticity

89 e receptors (AMPARs) mediate the majority of excitatory synaptic transmission and their function impa

90 eceptors (mGluR) are important modulators of excitatory synaptic transmission and therefore potential

91 pines belies the elaborate role they play in excitatory synaptic transmission and ultimately complex

92 ltifunctional aspect of PSD-95 in regulating excitatory synaptic transmission and unveil a novel form

93 bitory synaptic transmission, an increase in excitatory synaptic transmission, and concomitant increa

94 transcripts that regulate synaptic function, excitatory synaptic transmission, and dopamine signaling

95 ignaling improves episodic memory, increases excitatory synaptic transmission, and enhances long-term

96 these paralog proteins in neurodevelopment, excitatory synaptic transmission, and plasticity.

97 ignaling pathway proteins, enhanced cortical excitatory synaptic transmission, and restored dendritic

98 AMPA receptors are important for excitatory synaptic transmission, and their antagonists

99 tate (NMDA) receptors, the main mediators of excitatory synaptic transmission, are heterotetrameric r

100 rs (AMPARs), the principal mediators of fast excitatory synaptic transmission, are specifically excha

101 the long-term potentiation and depression of excitatory synaptic transmission, are widespread phenome

102 particular, in autism and implicate reduced excitatory synaptic transmission as a potential mechanis

103 r before or after disease induction restores excitatory synaptic transmission as well as presynaptic

104 AMPA receptors play a central role in basal excitatory synaptic transmission as well as synaptic mat

105 hly expressed in the CNS and are involved in excitatory synaptic transmission, as well as synaptic pl

106 sms and in particular through degradation of excitatory synaptic transmission associated with impaire

107 sly reported a physiological role of BRI2 in excitatory synaptic transmission at both presynaptic ter

108 iological role of Itm2b in the regulation of excitatory synaptic transmission at both presynaptic ter

109 rebellar cortex, activation of CB1R inhibits excitatory synaptic transmission at parallel fiber (PF)-

110 indicate that TRPV1 activation can modulate excitatory synaptic transmission at the first sensory sy

111 well as subsequent in vivo E2 treatment, on excitatory synaptic transmission at the hippocampal CA3-

112 ecifically to the postsynaptic modulation of excitatory synaptic transmission at the larval neuromusc

113 ored the strength of pathologically weakened excitatory synaptic transmission at the stress-sensitive

114 The impaired inhibitory (as well as excitatory) synaptic transmission at frequencies associa

115 This defect was specific for excitatory synaptic transmission, because no change in i

116 of neurosteroid E2 is to acutely potentiate excitatory synaptic transmission, but the mechanism of t

117 The neurotransmitter glutamate mediates excitatory synaptic transmission by activating ionotropi

118 postsynaptic membrane form the foundation of excitatory synaptic transmission by establishing the arc

119 Glutamate receptors mediate excitatory synaptic transmission by forming cation chann

120 The neurotransmitter glutamate mediates excitatory synaptic transmission by gating ionotropic gl

121 siological analysis demonstrates an enhanced excitatory synaptic transmission by increasing the relea

122 Excessive levels of Abeta disrupt excitatory synaptic transmission by promoting the remova

123 ts indicate that activation of Rac1 enhances excitatory synaptic transmission by recruiting AMPARs to

124 spinal cord slices with IFN-alpha suppressed excitatory synaptic transmission by reducing the frequen

125 o exist at presynaptic terminals and reshape excitatory synaptic transmission by regulating presynapt

126 The enhancement of excitatory synaptic transmission by TNFalpha is in fact

127 and suggest an important mechanism by which excitatory synaptic transmission can be dynamically modu

128 hese results suggest that the suppression of excitatory synaptic transmission can facilitate the appe

129 We conclude that excitatory synaptic transmission dependent on N-type and

130 In the mammalian brain, the specificity of excitatory synaptic transmission depends on rapid diffus

131 The strength of excitatory synaptic transmission depends partly on the n

132 1.5-fold increase in AMPA receptor-mediated excitatory synaptic transmission, dramatically altered t

133 act with neurons and blood vessels and shape excitatory synaptic transmission due to their abundant e

134 altered cytokine network, facilitated basal excitatory synaptic transmission, enhanced intrinsic neu

135 sis that an imbalance between inhibitory and excitatory synaptic transmission exists during the criti

136 Kainate receptors mediate fast, excitatory synaptic transmission for a range of inner ne

137 In the striatum, balanced excitatory synaptic transmission from multiple sources o

138 y, demonstrating a functional segregation of excitatory synaptic transmission from neuronal morpholog

139 e we describe the impact of this mutation on excitatory synaptic transmission from parallel and climb

140 cultured mouse hippocampal neurons, recorded excitatory synaptic transmission from transfected cells,

141 Long-term potentiation (LTP) of excitatory synaptic transmission has long been considere

142 Alteration in the inhibitory and excitatory synaptic transmission (I/E) balance is a fund

143 caused by imbalances between inhibitory and excitatory synaptic transmission (I/E).

144 etwork activity by postsynaptically reducing excitatory synaptic transmission in acute and organotypi

145 xazolepropionic acid) receptors mediate fast excitatory synaptic transmission in brain and underlie a

146 , whereas both stargazin and gamma-4 rescued excitatory synaptic transmission in cerebellar granule c

147 A(-/-) mice, indicating that potentiation of excitatory synaptic transmission in DA neurons is not ne

148 of a mechanism that balances inhibitory and excitatory synaptic transmission in developing neural ci

149 uperficial DH neurons suppressed spontaneous excitatory synaptic transmission in diabetic rats in gre

150 europeptide release from sensory neurons and excitatory synaptic transmission in dorsal horn neurons,

151 docannabinoid signaling in the regulation of excitatory synaptic transmission in frontal neocortex, a

152 MPAR trafficking, silent synapse number, and excitatory synaptic transmission in hippocampal and cort

153 a galanin receptor 1-triggered depression of excitatory synaptic transmission in indirect pathway nuc

154 that alpha-BTX-sensitive nAChRs mediate fast excitatory synaptic transmission in Kenyon cells in the

155 eases intrinsic excitability and spontaneous excitatory synaptic transmission in layer V pyramidal ne

156 opic glutamate receptors, which mediate most excitatory synaptic transmission in mammalian brains.

157 naptically expressed long-term depression of excitatory synaptic transmission in medium spiny neurons

158 suggest that the drug-induced enhancement of excitatory synaptic transmission in midbrain DA neurons,

159 -type glutamate receptors (AMPARs) regulates excitatory synaptic transmission in neurons.

160 opic glutamate receptor that governs most of excitatory synaptic transmission in neurons.

161 principal glutamate receptors mediating fast excitatory synaptic transmission in neurons.

162 Excitatory synaptic transmission in PMDS neurons can be

163 on or potentiation of AMPA receptor-mediated excitatory synaptic transmission in prefrontal cortex py

164 at fear extinction decreases the efficacy of excitatory synaptic transmission in projections from the

165 PACAP enhanced, however, excitatory synaptic transmission in projections from the

166 ose of natural or synthetic AAS strengthened excitatory synaptic transmission in putative ventral teg

167 rgely determines the strength of odor-evoked excitatory synaptic transmission in rat piriform cortica

168 neurons revealed no changes in inhibitory or excitatory synaptic transmission in response to PGE2 exp

169 modulation of TRPV1-mediated enhancement of excitatory synaptic transmission in response to PKC acti

170 ive forms of synaptic plasticity that reduce excitatory synaptic transmission in response to prolonge

171 s in the central nervous system that mediate excitatory synaptic transmission in response to the rele

172 ential, it is unknown whether toluene alters excitatory synaptic transmission in reward-sensitive dop

173 Furthermore, excitatory synaptic transmission in spinal cord slices a

174 frequency of action potentials, and enhanced excitatory synaptic transmission in spinal cord slices,

175 Furthermore, facilitated excitatory synaptic transmission in spinal dorsal horn n

176 gs revealed severely affected inhibitory and excitatory synaptic transmission in the amygdala, hippoc

177 and emotional behavior, has dual effects on excitatory synaptic transmission in the basolateral amyg

178 annels whose function is critical for normal excitatory synaptic transmission in the brain and whose

179 The majority of excitatory synaptic transmission in the brain occurs at

180 Glutamate receptors that mediate excitatory synaptic transmission in the brain show unusu

181 PARs) play a critical role in mediating fast excitatory synaptic transmission in the brain.

182 4 subunits that mediate the majority of fast excitatory synaptic transmission in the brain.

183 cid (AMPA) receptors mediate the majority of excitatory synaptic transmission in the brain.

184 utamate receptors (AMPARs) mediate most fast excitatory synaptic transmission in the brain.

185 a key mechanism to determine the strength of excitatory synaptic transmission in the brain.

186 nnels responsible for a majority of the fast excitatory synaptic transmission in the brain.

187 mate-gated cation channels that mediate fast excitatory synaptic transmission in the central nervous

188 channels are membrane proteins that mediate excitatory synaptic transmission in the central nervous

189 -Methyl-D-aspartate (NMDA) receptors mediate excitatory synaptic transmission in the central nervous

190 mediate a slow, Ca2+-permeable component of excitatory synaptic transmission in the central nervous

191 Glutamate receptors mediate the majority of excitatory synaptic transmission in the central nervous

192 s are ligand-gated ion channels that mediate excitatory synaptic transmission in the central nervous

193 ic glutamate receptors (iGluRs) mediate fast excitatory synaptic transmission in the central nervous

194 agues underlies much of our understanding of excitatory synaptic transmission in the central nervous

195 Ionotropic glutamate receptors mediate fast excitatory synaptic transmission in the central nervous

196 Ion channels activated by glutamate mediate excitatory synaptic transmission in the central nervous

197 Although, other forms of inhibitory and excitatory synaptic transmission in the circuit were unc

198 ) receptors (iGluRs) mediate the majority of excitatory synaptic transmission in the CNS and are esse

199 Fast excitatory synaptic transmission in the CNS is mediated

200 AMPA receptors mediate the majority of fast excitatory synaptic transmission in the CNS, and evidenc

201 Dendritic spines are the major sites of excitatory synaptic transmission in the CNS, and their s

202 mate receptors, which underlie a majority of excitatory synaptic transmission in the CNS, associate w

203 l membrane are the primary mediators of fast excitatory synaptic transmission in the CNS.

204 azolepropionic acid (AMPA) type mediate fast excitatory synaptic transmission in the CNS.

205 Glutamate receptors mediate the majority of excitatory synaptic transmission in the CNS.

206 ating a slow, calcium-permeable component of excitatory synaptic transmission in the CNS.

207 tion of microglia drives the facilitation of excitatory synaptic transmission in the dorsal horn, whi

208 Overall, we found that (2R,6R)-HNK enhances excitatory synaptic transmission in the hippocampus thro

209 r chaperone hsp90 (heat shock protein 90) in excitatory synaptic transmission in the hippocampus.

210 Fast excitatory synaptic transmission in the mammalian brain

211 s (iGluRs) that mediate the majority of fast excitatory synaptic transmission in the mammalian brain.

212 re tetrameric ion channels that mediate fast excitatory synaptic transmission in the mammalian brain.

213 receptors are the primary mediators of fast excitatory synaptic transmission in the mammalian CNS.

214 pathway-specific presynaptic enhancement of excitatory synaptic transmission in the NAc.

215 Most fast excitatory synaptic transmission in the nervous system i

216 utamate-gated ion channels that mediate fast excitatory synaptic transmission in the nervous system.

217 is a ligand-gated ion channel that mediates excitatory synaptic transmission in the nervous system.

218 Excitatory synaptic transmission in the nucleus accumben

219 -type glutamate receptors that underlie fast excitatory synaptic transmission in the SDH.

220 tioning blocked cocaine-evoked depression of excitatory synaptic transmission in the shell of the NAc

221 are involved in bi-directional regulation of excitatory synaptic transmission in the spinal cord SG r

222 R effects the expression of neuroligin 1 and excitatory synaptic transmission in the spinal cord, and

223 (2+) channels in primary sensory neurons and excitatory synaptic transmission in the spinal dorsal ho

224 o regional- and training-specific changes in excitatory synaptic transmission in the striatum.

225 have determined the actions of adenosine on excitatory synaptic transmission in the subiculum, the m

226 s are ligand-gated ion channels that mediate excitatory synaptic transmission in the vertebrate brain

227 Dendritic spines are the primary sites of excitatory synaptic transmission in the vertebrate brain

228 ndritic spines, which are the major sites of excitatory synaptic transmission in the vertebrate brain

229 tamate receptors (iGluRs) that mediate rapid excitatory synaptic transmission in the vertebrate brain

230 ant peptides in the LH to VTA projection, on excitatory synaptic transmission in the VTA and reward-s

231 ous study we showed that kisspeptin enhances excitatory synaptic transmission in these cells.

232 A, it is unknown whether leptin can modulate excitatory synaptic transmission in this brain region.

233 re we characterize the dynamic properties of excitatory synaptic transmission in two major intracorti

234 Here we report that nicotine potentiates excitatory synaptic transmission in ventral tegmental ar

235 We examined whether increased excitatory synaptic transmission in ventral tegmental ar

236 perience-dependent homeostatic plasticity of excitatory synaptic transmission in vivo.

237 hat disruption of LIS1 has direct effects on excitatory synaptic transmission independent of laminar

238 Excitatory synaptic transmission is accompanied by a loc

239 Excitatory synaptic transmission is altered during aging

240 ices of mouse somatosensory cortex show that excitatory synaptic transmission is markedly suppressed

241 Excitatory synaptic transmission is mediated by AMPA-typ

242 central nervous system, the majority of fast excitatory synaptic transmission is mediated by glutamat

243 mouse brain slices that alpha1-A(R)-mediated excitatory synaptic transmission is mediated by the iono

244 Excitatory synaptic transmission is typically studied un

245 gests the hypothesis that the suppression of excitatory synaptic transmission itself could facilitate

246 PGE2 (2 mum; 48 h) presynaptically increases excitatory synaptic transmission, leading to a hyperexci

247 Long-term depression (LTD) of excitatory synaptic transmission mediated by type 5 meta

248 d a decrease in the frequency of spontaneous excitatory synaptic transmission (mEPSCs) in neurons lac

249 In the brain, fast, excitatory synaptic transmission occurs primarily throug

250 michannels in resting states regulates basal excitatory synaptic transmission of hippocampal CA1 pyra

251 r neuronal differentiation, and impaired the excitatory synaptic transmission of NPC-derived neurons.

252 one rats had altered inhibitory and enhanced excitatory synaptic transmission of the principal neuron

253 perience induces a persistent enhancement of excitatory synaptic transmission on NAc shell D1+ neuron

254 hol session produced enduring enhancement of excitatory synaptic transmission onto dopamine D1 recept

255 s effect is mediated by the strengthening of excitatory synaptic transmission onto dopamine neurons t

256 on of insulin into the VTA, which suppresses excitatory synaptic transmission onto dopamine neurons,

257 rd, we hypothesized that leptin can decrease excitatory synaptic transmission onto dopamine neurons.

258 that GluN2D-containing NMDARs participate in excitatory synaptic transmission onto hippocampal intern

259 Notably, fear conditioning potentiates excitatory synaptic transmission onto these long-range p

260 Notably, we found that excitatory synaptic transmission onto these neurons was

261 t persistent long-term potentiation (LTP) of excitatory synaptic transmission onto ventral tegmental

262 Thus, enhanced CRF-induced potentiation of excitatory synaptic transmission onto VTA dopamine neuro

263 motes drug-seeking behaviors and potentiates excitatory synaptic transmission onto VTA dopamine neuro

264 Finally, oxA/hcrt-1-mediated excitatory synaptic transmission onto VTA neurons was no

265 ial dorsal horn neurons either by increasing excitatory synaptic transmission or by decreasing inhibi

266 c Ca2+ fluxes affects spontaneous and evoked excitatory synaptic transmission or synaptic plasticity.

267 Postsynaptic kainate receptors mediate excitatory synaptic transmission over a broad range of t

268 important consequences for the regulation of excitatory synaptic transmission, plasticity, epileptoge

269 Long-term potentiation (LTP) of excitatory synaptic transmission plays a major role in m

270 la pyramidal neurons shows an attenuation of excitatory synaptic transmission, presumably because of

271 nf transcription impaired inhibitory but not excitatory synaptic transmission recorded from layer V p

272 ic glutamate receptors (iGluRs) that mediate excitatory synaptic transmission, regulate neurotransmit

273 ioral function; however, the degree to which excitatory synaptic transmission relies on the normal op

274 hermore, nerve injury persistently increased excitatory synaptic transmission (spontaneous excitatory

275 loss of synaptic AMPA receptors and reduced excitatory synaptic transmission that corresponds with i

276 ependent translation of proteins involved in excitatory synaptic transmission that in turn drives the

277 ogical studies revealed impairments in basal excitatory synaptic transmission that involved both AMPA

278 Fast excitatory synaptic transmission that is contingent upon

279 Among diverse factors regulating excitatory synaptic transmission, the abundance of posts

280 As a key element in excitatory synaptic transmission, the receptor regulates

281 mulates ATP/adenosine release, and depresses excitatory synaptic transmission through activation of p

282 GABA release from A17s, regulate the flow of excitatory synaptic transmission through the rod pathway

283 uRs in the dorsal BNST induces depression of excitatory synaptic transmission through two distinct me

284 ligand-gated ion channels that mediate fast excitatory synaptic transmission throughout the central

285 utamate-gated ion channels that mediate fast excitatory synaptic transmission throughout the nervous

286 However, actions of serotonin on the excitatory synaptic transmission to LHb neurons have not

287 nipulating neurogenesis in adult mice alters excitatory synaptic transmission to mature dentate neuro

288 vestigated the effects of glucocorticoids on excitatory synaptic transmission to putative DR 5-HT neu

289 ABSTRACT: During excitatory synaptic transmission, various structurally u

290 ence suggests that anaesthetics also inhibit excitatory synaptic transmission via a presynaptic mecha

291 After blockade of ionotropic excitatory synaptic transmission, voltage-clamp recordin

292 Retinogeniculate excitatory synaptic transmission was also suppressed by

293 Similar results were obtained when excitatory synaptic transmission was eliminated in a low

294 This study shows that excitatory synaptic transmission was enhanced in the bed

295 ved on dendritic shafts, and the strength of excitatory synaptic transmission was unaffected, showing

296 f NL3 in WT or NL1 KO neurons did not affect excitatory synaptic transmission, whereas P0 knockdown o

297 s in the hippocampus for 7-10 days increases excitatory synaptic transmission, whereas short-term act

298 ptors mediates dopamine-evoked depression of excitatory synaptic transmission, which contributes to a

299 how that the protein is important for normal excitatory synaptic transmission, while its dysfunction

300 m presynaptic excitatory terminals regulated excitatory synaptic transmission within hippocampal CA3.