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1 regulating fluid secretion in the sublingual exocrine gland.
2 y and autonomic nervous system and endo- and exocrine glands.
3 ized by diminished secretory function of the exocrine glands.
4 secretion is utilized by epithelial cells of exocrine glands.
5 by a progressive chronic attack against the exocrine glands.
6 e (SjS), an autoimmune disease targeting the exocrine glands.
7 ic alterations in secretory cells of several exocrine glands.
8 e control of fluid and enzyme secretion from exocrine glands.
9 ent of salivary glands and potentially other exocrine glands.
10 epithelial barriers of mucous membranes and exocrine glands.
11 Loss of function leads to obstructed exocrine glands.
12 se involving lymphocytic infiltration of the exocrine glands.
13 eaf clipping, and application of fluids from exocrine glands.
14 nvolved in the generation and maintenance of exocrine gland acinar cells have not yet been establishe
19 helial mononuclear cell infiltrates, both in exocrine glands and in other parenchymal organs (kidney,
21 -1 immunoreactivity was also detected in the exocrine glands and was stronger than DAX-1 in the inner
22 borate cuticular crypts, supported by unique exocrine glands, and that these structures have been hig
23 Although IgG autoantibodies against various exocrine gland antigens have been described in IgG4-RD,
25 aryngeal glands (HPG), suggesting that these exocrine glands are sources of regurgitate proteins.
26 l of the intestinal epithelium as well as in exocrine glands associated with the gastrointestinal tra
27 s similar to CpMRP and assume therefore that exocrine gland-based defensive strategies, evolved by th
29 disease is an autoimmune disorder affecting exocrine glands, causing dry eyes and mouth and other mo
30 , exhibited extensive vacuolization of their exocrine gland cells, while the liver, brain, and muscle
31 ion, our data report a novel role of Irf6 in exocrine gland development and support a rationale for p
32 viated the need for surgical revision of the exocrine gland drainage and thus eliminated the morbidit
33 ver 3 wk post-AdV infection, consistent with exocrine gland dysfunction as a consequence of the infla
36 lymphocytic infiltration and destruction of exocrine glands, followed by systemic organ damage and B
37 show a requirement for the MC5-R in multiple exocrine glands for the production of numerous products,
39 d diabetes in these animals, we investigated exocrine gland function in NOD.B10.H2b mice, which have
44 likely calcium-dependent chloride channel of exocrine glands has led to a more detailed understanding
45 HAT-L4 was expressed in epithelial cells and exocrine glands in tissues including skin, esophagus, tr
46 sorder of unknown etiology, affects multiple exocrine glands including the salivary and lacrimal glan
47 ic mice results in widespread dysfunction of exocrine glands, including a marked decrease in sebum pr
48 ortantly, adenophages are present throughout exocrine glands, including lacrimal glands and mammary g
51 thelial cells that line the airway, gut, and exocrine glands; it is well established that CFTR plays
52 nifesting as lymphocytic infiltration of the exocrine glands leading to chronically impaired lacrimal
54 characterized by lymphocytic infiltration of exocrine glands, mainly salivary and lacrimal, resulting
55 c autoimmune disorder that typically affects exocrine glands--mainly labial and lacrimal--leading to
56 IL-1beta, as they occur in Sjogren syndrome exocrine glands, may impair the secretory function of th
61 Nonobese diabetic (NOD) mice develop an anti-exocrine gland pathology similar to human Sjogren syndro
62 they exhibited leukocyte infiltration of the exocrine glands, produced anti-nuclear autoantibodies, a
63 se activity, appearance of autoantibodies to exocrine gland proteins, and glandular lymphocytic focal
64 oimmune condition that primarily affects the exocrine glands, resulting in symptoms such as dry eyes
65 Ca(2+) indicators in specific cell types in exocrine glands revealed both similarities to and differ
66 defined circuit by which a single compound, exocrine gland-secreted peptide-1 (ESP1), enhances repro
68 lling, smooth muscle contraction, airway and exocrine gland secretion, and rhythmic movements of the
70 -Higueras et al., 2014], suggesting that the exocrine glands, similar to the endocrine, develop from
71 identify the role of IRF6 in development of exocrine glands, specifically the major salivary glands.
73 Characterizations of the myriad neuropteran exocrine gland systems, including prothoracic, metathora
74 oimmune disease involving salivary and other exocrine glands that leads to progressive lymphocytic in
75 ch as the secretions of airway epithelia and exocrine glands, the contraction of smooth muscles, and
76 twork in which each of the four layers is an exocrine gland tissue-specific gene co-expression networ
77 lytic enzyme activity contributes to loss of exocrine gland tolerance by generating abnormally proces
78 rome is a chronic autoimmune disorder of the exocrine glands with associated lymphocytic infiltrates