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1 AG-containing membranes provides a molecular explanation for why 1) DAG alone is sufficient to activa
2 sponse and tandem duplication and provide an explanation for why a large proportion of the RLK/Pelle
3 ecognized by MAb 2158 and offer a structural explanation for why a mismatched mutation at position 18
5 oxyanion hole becomes smaller, providing one explanation for why acylation may be less efficient foll
6 nd offspring.(1)(,)(2)(,)(3) The most common explanation for why adult play is so rare is that its fu
8 the water pulse and provides an alternative explanation for why ALD growth rates for this system dec
9 erved among species, our findings provide an explanation for why all dyneins move towards the minus e
10 are imposed on mate search, and provides an explanation for why Allee effects are often observed in
12 ncapsulation mechanism provides a convincing explanation for why almonds have a low metabolizable ene
14 idity models can be rejected or supported as explanations for why and how these three disorders overl
15 ogenic model discussed here also provides an explanation for why angioedema can occur at multiple sit
18 stabilized by ANGPTL4, providing a plausible explanation for why APOC2 is an activator of LPL, while
20 al malaria, and the results offer a possible explanation for why artemether provides less advantage t
22 ate our hypothesis, also suggest a potential explanation for why axonal microtubules deteriorate in n
23 nsing (DS), have been suggested as competing explanations for why bacterial cells use the local conce
24 l Niche Conservatism hypothesis is a leading explanation for why biodiversity increases towards the e
26 targets with >100 muM affinity, offering an explanation for why both MRG15 CD and Pf1 PHD1 domains a
33 al readout of each mutation provides a valid explanation for why cMyBP-C fails to work as a brake in
35 sducer (TPR-CHAT) binding sites providing an explanation for why crRNA binding facilitates TPR-CHAT b
36 from this alternative perspective offers an explanation for why deep CD19 compartmental depletion ma
38 are large aggregates, suggesting a possible explanation for why disease pathology does not always co
40 e variety of tissues and illuminate possible explanations for why diverse interventions can result in
41 particularly in the short term, may offer an explanation for why domesticators and breeders have real
43 n and its antiapoptotic function provides an explanation for why eIF2alpha kinase deficiency in disea
44 er modeling of chromosomes, and suggests new explanations for why elucidating the functional signific
45 al properties of mitotic cells, providing an explanation for why ER reorganization is necessary for m
46 ted HePTP/PP2A activity provides a molecular explanation for why ERK activity is sensitive to membran
47 oms as adaptive fail to provide parsimonious explanations for why even mild depressive symptoms impai
48 transplanted animals, providing a potential explanation for why exogenous IL-7 did not increase GVHD
49 eakest Allee effects and may thus provide an explanation for why extinctions due to Allee effects are
50 NA damage induced by HPV16 E7, providing one explanation for why FA patients are predisposed to HPV-a
52 n levels in fluorosed enamel and provides an explanation for why fluorosed enamel has a higher than n
53 sm in gammadelta T cells but also provide an explanation for why gammadelta T cells are less dependen
54 and that reassortment events can provide an explanation for why genetically related viruses with dif
55 ponses against HuNoV replication provide one explanation for why GII.4 infections are more widespread
56 ences in these contacts provide a structural explanation for why GluR2 L483Y and GluR3 L507Y are nond
58 m the target lysine and provide a structural explanation for why H3T3 phosphorylation and H3K9 acetyl
64 eptor after loss of sex steroids provides an explanation for why IL-6 is important for skeletal homeo
65 onses illustrate the need for a more unified explanation for why immunity is compromised in neonates.
66 immune escape variants, provides a molecular explanation for why immunotherapy for HBV infection may
68 existing literature and posits epidemiologic explanations for why investigators might have failed to
69 fication of gas5 as a 5'TOP gene provides an explanation for why it is a growth arrest specific trans
70 , we identified a systems-level, mechanistic explanation for why KRAS(G13D) cancers respond to EGFR i
73 use salivary proteins, and consider possible explanations for why lice have a complex salivary gland
74 However, we lack an ultimate, evolutionary explanation for why lifelong PA, particularly during mid
76 iently in the presence of Lis1, providing an explanation for why Lis1 is a required cofactor for most
77 nhibited state, which provides a mechanistic explanation for why Lis1 is required for efficient trans
78 r initiation provide a potential mechanistic explanation for why long-term use of low doses of NSAIDs
79 hat of complete MMR deficiency, providing an explanation for why loss of heterozygosity is not requir
80 results suggest a physiologically plausible explanation for why LTD induction is experimentally diff
81 sis regulation and then we consider possible explanations for why MCU-deficient mice are spared from
82 Our model offers a quantitatively robust explanation for why membrane bioenergetics are universal
83 ogen and its receptor on HBV thus provide an explanation for why men have a higher risk of HBV infect
84 nterkinetochore tension, thereby offering an explanation for why MI in mammals is so error-prone.
88 ns' metabolic theory provides a paradigmatic explanation for why most large-effect mutations are rece
89 nctions at middle promoters, and provides an explanation for why MotA can form non-specific multimers
91 mitochondrial function, providing potential explanations for why MTIF3 genetic variation at rs677859
92 cale orientation map provides a parsimonious explanation for why multivariate pattern analysis method
93 rate of GP Ibalpha degradation, providing an explanation for why mutation of their genes leads to def
94 lap in skeletal muscle and CNS (providing an explanation for why mutation of ZEB-1 alone has little e
95 protection function of BRCA1, and provide an explanation for why mutations within the BRCA1 RING doma
96 mputational analyses also provide a possible explanation for why N-debenzoyl-2'-deoxy-PTX is inactive
97 sical principle, this provides a theoretical explanation for why noninteger dimensions are useful in
98 ient loading state and identify a structural explanation for why only a select group of inhibitors di
100 d-selective region in vOT while providing an explanation for why other studies have found a lack of w
101 decay of the mRNA body, and this provides an explanation for why p38 MAPK regulates mRNA stability in
103 th VEGF-A upregulation offered a mechanistic explanation for why patients exhibit improved outcomes a
104 gA deficiency is an infrequent but important explanation for why patients with CD may be negative on
105 for finding the missed warning signs and an explanation for why people did not "connect the dots" wi
107 Pore constrictions provide a mechanistic explanation for why pit membrane thickness determines em
108 Hebbian synaptic plasticity and a functional explanation for why place cells become directionally sel
109 are localized in the chloroplast, a possible explanation for why plants have a larger immunophilin fa
110 In particular, the model provides a simple explanation for why PPC encodes reach targets in referen
111 Electrostatic targeting of Hsp70 provides an explanation for why preferential binding has been observ
112 ugh not without flaws, the model provides an explanation for why primary plastids have evolved so rar
114 IFN-gamma-induced apoptosis is not a viable explanation for why RB expression rescues DRB inducibili
116 nderlie viral evolution and provide a likely explanation for why relatively few types of plant DNA vi
120 by food odors, providing a possible unifying explanation for why sea turtles interact with marine pla
121 asC and rivR mutations do not provide a full explanation for why serotype M3 strains are associated w
125 of the mechanism of dissociation provides an explanation for why small molecules that bind at the AB/
126 ocks Mrc1 loading onto origins, providing an explanation for why so many mutants in DNA replication s
127 salt, temperature or osmolarity, but we lack explanations for why so many antiporters are needed and
128 ghts from this study may provide a plausible explanation for why some biogenic carbonates and carbona
129 one promotes CDK7i sensitivity, providing an explanation for why some cancers are more sensitive to C
131 und in many P-element constructs provides an explanation for why some gene function is retained.
133 diated by hER-alpha36 provide an alternative explanation for why some human breast cancers are resist
134 uccess as a breeder, offering one compelling explanation for why some individuals delay reproduction
135 te for the absence of Nef, thus providing an explanation for why some infectious molecular clones tha
139 rve cord (VNC), we uncovered a circuit-based explanation for why stimulation of posterior thoracic me
140 protein stability in cells and provide a new explanation for why stressed cells accumulate osmolytes.
142 e for G4DNA provides an additional molecular explanation for why such sequences are prevalent in the
145 for transcriptional activity, providing one explanation for why synergy between these factors is imp
146 Together, our data provide a mechanistic explanation for why the cap-proximal UI-RNA duplex inhib
147 large and fast movements provide a possible explanation for why the conduction pathways of a wide ra
148 ts microtubule shortening, thus providing an explanation for why the drug can counteract the phenotyp
149 lity to neutralizing antibody, suggesting an explanation for why the elicited neutralization was not
150 n would have suggested, offering a potential explanation for why the flawed power stroke mechanism ca
151 erlapping binding sites provides a potential explanation for why the G/C-rich Mad binding site consen
152 i organization between tissues, providing an explanation for why the Golgi is sufficiently robust to
153 timulatory activity of chitosan, offering an explanation for why the largest particles were nearly de
154 ctural basis for LCR function and provide an explanation for why the LCR core regions are so extremel
155 pace than is the mature form and provides an explanation for why the leader peptide is removed from t
157 is fails to provide an ultimate evolutionary explanation for why the male germline would tolerate mor
158 conformational transitions, and provides an explanation for why the modification robustly slows dive
159 hin the CAV1 complex, providing a structural explanation for why the mutant protein fails to homo-oli
162 he genome against transposons and suggest an explanation for why the piRNA pathway was retained in an
164 Therefore, these results provide a simpler explanation for why the proTAME-induced mitotic arrest i
165 tors of neurogenesis and provides a possible explanation for why the Q/R editing process exists.
166 -term premorbid level, providing a potential explanation for why the risks and benefits of lowering b
167 This visual analysis provides a possible explanation for why the salvage reactions in purine meta
170 Together, these studies provide a simple explanation for why the vast majority of BRCA2 mutants a
172 The simulation results provide some possible explanations for why the dolphin performed differently w
173 Results of the qualitative analysis suggests explanations for why the programme worked in some contex
174 cific T cells in Hu patients and provided an explanation for why their detection has been elusive.
175 n the primary tumor, providing a biochemical explanation for why their expression specifically suppre
176 The final section discusses several possible explanations for why there are often little or no conseq
177 acetylcholine receptor (nAChR) subunits, an explanation for why these currents get smaller is not av
178 an alternative, and possibly complementary, explanation for why these freshwater bottom dwellers emi
179 ay redundant roles in synapsis, providing an explanation for why these genes have evaded previous gen
180 ion by beta-Trcp, thus providing a molecular explanation for why these mutations cause beta-catenin a
181 rough the cholinergic system and provides an explanation for why these neuromodulators are involved i
182 quences in the human p53 gene, providing one explanation for why these sites are mutational hot spots
184 bow joints when attempting any movement, one explanation for why they were better at the 'reach up' t
187 ppressed in HPV-positive cells, providing an explanation for why this microRNA is targeted in HPV-pos
188 aerobic conditions and provides a reasonable explanation for why this mutant protein is functional un
190 th PXLE-containing proteins and providing an explanation for why Tibetans are not predisposed to eryt
192 growth factor receptor providing a possible explanation for why tumorigenesis was not altered after
194 rovenance of this complex fold and offers an explanation for why Wnts utilize both lipid- and protein
195 malian cardiomyocytes as well as a potential explanation for why zebrafish and newts, but not mammals
196 Zoep mutant phenotype, providing a plausible explanation for why Zoep genetically interacts with spt