ライフサイエンスコーパス: extracellular volume fraction

1 ion of extracellular space by calculation of extracellular volume fraction.

2 n to calculate global myocardial T1, T2, and extracellular volume fraction.

3 enhancement, native T1 relaxation times, and extracellular volume fraction.

4 portion to the square root of enlarged total extracellular volume fraction.

5 gression, leading to an apparent increase in extracellular volume fraction.

6 pattern of scar and quantify the myocardial extracellular volume fraction.

7 sis on the basis of assessment of myocardial extracellular volume fraction.

8 red ventriculo-arterial coupling, and higher extracellular volume fraction.

9 ement (LGE), T1 and T2 relaxation times, and extracellular volume fraction.

10 with the latter also associated with higher extracellular volume fraction.

11 and the extent of interstitial expansion via extracellular volume fraction.

12 en) exhibited increased myocardial fibrosis (extracellular volume fraction, 0.34 +/- 0.06 vs 0.29 +/-

13 adolinium enhancement, 0.90; T2 ratio, 0.79; extracellular volume fraction, 0.71; early gadolinium en

14 T1 (1225+/-30 to 1239+/-30 ms; P=0.02), and extracellular volume fraction (24.5+/-2.3% to 26.0+/-2.6

15 versus 1152+/-46 milliseconds; P<0.001) and extracellular volume fraction (26+/-5% versus 30+/-6%; P

16 ed regression led to an apparent increase in extracellular volume fraction (27.4+/-3.1% to 30.2+/-2.8

17 ly; P<0.001) and increased diffuse fibrosis (extracellular volume fraction, 27.4+/-2.2% versus 27.2+/

18 s; 95% CI: -69 to -5 ms; P = 0.026), indexed extracellular volume fraction (-3.9 g/m(2); 95% CI: -7.0

19 tauic), a cell size-dependent parameter, and extracellular volume fraction, a marker of interstitial

20 i) [adjusted z-score: 0.47 (0.09, 0.86)] and extracellular volume fraction [adjusted z-score: 0.79 (0

21 lar sodium concentration (C1, in mM) and the extracellular volume fraction (alpha) in grey and white

22 Extracellular volume fraction and tauic can track myocar

23 Extracellular volume fraction and tortuosity of the gran

24 n the cellular compartment, while changes in extracellular volume fraction and tortuosity, which are

25 itudinal strain, and T1 mapping to determine extracellular volume fraction and volume of cellular and

26 Measurements of extracellular volume fraction and water content were com

27 ric-mapping subclinical fibrosis (native-T1, extracellular volume fraction) and inflammation/edema (m

28 tification using native T1 relaxation times, extracellular volume fraction, and global longitudinal s

29 endently associated with wall thickness, T2, extracellular volume fraction, and late gadolinium enhan

30 l size, vascular volume fraction, intra- and extracellular volume fractions, and pseudo-diffusivity a

31 fraction, thus confirming the suitability of extracellular volume fraction as an in vivo measure of d

32 resonance measures of interstitial fibrosis (extracellular volume fraction), as well as serum biomark

33 High LV mass index, low native T1, and low extracellular volume fraction at baseline were all predi

34 The extracellular volume fraction, calculated from pre- and

35 Both post-contrast myocardial T1 time and extracellular volume fraction correlated with beta, the

36 ise CMR parameters, global myocardial T1 nor extracellular volume fraction differed significantly bet

37 c resonance (CMR) measurements of myocardial extracellular volume fraction (ECV) and late gadolinium

38 sodium concentration ([Formula: see text]), extracellular volume fraction (ECV) and the water fracti

39 In large cohorts, extracellular volume fraction (ECV) has been shown to qu

40 Extracellular volume fraction (ECV) is a marker for myoc

41 gadolinium enhancement (LGE), and automated extracellular volume fraction (ECV) maps per patient wer

42 The authors used CMR with extracellular volume fraction (ECV) measurement to chara

43 s through late gadolinium enhancement (LGE), extracellular volume fraction (ECV) measures by cardiac

44 Purpose To establish the sensitivity of extracellular volume fraction (ECV) quantification using

45 rdiac MRI T1 mapping sequences for measuring extracellular volume fraction (ECV) throughout the spect

46 d cardiac magnetic resonance measurements of extracellular volume fraction (ECV) to discover and quan

47 We measured the extracellular volume fraction (ECV) to quantify the extr

48 The extracellular volume fraction (ECV) was derived accounti

49 LV extracellular volume fraction (ECV) was predictive of co

50 LGE presence, extent, and pattern, and extracellular volume fraction (ECV) were evaluated accor

51 sis for native T1 relaxation times, GLS, and extracellular volume fraction (ECV) with respect to mort

52 ardiovascular risk factors on myocardial T1, extracellular volume fraction (ECV), and T2 at 3T in the

53 arditis as well as native T1, calculation of extracellular volume fraction (ECV), and T2 mapping (onl

54 ques permit quantification of the myocardial extracellular volume fraction (ECV), representing a surr

55 ental late Gadolinium enhancement (LGE), and extracellular volume fraction (ECV).

56 extracellular matrix expansion by measuring extracellular volume fraction (ECV).

57 vely quantify diffuse myocardial fibrosis as extracellular volume fraction (ECV).

58 brosis, which they correlated with increased extracellular volume fraction (ECV).

59 um enhancement [LGE], T1 and T2 mapping, and extracellular volume fraction [ECV] z scores) of patient

60 arker), native/postcontrast T1 mapping (with extracellular volume fraction [ECV]) to assess edema and

61 available indices of interstitial fibrosis (extracellular volume fraction [ECV]; N = 1172 and native

62 ocardial native (n) T1 (fibro-inflammation), extracellular volume fraction (fibrosis), and triglyceri

63 T2>52 ms, 78% for native T1>981 ms, 74% for extracellular volume fraction >0.24, and 100% for T2 rat

64 ighted imaging in 2 patients (13%), elevated extracellular volume fraction in 3 patients (20%), and e

65 conductivity of physiological saline and the extracellular volume fraction in brain tissue.

66 es a significant increase of skeletal muscle extracellular volume fraction in study participants with

67 Preterm-born young adults have greater extracellular volume fraction in the left ventricle that

68 Extracellular volume fraction in the right ventricular f

69 tricular dysfunction and prevented increased extracellular volume fraction, indicating that T1 mappin

70 s not affect either the TMA diffusion or the extracellular volume fraction, indicating that the enzym

71 uded (1) global longitudinal strain, and (2) extracellular volume fraction (log-rank 85).

72 between HCM and hypertension, over and above extracellular volume fraction, LV wall thickness and ind

73 pertrophy to establish whether native T1 and extracellular volume fraction may complement electrocard

74 nterstitium and quantify ECM expansion using extracellular volume fraction measures and other derange

75 The myocardial extracellular volume fraction (MECVF) has been used to d

76 The extracellular volume fraction of the RV free wall was me

77 BNP was associated with a 0.62% increment in extracellular volume fraction (p < 0.001), 0.011 increme

78 stant (P = .0007) and 14.3% in extravascular-extracellular volume fraction (P = .002) were seen after

79 late gadolinium enhancement and T(1) mapping/extracellular volume fraction, respectively.

80 lation results indicate that even for larger extracellular volume fractions than what is reported for

81 For two different geometries with the same extracellular volume fraction the geometry with the most

82 olume transfer coefficient K(trans), and the extracellular volume fraction v(e) were calculated for t

83 rd CMR parameters, global myocardial T1, and extracellular volume fraction values for assessing the a

84 longitudinal strain post-AVR, an increase in extracellular volume fraction was associated with worsen

85 Extracellular volume fraction was calculated.

86 Extracellular volume fraction was greater in preterm-bor

87 Extracellular volume fraction was higher in the LVEF 50%

88 Global extracellular volume fraction was statistically not diff

89 Extracellular volume fraction was unchanged, indicating

90 Extracellular volume fraction was used as a marker of di

91 Native T1 and extracellular volume fraction were significantly higher

92 Left ventricular native T1 values and extracellular volume fractions were higher in patients c