1 egionally-specific lens protein aggregation,
extracerebral amyloid formation, and supranuclear catara
2 aturation are based on the discrimination of
extracerebral and cerebral tissue layers, and they can e
3 The cranial circulation, both
extracerebral and cerebral, is innervated by fibers from
4 Extracerebral and intraventricular CSF was readily quant
5 Cerebral and
extracerebral binding levels were not correlated with ea
6 Large variability in cerebral and
extracerebral binding was observed among individuals.
7 No group-level differences were found in
extracerebral binding.
8 ranial SCG targets, such as pineal gland and
extracerebral blood vessels (bv).
9 he sympathetic innervation of representative
extracerebral blood vessels [internal carotid artery (IC
10 TH protein associated with the
extracerebral blood vessels was also significantly decre
11 TH levels in the SCG and
extracerebral blood vessels were determined by Western b
12 the heart, external carotid artery, and the
extracerebral blood vessels, as well as estrogen recepto
13 gets, the external carotid artery, heart and
extracerebral blood vessels.
14 Unlike pineal gland and
extracerebral bv, the external carotid artery, an extrac
15 findings provide a neural mechanism by which
extracerebral cephalic blood flow couples to brain event
16 pointense) white matter, and ventricular and
extracerebral cerebrospinal fluid (CSF).
17 ability in a brain preparation isolated from
extracerebral compartments.
18 We prospectively captured all
extracerebral components of the Sequential Organ Failure
19 nt arms and was more closely associated with
extracerebral distant metastases (P = .016) than with is
20 Cyclosporin A (CsA) is well known, for its
extracerebral effect, as an immunosuppressant in organ t
21 nd Janeway lesions were associated with more
extracerebral emboli (75.0% vs 31.8%, P = .02).
22 Theories rarely unify brain measures with
extracerebral factors or capture heterogeneity in indivi
23 Seizure induction in the absence of
extracerebral factors promoted the release of IL-1beta f
24 s induce brain inflammation independently on
extracerebral factors.
25 -714 uptake throughout the body could assess
extracerebral inflammation in post-COVID syndrome.
26 rther studies using [(18)F]DPA-714 to assess
extracerebral inflammation should consider these kinetic
27 n (r = -0.49, P = 0.005), both in intra- and
extracerebral lesions.
28 intensity, underscores the critical role of
extracerebral mechanisms in migraine pathophysiology.
29 largest lesion (p = 0.0037), and presence of
extracerebral metastases (p = 0.0017), were independent
30 ET showed heterogeneous uptake in intra- and
extracerebral metastatic lesions in melanoma patients.
31 However, the prevalence of
extracerebral multiple organ dysfunction in postcardiac
32 all 12 dogs survived to 96 hrs without gross
extracerebral organ damage (p < .0001).
33 and total brain histologic damage scores and
extracerebral organ damage were assessed at 96 hrs.In no
34 diac arrest patients; 96% had some degree of
extracerebral organ dysfunction and 66% had severe dysfu
35 ts of this study support the hypothesis that
extracerebral organ dysfunction is common and associated
36 The most common
extracerebral organ failures were cardiovascular (i.e.,
37 nd 66% had severe dysfunction in two or more
extracerebral organ systems.
38 rolonged CPCR in dogs preserves viability of
extracerebral organs and improves outcome.
39 Attenuation of water content of
extracerebral organs with hypertonic saline treatment ma
40 0, severe damage), and morphologic damage of
extracerebral organs.
41 us levels of [(18)F]DPA-714 uptake in the 10
extracerebral organs.
42 e optimal pharmacokinetic model in different
extracerebral organs.
43 nd the only blood in the jugular bulb was of
extracerebral origin.
44 may reflect possible early extrastriatal and
extracerebral pathology of PD.
45 re prion diseases are usually transmitted by
extracerebral prion infection, but clinical disease resu
46 progression-free survival, and freedom from
extracerebral progression in univariable and multivariab
47 study was to explore the applicability of an
extracerebral reference region for the quantification of
48 ry constant or total distribution volume for
extracerebral regions.
49 Concordant brain and
extracerebral responses support use of pembrolizumab to
50 The highest
extracerebral Sequential Organ Failure Assessment score
51 le organ dysfunction (defined as the highest
extracerebral Sequential Organ Failure Assessment score)
52 nts in the MLPT group (all P </= .01), while
extracerebral space was larger (P < .0001).
53 bral tissue (82+/-14 microM) with respect to
extracerebral tissue (30+/-7 microM).
54 measured in cerebral tissue (56%+/-10%) and
extracerebral tissue (62%+/-6%).
55 ts, where the top layer (layer 1) represents
extracerebral tissue (scalp, skull, dura mater, subarach
56 concentration and saturation in cerebral and
extracerebral tissue of adult human subjects, where the
57 on of images was performed by normalizing to
extracerebral tissue.
58 e efficiently reducing lipid disturbances in
extracerebral tissues.