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1 ul in identifying disease (1 prostate bed, 3 extraprostatic).
2 expressed prostatic secretions from men with extraprostatic and organ-confined prostate cancers ident
4 nd 1994, 1369 non-stage A1 and 423 advanced (extraprostatic) cases of prostate cancer were diagnosed.
6 0.06-4.6) ng/mL, 56 of 86 patients (65%) had extraprostatic disease (pT3), 39 of 86 (45%) had a Gleas
8 isease (pT2) (1477 of 2160; 68%), those with extraprostatic disease at MRI (102 of 1477; 7%) were at
9 d better outcomes than those with concordant extraprostatic disease at MRI and pathologic analysis: 1
11 rther, alternative modalities of determining extraprostatic disease must be investigated beyond the c
14 ivity of this pathway may be selected by the extraprostatic environment or, as supported by our data,
15 5% CI: 1.2, 2.1; P = .003); and suspicion of extraprostatic extension (EPE) (HR, 2.18; 95% CI: 1.1, 4
17 tures associated with pathologically defined extraprostatic extension (EPE) of prostate cancer and to
18 1.6; P < .001), SVR (OR, 6.2; P = .02), and extraprostatic extension (EPE) scores of 2 (OR, 9.3; P <
21 predicted larger tumor volumes (P < 0.001), extraprostatic extension (P = 0.003), and seminal vesicl
26 (kappa = 0.266-0.439); and fair for definite extraprostatic extension on T2-weighted images (kappa =
27 was associated with cancers characterized by extraprostatic extension or distant metastases (stage C
29 ely selected patients with positive margins, extraprostatic extension or seminal vesicle invasion, bu
30 ry, European Society of Urogenital Radiology extraprostatic extension score, nodes) fitted to the tra
31 vesicle invasion, positive surgical margins, extraprostatic extension) and salvage radiotherapy with
32 and Data System score, index lesion burden, extraprostatic extension, and preoperative guided biopsy
33 nical stage, Gleason score, surgical margin, extraprostatic extension, and seminal vesicle invasion,
35 in patients with either positive margins or extraprostatic extension, its effect on cause-specific m
36 inical and pathologic findings (grade group, extraprostatic extension, nodal involvement) relevant fo
37 ith pathologically advanced prostate cancer (extraprostatic extension, positive surgical margins, or
38 progression, as APCs exhibit lower rates of extraprostatic extension, seminal vesical invasion and l
39 thological features including Gleason score, extraprostatic extension, status of surgical margins, an
40 pecific antigen level greater than 10 ng/mL, extraprostatic extension, tumor volume more than 20%, ca
43 gan-confined disease, and 20 (70%) of 30 had extraprostatic extension; 11 (37%) of the 30 had positiv
45 state, including bed and seminal vesicle, or extraprostatic, including all lymph nodes, bone, or soft
47 .4%, and 70.3% for the lesion, prostate, and extraprostatic levels, respectively, with associated Fle
53 e region; and 83.3%, 75.0%, and 83.3% in the extraprostatic region for readers 1, 2, and 3, respectiv
58 hibits only limited expression in benign and extraprostatic tissues, and thus represents an ideal tar
60 mental lineage, arising from either intra or extraprostatic tumour cell populations, at early and lat
61 h nodes originate from evolutionary advanced extraprostatic tumour cells rather than less advanced ce