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1 tion (standard definition plus VF with three extrastimuli).
2 uction, termination, and resetting by single extrastimuli.
3 requency, and atrial vulnerability to atrial extrastimuli.
4 ion latency, and AF incidence from premature extrastimuli.
5 extrastimuli or VF induced with two or fewer extrastimuli.
6 e-dimensional intraoperative cardiac maps of extrastimuli and beats during 44 nonsustained VTs and th
7 lectrogram duration (DeltaED) in response to extrastimuli and increased S1S2 coupling intervals at wh
8 ned reentrant beats were induced with single extrastimuli, and the complete reentrant loop was visual
9 e AF inducibility by high right atrial (HRA) extrastimuli (APDs).
10 on of polymorphic VT or VF, even with double extrastimuli, appears less relevant than induction of mo
11  400 ms) and followed by one to three atrial extrastimuli at one to four pacing sites in the right at
12 to the baseline stimulation, with the atrial extrastimuli being delivered at the pacing site responsi
13 ed the vulnerable window during which single extrastimuli could induce TdP in both models.
14  to the His bundle could be advanced by late extrastimuli delivered at the anterior and posterior app
15                            In contrast, late extrastimuli delivered at the inferoparaseptal mitral an
16                                       Single extrastimuli delivered during sustained epicardial VT in
17  flutter acceleration were induced by single extrastimuli delivered in the isthmus between the tricus
18 fractory periods were determined with single extrastimuli delivered in the tricuspid annulus-eustachi
19                                              Extrastimuli dynamically modulated each characteristic o
20 ard protocol consisting of single and double extrastimuli followed by burst pacing was performed from
21 timulation protocol (3 drive cycle length, 3 extrastimuli >/=200 ms, and burst>/=2 sites) was repeate
22 L, AF+AFLs and AF, while AF vulnerability to extrastimuli increased in parallel.
23                  Sites activated last during extrastimuli initiating nonsustained or sustained VTs oc
24 cible VF, especially with triple ventricular extrastimuli, may merit reconsideration.
25 r polymorphic VT induced with three or fewer extrastimuli or VF induced with two or fewer extrastimul
26        We hypothesized that single premature extrastimuli (S(2)) insufficient to induce reentry produ
27                 The first, second, and third extrastimuli (S2 through S4) conducted with progressivel
28                                   Programmed extrastimuli showed evidence of a decremental accessory
29                  Also, Group I required more extrastimuli to induce VT in PES 1 (2 [1-4] versus 2 [1-
30                                       Atrial extrastimuli were delivered in the TA-ER isthmus during
31 single, double, and triple right ventricular extrastimuli were delivered to refractoriness.