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1 ased alveolar permeability and contralateral extravascular lung water.
2 vimetric method and by direct measurement of extravascular lung water.
3 ting a 169 +/- 166 mL increase in nonindexed extravascular lung water.
4 was performed to record the value of indexed extravascular lung water.
5 iac index, pulmonary blood volume index, and extravascular lung water.
6 ema, which can be assessed by measurement of extravascular lung water.
7 tual extravascular lung water, and predicted extravascular lung water.
8 tment with intravenous beta-agonists reduces extravascular lung water.
9 erval, 0.65-0.94) was larger than for actual extravascular lung water (0.72; confidence interval, 0.5
10 hough the area under the curve for predicted extravascular lung water (0.8; confidence interval, 0.65
11 acid versus hydrochloric acid showed higher extravascular lung water (1,424 +/- 419 vs. 574 +/- 195
12 multiple regression model, the ratio between extravascular lung water and end-expiratory lung volume
13 for lung edema and has been shown to reduce extravascular lung water and improve lung function in mo
16 drome, to determine the relationship between extravascular lung water and other markers of lung injur
17 respiratory distress syndrome (ARDS) reduced extravascular lung water and plateau airway pressure.
19 , PaO2/Fio2 ratio, oxygenation index, actual extravascular lung water, and predicted extravascular lu
20 iple logistic regression analysis, predicted extravascular lung water but not actual extravascular lu
22 l showed a pattern consistent with increased extravascular lung water (diffuse, bilateral, symmetrica
23 predicted or actual body weight for indexing extravascular lung water does not lead to independence o
25 and subsequently every 6 hours, we recorded extravascular lung water, end-expiratory lung volume, lu
26 nd we quantified lung injury in terms of the extravascular lung water (EVLW) content, filtration coef
28 hypothesized that it could be improved using extravascular lung water (EVLWi) and plasma biomarkers o
29 o abrogate significantly the accumulation of extravascular lung water evoked by 6-hour exposure to en
30 ody weight, improves the predictive value of extravascular lung water for survival and correlation wi
31 atio were lower in patients with a change in extravascular lung water >/= 10% than in patients with a
34 lly contributed to prediction of a change in extravascular lung water >/= 10%, independent of the pre
37 lowing endotoxin-induced lung injury reduced extravascular lung water, improved lung endothelial barr
39 the low tidal volume group, as was decreased extravascular lung water in the uninstilled lung in the
44 was 70% in patients with a maximum value of extravascular lung water index >21 mL/kg and 43% in the
46 distress syndrome episode (maximum value of extravascular lung water index and maximum value of pulm
47 ght, p < 0.001 [t-test] for maximum value of extravascular lung water index and median [interquartile
48 rome might be associated with an increase in extravascular lung water index and pulmonary vascular pe
58 In multivariate analyses, maximum value of extravascular lung water index or maximum value of pulmo
61 f cardiac index, global end-diastolic index, extravascular lung water index, and stroke volume index,
66 taneous breathing trial-induced increases in extravascular lung water indexed for ideal body weight,
67 nary edema were 0.89 (95% CI, 0.78-0.99) for extravascular lung water indexed for ideal body weight,
68 ded pulmonary artery occlusion pressure, the extravascular lung water indexed for ideal body weight,
70 50 eligible patients, 132 patients (88%) had extravascular lung water indexed to predicted body weigh
84 e compared between patients with a change in extravascular lung water <10% and patients with a change
86 ding upper limits for fluid resuscitation of extravascular lung water (<10 mL/kg) and global end-dias
95 to predicted body weight, females had a mean extravascular lung water of 9.1 (SD=3.1, range: 5-23) mL
96 tion, intrapulmonary shunting, and increased extravascular lung water (p < .05 compared with baseline
97 ng strain (p < 0.001; adjusted R2, 0.18) and extravascular lung water (p < 0.001; adjusted R2, 0.11).
98 ve analysis indicated that EVLWp, Vd/Vt, and extravascular lung water (p = .0005, .009, and .013, res
100 o detect small short-term changes of indexed extravascular lung water secondary to bronchoalveolar la
101 iately after bronchoalveolar lavage, indexed extravascular lung water significantly increased from 12
104 lung water is a better predictor than actual extravascular lung water to identify patients at risk fo
108 ribe and assess the clinical significance of extravascular lung water variations after pulmonary enda
109 cted extravascular lung water but not actual extravascular lung water was a predictor of mortality wi
112 bronchoalveolar lavage, the value of indexed extravascular lung water was significantly different fro
113 s of lung injury, and to examine if indexing extravascular lung water with predicted body weight (EVL