ライフサイエンスコーパス: faecalis

1 n opsonophagocytosis of bacteria including E faecalis.

2 om temperature in comparison to Enterococcus faecalis.

3 regulator of antimicrobial resistance in E. faecalis.

4 s was more effective against E. coli than E. faecalis.

5 regulator of cephalosporin resistance in E. faecalis.

6 ls, yeast, Escherichia coli and Enterococcus faecalis.

7 deficient (Il10(-/-)) mice with Enterococcus faecalis.

8 he bioactivities of peptides generated by E. faecalis.

9 imicrobial activity for P. gingivalis and E. faecalis.

10 tease-secreting enteric microbe Enterococcus faecalis.

11 ns, Actinomyces naeslundii, and Enterococcus faecalis.

12 ns Clostridioides difficile and Enterococcus faecalis.

13 contributes to intestinal colonization by E. faecalis.

14 epatitis have increased faecal numbers of E. faecalis.

15 s of bacteriophages that target cytolytic E. faecalis.

16 oli, Pseudomonas aeruginosa and Enterococcus faecalis.

17 C mice were challenged with a high dose of E faecalis 12 hours after intraperitoneal administration o

18 microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution B: Staphyloco

19 we investigated the ability of Enterococcus faecalis 2/28, isolated from artisan cheese, to release

20 it exotoxin that is secreted by Enterococcus faecalis(5,6)-as a cause of hepatocyte death and liver i

21 e, we report the cryo-EM structure of the E. faecalis 70S ribosome to a global resolution of 2.8 angs

22 Enterococcus faecalis, a Gram-positive bacterium, and Candida albican

23 zation of collateral effects in Enterococcus faecalis, a gram-positive opportunistic pathogen.

24 Enterococcus faecalis, a leading cause of hospital-acquired infection

25 omologues encoded by S. aureus USA300 and E. faecalis acetylate spermidine, spermine and norspermidin

26 E. faecalis also colonizes the female reproductive tract, a

27 us, Pseudomonas aeruginosa, and Enterococcus faecalis although PSO had an antimicrobial activity agai

28 Multidrug-resistant Enterococcus faecalis, an opportunistic human pathogen, evolves resis

29 O-1, were first developed using Enterococcus faecalis, an organism that has previously been shown to

30 mmon species, Escherichia coli, Enterococcus faecalis and Bacteroides vulgatus, did not suggest a gen

31 apeutic and preventive strategies against E. faecalis and beyond.

32 er, the sera conferred protection against E. faecalis and E. faecium strains in a mouse infection mod

33 genic E. coli isolates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within 4 a

34 the 2 sera was observed against different E. faecalis and E. faecium strains.

35 ach with less than 28 SNP differences, of E. faecalis and E. hormaechei.

36 ilm and faecal samples included Enterococcus faecalis and Enterobacter hormaechei.

37 rains of Staphylococcus aureus, Enterococcus faecalis and Enterococcus faecium.

38 mM), the toxicity of 10 mg L(-1) PCP for E. faecalis and fresh anaerobic sludge was detected in 10 m

39 differences among seven chlorophenols to E. faecalis and fresh mixed anaerobic sludge were elucidate

40 be was selected from 16S rRNA sequence of E. faecalis and immobilized on a gold electrode surface in

41 n NICU organisms including K. oxytoca and E. faecalis and increases in common adult organisms includi

42 cy against vancomycin resistant Enterococcus faecalis and methicillin-resistant Staphylococcus aureus

43 nuclear neutrophils to readily clear both E. faecalis and multidrug-resistant E. faecium.

44 ing of how (p)ppGpp promotes virulence in E. faecalis and other bacterial pathogens is still lacking.

45 g overproduction of PrgB-like adhesins in E. faecalis and other clinically-important Gram-positive sp

46 For Enterococcus faecalis and other enterococcal species, revised breakpo

47 Antibiotics eliminated collagenolytic E faecalis and P mirabilis but did not reduce tumor format

48 elated with the presence of collagenolytic E faecalis and Proteus mirabilis.

49 to characterize the infection dynamics of E faecalis and show that infected wounds result in 2 diffe

50 teria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Escherichia coli

51 ith host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus aureus) within

52 sitive ( Staphylococcus aureus, Enterococcus faecalis) and Gram negative bacteria (e.g., Escherichia

53 as developed for Gram-positive (Enterococcus faecalis) and Gram-negative (Escherichia coli) reference

54 SA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bacteria Escheri

55 innocua, Staphylococcus aureus, Enterococcus faecalis, and Bacillus anthracis, on samples similar to

56 including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resistant strains

57 idis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and three associated

58 e facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge, with or with

59 .00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.

60 herichia coli and Gram-positive Enterococcus faecalis applied during the first 7 months of life did n

61 was assessed by application on Enterococcus faecalis as a model organism for Gram-positive bacteria.

62 We undertook whole genome sequencing of E. faecalis associated with bloodstream infection in the UK

63 tection against antimicrobial peptides in E. faecalis at a significant cost in bacterial fitness.

64 ch determined the anatomic distribution of E faecalis at the site of the anastomosis and within tumor

65 and two AR bacterial strains (E. coli and E. faecalis, at 10(5) CFU mL(-1)) were spiked in real WW.

66 aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptococcus pneumoni

67 reus ATCC 29213, 0.016 to 0.12 mug/ml for E. faecalis ATCC 29212, 0.008 to 0.03 mug/ml for S. pneumon

68 TCC 29213, 0.25 to 2 mug/ml for Enterococcus faecalis ATCC 29212, 1 to 4 mug/ml for Escherichia coli

69 ococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli DH5alpha, onto

70 These findings indicate that E faecalis avoids the complement phagocytosis by the AP vi

71 Monomicrobial E. faecalis bacteremia (hazard ratio [HR], 3.60; 95% confid

72 ic gentamicin for uncomplicated Enterococcus faecalis bacteremia in children.

73 A total of 344 patients with E. faecalis bacteremia were included, all examined using ec

74 paring clinical outcomes of children with E. faecalis bacteremia without endocarditis receiving ampic

75 Monomicrobial E. faecalis bacteremia, community acquisition, prosthetic h

76 lence of 26% definite IE in patients with E. faecalis bacteremia, suggesting that echocardiography sh

77 ests that for children with uncomplicated E. faecalis bacteremia, the addition of low-dose gentamicin

78 should be considered in all patients with E. faecalis bacteremia.

79 ned the prevalence of IE in patients with E. faecalis bacteremia.

80 ine the prevalence of IE in patients with E. faecalis bacteremia.

81 we included 647 consecutive patients with E. faecalis bacteremia.

82 rditis (IE) among patients with Enterococcus faecalis bacteremia.

83 cardiography in consecutive patients with E. faecalis bacteremia.

84 Newly isolated Enterococci faecalis bacterial strains AIM06 (DSM100702) and SR14 (D

85 We report here the identification of the E. faecalis bacteriocin, EntV, produced from the entV (ef10

86 this phage and show that it can eradicate E. faecalis biofilms formed in vitro on a standard polystyr

87 ation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation of sewage-sour

88 ing disruption of intestinal homeostasis, E. faecalis can overgrow, cross the intestinal barrier, and

89 investigations demonstrate that GelE from E. faecalis can regulate enteric epithelial permeability vi

90 ts from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integration.

91 Enterococcus faecalis Cas1-Cas2 selectively binds to a splayed 30-bas

92 EF508 exhibited high binding affinity to E. faecalis cells (K(D)-value: 37 nM) and successfully disc

93 of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three surface adhe

94 E. faecalis co-exists with Escherichia coli and other patho

95 ary to identify novel genes important for E. faecalis colonization and persistence in the vaginal tra

96 Although factors promoting E. faecalis colonization of intestines are not fully known,

97 of OG1RF_12399-12402 is not required for E. faecalis colonization of the mouse intestine but is asso

98 dentification of genetic determinants for E. faecalis commensal and pathogenic interactions with M. s

99 Here, we show that the dynamics of E. faecalis communities exposed to antibiotics can be surpr

100 n diet and given antibiotics, surgery, and E faecalis compared with only 30% of mice fed the standard

101 In Enterococcus faecalis, conjugation of a Cas9-targeted plasmid was enh

102 resence of cytolysin-positive (cytolytic) E. faecalis correlated with the severity of liver disease a

103 In addition, the composition ratio of E. faecalis could be decreased in multispecies microecology

104 nting colonization by multidrug-resistant E. faecalis could therefore be a valuable approach towards

105 ally regulate cephalosporin resistance in E. faecalis, dependent on the kinase activity of IreK.

106 ef-biosensor was successfully applied for E. faecalis detection in human samples.

107 gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in secreted bacteri

108 A specific E. faecalis DNA probe was selected from 16S rRNA sequence o

109 present study, an Enterococcus faecalis (E. faecalis) DNA biosensor (ef-biosensor) was fabricated to

110 In the present study, an Enterococcus faecalis (E. faecalis) DNA biosensor (ef-biosensor) was

111 Enterococcus faecalis (E. faecalis) is one of the indicator bacteria

112 ccus gordonii (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolates after trea

113 his approach is demonstrated on Enterococcus faecalis (E. faecalis), which served as target in eleven

114 egionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, antivirulence, an

115 le-molecule FRET methods to the Enterococcus faecalis (Efa) Cas1-Cas2 system to establish a kinetic f

116 ) of the opportunistic pathogen Enterococcus faecalis, encodes a 6-phospho-beta-glucosidase (GenA) an

117 of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in endocarditis a

118 t EfbA is an important factor involved in E. faecalis endocarditis and that rEfbA immunization is eff

119 ereus group, Enterococcus spp., Enterococcus faecalis, Enterococcus faecium, Staphylococcus spp., Sta

120 athogens Staphylococcus aureus, Enterococcus faecalis, Enterococcus faecium, Streptococcus pyogenes,

121 Finally, we show that an E. faecalis epa mutant strain is deficient in intestinal co

122 aomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O157:H7, Salmone

123 and frequently cocolonizes with Enterococcus faecalis, Escherichia coli, Providencia stuartii, and Mo

124 We further describe that E faecalis evades the AP by recruitment of FH on its surfa

125 that hsdS allelic variations in Enterococcus faecalis exert significant impact on gene expression.

126 fferentiation of DCs were not affected by E. faecalis, expression of the autophagy-related proteins A

127 us (MRSA), vancomycin-resistant Enterococcus faecalis/faecium (VREfc/VREfm), and ciprofloxacin-resist

128 resisting immune clearance, contributes to E faecalis fitness.

129 osition in vancomycin-resistant Enterococcus faecalis following the growth in presence of vancomycin

130 Furthermore, S. aureus and E. faecalis form biofilms normally without polyamines, and

131 ue: 37 nM) and successfully discriminated E. faecalis from 20 different Enterococcus and non-Enteroco

132 hia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodrat (Neotoma l

133 To identify and characterize E. faecalis genes that are key to intestinal colonization,

134 Fibrobacteres phylum and 12 genera in the E. faecalis group and antibiotics group were lower than tha

135 12 genera were uniquely identified in the E. faecalis group on d 14 and 28.

136 ary treatments: the basal diet group, the E. faecalis group, and the antibiotic group on d 0, 14, and

137 Bacterial abundance and diversity in the E. faecalis group, bacterial diversity in the antibiotic gr

138 uire polyamines for growth, S. aureus and E. faecalis grow normally over multiple subcultures in the

139 Here we show that E. faecalis harbouring pPD1 replaces indigenous enterococci

140 teria Staphylococcus aureus and Enterococcus faecalis have lost either all or most polyamine biosynth

141 omone-responsive, conjugative plasmids of E. faecalis have retained Prg-like surface functions over e

142 gens, Escherichia coli), except Enterococcus faecalis, human milk was more antimicrobial than bovine

143 nisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues.

144 in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of GAC biosynthe

145 llowing vaginal colonization, we observed E. faecalis in vaginal, cervical, and uterine tissue.

146 ile for the bacterial indicator Enterococcus faecalis, inactivation results were comparable across we

147 When pulsed with ovalbumin (OVA), the E. faecalis-induced DCs showed reduction in CD4(+) OVA-spec

148 tor beta1 [TGF-beta1]) were suppressed in E. faecalis-induced DCs, while IL-1beta, tumor necrosis fac

149 investigate the role of this receptor in E. faecalis-induced permeability.

150 The pathogenesis of E. faecalis infection relies in part on its capacity to col

151 d with enhanced survival from M. luteus + E. faecalis infection.

152 tial targets for interventions to prevent E. faecalis infections.

153 eat a broad range of antibiotic-resistant E. faecalis infections.

154 The present study examined how E. faecalis influences the differentiation of murine bone m

155 permits growth in gluconate, facilitates E. faecalis intestinal colonization, and exacerbates coliti

156 nts of cholate and lysozyme resistance in E. faecalis, IreK was the only one found to be required for

157 Enterococcus faecalis is a commensal and pathogen of humans and insec

158 Enterococcus faecalis is a commensal bacterium of the human intestine

159 Enterococcus faecalis is a Gram-positive commensal bacterium native t

160 Enterococcus faecalis is a gram-positive organism responsible for ser

161 Enterococcus faecalis is a human intestinal pathobiont with intrinsic

162 Since Enterococcus faecalis is a natural heme auxotroph and cause of bloods

163 from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding protein.

164 In Manduca sexta, E. faecalis is an infrequent member of the commensal gut co

165 The Gram-positive bacterium Enterococcus faecalis is both a colonizer of the gastrointestinal tra

166 Enterococcus faecalis is both a common commensal of the human gastroi

167 Enterococcus faecalis is considered to be the most important species

168 ent tyrosine decarboxylase from Enterococcus faecalis is followed by transformation of dopamine to m-

169 Enterococcus faecalis is frequently associated with polymicrobial inf

170 positive opportunistic pathogen Enterococcus faecalis is frequently responsible for nosocomial infect

171 E. faecalis is intrinsically resistant to several antibioti

172 mechanisms of cephalosporin resistance in E. faecalis is lacking.

173 Enterococcus faecalis is one of the most frequently isolated bacteria

174 red for pathogenesis, GIT colonization by E. faecalis is poorly understood.

175 Expansion of intestinal Enterococcus faecalis is sufficient to exacerbate ethanol-induced liv

176 Enterococcus faecalis is the third most frequent cause of infective e

177 Enterococcus faecalis (E. faecalis) is one of the indicator bacteria that have bee

178 U gene pairs are widely distributed among E. faecalis isolates and other enterococcal and staphylococ

179 This revealed that 6 E. faecalis isolates behaved in a commensal manner with no

180 The virulence of 28 E. faecalis isolates representing 24 multilocus sequence ty

181 ivation mechanism may be active against Ent. faecalis, it is not for the sewage-source organisms.

182 gelatinase-and-cytolysin-negative strain E. faecalis JH2-2.

183 species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pneumoniae, and

184 l isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudomonas aerugin

185 aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pneumoniae, Escherichia coli, and S

186 es indigenous enterococci and outcompetes E. faecalis lacking pPD1.

187 ned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase gene (gelE).

188 robiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clostridium orbisc

189 A comparison of the E. faecalis LiaR, E. faecium LiaR, and the LiaR homolog fro

190 Enterococcus faecalis, long implicated in serious systemic infections

191 on of conformational changes in Enterococcus faecalis MDD that describe sequential steps in an induce

192 mevalonate diphosphate decarboxylase from E. faecalis (MDDEF).

193 Thus, E. faecalis modulates its local environment by contributing

194 Analyses of E. faecalis mutants exhibiting defects in antimicrobial res

195 report the GIT colonization phenotypes of E. faecalis mutants lacking selected sortase-dependent prot

196 sis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2 clinically av

197 The 70S ribosome structure of E. faecalis now extends our knowledge of bacterial ribosome

198 We adapted E. faecalis OG1RF (a laboratory strain) and S613TM (a clini

199 Here, we expressed and purified the E. faecalis OG1RF EfbA and confirmed that this protein bind

200 bial peptide activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infecti

201 i actively prevents urease enhancement by E. faecalis, P. stuartii, and E. coli Importantly, these in

202 ggest that multiple factors contribute to E. faecalis persistence in the reproductive tract.

203 trans isomerase, as well as the Enterococcus faecalis polysaccharide diheteroglycan, are able to indu

204 t can be exploited to alter heterogeneous E. faecalis populations.

205 r mobile genetic elements shapes in vitro E. faecalis populations.

206 Multidrug-resistant Enterococcus faecalis possess numerous mobile elements that encode vi

207 robiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant GIT.

208 avA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important in experimenta

209 Enterococcus faecalis PrgA, encoded by the conjugative plasmid pCF10,

210 , Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm formation in

211 It is concluded that E. faecalis promotes the differentiation of bone marrow ste

212 Secreted E. faecalis proteins induced permeability in epithelial cel

213 Secreted E. faecalis proteins induced permeability in the colonic ep

214 enteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.

215 as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebsiella pneumon

216 ications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphylococcus aureus

217 ses (Brochothrix thermosphacta, Enterococcus faecalis, Pseudomonas fluorescens, Salmonella typhimuriu

218 l carbohydrate metabolism in general, and E. faecalis PTS-gluconate in particular, during inflammatio

219 ine N-acetyltransferases in S. aureus and E. faecalis represent a new paradigm for bacterial polyamin

220 ort chains in SCV S. aureus and wild-type E. faecalis results in reduced growth rate but provides res

221 For E. faecalis, results were similar in original water samples

222 logs from Bacillus subtilis and Enterococcus faecalis retain this ability.

223 st identified from the pathogen Enterococcus faecalis S613.

224 We show that E. faecalis significantly augments E. coli biofilm growth a

225 is the more preferred substrate, and that E. faecalis SSAT is almost as efficient as human SSAT with

226 of Gram-positive facultative anaerobes (Ent. faecalis, Staph. aureus, and Strep. bovis).

227 heart caused by streptococcus, Enterococcus faecalis, Staphylococcus aureus, or coagulase-negative s

228 Colonization with an E. faecalis strain carrying a conjugation-defective pPD1 mu

229 y is a phosphotransferase system (PTS) in E. faecalis strain OG1RF that phosphorylates gluconate and

230 The Enterococcus faecalis strain, which was not inhibited, produced reute

231 protein named AfIP-1A/1B from an Alcaligenes faecalis strain.

232 olytic rodent-derived strain of Enterococcus faecalis (strain E2), and on the second day they were gi

233 efined bacterial consortia containing the E. faecalis strains and measured inflammation and bacterial

234 intestinal epithelial cell lines with the E. faecalis strains and measured intracellular bacterial su

235 Enterococcus faecalis strains are resident intestinal bacteria associ

236 e intestine, pPD1 is transferred to other E. faecalis strains by conjugation, enhancing their surviva

237 aecium strains but none of five Enterococcus faecalis strains consistently developed resistance at al

238 We generated E. faecalis strains containing deletions/point mutations in

239 g exposure to CCM from parental or mutant E. faecalis strains indicated paracellular permeability.

240 ion of several bacteriophages that target E. faecalis strains isolated from the oral cavity of patien

241 lly in the presence of gluconate and that E. faecalis strains lacking, or harboring a single point mu

242 The E. faecalis strains were from two sequence types (ST191 and

243 For E. faecalis strains, only GS exhibits a rapid bactericidal

244 ealer to multispecies bacteria (Enterococcus faecalis, Streptococcus gordonii, Actinomyces naeslundii

245 terial composition in the colon, enhanced E. faecalis survival within macrophages, and increased proi

246 a coli Symbio and Gram-positive Enterococcus faecalis Symbio or placebo from week 5 until the end of

247 portunistic nosocomial pathogen Enterococcus faecalis synthesizes a specific lysoform lipoprotein (N-

248 fy LiaR-independent pathways in Enterococcus faecalis that regulate cell membrane adaptation in respo

249 of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway, mevalonate di

250 In Enterococcus faecalis, the regulatory nucleotides pppGpp and ppGpp, c

251 f SrtA is key to the ability of Enterococcus faecalis to bind mucin (a major component of the GIT muc

252 tudy, we investigated the contribution of E. faecalis to mixed-species infection when iron availabili

253 The resistance of E. faecalis to several classes of antibiotics and its capac

254 E. faecalis tolerates high concentrations of GIT antimicrob

255 Interestingly, we observe that E. faecalis transiently maintains CRISPR targets despite ac

256 as Corynebacterium striatum and Alcaligenes faecalis, typically considered commensals or contaminant

257 earch demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics enhanced growth

258 microbiota in the distal gut of pigs fed E. faecalis UC-100 substituting antibiotics, this study ass

259 We show that E. faecalis upregulates OG1RF_12399 transcription specifica

260 ed against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.

261 ver, little is known about the mechanisms E. faecalis uses to colonize and compete for stable gastroi

262 concentrated conditioned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase gen

263 : (i) the accretion of mobile elements in E. faecalis V583 renders it incompatible with commensal str

264 Gelatinase (GelE) purified from E. faecalis V583 was used to confirm the ability of this pr

265 use vaginal colonization model to explore E. faecalis vaginal carriage and demonstrate that both vanc

266 known about specific factors that promote E. faecalis vaginal colonization and subsequent infection.

267 group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 microg/mL).

268 ur results show a strong C3b deposition on E faecalis via both the CP and the LP but not through the

269 irulence map that explains enhancement in E. faecalis virulence and contributes to a deeper comprehen

270 s, we hypothesized that (p)ppGpp mediates E. faecalis virulence through regulation of metal homeostas

271 nvestigation indicated that, unlike other E. faecalis virulence traits, phage03-like elements were fo

272 jor role in control of ace expression and E. faecalis virulence.

273 (MRSA) and vancomycin-resistant Enterococcus faecalis (VRE), and another undergoes spontaneous cycliz

274 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial pathogen(1,2

275 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial pathogen1,2.

276 of vancomycin-resistant and -susceptible E. faecalis (VSEfs), which has important implications for i

277 D) determined for HAdV 41, Phi X 174, and E. faecalis was 35 GU/muL, 1 GU/muL, and 5 x 10(3) GU/muL (

278 prostates challenged with either UPEC or E. faecalis was equal or greater than primary infection des

279 nic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditis elegans mod

280 Ultrastructurally, E. faecalis was identified in single-membrane vacuoles, som

281 Under sterile conditions, 1 muL Enterococcus faecalis was inoculated inside the implants, and abutmen

282 f solar exposure for AR E. coli, while AR E. faecalis was more resistant to the disinfection process

283 Higher sorption of DOM to E. faecalis was roughly correlated with higher photoinactiv

284 e, using a laboratory strain of Enterococcus faecalis, we developed a novel Caenorhabditis elegans mo

285 nmental lytic phages with tropism against E. faecalis, we found that these phages require the enteroc

286 ia coli (UPEC) or Gram-positive Enterococcus faecalis, we used a mouse transurethral instillation mod

287 alis and the endodontic species Enterococcus faecalis were grown to early log phase and inoculated on

288 AcpAs of Lactococcus lactis and Enterococcus faecalis were inactive.

289 teicoplanin-susceptible (VanB phenotype) E. faecalis were responsible for major and minor errors.

290 rcine gut in response to diets containing E. faecalis were similar to the response to which containin

291 (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolates after treatment with an

292 is demonstrated on Enterococcus faecalis (E. faecalis), which served as target in eleven rounds of ce

293 1, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygiene.

294 from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRISPR locus, ter

295 ophages can specifically target cytolytic E. faecalis, which provides a method for precisely editing

296 gle very major error was obtained against E. faecalis, while vancomycin-intermediate S. aureus (VISA)

297 Incubation of E. faecalis with heme increased growth and restored catalas

298 Our findings link cytolytic E. faecalis with more severe clinical outcomes and increase

299 del of colonization of the mouse gut with E. faecalis, without disrupting the microbiota, to evaluate

300 y describe a mouse model for investigating E faecalis wound infection determinants, and suggest that