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1 r titin (TTN) are the leading known cause of familial dilated cardiomyopathy.
2 iciency is probably the most common cause of familial dilated cardiomyopathy.
3 2 different SCN5A mutations associated with familial dilated cardiomyopathy.
4 ), which has previously been associated with familial dilated cardiomyopathy.
5 proteins have been recorded in patients with familial dilated cardiomyopathy.
6 aluations were performed in 21 kindreds with familial dilated cardiomyopathy.
7 lioma, split-hand/foot deformity type 1, and familial dilated cardiomyopathy.
8 udied in wild-type mice and a mouse model of familial dilated cardiomyopathy.
9 study is to identify novel mutations causing familial dilated cardiomyopathy.
10 e, familial hypertrophic cardiomyopathy, and familial dilated cardiomyopathy.
11 unt for approximately 10 percent of cases of familial dilated cardiomyopathy and are particularly pre
12 lament-Ca(2)(+) responsiveness in a model of familial dilated cardiomyopathy and improve cardiac func
13 AG3 are associated with an increased risk of familial dilated cardiomyopathy, and common variants are
15 C (LMNA) gene mutations are a known cause of familial dilated cardiomyopathy, but the precise mechani
21 and that two lamin A mutants associated with familial dilated cardiomyopathy, E203G and E203K, exhibi
22 imed to characterize the clinical profile of familial dilated cardiomyopathy (FDC) in the families of
24 ted cardiomyopathy occurring in families, or familial dilated cardiomyopathy (FDC), may occur in 20%
28 arch comparing the survival of children with familial dilated cardiomyopathy (FDCM) to that of childr
32 fy the causal variant in a large family with familial dilated cardiomyopathy of unknown pathogenesis.
33 ecause of prominent genetic heterogeneity in familial dilated cardiomyopathy, recognition of pathogen
34 S for mutations in 116 DCM families from the Familial Dilated Cardiomyopathy Registry and in 309 subj
38 pathological remodeling in a mouse model of familial dilated cardiomyopathy with established disease
39 rovided for a novel therapeutic approach for familial dilated cardiomyopathy, with potential applicat