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1 was eliminated after adjustment for the PCF1-Fasted.
2 113.11, P < 0.001) after being fed than when fasted.
3 n rate compared to patients that were orally fasted.
4 d beta5 were lower (P<0.05) for fed than for fasted.
5 ion of hepatic lipid biosynthetic genes when fasted.
6 gnificantly reduced in the people with T2DM (Fasted: 0.0084+/-0.0067 [Control] versus 0.0016+/-0.0014
11 ucagon ratio, and HOMA-insulin resistance in fasted adult DKO pigs and blood glucose and C-peptide ch
12 sphorylation in endothelial cells, overnight-fasted adult male rats received continuous GLP-1 infusio
14 ions in a nationally representative group of fasted adults in the US population in NHANES samples fro
15 than the GM C(trough) of 0.83 mg/L (26%) in fasted adults on dolutegravir 50 mg once-daily; in child
17 which was 54% lower than the GM C(trough) in fasted adults; and in those 30 kg to less than 40 kg tak
22 erwent small intestinal motility (manometry; fasted and fed contraction frequency, phase III time) an
26 oxo1, mice adapted appropriately to both the fasted and fed state, and insulin suppressed hepatic glu
27 bolic and behavioral differences between the fasted and fed state, and reshapes the way nutrients are
30 spillover of chylomicron-triglyceride in the fasted and fed states, and FA stored in liver lipid drop
36 ignificant lower blood glucose levels in the fasted and post-prandial states, indicating a role for p
37 psies was used to measure MPS under baseline fasted and postprandial conditions in both a rested (res
39 oxidation, and net liver fat content in the fasted and postprandial states, we used stable-isotope t
40 lite profiling, we identified changes during fasted and refed conditions in a diverse array of mitoch
42 t with this model have been reported in both fasted and refed mice, however the impact of the fasting
46 ression were assessed in the postabsorptive (fasted) and postprandial (fed; 480 kcal, 20 g protein) s
47 tomated fit performed reliably in untreated, fasted, and ketamine-xylazine-treated mice, with no stat
51 ugh the citric acid cycle first increased in fasted animals from 35 to 71% for glucose and from 17 to
53 l activation and metabolism, as T cells from fasted animals had low glucose uptake and decreased abil
55 her leptin addition to cultured T cells from fasted animals or leptin injections to fasting animals w
56 st of these studies were performed utilizing fasted animals prior to perfusion so that a simplified m
57 ution from exogenous lactate did increase in fasted animals, but its overall contribution remained sm
60 e short-term survival during hypoglycemia in fasted animals, the CRR must overcome this energy-sparin
61 the diencephalic area of old animals and in fasted animals, whereas NPY increased sharply; (d) centr
64 revealed that mIndy was induced in livers of fasted as well as in high-fat-diet-streptozotocin diabet
65 d 13,298 viral hepatitis negative adults who fasted at least 4 hours using the nonalcoholic fatty liv
66 insulin sensitive than WT controls; however, fasted Atp7b (-/-) mice exhibited hypoglycemia after adm
68 controls, superfused hypothalamic slices of fasted birds treated with CART-peptide showed a signific
69 erences in average daily oxygen consumption, fasted blood glucose or plasma free fatty acids, but fas
76 d dried blood spots in a follow-up of 13,879 fasted children aged 11.5 years (interquartile range 11.
77 mpact of fuel type, an independent overnight fasted cohort was scanned before and after administratio
78 his assumption, TG levels decreased in CM of fasted compared with nonfasted CM-Plin5 mice indicating
79 aim of this study was to measure the basal (fasted) concentrations of unmetabolized folic acid in th
81 lucose uptake in BAT under thermoneutral and fasted conditions, a response that was further potentiat
82 rabinoside were twice as bioaccessible under fasted conditions, suggesting lipid-rich matrices select
85 3-(3)H]glucose, and [U-(14)C]alanine in 20 h-fasted conscious ZDF and their lean littermates (ZCL) un
87 e congenic parental strain (Ahr(Fx/Fx)), non-fasted Cre(Alb)Ahr(Fx/Fx) mice exhibit a 4-fold increase
92 were elevated in SI stem/progenitor cells of fasted etoposide-treated mice, which importantly correla
95 tent increase in IN1 interneuron activity in fasted flies that decreased proportionally in response t
100 en subjected to calorie restriction and then fasted for 23 hours, the L-Ghr (-/-) mice, but not the F
103 oups that were placed on a normal chow diet, fasted for 24 hours, or fasted for 24 hours and then fed
104 nduced gut barrier dysfunction, WT mice were fasted for 48 hours +/- IAP supplementation in the drink
105 1) (non-satiation feeding, NSF) for 4 weeks, fasted for 4d (F) and then fed to satiation (SF) for 21d
111 patic content of long-chain acylcarnitine in fasted Gpat1(-/-) mice was 3-fold higher than in control
116 0.047%/hour) compared with rates observed in fasted healthy controls (0.039%/hour; 95% CI, 0.029% to
118 ine modulates duodenal protein metabolism in fasted healthy humans, but its effects in a fed state re
120 herefore, a case-control study including 120 fasted, healthy, age- and gender matched subjects with/w
121 metastases) to oral ceritinib 750 mg per day fasted (in 21 day treatment cycles) or chemotherapy (int
122 tite and snack intake of postprandial versus fasted intranasal insulin administration to the brain in
123 pl] concentrations was analyzed in overnight-fasted lean and obese individuals subjected to a whole-m
124 ncentrations approximately 8-fold in 48-hour-fasted lean rats, and this normalization of plasma lepti
126 association study (GWAS) of circulating non-fasted lipoprotein lipid traits in the UK Biobank (UKBB)
127 s OXPHOS in fasted liver and may explain how fasted liver adapts to increased substrate oxidation.
128 MT1, specifically PRMT1V2, was stabilized in fasted liver and hepatocytes treated with glucagon, in a
129 drial transcription that regulates OXPHOS in fasted liver and may explain how fasted liver adapts to
130 etabolism and lipid oxidation, is induced in fasted liver mitochondria and implicated in metabolic sy
137 oxytocin (24 IU) administered to 29 healthy, fasted male subjects on glucose homeostasis measured by
140 Feeding and administration of glucose in fasted mice activated the vagal tone without affecting b
141 -ribosylated BiP in the inactive pancreas of fasted mice and a rapid decline in this modification in
143 sociated with reduced hepatic cholesterol in fasted mice and reduced bile acids (BAs) in feces, with
144 prostate tissue is stimulated by glucose in fasted mice and that release of Zn(II) can be monitored
146 In contrast, liver mitochondria from fed and fasted mice expressed little Acot activity, which was co
150 es of ad libitum-fed, diet-induced obese and fasted mice to the anorectic actions of leptin were exam
153 nbiased and targeted metabolomic analysis of fasted mice with a conditional knockout of ACOT7 in the
155 augmented histamine release in the cortex of fasted mice within a time window compatible to its anore
157 ophagy genes and inhibited autophagy even in fasted mice, and feeding-mediated inhibition of macroaut
158 nregulation of GLUT4 expression in vitro, in fasted mice, and in mice subjected to diet-induced obesi
159 patic Foxo1 mRNA and FOXO1 protein levels in fasted mice, as well as fasting blood glucose levels.
161 loss of CREBH decreased hepatic Fsp27beta in fasted mice, suggesting that CREBH plays a critical role
162 as with calorie-free "food," in both fed and fasted mice, suggesting that it is driven by the act of
163 neered lung and pancreatic cancer tumours in fasted mice, the contribution of circulating lactate to
164 hromatography MS/MS to analyze the hearts of fasted mice, we observed decreased BCAA-catabolizing enz
174 y in 14 subjects with metabolic syndrome who fasted (no eating or drinking) from dawn to sunset for m
177 to increased adipose tissue lipolysis, when fasted or fed a high-fat low-carbohydrate ketogenic diet
178 Principal components analysis of either fasted or fed-state metabolites identified one factor af
183 ter (D(h)) of 24.6 +/- 0.4 nm examined under fasted (pH 2) and fed (pH 5) gastric conditions using na
185 lood glucose or plasma free fatty acids, but fasted plasma insulin and the homeostatic model assessme
186 nborn defects in fatty acid oxidation and of fasted PPARalpha-deficient mice and while death was unaf
188 asis defect correlates with the inability of fasted RagA(GTP/GTP) neonates to trigger autophagy and p
190 4E with eIF4G was maintained in the liver of fasted rats as well as in serum-deprived mouse embryo fi
197 in RAAS signaling between the stimulated and fasted rats, suggesting that acute hyperinsulinemia incr
200 ts analysis factor in the fasted state (PCF2-Fasted), reflected a wide range of acylcarnitines and wa
201 was also 45% lower (P<0.05) for fed than for fasted, regardless of dietary protein and energy manipul
203 metabolism to fatty acid oxidation, and the fasted response occurs much more rapidly in pregnant wom
205 n to fed subjects (Fed-Ghrelin) and fasting (Fasted-Saline) significantly increased the appeal of hig
207 total cholesterol to HDL cholesterol in the fasted serum (P = 0.03) and postprandial serum (P = 0.01
208 d hepatic and circulating levels of FGF21 in fasted SIRT1 LKO mice were associated with reduced hepat
209 k1 expression were reduced in hypothalami of fasted Snord116 paternal knockout (Snord116p-/m+) mice.
213 principal components analysis factor in the fasted state (PCF1-Fasted), which was heavily weighted b
214 principal components analysis factor in the fasted state (PCF2-Fasted), reflected a wide range of ac
218 sis were obtained while patients were in the fasted state and every 30 min for 4 h following meal ing
219 gulation of fatty acid oxidation both in the fasted state and in mice consuming a high-fat, low-carbo
220 h increased levels of lymphatic GLP-1 in the fasted state and increased levels of intestinal Akkerman
221 ty acid oxidation and gluconeogenesis in the fasted state and lipogenesis and glycolysis in the fed s
225 ice were similar to wild-type animals in the fasted state but paradoxically decreased after refeeding
226 ed less NLRP3 inflammasome activation in the fasted state compared with that in refed conditions.
227 istration in the postprandial but not in the fasted state decreased appetite as well as intake and ra
228 nsulin-resistant and obese db/db mice in the fasted state displayed elevated lipogenic gene expressio
229 measurement and manometry were performed in fasted state for 20 minutes and for 90 minutes following
231 329 variables measured in 15 subjects in the fasted state identified one factor, the principal compon
235 ituated amounts of protein intake affect the fasted state of, and the stimulatory effect of a protein
236 rticipants (14 female, 16 male; in overnight fasted state on both scanning days) performed a willingn
238 04% compared with 0.039% +/- 0.007%/h in the fasted state to 0.062% +/- 0.005% compared with 0.057% +
239 04% compared with 0.039% +/- 0.007%/h in the fasted state to 0.062% +/- 0.005% compared with 0.057% +
242 its oral bioavailability was measured in the fasted state with high and low oral doses, before and af
243 orted to protect against hypoglycemia in the fasted state, but previous data have not linked the two.
250 uantitative analysis reveals that during the fasted state, the contribution of glucose to tissue TCA
251 ning mouse livers, we have found that in the fasted state, the Mediator complex exists primarily as a
262 ional MRI (fMRI) scans were performed in the fasted state; the blood oxygen level-dependent (BOLD) re
264 es metabolic rate and food intake in fed and fasted states and suppresses glucose production without
265 food valuation signals differ in the fed and fasted states between persons with high dietary disinhib
268 describing the perfused livers under fed and fasted states, the proposed approach successfully determ
276 glucose level also was observed in overnight-fasted, streptozotocin-treated ghrelin receptor-null mic
279 chemical markers were performed in overnight-fasted subjects before and after the 6-mo treatment peri
280 led alanine into another subgroup of 60-hour fasted subjects to increase rates of alanine turnover, s
284 ient (RQ) by indirect calorimetry during the fasted to fed transition (e.g. mixed meal challenge) or
285 bese mice and during the transition from the fasted to the fed state, suggesting that the regulation
287 In all participants, FRS decreased from the fasted to the satiated state in the cingulate (P < 0.005
295 changes in pol III occupancy in the liver of fasted versus refed wild-type mice are largely confined
297 fold induction of Dies1 transcript in WAT of fasted vs. fed mice, suggesting a role for Dies1 in nutr
299 ts analysis factor in the fasted state (PCF1-Fasted), which was heavily weighted by the PA:OA ratio o
300 As a model for a migrating songbird, we fasted zebra finches (Taeniopygia guttata) that had been