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1 causative agent of Pierce's disease (Xylella fastidiosa).
2 ers, phytoplasmas, spiroplasmas, and Xylella fastidiosa.
3 aused by the Gram-negative bacterium Xylella fastidiosa.
4 cquisition, retention, and inoculation of X. fastidiosa.
5 egy for the control of diseases caused by X. fastidiosa.
6 antivirulence effects of XfDSF itself in X. fastidiosa.
7 ysis of Xfas53, a temperate phage of Xylella fastidiosa.
8 ve regulation of DSF synthesis by RpfF in X. fastidiosa.
9 ransmit the bacterial plant pathogen Xylella fastidiosa.
10 targets for preventing diseases caused by X. fastidiosa.
11 ted decreased symptoms after infection by X. fastidiosa.
12 the valve in sharpshooter vectors of Xylella fastidiosa.
13 A similar protein has been found in Xylella fastidiosa, a bacterium that infects grapes, citrus and
14 Here we show that OMVs produced by Xylella fastidiosa, a xylem-colonizing plant pathogenic bacteriu
16 xylem-limited pathogenic bacterium, Xylella fastidiosa, acts as a priming stimulus in Vitis vinifera
18 ll Xanthomonas species as well as in Xylella fastidiosa and the human pathogen, Stenotrophomonas malt
19 ell into the active site of Ohr from Xylella fastidiosa and were efficiently reduced by the recombina
21 only xylem-feeding specialists vectors of X. fastidiosa (and probably P. syzygii), when many leafhopp
22 f X. campestris were replaced by those of X. fastidiosa, and the contribution of each gene to the ind
23 plants and vectoring to new host plants, X. fastidiosa apparently coordinates these traits in a popu
24 ylem network, the downward spread of Xylella fastidiosa bacteria in grape stems was modeled, and reve
26 and inoculation mechanisms are linked to X. fastidiosa biofilm formation and fluid dynamics in the f
27 Citrus Tristeza Virus (CTV) and iii) Xylella fastidiosa, both causing great economic loss worldwide.
28 approach to characterize the secretome of X. fastidiosa, both in vitro and in planta, and identified
29 OMV production thus is a strategy used by X. fastidiosa cells to adjust attachment to surfaces in its
30 or-dependent quorum-sensing system, and a X. fastidiosa DeltarpfF mutant in which quorum signaling wa
33 ysaccharides, are important in regulating X. fastidiosa gene expression and mediating vector transmis
35 sA as one of the pathogenicity factors of X. fastidiosa in grapevines that leads to leaf scorching an
38 the vector-borne bacterial pathogen Xylella fastidiosa in olive hosts, which was first identified in
43 ion of the lethal bacterial pathogen Xylella fastidiosa into healthy crop plants, causing economicall
49 lent response in grapevines observed when X. fastidiosa is disrupted for production of PrtA, and that
50 e strains from two distinct subspecies of X. fastidiosa is indicative of recent horizontal transfer,
55 dition of 50 to 100 microM Cu to standard X. fastidiosa media increases biofilm, while higher concent
56 ures of xylem vessels, we discovered that X. fastidiosa migrates via type IV-pilus-mediated twitching
58 ombination between a widespread family of X. fastidiosa P2-related prophage elements and a podophage
61 egrading enzymes likely to be produced by X. fastidiosa (polygalactuoronase and endo-1,4- beta -gluca
62 ing enzyme impacts on PMP and how a small X. fastidiosa population, introduced into grapevines by ins
65 P31758 are the mostly closely related non-X. fastidiosa proteins to most of the Trb proteins encoded
66 e history of advances in research on Xylella fastidiosa provides excellent examples of how paradigms
71 es of DPP-7 were found in genomes of Xylella fastidiosa, Shewanella putrefaciens, and P. gingivalis.
72 rization of the first virulent phages for X. fastidiosa, siphophages Sano and Salvo and podophages Pr
73 ment measures have caused some slowing of X. fastidiosa spread, stronger measures will be required to
75 Xfas53 was isolated from supernatants of X. fastidiosa strain 53 and forms plaques on the sequenced
76 to that of pXFAS01 from X. fastidiosa subsp. fastidiosa strain M23; the two plasmids vary at only 6 n
77 s of ~4 x 10(-12) ml cell(-1) min(-1) for X. fastidiosa strain Temecula 1 and ~5 x 10(-10) to 7 x 10(
80 almost identical to that of pXFAS01 from X. fastidiosa subsp. fastidiosa strain M23; the two plasmid
81 5) was present in the Riv5 strain of Xylella fastidiosa subsp. multiplex isolated from ornamental plu
82 omosomal and plasmid (pXF51) sequences of X. fastidiosa subsp. pauca strain 9a5c and more distant sim
83 k Decline Syndrome (OQDS), caused by Xylella fastidiosa subspecies pauca, affected the Salento olive
84 es hxfA and hxfB to a DeltarpfF strain of X. fastidiosa, suggesting that RpfF is involved in XfDSF se
85 campestris, Xanthomonas oryzae, and Xylella fastidiosa T2S also occurs in nonpathogenic bacteria, fa
87 subsequent to the introduction of subspecies fastidiosa to the United States in the late 19(th) centu
88 gely on the ability of the bacterium Xylella fastidiosa to use cell wall-degrading enzymes (CWDEs) to
91 of stems infected with the bacterium Xylella fastidiosa was compared with the PMP of healthy stems.
94 oic acid (c2-HDA), a DSF produced by Xylella fastidiosa, was the most potent among those tested, repr
95 rs of the plant pathogenic bacterium Xylella fastidiosa Wells et al. (Xanthomonadales: Xanthomonadace
96 es of the phytopathogenic bacterium, Xylella fastidiosa, which causes serious disease in plants, incl
97 olysaccharides mediate gene regulation in X. fastidiosa, which results in phenotypic changes required
101 ation of the xylem-limited bacterium Xylella fastidiosa (Xf) within xylem vessels is the sole factor
102 ease (PD) of grapevines is caused by Xylella fastidiosa (Xf), a xylem-limited gamma-proteobacterium t
103 to protect against the gram-negative Xylella fastidiosa (Xf), which causes diseases in multiple plant
104 ented by its own DSF, the DSF produced by X. fastidiosa (XfDSF) did not restore expression of the XfD
106 of this chimera to target the Xf subspecies fastidiosa (Xff), which causes Pierce disease in grapevi