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1 chemo- and/or radiation therapy included the fastigial and interposed nuclei (NF and NI).
2                                         Each fastigial cell type is connected with a specific set of
3  by modular synaptic connections of distinct fastigial cell types with posturomotor, oromotor, positi
4 oth buds, thymic epithelium, pontine nuclei, fastigial cerebellar nuclei, and cerebral cortex.
5 ce of brainstem afferents terminating in the fastigial cerebellar nucleus and/or ventrolateral reticu
6 e, we find that neurons of the mouse medial (fastigial) cerebellar nuclei (mCbN), which fire spontane
7                    Our results indicate that fastigial control of hippocampal seizures does not requi
8 l raphe nuclei (DRN) send projections to the fastigial deep cerebellar nuclei (fDCN) and that photost
9                         Conversely, however, fastigial excitation robustly attenuated hippocampal sei
10 alyses of vermis output neurons in the mouse fastigial (medial cerebellar) nucleus, we identify five
11 mand optogenetic excitation of glutamatergic fastigial neurons either ipsilateral or contralateral to
12                Though Purkinje cell input to fastigial neurons is inhibitory, direct optogenetic inhi
13                                  Glycinergic fastigial neurons make functional projections to vestibu
14                                   Excitatory fastigial neurons project to a large number of downstrea
15        We find, as previously reported, that fastigial neurons project to numerous brain regions via
16 ind that on-demand optogenetic excitation of fastigial neurons projecting to the central lateral nucl
17 tral lateral nucleus of the thalamus-but not fastigial neurons projecting to the reticular formation,
18                                  We examined fastigial neurons projecting to the superior colliculus,
19        In contrast, on-demand stimulation of fastigial neurons targeting the central lateral nucleus
20 ter when selectively targeting glutamatergic fastigial neurons than when an approach that lacked cell
21 nsitivity in the vestibular (VN) and rostral fastigial nuclei (FN) using high-frequency (2 Hz) oscill
22                         Projections from the fastigial nuclei to the periaqueductal grey area were re
23  flocculus, ventral paraflocculus, bilateral fastigial nuclei, and uvula.
24 ostral contralateral anterior interposed and fastigial nuclei, but did not label cells within the ves
25 tor output neurons in the medial cerebellar (fastigial) nuclei are in fact glycinergic, not glutamate
26            The caudal part of the cerebellar fastigial nucleus (CFN) influences the horizontal compon
27 s executed during inactivation of the caudal fastigial nucleus (cFN) on one side of the cerebellum.
28     Electrical stimulation of the cerebellar fastigial nucleus (FN) elevates regional cerebral blood
29 at, electrical stimulation of the cerebellar fastigial nucleus (FN) for 1 h reduces the volume of foc
30     Electrical stimulation of the cerebellar fastigial nucleus (FN) in spontaneously hypertensive (SH
31                               The cerebellar fastigial nucleus (FN) was electrically stimulated for 1
32 cited, would, like stimulation of cerebellar fastigial nucleus (FN), produce central neurogenic neuro
33 am projection of vermal Purkinje cells - the fastigial nucleus - in order to determine whether increa
34                                   The caudal fastigial nucleus also is necessary for the recovery of
35                              Both the caudal fastigial nucleus and the flocculus/paraflocculus are ne
36 rea) to rostral [vasopressor] regions of the fastigial nucleus and ventral reticular formation were r
37 bitory, direct optogenetic inhibition of the fastigial nucleus had no effect on seizure duration.
38 t that stimulating excitatory neurons in the fastigial nucleus may be a promising approach for therap
39 c inhibition of glutamatergic neurons in the fastigial nucleus of the cerebellum does not alter hippo
40 he pontomedullary junction as well as in the fastigial nucleus of the cerebellum.
41 c excitation of glutamatergic neurons in the fastigial nucleus successfully inhibits hippocampal seiz
42 s solitarius, the ventrolateral medulla, the fastigial nucleus, and the hypothalamus.
43 ulations via their projections to the caudal fastigial nucleus, and uncovered a simple-spike populati
44 f reference frames for motion in the rostral fastigial nucleus, in contrast to cells in the vestibula
45 ing that the posterior vermis and the caudal fastigial nucleus, to which it projects, provide a signa
46  by electrical stimulation of the cerebellar fastigial nucleus, we found that prohibitin is upregulat
47 l of the deep cerebellar nuclei, the rostral fastigial nucleus, were compared with those in the brain
48 al anterior interpositus nucleus and rostral fastigial nucleus.
49  consisting of the cerebellar vermis and the fastigial nucleus.
50 emoreceptor sites, with the exception of the fastigial nucleus.
51 lus, and the oculomotor region of the caudal fastigial nucleus.
52 ermine whether increases and/or decreases in fastigial output can underlie seizure cessation.
53          Rather, selective modulation of the fastigial output channel to the central lateral thalamus
54  not require simultaneous modulation of many fastigial output channels.
55 approach to selectively label and manipulate fastigial outputs.
56 nd selective neuronal loss within the mature fastigial pathway of the deep cerebellar nuclei (DCN), a