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1 and methyl palmoxirate (MP; an inhibitor of fat metabolism).
2 atty acid oxidation products (preference for fat metabolism).
3 e did not evaluate alterations to energy and fat metabolism.
4 cell cycle control, cytokine signaling, and fat metabolism.
5 ance occurs may be related to alterations in fat metabolism.
6 iabetes and may be related to alterations in fat metabolism.
7 athetic overactivity and regulation of brown fat metabolism.
8 cultured pancreatic islets to regulate their fat metabolism.
9 tions between nutritional Cu homeostasis and fat metabolism.
10 capacity and downregulated genes involved in fat metabolism.
11 ng that the ills of inactivity extend beyond fat metabolism.
12 the body is not sufficiently thrifty in its fat metabolism.
13 pathway and linolenic acid, cholesterol, and fat metabolism.
14 the benefits of exercise, at least regarding fat metabolism.
15 ssociated with gastrointestinal motility and fat metabolism.
16 rotein plays a role in controlling sugar and fat metabolism.
17 tinct germline anomalies affect lifespan and fat metabolism.
18 ncreased glycolysis, energy expenditure, and fat metabolism.
19 ed a possible role of mIndy in human hepatic fat metabolism.
20 is coupled with the production of energy via fat metabolism.
21 icating a complex link between FTO, IRX3 and fat metabolism.
22 boundaries and regulate different aspects of fat metabolism.
23 hormone that also plays a regulatory role in fat metabolism.
24 mitochondrial H(2)O(2) produced by increased fat metabolism.
25 organism's health, from immune regulation to fat metabolism.
26 c processes including glucose regulation and fat metabolism.
27 ed in the control of glucose homeostasis and fat metabolism.
28 a diet of 20% butter fat would have altered fat metabolism.
29 important for PGC-1alpha function and brown fat metabolism.
30 matase activity, and seems to be linked with fat metabolism.
31 bile salt interactions are likely to play in fat metabolism.
32 transcription factors and genes involved in fat metabolism.
33 ver have a novel direct effect on hepatocyte fat metabolism.
34 ggesting that endocannabinoids also regulate fat metabolism.
35 sclerosis, circulating lipids and intestinal fat metabolism.
36 iabetes and may be related to alterations in fat metabolism.
37 Mir-14 also regulates fat metabolism.
38 ng development, and in stress resistance and fat metabolism.
39 egulators of insulin action and of sugar and fat metabolisms.
40 a sympathetically-mediated increase in brown fat metabolism; (2) reduced thermogenesis probably contr
42 linking obesity-associated inflammation with fat metabolism and adaptation to energy stress in aggres
50 n both the mammalian stress response, and in fat metabolism and energy regulation, making it of incre
53 ithin the DGAT1 gene with known functions on fat metabolism and inflammatory response in dairy cattle
55 ncreatic lipase (PL) plays a central role in fat metabolism and is a validated target for weight loss
56 amma) is a transcription factor important in fat metabolism and is emerging as an important regulator
58 The mechanisms involve a metabolic shift to fat metabolism and ketone production, and stimulation of
61 had greater expression of genes involved in fat metabolism and lower expression of genes involved in
64 ated receptor-alpha), and a key regulator of fat metabolism, and 2 downstream genes: fat-5 and acs-2
66 protein response and altered host energy and fat metabolism, and eventually reduced survival to polym
69 echanisms underlying olfactory regulation of fat metabolism, and suggests the association between olf
70 cific fatty acids and physiological roles of fat metabolism are important subjects that are still poo
71 cetone levels that indicate intensified body fat metabolism, as validated by parallel venous blood be
72 munity, inflammation, glucose metabolism and fat metabolism, as well as on brain and heart function.
73 istance in muscle contributes to the altered fat metabolism associated with type 2 diabetes, but tiss
74 ga-6 fatty acids and regulatory functions of fat metabolism beyond its well-known role in energy stor
75 aining in body fat, resting carbohydrate and fat metabolism, blood pressure, VO(2max) and upper body
78 ous developmental input regulates intestinal fat metabolism by engaging mTORC2 signaling to promote t
80 and that this lavish approach for regulating fat metabolism contributes to the high incidence of pre-
81 rs for renal disease (arterial hypertension, fat metabolism disorder, and hyperglycemia) were not ass
82 ealthy humans, unlike rodents, regulation of fat metabolism does not require an adaptive response at
85 at cortisol may play a key role in increased fat metabolism during highly energetically demanding per
86 ndrial enzymes involved in cellular defense, fat metabolism, energy supply, and chaperones were ident
88 the dysregulation of a subset of glucose and fat metabolism genes which increase both glucose uptake
90 spectrum of peripheral functions, including fat metabolism, genetic examples are still needed to ill
91 physiological processes including apoptosis, fat metabolism, glucose homeostasis, and neurodegenerati
92 the assessment of food quality and signal to fat metabolism, growth, feeding behavior, reproduction,
96 symposium in May 2018 entitled "Obesity and Fat Metabolism in HIV-infected Individuals." Mechanisms
100 e coordinated regulation of carbohydrate and fat metabolism in liver by mechanisms that are independe
101 ncreases the size of its midgut and enhances fat metabolism in order to provide the energy needed for
102 roup of regulators that regulate glucose and fat metabolism in peripheral tissues and modulate inflam
108 ta on single substrate (pyruvate, succinate, fat) metabolism in both normal diet (CON) and high-fat d
109 n of gene expression involved in glucose and fat metabolism, including glucose transporter-4, hexokin
110 cond, it can improve body mass, glucose, and fat metabolism, increase insulin sensitivity, and decrea
111 ation of genes of mitochondrial function and fat metabolism indicated increased thermogenic activity,
112 regulated functional pathways for long-chain fat metabolism, indicating that these two enzymes are li
113 nown for its role in signaling satiation and fat metabolism-is increased in the lungs of obese mice a
114 CoA carboxylase, a key regulator of cellular fat metabolism known to be a part of the cytosolic subst
116 e show that this feeding behavior depends on fat metabolism mediated by the SREBP-SCD pathway, an ace
118 trengthens the pathophysiologic link between fat metabolism, mitochondria and Parkinson's disease.
119 tional roles of the lymphatic vasculature in fat metabolism, obesity, inflammation, and the regulatio
121 vering biosynthetic functions of a conserved fat metabolism pathway, alpha-oxidation, using C. elegan
124 es conveying larger body size and changes in fat metabolism, predisposing their QTP cold adaptation.
125 , have emerged as key players in glucose and fat metabolism previously thought controlled largely by
126 re hypophagic, suggesting that IGFII affects fat metabolism rather than feeding behavior in adult mic
127 a chromatin binding was reoriented away from fat metabolism-regulating genes when stimulated in the p
129 developmental transitions (EcR activity) and fat metabolism (SREBP activity) during the larval-pupal
131 t that ABCA7 plays a novel role in lipid and fat metabolism that Abca7-/- mice can be used to elucida
132 al gene expression, twitching phenotype, and fat metabolism through RNA interference toward gfp, sbp-
133 logical processes from insulin secretion and fat metabolism to cellular proliferation and differentia
134 transported from the intestine, the site of fat metabolism, to the oocytes yolk, which is a lipoprot
135 se overexpression (mCK-hLPL), which augments fat metabolism, to WLM and weight regain and found that
136 ansactivating isoform C/EBPbeta-LAP promotes fat metabolism under normal feeding conditions and expan
138 Most importantly, we found that the body fat metabolism was especially pronounced for most volunt
140 aging livers have alterations in glucose and fat metabolism, we examined a possible role of SIRT1 in
141 ne the effect of an ACC2 deletion on hepatic fat metabolism, we studied the regulation of the enzymes
143 ocyte hypertrophy, glycerol permeability and fat metabolism were studied in adipocytes isolated from
144 on or steatosis, due to dysregulated hepatic fat metabolism, which can progress to nonalcoholic steat
145 /PGC1alpha and Id1/Ebf2 in controlling brown fat metabolism, which has significant implications in th
146 ne concentrations, and 2) inadequate hepatic fat metabolism, which leads to steatosis (fatty accumula
147 conclude that TM6SF2 is a regulator of liver fat metabolism with opposing effects on the secretion of
148 xhibit postnatal growth retardation, altered fat metabolism with profound hyperglycerolemia and eleva
149 usly affects multiple aspects of glucose and fat metabolism within the muscle, which can adversely af
150 not all olfactory neurons actively regulate fat metabolism without affecting eating behaviors in Cae