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1 o and fasting insulin levels, but lower body fat).
2 idate alternative parameters to assess liver fat.
3 etect hepatic steatosis and quantify hepatic fat.
4 vels, indicating metabolically dysfunctional fat.
5 l, which focused on the reduction of dietary fat.
6 specifically promoting lipolysis in visceral fat.
7 e, comparable activity, and equivalent fecal fat.
8 role in cold-induced thermogenesis in brown fat.
9 r population leads to reduced innervation in fat.
10 ed in liver and down-regulated in muscle and fat.
11 and tuft cell markers were downregulated in fat-1 mice in response to EtOH, while defense responses
13 5% of energy; P = 0.02] and monounsaturated fat [12.4 wk (95% CI: 4.2, 20.5) per 5% of energy; P = 0
14 , 30% fat), high-fat overfeeding (HFOF) (60% fat, 20% carbohydrate), and high-carbohydrate overfeedin
15 ndependent respiration in subcutaneous white fat, 3) change the gut microbiota composition, and 4) pr
17 ollow-up, the HIV+ patients gained more body fat (8.6% +/- 0.7%) than the control patients (4.5% +/-
18 RNA2 signaling may activate glycolytic beige fat, a subpopulation of beige adipocytes mediated by GAB
21 urenine metabolism associated with abdominal fat accumulation to be a potential source of inflammatio
25 aSAT and correlated with a decrease in body fat and an increase in soleus and hepatic fat content (p
27 A close interplay between the peri-brachial fat and brachial dilatation can be translated into novel
29 ulation, PNPLA3 I148M and higher NAFLD liver fat and fibrosis scores were associated with increased l
30 ons in multiple pathways regulating glucose, fat and glycogen metabolism in Gprc6a(Liver-cko) mice.
31 at ages 5, 10, and 16/17 y; with percentage fat and percentage lean body mass at age 16/17 y; and wi
32 rographs of digesta also suggest the size of fat and protein in gastric phase to be smaller than in i
36 have demonstrated continued abnormalities in fat and/or lipid storage in PLWH treated with newer drug
40 t parameters of soy drink including protein, fat, and polyphenol content kept consistent during the s
42 rich nations, diets high in processed foods, fat, and sugar can contribute to chronic inflammatory co
44 us) also completed daily journals and a body fat assessment via dual-energy x-ray absorptiometry.
45 , in addition to the omental milky spots and fat-associated lymphoid clusters, in mice, the serous su
46 e of the proton peak area predominantly from fat at 56 days, mature after 270 days to be more yellow,
47 ociated with childhood BMIZ, percentage body fat at age 16/17 y, and a MetS score at age 16/17 y.
49 ondrial activity in mouse adipocytes and fly fat bodies with downregulated PI3K, which were confirmed
50 ncluding vitellogenin (Vg) production in the fat body and Vg uptake by maturing oocytes, is of great
51 e we report a perinuclear MTOC in Drosophila fat body cells that is anchored by the Nesprin homologue
52 glands from partially engorged females; (iv) fat body from partially and fully engorged females; and
56 also show that Dot1l is induced during brown fat cell differentiation and by cold exposure and that D
57 ) contains mitochondria-enriched thermogenic fat cells (brown adipocytes) that play a crucial role in
58 how consumption of low-fat milk and regular-fat cheese enriched in gamma-aminobutyric acid (GABA) in
61 tion of early childhood cow's milk volume or fat consumption with fracture risk in later childhood.
64 ibers also led to reductions in overall body fat content and improved glucose metabolism in mice fed
65 linked to the moisture loss increasing meat fat content and the culinary fat addition whose FA compo
66 gests that a 30% relative reduction in liver fat content as assessed by magnetic resonance imaging-pr
69 explore the possibility of predicting total fat content in whole dried cocoa beans at a single bean
70 study were to evaluate whether the volume or fat content of cow's milk consumed at 1-3 years of age w
73 on in neuromuscular diseases, and changes in fat content precede changes in function, which suggests
74 ns in Pro-C3, ELF, and cT1, but not in liver fat content, 7alpha-hydroxy-4-cholesten-3-one, or ALT, w
75 ared with <3 servings/d, irrespective of its fat content, while maintaining energy intake has no effe
76 was supplemented to ewes, and milk yield and fat content-fatty acid (FA) and phospholipid (PL) compos
79 s during heterogeneous and transient flow of fat crystal dispersion demonstrated that local constitut
80 that, Frizzled/Vang signaling couples to the Fat/Dachsous PCP directional signal in opposite directio
81 rend = 0.74) for 2 or more servings/d of low-fat dairy other than yogurt relative to <1 serving/mo an
83 et, which is rich in fruits, vegetables, low-fat dairy, and fiber and has low levels of saturated fat
85 identified PDGFD as a likely causal gene for fat deposition in the tails of sheep through transcripto
86 ]: 2.91-4.23) compared with those in the low-fat diet (1.16%; 95% CI: 0.80 to 1.98) with a difference
89 hese with the effects of a prototypical high-fat diet (HFD) as well as cessation of exposure on pheno
92 e membrane (MFGM-PL) supplementation to high-fat diet (HFD) rats during pregnancy and lactation could
93 .Leiden mice received 16 weeks either a high-fat diet (HFD) to induce obesity, or chow as reference g
97 uninephrectomy [UniNx]) in mice reduced high-fat diet (HFD)-induced adipose tissue inflammation, ther
102 t to improve liver damage in mice fed a high-fat diet and in mice fed a methionine-choline-deficient
103 cer risk factors, such as high-sugar or high-fat diet and inflammation, impact cell competition-based
105 subcutaneous fat (VAT/SAT) ratio after high-fat diet challenge, in comparison to their wild-type cou
106 viously determined that male mice fed a high-fat diet exhibit macrophage infiltration into the hypoth
107 RNA2 signaling is activated after acute high fat diet feeding and this effect is manifested through b
108 athy was evaluated in rats fed a 45% kcal as fat diet for 8 weeks before administering streptozotocin
110 7A) mice that were fed either a chow or high-fat diet showed similar weight gain as the wild-type lit
111 tic transcriptional response in mice on high-fat diet treated with metformin was largely ablated by A
112 57BL/6J mice, 33 weeks old), fed with a high fat diet which increases adipose tissue favouring overwe
114 randomly assigned to receive chow diet, high fat diet with sugar in drinking water (Western diet- WD)
115 axonomic changes specific to the healthy low-fat diet) and others tracked with weight loss (7 taxonom
116 o groups and fed either i) control, CON (45% fat diet) ii) CON + MINO, iii) OLZ (45% fat diet with OL
118 sm in mice fed either regular chow or a high-fat diet, demonstrating that these metabolic effects are
120 endothelium-specific knockout mice and high-fat diet-fed mice to assess the role of endothelial AKAP
122 ion in both models, disturbed flow- and high fat diet-induced atherosclerosis, whereas Nck2 deletion
123 ic genes in BAT, and are protected from high-fat diet-induced obesity and development of insulin resi
124 treptozocin-induced type 1 diabetes and high fat diet-induced type 2 diabetes mouse models and liver-
134 d that when chimeric animals were fed a high-fat-diet, animals with low levels of chimerism showed a
136 cific MyD88 or IRAK2 deficiency reduced high-fat-diet-induced weight gain, increased energy expenditu
138 te in adipose and muscle tissues during high-fat diets and contribute to a state of local inflammatio
140 with rodent work showing that high saturated fat diets increase gliosis and neuroinflammation in rewa
141 ection from weight gain on standard and high-fat diets, and an adiposity-dependent improvement in glu
144 ) was related to traditional measurements of fat distribution, such as total fat mass at DXA, viscera
145 ton relaxation were studied in water-in-milk fat emulsions during in situ cooling from 40 degrees C t
146 Here, we provide evidence that a thermogenic fat-epithelial cell axis regulates intestinal disease to
147 by magnetic resonance imaging-proton density fat fraction (MRI-PDFF) from baseline may be associated
148 nd magnetic resonance imaging proton density fat fraction (MRI-PDFF) in the detection of NASH in indi
149 patic steatosis quantified by proton density fat fraction (PDFF) on magnetic resonance imaging (MRI)
152 elated with fat fraction values; the optimal fat fraction threshold was determined to differentiate c
159 fruits, vegetables, whole grains, low-fat or fat-free dairy products, lean protein sources, nuts, see
162 diet group and by 7.02 mumol per kilogram of fat-free mass per minute (95% CI, 3.21 to 10.84) in the
163 from baseline, by 7.04 mumol per kilogram of fat-free mass per minute (95% confidence interval [CI],
164 (95% CI, 2.41 to 8.33) mumol per kilogram of fat-free mass per minute in the two groups, respectively
165 ng integrase inhibitor-based regimens), with fat gain due to restoration to health in antiretroviral
166 whether maternal polar lipids-enriched milk fat globule membrane (MFGM-PL) supplementation to high-f
169 of dairy products, whether low or regular in fat, has no overall effect on daytime ambulatory BP comp
170 sease (obesity/prediabetes) via chronic high-fat (HF) diet and modeled VCID via unilateral common car
172 nutrition was tested using chow-fed and high-fat (HF)-fed SkM-specific AMPKalpha1alpha2 knockout (mdK
173 e littermates to a fast food-mimicking, high-fat high-sucrose diet and profiled the metabolic phenoty
174 rd overfeeding (STOF) (50% carbohydrate, 30% fat), high-fat overfeeding (HFOF) (60% fat, 20% carbohyd
177 operative dietary prehabilitation with a low-fat, high-fiber diet reverses the impact of Western diet
182 loyed effectively for quantification of lard fat in butter fat samples with easy, robust, effective,
183 At 24 wk, percent energy from saturated fat increased from baseline in the HF group by 3.6%, (95
186 work mapped the distribution of neck muscle fat infiltration (MFI) in the deep cervical extensor mus
189 n AP cortisol, SAE cortisol, K10 scores, and fat intake among female participants and athletes were d
192 nalysis, and mediation analysis with CHO and FAT intakes as exposures and cg00574958 methylation as t
193 aimed to elucidate whether carbohydrate and fat intakes modulate cg00574958 methylation and the risk
195 ymes, fetuin-A, body composition, pancreatic fat, intramyocellular lipids, fecal SCFAs, blood pressur
199 mel milk samples) were analyzed for protein, fat, lactose and total solids by near and mid-infrared t
201 etion of adipose OGT causes a rapid visceral fat loss by specifically promoting lipolysis in visceral
204 all improvement in both BMI (-0.9+/-0.6) and fat mass (FM: -2.3+/-1.5), while lean body mass was pres
205 5.6%, respectively, P = 0.123), reduction in fat mass (mean difference, -1.537 kg; 95% CI, -2.947 to
206 ed food intake and body weight and preserved fat mass and lean mass during cachexia and LPS-induced a
207 induced urinary glucose loss include reduced fat mass and more ketone bodies as additional fuel.
209 surements of fat distribution, such as total fat mass at DXA, visceral and subcutaneous adipose tissu
213 ubjects was without effect on body weight or fat mass, but improved several measures of glucose homeo
215 een races and was positively correlated with fat-mass loss, but not with weight regain, overall.
216 orbance spectroscopy of samples flanking the fat, may be an important factor in cellular hydration an
218 s LC which influences the amount and type of fat migration to particle surface resulting in varying w
219 study aimed to assess how consumption of low-fat milk and regular-fat cheese enriched in gamma-aminob
221 ernational guidelines that recommend reduced-fat milk for children might not lower the risk of childh
225 lation of the additives in liver and adipose fat of 91 to 120,000 times the rate from the natural die
226 acterize ASCs isolated from the subcutaneous fat of domestic pigs (pASCs) and examine the effect of h
228 hasize fruits, vegetables, whole grains, low-fat or fat-free dairy products, lean protein sources, nu
229 d weight similarly, showed no differences in fat or lean mass accumulation, and displayed no changes
231 ales were fed control diet (CD; 10%kcal from fat) or HFD (60%kcal from fat) starting at post-natal da
232 r lipids, an increase in milk (total and low-fat) or yogurt consumption was positively associated wit
233 of concern (i.e., sugars, sodium, saturated fat, or energy) according to Chilean nutrient thresholds
234 ing (STOF) (50% carbohydrate, 30% fat), high-fat overfeeding (HFOF) (60% fat, 20% carbohydrate), and
236 survival of mice when injected into mammary fat pad of syngeneic mice, and demonstrated synergy when
237 or tissues were implanted into interscapular fat pads of NSG mice, and mice were given injections of
239 on full-fat dairy products high in saturated fat, particularly fermented dairy foods, demonstrates so
240 ight, body mass index (BMI), percentage body fat (PBF), and waist, hip, arm, and thigh circumferences
244 ts that differed in their relative levels of fat, protein and fibre content: an insect diet (low cont
245 ) of relative intake from the macronutrients fat, protein, carbohydrates, and sugar in over 235,000 i
247 ansverse relaxation values for the water and fat proton fractions and a higher relative %age of the p
248 on on crystallization kinetics and water and fat proton relaxation were studied in water-in-milk fat
249 To assess whether MRI with multipoint Dixon fat quantification allows for noninvasive differentiatio
251 f milk (p < 0.001); for protein (r >= 0.96), fat (r >= 0.99), lactose (r = 0.82) and total solids (r
252 insulin resistance [HOMA-IR]), trunk-to-leg fat ratio, resting energy expenditure, respiratory quoti
253 set of sensory neurons that act upstream of fat regulation as well as of central sleep-controlling n
259 ely for quantification of lard fat in butter fat samples with easy, robust, effective, low-cost and r
263 Guidelines recommend reducing saturated fat (SFA) intake to decrease cardiovascular disease (CVD
266 the AITC surface exchange rate and the AITC fat solubility, an overall picture of the factors influe
272 ycemic index of diet; lower intakes of trans fat, sugar-sweetened beverages/fruit juices, and red/pro
273 L enrichment without compromising total milk fat, suggesting strategies to improve dairy animals' mil
274 axation was observed in an artery with white fat (superior mesenteric artery) and in aorta from both
275 p 1 consisted of precontrast T1-weighted and fat-suppressed T2-weighted images (no contrast agent).
277 lated breath-hold examination sequences with fat suppression, performed between 2011 and 2015 were in
278 lting from the apple diet contained 50% more fat than larvae fed the fruit and spent grain mixtures.
279 sential amino acids can 1) promote the brown fat thermogenic program and fatty acid oxidation, 2) sti
282 d 2,4-di-tert-butyl-phenol (DBP), in humans (fat tissues, serum, urine, breast milk, and fingernails)
284 ally, despite harboring highly dysfunctional fat, transgenic mice display massive beta-cell hyperplas
285 t (KO) have a lower visceral to subcutaneous fat (VAT/SAT) ratio after high-fat diet challenge, in co
286 he effect of 2 healthy dietary patterns (low-fat versus Mediterranean diet) on the incidence of cardi
287 e were no differences in gender, BMI, % body fat, visual acuity or contrast sensitivity between those
289 Reductions in extraocular muscle, orbital fat volume, or both were observed in 6 patients in the t
291 ces with visceral and subcutaneous abdominal fat volumes, LSI, and FLD were assessed in linear and lo
299 esions endocardially and epicardially though fat while acutely sparing nearby the coronary arteries.