ライフサイエンスコーパス: fatty

1 The branched chain fatty acid (BCFA) concentration (15 to 18 ug) was almost

2 nted to ewes, and milk yield and fat content-fatty acid (FA) and phospholipid (PL) composition-were m

3 s of CE and stimulated triacylglycerol (TAG)/fatty acid (FA) cycling in WAT through impacting lipogen

4 by acyl-CoA thioester binding that optimizes fatty acid (FA) metabolism in response to environmental

5 The fatty acid (FA) profile of the CNO indicates an importan

6 ), the rate-limiting enzyme of mitochondrial fatty acid (FA) transport, is repressed by hypoxia-induc

7 This study assessed the impact of fatty acid (FA) type, phospholipid concentration on MM f

8 odules revealed highly activated glycolysis, fatty acid (FA), 2-monoacylglycerol (2-MAG), and membran

9 We previously showed omega-3 fatty acid (omega-3)-mediated repair of unfolded protein

10 Addition of a C16 fatty acid (palmitate) to lipid A by the outer membrane

11 Findings on prenatal polyunsaturated fatty acid (PUFA) intake and child wheeze and asthma hav

12 rRNA amplicon sequencing and for short-chain fatty acid (SFCA) analysis.

13 ated ambient temperature on ruminal volatile fatty acid (VFA) dynamics and rumen epithelium gene expr

14 rate-limiting enzyme of the very-long-chain fatty acid (VLCFA) beta-oxidation pathway in peroxisomes

15 17B12) as a human hub of the very-long-chain fatty acid (VLCFA) synthesis pathway and core interactor

16 a substantial fraction of the unusual trans fatty acid 16:1(Delta3trans) or 16:1t.

17 nescence, nonribosomal peptide biosynthesis, fatty acid activation, and beta-lactone formation.

18 nowledge, the first to indicate that the n-6 fatty acid AdA effectively blocks production of LTB(4) b

19 hibition of the anandamide-degrading enzyme, fatty acid amide hydrolase (FAAH), prolongs the regulato

20 hrough inhibition of its degradative enzyme, fatty acid amide hydrolase (FAAH), restored both synapti

21 iological processes, which are terminated by fatty acid amide hydrolase (FAAH).

22 were used for chemical gross composition and fatty acid analysis.

23 Mitochondrial function and fatty acid and glucose metabolism were impaired in HF-pa

24 While the pathways of diatom fatty acid and TAG synthesis appear to be well conserved

25 es hydrogen peroxide and the polyunsaturated fatty acid arachidonic acid are among the earliest known

26 HL) protein regulates lipid mobilization and fatty acid beta-oxidation during seed germination and se

27 he pathways of TAG catabolism and downstream fatty acid beta-oxidation have not been characterised in

28 to its inhibitive effects on the enzymes in fatty acid beta-oxidation.

29 Recent studies demonstrate that adipose fatty acid binding protein (FABP4) promotes obesity-asso

30 insights through quantitative exploration of fatty acid biosynthesis processes for optimal biofuels,

31 , mtACP, as a key component of mitochondrial fatty acid biosynthesis, is important in generating the

32 f acetyl-CoA carboxylase and polyunsaturated fatty acid biosynthesis.

33 n an increase in the circulating short-chain fatty acid butyrate and pain improvement following FMT.

34 ith elevated expression of genes involved in fatty acid catabolism.

35 presence of elevated amounts of unsaturated fatty acid chains.

36 vity (RSA), oxidative stability index (OSI), fatty acid composition and Maillard reaction products (M

37 resent a great heterogeneity of seed oil and fatty acid composition, accumulating Very Long Chain Fat

38 ), without altering seed quality traits like fatty acid composition, glucosinolates, oil and protein

39 cid starvation response and altered cellular fatty acid composition.

40 doplasmic reticulum omega-oxidation, a minor fatty acid degradation pathway known to be stimulated by

41 be explained by genetic variation within the fatty acid desaturase (FADS) gene cluster that is associ

42 74547 within the FADS1 gene, encoding FADS1 (fatty acid desaturase 1), with risk of several cardiovas

43 iation study (GWAS) revealed variants in the fatty acid desaturase 3 (FADS3) gene to be significantly

44 e dehydrogenases, a fatty-acid-CoA ligase, a fatty acid desaturase and associated oxidoreductases.

45 c acid (11Me-12t-18:1) by a newly identified fatty acid desaturase, UfaD.

46 s, laccases (AA1_1), xylanases (GH10, GH11), fatty acid desaturases and tannases.

47 (3-MCPDEs) and 2-monochloropropane-1,3-diol fatty acid esters (2-MCPDEs), and semi-quantification of

48 antification of 3-monochloropropane-1,2-diol fatty acid esters (3-MCPDEs) and 2-monochloropropane-1,3

49 MCPDEs), and semi-quantification of glycidyl fatty acid esters (GEs) in edible oils.

50 a highly active bacterial enzyme capable of fatty acid hydroxylation at a >3,000 min(-1) turnover ra

51 Erucic acid is an unwanted fatty acid in oil.

52 ile in X. muta there was higher abundance of fatty acid metabolic pathways.

53 olism', 'ceramides', 'creatine metabolism', 'fatty acid metabolism (acyl carnitine, polyunsaturated)'

54 molecular signatures indicative of enhanced fatty acid metabolism (FAM).

55 e pathways for fuel excess, the glycerolipid/fatty acid metabolism and the polyol pathway, were found

56 ately connected to intracellular pathways of fatty acid metabolism and therefore uniquely poised to c

57 EAE susceptibility, implicating short-chain fatty acid metabolism as a key element conserved across

58 substrate utilization from oxygen-consuming fatty acid metabolism toward oxygen-sparing glycolysis a

59 ncoupling of LDs from mitochondria, reducing fatty acid metabolism while increasing LD-bacterial cont

60 mediated ripening, including AOX, TCA cycle, fatty acid metabolism, amino acid metabolism, organic ac

61 val effects, primarily through angiogenesis, fatty acid metabolism, and hemopoesis pathways.

62 an uncharacterized branch of polyketide and fatty acid metabolism, encoding a large diversity of bio

63 n metabolic pathways, carbon metabolism, and fatty acid metabolism, likely descriptive of the epigene

64 rate-limiting enzyme for omega-3 and omega-6 fatty acid metabolism.

65 was an effective means to confirm the three fatty acid moieties of the TAGs, leading to the rapid an

66 ves were engineered through conjugation with fatty acid moieties that are known to exhibit high bindi

67 iander contains petroselinic acid, an isomer fatty acid of oleic acid.

68 Mitochondrial fatty acid oxidation (FAO) contributes to the proton mot

69 Increased fatty acid oxidation (FAO) has long been considered a cu

70 in this process through directly activating fatty acid oxidation (FAO) in the ground-state ESCs.

71 receptor CD36, accumulated lipids, and used fatty acid oxidation (FAO) instead of glycolysis for ene

72 Fatty acid oxidation (FAO) is a key bioenergetic pathway

73 etic organ: lymph gland, we demonstrate that Fatty Acid Oxidation (FAO) is essential for the differen

74 lism involving oxidative phosphorylation and fatty acid oxidation (FAO) with substantial accumulation

75 Fatty acid oxidation activity and tricarboxylic acid (TC

76 lmost exclusively dependent on mitochondrial fatty acid oxidation as a consequence of mitochondrial c

77 CP1) in brown and beige adipocytes uncouples fatty acid oxidation from ATP generation in mitochondria

78 18, in regulating the expression of genes in fatty acid oxidation in humanized livers through its int

79 on of genes associated with mitochondria and fatty acid oxidation in RYR1 mutants when compared with

80 if nervonic acid alters markers of impaired fatty acid oxidation in the liver.

81 hway for IL-10 production, shifting from the fatty acid oxidation pathway conventionally utilized for

82 romote the brown fat thermogenic program and fatty acid oxidation, 2) stimulate uncoupling protein 1

83 pecific genes involved in TAG hydrolysis and fatty acid oxidation, and that PA relieves AHL4-mediated

84 in MCSFA-HFD, accompanied by increased basal fatty acid oxidation, maintained glucose metabolic flexi

85 reas OXPHOS(low) BAP1 mutant UM cells employ fatty acid oxidation.

86 ies were carried out in the gain-of-function fatty acid oxygenation 2 (fou2) mutant that, even when u

87 phingomyelins with fully saturated sphingoid-fatty acid pairs (RR Q4 versus Q1 = 3.15; 95% CI: 1.75,

88 Exposure of macrophages to the saturated fatty acid palmitate increased glycolysis and HIF-1alpha

89 ctroscopy, Pb is shown to be associated with fatty acid phosphate, sulfate, chloride, and carboxylate

90 biosynthesis, is important in generating the fatty acid precursor of lipoic acid biosynthesis.

91 by biosynthetic pathway, defined by (1) the fatty acid precursor, including linoleic acid (LA), arac

92 The fatty acid precursors had estimated mean concentrations

93 of FO and rapeseed oil (RO) with a specific fatty acid profile in broodstock diets, without altering

94 Extraction yield, fatty acid profile, nutritional impact and cholesterol c

95 g season with two diets containing different fatty acid profiles and their effects on reproductive ho

96 We compared the fatty acid profiles of the ovary to those of the mantle

97 and ILC3s through its cognate receptor free fatty acid receptor 2 (FFAR2).

98 ecular sensors for detection of two putative fatty acid receptors, G protein-coupled receptor 120 (GP

99 he regulation of important genes involved in fatty acid regulation, energy production, and hormone-me

100 t formulation since it presented the highest fatty acid retention (68.51, 65.47, 47.27, 53.68, 52.95,

101 ietary carotenoids and long-term RBC omega-3 fatty acid status, as well as common secondary clinical

102 We developed a detailed kinetic model of the fatty acid synthase (FAS) of Escherichia coli and paired

103 cruiting the histone demethylase Lsd1 to the fatty acid synthase gene promoter.

104 Correlating with this, administration of a fatty acid synthase inhibitor, cerulenin, also alleviate

105 ynthesis, one in the cytoplasm (catalyzed by fatty acid synthase, FASN) and one in the mitochondria (

106 cofactor protein that is required by Type II fatty acid synthases (FASs).

107 malonyl-CoA, the rate-limiting substrate for fatty acid synthesis (FAS), is produced in the soma and

108 We propose that Myc-regulated fatty acid synthesis is a valid target for therapy and/o

109 nd transcriptomics analysis, we identify the fatty acid synthesis pathway as a druggable susceptibili

110 nctions to the germline; there it is used in fatty acid synthesis to critically support embryonic dev

111 cycle were all significantly decreased, but fatty acid synthesis transcripts were increased, compare

112 Cells harbor two systems for fatty acid synthesis, one in the cytoplasm (catalyzed by

113 ilities afforded by functional redundancy in fatty acid synthesis.

114 Sciadonic acid (Scia) is a Delta5-olefinic fatty acid that is particularly abundant in edible pine

115 yltransferase, ACAT1) transfers a long-chain fatty acid to cholesterol to form cholesteryl esters tha

116 Fatty acid transport protein 4 (FATP4), a transmembrane

117 Docosahexaenoic acid (DHA) is a omega-3 fatty acid typically obtained from the diet or endogenou

118 sociated with overexpression of the critical fatty acid uptake and de novo lipogenesis genes Pparg, M

119 k among circadian rhythms, hypoxia response, fatty acid uptake, and NAFLD.

120 ilization, further activating the process of fatty acid uptake.

121 6 promoter to augment expression and enhance fatty acid uptake.

122 intermediate was a methylated diunsaturated fatty acid, (10E,12E)-11-methyloctadeca-10,12-dienoic ac

123 cates altered metabolic fluxes (Krebs cycle, fatty acid, carbohydrate, amino acid metabolism).

124 nstrated that nervonic acid, a C24:1 omega-9 fatty acid, predominantly acylated to sphingolipids, inc

125 GPR84 is a medium chain free fatty acid-binding G-protein-coupled receptor associated

126 n P2 is a peripheral membrane protein of the fatty acid-binding protein family that functions in the

127 Fatty acid-derived volatile organic compounds (FA-VOCs)

128 Incubation of organoids with free fatty acid-enriched media resulted in structural changes

129 Notably, inhibiting cPLA2 synergizes with fatty acid-free diet to restore immunogenicity and selec

130 y to detect receptors' locations and monitor fatty acid-induced receptor redistribution demonstrates

131 plexed SERS imaging platform in the study of fatty acid-receptor interactions and might provide funct

132 food products with lower saturated and trans-fatty acid.

133 s, we propose C15:0 as a potential essential fatty acid.

134 al. show how oncogene-driven fatty-acid synthesis favors Treg cells over effector T c

135 hydrogenases, two aldehyde dehydrogenases, a fatty-acid-CoA ligase, a fatty acid desaturase and assoc

136 f cell signaling and apoptosis-related ions [fatty acids (341.2100 and 382.3736 m/z) and glycerophosp

137 Free fatty acids (FA) are a vital component of cells and are

138 only, TAG contains 18-carbon polyunsaturated fatty acids (FA), but plants also produce oils with uniq

139 gradation of lipids in the fillets were free fatty acids (FFA), lipid hydroperoxides (PV) and thiobar

140 We find that acyl-CoAs, rather than free fatty acids (FFAs), are the preferred substrate for CvFA

141 he hydrolytic cleavage of TGs generates free fatty acids (FFAs), which can serve as energy substrates

142 This led to elevated plasma free fatty acids (FFAs), which were transported to the adipos

143 Production of hydroxy fatty acids (HFAs) in transgenic crops represents a prom

144 levels of long chain omega-3 polyunsaturated fatty acids (LC omega-3 PUFA) have been linked to major

145 concentrations of long-chain polyunsaturated fatty acids (LC-PUFAs), while in the center region marin

146 crobes, increasing production of short chain fatty acids (mainly acetate and lactate) and favoring gr

147 Medium-chain fatty acids (MCFAs) have in rodents been shown to have p

148 ls had higher amounts of n-3 polyunsaturated fatty acids (n-3 PUFA).

149 Omega-3 fatty acids (n-3 PUFAs) are essential for the functional

150 In addition, omega-3 polyunsaturated fatty acids (n-3 PUFAs) have been reported to act as bot

151 eneficial effects of omega-3 polyunsaturated fatty acids (n-3 PUFAs) in cardiovascular prevention hav

152 despite increasing circulating nonesterified fatty acids (NEFA), the main substrate for synthesis of

153 Herein we report that electrophilic nitro-fatty acids (nitro-oleic acid and nitro-conjugated linol

154 Supplementation with omega-3 polyunsaturated fatty acids (omega-3 PUFA) and low-dose aspirin (ASA) ha

155 nces in 22:0, 18:1 cis 9, and 14:0 13-methyl fatty acids (P <= 0.011).

156 was almost twice as much as polyunsaturated fatty acids (PUFA) concentration found in the polar lipi

157 eived a diet enriched in n-3 polyunsaturated fatty acids (PUFA), known to enhance nutritional quality

158 Polyunsaturated fatty acids (PUFAs) form cellular, mitochondrial, retina

159 sts supplementation with n-3 polyunsaturated fatty acids (PUFAs) may be efficacious for treatment of

160 , we show that the levels of polyunsaturated fatty acids (PUFAs), especially linoleic acid (LA) and e

161 drying caused a relative loss of short chain fatty acids (SCFA).

162 atment, PLPE increased levels of short-chain fatty acids (SCFAs) by enhancing abundance of SCFA-produ

163 ion of gut bacteria that produce short-chain fatty acids (SCFAs) through gut fermentation of fiber, i

164 Short chain fatty acids (SCFAs; e.g., acetate, propionate, and butyr

165 Magnaporthe oryzae, requires very-long-chain fatty acids (VLCFAs), which act as mediators of septin o

166 In PDAC, obesity and excess fatty acids accelerate tumor growth and increase metasta

167 -beta-treated PASMCs, glucose, glutamine and fatty acids all contributed carbons to the TCA cycle.

168 ntly higher concentrations of non-esterified fatty acids and beta-hydroxybutyrate than mid-postpartum

169 kin-1beta), rebalanced levels of short-chain fatty acids and bile acids, improved gut barrier integri

170 variety of distinct lipophilic moieties like fatty acids and cholesterol increases ASO accumulation a

171 types of PCMs, a specific focus is placed on fatty acids and fatty alcohols for their natural availab

172 d contributes to the de novo biosynthesis of fatty acids and Leu; peroxisome-localized ACN1 enables t

173 s provides not only amino acids, but sugars, fatty acids and nucleotides for biosynthesis, conferring

174 Among cutin compounds, omega-hydroxy fatty acids and polyhydroxy-fatty acids were specificall

175 tically with CD36 in sensing polyunsaturated fatty acids and promoting Ca(2+) mobilization, further a

176 l biomolecules (e.g. omega-3 polyunsaturated fatty acids and sterols) render Synechococcus poor prima

177 ced autophagy and promoted beta-oxidation of fatty acids and stimulated gene expression of acyl-CoA d

178 The heart primarily consumed fatty acids and, unexpectedly, little glucose; secreted

179 Short-chain fatty acids are processed from indigestible dietary fibe

180 Specifically, exogenous polyunsaturated fatty acids are rapidly incorporated into membrane lipid

181 stimulated by exposure to nonesterified free fatty acids at concentrations observed in obese subjects

182 esses sn-1 specificity, with polyunsaturated fatty acids at the sn-2 position generating polyunsatura

183 Furthermore, short-chain fatty acids butyrate and propionate protect against hepa

184 (slight differences only in some particular fatty acids distributions: C10:0, C12:0 and C22:0) conce

185 c reprogramming that enhances utilisation of fatty acids for energy generation.

186 or rapid thermal dissipation and unsaturated fatty acids for membrane fluidity.

187 sed and increased, respectively, the flux of fatty acids from phospholipids and galactolipids into tr

188 Organoids incubated with free fatty acids had gene expression signatures similar to th

189 alcohols in O(1) class and subsequently to n-fatty acids in O(2) class.

190 c dates from each of the C(16:0) and C(18:0) fatty acids in pottery vessels provide an internal quali

191 d increased circulating levels of individual fatty acids in the plasma of Hi-Myc mice and human subje

192 grown in a light-dark cycle synthesized more fatty acids in their seeds.

193 lly with reference to the potential of these fatty acids in weight loss maintenance.

194 s, and that the specificity for longer chain fatty acids is regulated by a conformational change in t

195 n part through the production of short-chain fatty acids leading to Treg cell development, and merits

196 rain synthesized two structures with the C16 fatty acids located in acyloxyacyl linkage at the 2' and

197 olated, incubated with fluorescently labeled fatty acids or glucose analog, and analyzed by confocal

198 indolepropionic acid, serotonin, short chain fatty acids or tauroursodeoxycholic acid shows a similar

199 ing palmitic (C(16:0)) and stearic (C(18:0)) fatty acids purified by preparative gas chromatography(5

200 of lipoprotein subclasses; and 81 lipid and fatty acids ratios.

201 is of IHTG and partitioning of the resulting fatty acids toward ketogenesis (+232%) due to reductions

202 The strongest association of individual fatty acids were for palmitoleic acid (25.3 wk; 95% CI:

203 Sixteen fatty acids were identified in PSO.

204 Fatty acids were measured at visit 1 (1987-1989); and co

205 in), free fatty acids, or galactolipid-bound fatty acids were poor substrates.

206 Thirty-four fatty acids were quantified and their concentrations and

207 s, omega-hydroxy fatty acids and polyhydroxy-fatty acids were specifically affected, while the reduct

208 il (Coil) contains 50-60% of polyunsaturated fatty acids which are susceptible to oxidation.

209 We determined serum-PC fatty acids with gas chromatography.

210 evel analyses of associations of DNL-related fatty acids with incident T2D.

211 id composition, accumulating Very Long Chain Fatty Acids with industrial applications.

212 fying terpenoids embedded in a fluid matrix (fatty acids) comprising nonpolar and polar portions serv

213 groups, as well as all PUFA (polyunsaturated fatty acids) proportions.

214 s as secretagogues (it does so partially for fatty acids).

215 The health promoting omega-3, -7, and -5 fatty acids, alpha-linolenic acid (ALA), docosahexaenoic

216 hatic hydrocarbon resin), including alkanes, fatty acids, amides, and tackifying terpenoids embedded

217 Of the 51 lipids, fatty acids, and low-molecular-weight metabolites, 21 we

218 provided 230 metabolite measures: 51 lipids, fatty acids, and low-molecular-weight metabolites; 98 li

219 icate a potential role of AeOBP22 binding to fatty acids, and that the specificity for longer chain f

220 lular metabolism by suppressing oxidation of fatty acids, and thus adapts the cells to an avascular l

221 Metabolic analysis indicated changes in fatty acids, antioxidant capacity, biochemicals related

222 n triglycerides (MCT), containing C(8)-C(12) fatty acids, are used to treat several pediatric disorde

223 holipase A(2) (sPLA(2)) enzymes release free fatty acids, including arachidonic acid, and generate ly

224 discuss microbial regulation of short-chain fatty acids, neurotransmitters, as-yet-uncharacterized b

225 rition through the production of short-chain fatty acids, nitrogen recycling, and amino acid producti

226 while acyl-ACP (acyl carrier protein), free fatty acids, or galactolipid-bound fatty acids were poor

227 ches, permeation enhancers such as alkanols, fatty acids, prodrugs, and vesicular delivery for steroi

228 ow that the START domain binds to long-chain fatty acids, products of Them1's enzymatic reaction, as

229 elta(13)C and Delta(13)C values of the major fatty acids, provides chemical evidence for milk, meat,

230 .3 and 37.4% of alpha-linolenic and linoleic fatty acids, respectively.

231 ates an important content of polyunsaturated fatty acids, the most important being eicosapentaenoic a

232 ification of avocado puree with some hydroxy fatty acids, which deserved marked attention due to thei

233 precursors of acetyl-CoA are converted into fatty acids.

234 riven maternal investments, particularly key fatty acids.

235 tabolism through increased beta-oxidation of fatty acids.

236 , and marked reductions in cecal short-chain fatty acids.

237 idence interval (CI): 1.16 - 1.49%) of total fatty acids.

238 ted by esterifying isosorbide with sunflower fatty acids.

239 convoluting the nutritional effects of trans-fatty acids.

240 associated with the incorporation of unusual fatty acids.

241 nabolic flux from glucose to cholesterol and fatty acids.

242 aerobic fermentation to generate short-chain fatty acids.

243 ading to increased production of unsaturated fatty acids.

244 -carbon units into production of short-chain fatty acids.

245 dividual TFA levels as a percentage of total fatty acids.

246 ns allows for the determination of the lipid fatty acyl tail identities and positions, which is not p

247 curs when phospholipids with polyunsaturated fatty acyl tails are oxidized in an iron-dependent manne

248 complex that efficiently converts long chain fatty acyl-ACP/fatty acyl-CoA into hydrocarbon.

249 te entrances for each of the two substrates, fatty acyl-CoA and diacylglycerol.

250 ficiently converts long chain fatty acyl-ACP/fatty acyl-CoA into hydrocarbon.

251 cerol (TAG) synthesis, the esterification of fatty acyl-CoA to diacylglycerol.

252 enzyme A (CoA) diphosphatase that hydrolyzes fatty acyl-CoA to yield acyl 4'-phosphopantetheine.

253 volutionarily conserved ER protein FIT2 as a fatty acyl-coenzyme A (CoA) diphosphatase that hydrolyze

254 an intracellular transporter and buffer for fatty-acyl-CoA and is active in membrane assembly.

255 xylic acids and derivatives, polyketides and fatty acyls.

256 specific focus is placed on fatty acids and fatty alcohols for their natural availability, low toxic

257 teryl esters were positively correlated with fatty fish.

258 take ameliorating effects of alcohol-smoking-fatty food.

259 s DCS1 are effective for the preservation of fatty foods against lipid oxidation.

260 abetic milieu, including high glucose and/or fatty levels, and by the ablation of genes associated wi

261 ailure, but early risk factors for adulthood fatty liver are poorly understood.

262 Nonalcoholic fatty liver disease (NAFLD) affects a quarter of the adu

263 esistance), and the presence of nonalcoholic fatty liver disease (NAFLD) and nonalcoholic steatohepat

264 eity captured under the acronym nonalcoholic fatty liver disease (NAFLD) and provide suggestions on t

265 Nonalcoholic fatty liver disease (NAFLD) encompasses a range of condi

266 ), Forns (n = 122,419), and the nonalcoholic fatty liver disease (NAFLD) fibrosis scores (NFS, n = 13

267 eded to monitor key features of nonalcoholic fatty liver disease (NAFLD) in children that relate to i

268 Non-alcoholic fatty liver disease (NAFLD) is a frequent condition in o

269 In HIV-uninfected patients, nonalcoholic fatty liver disease (NAFLD) is associated with incident

270 Nonalcoholic fatty liver disease (NAFLD) is the most common pediatric

271 NASH) is a progressive form of Non-alcoholic fatty liver disease (NAFLD), a chronic liver disease wit

272 ciated with reduced severity of nonalcoholic fatty liver disease (NAFLD), based on histologic analysi

273 development of type 2 diabetes, nonalcoholic fatty liver disease (NAFLD), or cardiovascular disease.

274 ion against the development of non-alcoholic fatty liver disease (NAFLD).

275 a number of diseases, including nonalcoholic fatty liver disease (NAFLD).

276 ned popularity in management of nonalcoholic fatty liver disease (NAFLD).

277 which manifests in the liver as nonalcoholic fatty liver disease (NAFLD).

278 d metabolism and development of nonalcoholic fatty liver disease (NAFLD).

279 clock and promotes spontaneous non-alcoholic fatty liver disease (NAFLD).

280 nd fibrosis in mouse models of non-alcoholic fatty liver disease and advanced fibrosis, as well as to

281 apidly increasing prevalence of nonalcoholic fatty liver disease and its aggressive form, nonalcoholi

282 Nonalcoholic fatty liver disease is the most prevalent liver disease

283 -choline-deficient diet causing nonalcoholic fatty liver disease or to Lieber DeCarli diet causing et

284 , and SERPINA1) and key pathways involved in fatty liver disease pathobiology.

285 of non-alcoholic chronic liver failure from fatty liver disease to steatohepatitis are unavailable.

286 ncreasing rates of obesity and non-alcoholic fatty liver disease(2-4).

287 o cardiovascular disease, diabetes mellitus, fatty liver disease, and cancer.

288 uctural changes associated with nonalcoholic fatty liver disease, such as decay of bile canaliculi ne

289 ese findings may also apply to non-alcoholic fatty liver disease, which shares similar pathological a

290 ave only examined patients with nonalcoholic fatty liver disease.

291 of metabolic syndrome, such as non-alcoholic fatty liver disease.

292 e previously been implicated in nonalcoholic fatty liver disease.

293 ing population with increasing prevalence of fatty liver disease.

294 ated with the development of risk factors of fatty liver in adulthood.

295 The fatty liver index (FLI), a noninvasive steatosis biomark

296 Fatty liver is a preventable cause of liver failure, but

297 Fatty liver was determined by ultrasound during the last

298 tes, usually accompanied by dyslipidemia and fatty liver, as seen in lipodystrophies.

299 sy showed low endocardial voltages and fibro-fatty replacement in areas of late gadolinium enhancemen

300 The fatty tissue and carbonate shells were assessed for accu