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1 trols, P = 1.6 x 10(-9) versus MMR-unrelated febrile seizures).
2 7%, 47-66) of those children had a prolonged febrile seizure.
3 l appearing child who presents with a simple febrile seizure.
4 hannels may also predispose some children to febrile seizures.
5 ated with autosomal dominant transmission of febrile seizures.
6 ptor, is linked to generalized epilepsy with febrile seizures.
7 sociated with childhood absence epilepsy and febrile seizures.
8 eizures described in 393 reports included 94 febrile seizures.
9 ile convulsions, one had PFC and one had non-febrile seizures.
10 itive problems in individuals suffering long febrile seizures.
11 gions failed to show evidence for linkage to febrile seizures.
12  Eighteen affected individuals had recurrent febrile seizures.
13 s findings and rarely a history of childhood febrile seizures.
14 atients, only one had a history of childhood febrile seizures.
15    Of these, nine had a history of childhood febrile seizures.
16 bellar involvement, and 6 of 8 had childhood febrile seizures.
17 y and with history of hospital admission for febrile seizures.
18 uring childhood, including both epilepsy and febrile seizures.
19 ia-induced convulsions, a model of pediatric febrile seizures.
20 in a 5year-old with refractory epilepsy post-febrile seizures.
21  vaccination and controls with no history of febrile seizures.
22 to 6 years with FSE and controls with simple febrile seizures.
23 and reduced the frequency of spontaneous and febrile seizures.
24 prevention of epilepsy in some children with febrile seizures.
25 , but there is an association with childhood febrile seizures.
26 ) and without (overall n = 803) a history of febrile seizures.
27 , no association was found for rs7587026 and febrile seizures.
28 s for the management of children with simple febrile seizures.
29 ween mesial temporal sclerosis and prolonged febrile seizures.
30 n family with childhood absence epilepsy and febrile seizures.
31  are not a transient effect of the prolonged febrile seizures.
32 nts, in the majority of children with simple febrile seizures.
33 be evident in human children after prolonged febrile seizures.
34 ossible epileptic seizures and 220 as having febrile seizures.
35          43 148 individuals had a history of febrile seizures, 10 355 had epilepsy, and 1696 had both
36 tinct acute encephalopathy syndromes, simple febrile seizures (14), other seizures (16), acute ataxia
37 pilepsy was lower in patients with prolonged febrile seizures (14.3%, 6.3-29.4) and survivors of acut
38 ines is associated with an increased risk of febrile seizures 7 to 10 days following immunization amo
39 s associated with a 2-fold increased risk of febrile seizures 7 to 10 days following immunization com
40 acteristics including epilepsy (13/29, 45%), febrile seizures (7/29, 25%), generalized tonic-clonic s
41 ine was associated with an increased risk of febrile seizures 8 to 14 days after vaccination (relativ
42 e generalised or partial seizures (excluding febrile seizures), a new-onset focal neurological defici
43              We excluded infants with simple febrile seizures, acute provoked seizures, known acquire
44                                              Febrile seizures affect 2-4% of all children and have a
45  vaccination, 9 children were diagnosed with febrile seizures after the first (5.5 per 100,000 person
46                             Children who had febrile seizures after vaccination were followed to iden
47 iated with vaccination, the children who had febrile seizures after vaccination were not found to be
48 is family, defining the disease phenotype as febrile seizures alone.
49 clerosis findings and a history of childhood febrile seizures and (b) no MR mesial temporal sclerosis
50                     The relationship between febrile seizures and childhood immunizations is explored
51 ing the NaV1.1-R1648H mutation, which causes febrile seizures and epilepsy in humans.
52  were categorised according to occurrence of febrile seizures and epilepsy, before entering the follo
53 g of KCC2, found previously in patients with febrile seizures and epilepsy, has been demonstrated to
54 g the relationship of animal models to human febrile seizures and epilepsy.
55 g syntaxin-1B, that are associated with both febrile seizures and epilepsy.
56 r additional CPA6 mutations in patients with febrile seizures and focal epilepsy, which encompasses t
57  effect of nimodipine in an in vivo model of febrile seizures and found that this drug dramatically r
58 ave been identified that are associated with febrile seizures and generalized seizures with febrile s
59 ly healthy infant who presented with complex febrile seizures and improved on oseltamivir without neu
60 rrent knowledge of the mechanisms underlying febrile seizures and of changes in neuronal gene express
61         Combining a new rat model of complex febrile seizures and patch clamp techniques, we determin
62 a; (3) paroxysmal extreme pain disorder; (4) febrile seizures and recently (5) small fibre sensory ne
63                                        These febrile seizures and their potential contribution to the
64 fractory epilepsies, often with a history of febrile seizures and with frequent hippocampal atrophy a
65 s in GABRG2 have been associated with simple febrile seizures and with genetic epilepsy syndromes, in
66 mutations in human CPA6 were associated with febrile seizures and/or temporal lobe epilepsy.
67 nce of megalencephaly and ventriculomegaly, (febrile) seizures and autism-like behaviour.
68 trols, P = 1.2 x 10(-9) versus MMR-unrelated febrile seizures) and the measles virus receptor CD46 (r
69 ing for epilepsy type, sex, site, history of febrile seizure, and age at last observation.
70 ayed speech, a history of febrile and/or non-febrile seizures, and a wide-based, spastic, and/or stif
71 ced threshold and accelerated propagation of febrile seizures, and decreased threshold to flurothyl-i
72 oportion of viral infections associated with febrile seizures, and may be the primary cause of the se
73 obe epilepsy with hippocampal sclerosis with febrile seizures, and open avenues for investigation of
74 plained 2.8% of the variance in liability to febrile seizures, and the single nucleotide polymorphism
75                  A reduction in post-vaccine febrile seizures appears to be present since the introdu
76                                              Febrile seizures are associated with increased brain tem
77           Several rarer epilepsies featuring febrile seizures are caused by mutations in SCN1A, which
78 oseola occurs in a minority of patients, and febrile seizures are infrequently associated with primar
79 dwide incidence as high as 6.7% of children, febrile seizures are one of the most common reasons for
80  but the mechanisms underlying generation of febrile seizures are poorly understood.
81                                              Febrile seizures are the most common form of childhood s
82                                              Febrile seizures are the most common seizure disorder in
83                                              Febrile seizures are the most common seizure type in you
84                                              Febrile seizures are the most common type of development
85                   Seizures induced by fever (febrile seizures) are the most common type of pathologic
86 utations in these channels are implicated in febrile seizures associated with vaccination; and impair
87 obe epilepsy with hippocampal sclerosis with febrile seizures at the sodium channel gene cluster on c
88                                The number of febrile seizures attributable to the administration of D
89 A total of 7811 children were diagnosed with febrile seizures before 18 months, of whom 17 were diagn
90 d age-related seizures in early life such as febrile seizures, benign focal neonatal and infantile se
91 nsus febrile seizure phenotype, and none had febrile seizures beyond 6 years of age.
92 ing gamma2 subunit mutations associated with febrile seizures but not of wild-type alpha1beta2gamma2
93 eased risk of childhood seizures, especially febrile seizures, but not epilepsy.
94 rtussis vaccine carries an increased risk of febrile seizures, but whether this risk applies to the a
95 minent febrile seizures plus and early onset febrile seizures-but included more adults with focal epi
96                                              Febrile seizures can arise in response to fevers induced
97 on scans comparing children with MMR-related febrile seizures, children with febrile seizures unrelat
98 ry clinical discharge diagnoses (eg, sepsis, febrile seizures, dehydration, or other non-respiratory
99                                              Febrile seizure developed in 5 children the day after re
100  alterations in synaptic communication after febrile seizures does not support the prevalent view of
101 dies have shown that adult rats who suffered febrile seizures during development reveal memory impair
102 g a single episode of experimental prolonged febrile seizures during early postnatal development.
103 e series (SCCS) study based on children with febrile seizures during follow-up of the cohort.
104 erall, children did not have higher risks of febrile seizures during the 0 to 7 days after the 3 vacc
105                                        Early febrile seizures, episodes of status epilepticus as well
106 resenting with recurrent prolonged (>10 min) febrile seizures; febrile or afebrile status epilepticus
107 d infants with new-onset epilepsy or complex febrile seizures from IPCHiP centres, who were younger t
108 cal models for the prediction of the risk of febrile seizure (FS) and FS recurrence.
109 lepsy phenotypes, including Dravet syndrome, febrile seizures (FS) and genetic epilepsy with FS plus.
110 ts from an epilepsy GWAS meta-analysis and a febrile seizures (FS) GWAS are significantly more enrich
111                       Experimental prolonged febrile seizures (FS) lead to structural and molecular c
112 AE), juvenile myoclonic epilepsy (JME), pure febrile seizures (FS), generalized epilepsy with febrile
113                           Fever can provoke "febrile" seizures (FS).
114 urthermore, compared to controls with simple febrile seizures, FSE subjects with normal acute MRI had
115                                              Febrile seizures (FSs) are common neurological disorders
116                                              Febrile seizures (FSs) are the most common convulsion in
117                                 Whether long febrile seizures (FSs) can cause epilepsy in the absence
118                                 Whereas most febrile seizures (FSs) carry a benign outcome, a subpopu
119                                              Febrile seizures (FSs) constitute the most prevalent sei
120 lenges due to a recognized increased risk of febrile seizures (FSs) when used as the first dose in th
121                          In prior studies of febrile seizures (FSs), prolonged FSs were defined, abse
122  a subset of children experiencing prolonged febrile seizures (FSs), the most common type of childhoo
123 blems, including asthma, hyperbilirubinemia, febrile seizures, gastroenteritis, and, together with th
124 accelerated forgetting rate in the prolonged febrile seizure group.
125 articipant (2.9%, 0.5-14.5) in the prolonged febrile seizures group developed temporal lobe epilepsy
126  were distinctly associated with MMR-related febrile seizures, harboring the interferon-stimulated ge
127                                              Febrile seizures have been suspected to have a genetic b
128  was raised in individuals with a history of febrile seizures (hazard ratio [HR] 1.12, 95% CI 1.08-1.
129 in these families were generally benign, and febrile seizure history was infrequent (9.8%).
130 included associations of SCN1A with "complex febrile seizures" (HP: 0011172; p = 2.1 x 10(-5)) and "f
131                           After experimental febrile seizures (i.e., early in the epileptogenic proce
132 conducted a genome-wide association study of febrile seizures in 7635 cases and 83 966 controls ident
133 tly increased following experimental complex febrile seizures in developing rats, without a change in
134  (exacerbated by mutation) may contribute to febrile seizures in GEFS+ and perhaps normal individuals
135  Furthermore, four loci were associated with febrile seizures in general, implicating the sodium chan
136 ation, R82Q, that is associated with CAE and febrile seizures in humans.
137 crocephaly, intrauterine growth retardation, febrile seizures in infancy, impaired speech, stereotypi
138 V-6 infection is a common cause of fever and febrile seizures in infants.
139 y reduces both the incidence and duration of febrile seizures in rat pups, suggesting new possibiliti
140 abolishes the long-term effects of prolonged febrile seizures in the developing brain.
141 plication of CB1 receptor antagonists during febrile seizures in vivo blocked the seizure-induced per
142 netic variants influencing susceptibility to febrile seizures, including two loci specifically associ
143  occluded by previous in vivo fever-induced (febrile) seizures, indicating a common pathway.
144 arge Utah family with 21 members affected by febrile seizures inherited as an autosomal dominant trai
145 els, in which mild impairment predisposes to febrile seizures, intermediate impairment leads to GEFS+
146                         Experimental complex febrile seizures involving the immature rat hippocampus
147                                     Familial febrile seizures is caused by mild loss-of-function muta
148 course of an epilepsy; (ix) the prognosis of febrile seizures is generally good, with ~6-7% developin
149 ure rodent studies of experimental prolonged febrile seizures, isoform-specific changes in the expres
150 ping revealed significant evidence for a new febrile seizure locus (FEB3) on chromosome 2q23-24 with
151                                              Febrile seizures may be a complication in young children
152 A322D)beta2gamma2 receptors, suggesting that febrile seizures may be produced by a temperature-induce
153 lities in 26 children soon after a prolonged febrile seizure (median: 37.5 days) and compared their r
154  recommended schedule presents with a simple febrile seizure, minimal intervention should be the norm
155 tinct acute encephalopathy syndromes, simple febrile seizures (n = 14), other seizures (n = 16), acut
156 drawal, age at onset of epilepsy, history of febrile seizures, number of seizures before remission, a
157                                              Febrile seizures occurred after dose 1 of MMR vaccine at
158 servational study found an increased risk of febrile seizure on the day of or 1 day after vaccination
159 facilitate the enduring effects of prolonged febrile seizures on neuronal and network excitability.
160 ever generates seizures, the effects of long febrile seizures on neuronal function and the potential
161    There are significantly elevated risks of febrile seizures on the day of receipt of DTP vaccine an
162 ion was associated with an increased risk of febrile seizures on the day of the first 2 vaccinations
163 ine was associated with an increased risk of febrile seizures only on the day of vaccination (adjuste
164 ith prominent deja vu and without antecedent febrile seizures or magnetic resonance imaging abnormali
165 features were associated with BSN, including febrile seizures (p = 1.26e-06) and behavioral disinhibi
166 ased with a growing number of admissions for febrile seizures (p<0.0001) and with later onset of chil
167 ts with neuropsychiatric disease compared to febrile seizure patients in a general population sample.
168 ygenic risk of febrile seizures was lower in febrile seizure patients with neuropsychiatric disease c
169 ed with fever, consistent with the consensus febrile seizure phenotype, and none had febrile seizures
170 pilepsy, including generalized epilepsy with febrile seizures plus (GEFS+ type 2), severe myoclonic e
171 sorders, including generalized epilepsy with febrile seizures plus (GEFS+) and severe myoclonic epile
172 yndromes including generalized epilepsy with febrile seizures plus (GEFS+) and severe myoclonic epile
173 forms of epilepsy, generalized epilepsy with febrile seizures plus (GEFS+) and severe myoclonic epile
174 gada syndrome) and generalized epilepsy with febrile seizures plus (GEFS+) associated with C-terminal
175 tations that cause generalized epilepsy with febrile seizures plus (GEFS+) have been identified previ
176                        Genetic epilepsy with febrile seizures plus (GEFS+) is an autosomal dominant f
177                    Generalized epilepsy with febrile seizures plus (GEFS+) is caused by missense muta
178                    Generalised epilepsy with febrile seizures plus (GEFS+) is the most studied famili
179 re associated with the genetic epilepsy with febrile seizures plus (GEFS+) spectrum disorders in huma
180  two families with generalized epilepsy with febrile seizures plus (GEFS+) type 2.
181 ile seizures (FS), generalized epilepsy with febrile seizures plus (GEFS+), and Dravet syndrome (DS)/
182 X, associated with generalized epilepsy with febrile seizures plus (GEFS+), created a loss of functio
183 isorders including generalized epilepsy with febrile seizures plus (GEFS+),(7) severe myoclonic epile
184 d in families with generalized epilepsy with febrile seizures plus (GEFS+).
185 minantly inherited generalized epilepsy with febrile seizures plus (GEFS+).
186 identified, results in genetic epilepsy with febrile seizures plus (GEFS+).
187 ilepsy syndrome termed genetic epilepsy with febrile seizures plus (GEFS+).
188 f classical GEFS+ families-such as prominent febrile seizures plus and early onset febrile seizures-b
189 bility alleles for generalized epilepsy with febrile seizures plus and juvenile myoclonic epilepsy.
190 forms of epilepsy, generalized epilepsy with febrile seizures plus and severe myoclonic epilepsy of i
191 brile seizures and generalized seizures with febrile seizures plus pedigrees.
192 enetic disorder termed "Genetic Epilepsy and Febrile Seizures Plus" (GEFS(+)).
193 f epilepsy (GEFS+, generalized epilepsy with febrile seizures plus) in humans has been accounted for
194                 In generalized epilepsy with febrile seizures plus, an autosomal dominant epilepsy sy
195  absence epilepsy, generalized epilepsy with febrile seizures plus, and Dravet syndrome or severe myo
196 ibility allele for generalized epilepsy with febrile seizures plus, are also potentiated by these DHP
197  epilepsy syndrome generalized epilepsy with febrile seizures plus, which includes a spectrum of seiz
198 a primary cause of generalized epilepsy with febrile seizures plus.
199 es associated with generalized epilepsy with febrile seizures plus.
200 otypes associated with genetic epilepsy with febrile seizures plus.
201 ely milder syndrome of genetic epilepsy with febrile seizures plus.
202 l gene SCN1B linked to genetic epilepsy with febrile seizures plus.
203                      In a minority of cases, febrile seizures precede later development of epilepsy.
204                                              Febrile seizures probably do not represent a homogeneous
205 esistant strain of Streptococcus pneumoniae, febrile seizure rates following measles-mumps-rubella-va
206 ondition, the precise cellular mechanisms of febrile seizures remain unclear.
207                                              Febrile seizures represent a serious adverse event follo
208                                              Febrile seizures represent the most common type of patho
209       Children with a history of a prolonged febrile seizure show signs of acute hippocampal injury o
210 acute event or the duration of the prolonged febrile seizure, suggesting that the observed effect is
211         As compared with other children with febrile seizures that were not associated with vaccinati
212 ng that, in the hippocampus of rats that had febrile seizures, the long-lasting enhancement of the wi
213                Despite the high incidence of febrile seizures, their contribution to the development
214 usion, this largest genetic investigation of febrile seizures to date implicates central fever respon
215 h includes a spectrum of seizures types from febrile seizures to Dravet syndrome.
216  MMR-related febrile seizures, children with febrile seizures unrelated to vaccination and controls w
217                             The incidence of febrile seizures was 8 of 26 062 (0.031%) prior to initi
218                    However, a higher risk of febrile seizures was found on the day of the first (HR,
219 2(K289M), and gamma2(Q351X)] associated with febrile seizures was highly temperature dependent.
220     Finally, we found that polygenic risk of febrile seizures was lower in febrile seizure patients w
221 mutation [alpha1(A322D)] not associated with febrile seizures was not highly temperature dependent.
222        Using an appropriate-age rat model of febrile seizures, we investigated the acute and chronic
223 dissect out variants related to a history of febrile seizures, we tested cases with mesial temporal l
224 intrinsic firing may play a critical role in febrile seizures, we tested the effect of nimodipine in
225  found that higher polygenic risk scores for febrile seizures were associated with epilepsy and with
226 neurologic deficit, and history of childhood febrile seizures were recorded and correlated with MR fi
227                                              Febrile seizures were reported in only 2 patients (9.5%)
228          In a cohort of 172 individuals with febrile seizures, who did not develop epilepsy during pr
229  and GABRG2) have been identified that cause febrile seizures with or without epilepsy.
230                   Children with epilepsy and febrile seizures-with and without concomitant epilepsy-a
231                        Relative incidence of febrile seizures within 0 to 7 days (0, 1-3, and 4-7 day

 
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