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1 r humanized (ex-GF mice colonized with human fecal microbiota).
2 protocol for 16S ribosomal RNA sequencing of fecal microbiota.
3 ffects of co-habitation on skin than oral or fecal microbiota.
4 itivity to colitis and skewed composition of fecal microbiota.
5 ative PCR to monitor and quantify changes in fecal microbiota.
6 ing clinical challenge mostly originating in fecal microbiota.
7  agents that relatively spare the indigenous fecal microbiota.
8 gi, with mixed effects on rumen bacteria and fecal microbiota.
9 ice with colitis, via TLR4, and alters their fecal microbiota.
10 ficant alterations in the composition of the fecal microbiota.
11  is associated with differences in the human fecal microbiota.
12 es and in fecal preparations of mixed murine fecal microbiota.
13 an expansion of the genus Allobaculum in the fecal microbiota.
14                                              Fecal microbiota 16S rRNA genes, immune cell distributio
15 ced overlap with the deeply sequenced twins' fecal microbiota (18.3 +/- 0.3%, 15.3 +/- 0.3%).
16 th MaR, but not GaM, significantly decreased fecal microbiota abundance and diversity, and expanded t
17 assic approaches, we found no differences in fecal microbiota abundance or composition between patien
18 s and pectinase modulated the composition of fecal microbiota after in vitro fermentation, increasing
19                                              Fecal microbiota amplicon sequencing from matched indivi
20                                              Fecal microbiota analyses were performed on patients' st
21                                              Fecal microbiota analysis of 3 successful FT recipients
22                                              Fecal microbiota analysis showed reduced bacterial richn
23                                              Fecal microbiota analysis was performed using 16S riboso
24  levels of tryptophan and composition of the fecal microbiota, analyzed by 16S ribosomal DNA amplicon
25           Here, we explore links between the fecal microbiota and abdominal adiposity using body comp
26 amily member 3 alpha gene (REG3A) alters the fecal microbiota and affects development of colitis in m
27  association between low diversity childhood fecal microbiota and increased diabetes risk.
28                    The aim is to investigate fecal microbiota and metabolites at different ages in in
29 n child health and might modulate changes in fecal microbiota and metabolites.
30              We characterized changes in the fecal microbiota and metabolome to identify the mechanis
31 ctors, potentially through modulation of the fecal microbiota and metabolome.
32               We simultaneously measured the fecal microbiota and multiple environmental and host-rel
33 te temporal changes in the concentrations of fecal microbiota and short-chain fatty acids (SCFAs) in
34 dietary protein increased the density of the fecal microbiota and the severity of colitis in SPF mice
35 to 12 mo alters the 16S configuration of the fecal microbiota as read out by amplicon sequence varian
36                                 Diversity of fecal microbiota, assessed by Shannon index, did not dif
37        We carried out 16S-based profiling of fecal microbiota, assessed the expression of bile acid m
38                       The composition of the fecal microbiota associated with serum levels of tryptop
39 crolimus, and mycophenolate mofetil modified fecal microbiota at the family level in each experimenta
40                                              Fecal microbiota B-diversity segregated patients accordi
41 crobial responses were tested using in vitro fecal microbiota batch fermentations of five donors.
42 n was associated with the composition of the fecal microbiota, but not with constipation or colonic t
43         Metagenomic phylogenetic analysis of fecal microbiota by deep sequencing revealed considerabl
44                              We profiled the fecal microbiota by pyrosequencing the gene encoding 16S
45 iltrates from donor stool (FFT), rather than fecal microbiota, can be sufficient to restore normal st
46  results in reduced IL-18 levels and altered fecal microbiota characterized by expanded representatio
47                       Prednisolone disrupted fecal microbiota community structure, decreased Bacteroi
48 oos and hosted a strikingly low diversity of fecal microbiota compared to individuals born in natural
49   We did not find significant differences in fecal microbiota composition among patients with differe
50                                              Fecal microbiota composition analysis was performed in a
51         In the subcohort of 42 children with fecal microbiota composition analysis, the children with
52 g 16S ribosomal gene sequencing, we assessed fecal microbiota composition and diversity, as measured
53                                              Fecal microbiota composition and inferred function, feca
54                               Differences in fecal microbiota composition between groups were analyze
55 rked differences in the small intestinal and fecal microbiota composition between mice housed on clea
56                                  Analysis of fecal microbiota composition by 16S ribosomal RNA gene s
57                  One year after surgery, the fecal microbiota composition differed between CD patient
58                              The role of the fecal microbiota composition for the postoperative disea
59 lock component, abolished rhythmicity in the fecal microbiota composition in both genders.
60                                          The fecal microbiota composition of CD patients, analyzed by
61                                Secondly, the fecal microbiota composition of the African swine fever
62 ed the impact of initial alpha-diversity and fecal microbiota composition on beta-diversity instabili
63                                  Analysis of fecal microbiota composition showed an increase in bacte
64 e adjusted for confounding factors, we found fecal microbiota composition to be associated with devel
65 s of metabolic disease and inflammation, and fecal microbiota composition were assessed.
66 ces in asthma characteristics, food diaries, fecal microbiota composition, and levels of serum inflam
67 .Fecal and serum menaquinone concentrations, fecal microbiota composition, and plasma and fecal cytok
68        In this study, we investigated if the fecal microbiota composition, determined by a high throu
69 lly, delta-infected GSH exhibited an altered fecal microbiota composition, marked by increased inflam
70                                              Fecal microbiota composition, measured by 16S rRNA seque
71 ssed nutrient intake, food-related behavior, fecal microbiota composition, microbial fermentation, an
72 eral and adipose tissue insulin sensitivity, fecal microbiota composition, plasma and fecal SCFA, ene
73 , lifetime antibiotic use after weaning, and fecal microbiota composition.
74 ns in energy balance, blood metabolomics and fecal microbiota composition.
75 ndry and metagenomics to show that the human fecal microbiota consists largely of taxa and predicted
76 from healthy donors or were given autologous fecal microbiota (control); each transplant was administ
77                                              Fecal microbiota did not change significantly across all
78  and B-diversity of the 16S configuration of fecal microbiota did not differ between the 2 groups.
79 ently, here we demonstrate that the phyla of fecal microbiota differ substantially between L1-KO mice
80                                              Fecal microbiota diversity and richness were highest in
81                             In this setting, fecal microbiota, evaluated by 16S rRNA gene amplicon se
82 here has been growing interest in the use of fecal microbiota for the treatment of patients with chro
83                                              Fecal microbiota for transplantation (FMT) is being stud
84                              We transplanted fecal microbiota from adult female twin pairs discordant
85        Finally, we found that transplants of fecal microbiota from age-matched APPPS1-21 male mice in
86 tics and subsequently transplanted them with fecal microbiota from Casp1(-/-) mice based on a cohousi
87 ht to investigate whether transplantation of fecal microbiota from drug-free patients with schizophre
88 aised under standard conditions; exposure of fecal microbiota from L2-IL1B mice fed the HFD to L2-IL1
89                                          The fecal microbiota from mothers and their babies differed
90 T hosts that were recolonized with dysbiotic fecal microbiota from Nod2-deficient mice.
91 ecal microbiota from the mice which received fecal microbiota from patients with constipation also up
92                               In this study, fecal microbiota from patients with constipation and hea
93                      The mice which received fecal microbiota from patients with constipation present
94                                              Fecal microbiota from REG3A-TG mice protect non-TG mice
95 The results revealed that transplantation of fecal microbiota from schizophrenic patients into antibi
96                                   Moverover, fecal microbiota from the mice which received fecal micr
97                         We characterized the fecal microbiota from these two species by pyrosequencin
98 TMOP were transmissible by transplanting the fecal microbiota from TMOP-treated mice, indicating that
99                           Transplantation of fecal microbiota from women with PCOS or B. vulgatus-col
100  from different studies of the Western adult fecal microbiota generally clustered by study, and the 1
101                       Variation in the human fecal microbiota has previously been associated with bod
102 lonized with the patients' and the controls' fecal microbiota, highlighting 78 differentially enriche
103 015), and gamma-proteobacteria domination of fecal microbiota (HR, 2.64; 95% CI, 1.10-5.65; P = 0.031
104                                Assessment of fecal microbiota identifies patients at highest risk for
105     This study compared the structure of the fecal microbiota in 29 mestizo children aged 7-18 years,
106  provide novel insights into the role of the fecal microbiota in cardio-metabolic disease with clear
107                      We found that dysbiotic fecal microbiota in FA infants evolved compositionally o
108 circadian rhythmicity and composition of the fecal microbiota in mice.
109 of wheat and ATIs on severity of colitis and fecal microbiota in mice.
110 his study, we determine the structure of the fecal microbiota in mothers and neonates according to ma
111          We compared the colonic mucosal and fecal microbiota in patients with chronic constipation a
112    Anti-TNF therapy shifted the diversity of fecal microbiota in patients with IBD, but not with rheu
113 to determine its effects on symptoms and the fecal microbiota in patients with IBS.
114                                     Baseline fecal microbiota in patients with MPR were enriched with
115 ubstituting antibiotics, this study assessed fecal microbiota in pigs from different dietary treatmen
116 icrobiota are altered in MS by comparing the fecal microbiota in relapsing remitting MS (RRMS) (n = 3
117 one prebiotic formula on the same inoculated fecal microbiota in two frequently used batch systems: p
118 es from the mice transplanted with patients' fecal microbiota increased both kynurenic acid synthesis
119 to colonize GF mice shows a direct effect of fecal microbiota independent of active liver inflammatio
120   In conclusion, the structure of the infant fecal microbiota is affected by the maternal H. pylori s
121                                              Fecal microbiota is often used as a proxy for understand
122 s support the notion that disruptions to the fecal microbiota may help explain the observed effects o
123 ntries, which correlates with alterations in fecal microbiota (microflora) and widespread use of anti
124 spread use of antibiotics and alterations in fecal microbiota ("microflora").
125 emerging under ciprofloxacin pressure in the fecal microbiota, no proof of selection of quinolone-res
126               Therapeutic transplantation of fecal microbiota normalizes brain lesion-induced dysbios
127    A similar shift in the composition of the fecal microbiota occurred after a few months in TG mice
128 iple antimicrobial resistance genes), in the fecal microbiota of 147 mothers and their children sampl
129                                          The fecal microbiota of 2-month-old infants was considerably
130 acterize bacterial strain composition in the fecal microbiota of 37 U.S. adults sampled for up to 5 y
131 rtium of bacterial strains cultured from the fecal microbiota of a 6-month-old stunted Malawian infan
132 nce during the first 3 wk of life within the fecal microbiota of an infant born at 28-wk gestation.
133 sequencing, plasma/urine metabolomes and the fecal microbiota of Angus steers grazing non-toxic or E+
134                   In a longitudinal study of fecal microbiota of children from 5 weeks through 6 to 1
135                                          The fecal microbiota of domestic and wild suids was analyzed
136 ly, these data suggest that abundance in the fecal microbiota of facultative anaerobes, such as S. Ty
137 irus-like particles (VLPs) purified from the fecal microbiota of five healthy humans.
138                                          The fecal microbiota of Fut2(-) mice that lack fucosylated h
139 in (150 mg 4 times daily for 10 days) on the fecal microbiota of healthy humans for a period of 1 yea
140 al 16S ribosomal RNA gene sequences from the fecal microbiota of humans and 59 other mammalian specie
141                Autologous transplantation of fecal microbiota of Ldlr(-/-) mice served as control.
142                                          The fecal microbiota of mice that express hREG3A had a signi
143    Here we analyzed circadian rhythms in the fecal microbiota of mice using deep sequencing, and foun
144  pyridoxamine did not produce alterations in fecal microbiota of mice.
145                      Characterization of the fecal microbiota of patients undergoing allogeneic hemat
146                              We profiled the fecal microbiota of patients with CDAD (both initial and
147 reduced Clostridia abundance distinguish the fecal microbiota of pre-IBD patients from IBS patients.
148 lic consequences and specific effects on the fecal microbiota of protein and zinc deficiency were pro
149                                          The fecal microbiota of the hospitalized subjects had abnorm
150                                          The fecal microbiota of the whole group of patients with cir
151                                          The fecal microbiota of vaginally delivered infants (both ne
152                     However, the profiles of fecal microbiota only partially replicate those of the m
153 t received 100 capsules containing their own fecal microbiota or placebo until month 14.
154           Although no significant changes in fecal microbiota or SCFAs were observed during enteral f
155                      E+ grazing affected the fecal microbiota profile; +THI conditions modulated the
156                                              Fecal microbiota profiles from overweight (n = 29) and o
157 ne pre-treatment rectal gene expression, and fecal microbiota profiles, in 206 pediatric UC patients
158                       The groups had similar fecal microbiota profiles, serum markers of inflammation
159  significantly disturbed composition whereas fecal microbiota recovered a similar composition to cont
160 on deficiency altered the composition of the fecal microbiota, reduced mucosal barrier function, and
161         Reconstitution of GF mice with naive fecal microbiota reinstated morphine analgesic tolerance
162 roach begins by identifying uncultured human fecal microbiota samples that transmit immune phenotypes
163                  Sigmoid colonic mucosal and fecal microbiota samples were collected from 25 healthy
164                                           AN fecal microbiota showed a compositional predominance of
165                                              Fecal microbiota showed covariation with mucosal adheren
166                                          Two fecal microbiota signatures (Clostridium and Klebsiella
167              SIRT1 iKO mice also had altered fecal microbiota starting at 4-6 months of age compared
168  gnotobiotic mice colonized with a synthetic fecal microbiota, StrainR2 resolves strain abundances wi
169 NA-based analysis of the T-bet(-/-)Rag2(-/-) fecal microbiota suggest that the structure of the endog
170        We identified specific communities of fecal microbiota that are associated with the bioavailab
171 impact on the viability and diversity of the fecal microbiota that is recovered in the laboratory.
172  systematic compositional differences in the fecal microbiota that occurred with age and between West
173                                              Fecal microbiota transfer was associated with partial co
174 scuss how probiotics, prebiotics, synbiotic, fecal microbiota transfer, polyphenols, specific diets,
175 cal microbiome and were partially rescued by fecal microbiota transfer.
176                                              Fecal microbiota transfers from Tie2-Cre Arg1fl/fl mice
177                 We applied our strategy to a fecal microbiota transplant (FMT) donor stool using mult
178      We aimed to investigate the outcomes of fecal microbiota transplant (FMT) for relapsing CDI usin
179                                              Fecal Microbiota Transplant (FMT) has shown some success
180 pose of this article is to review the use of fecal microbiota transplant (FMT) in the treatment of pe
181                                              Fecal microbiota transplant (FMT) is a highly efficaciou
182                                              Fecal microbiota transplant (FMT) is recommended for Clo
183                                              Fecal microbiota transplant (FMT) reverses the course of
184  metronidazole, vancomycin, fidaxomicin, and fecal microbiota transplant (FMT).
185 mall intestinal cell shedding, whereas adult fecal microbiota transplant alone had no effect.
186 ulate various microbial communities, such as fecal microbiota transplant and bioreactor systems' opti
187 ehavioral symptoms in children with ASD, and fecal microbiota transplant could transform the dysbioti
188                                     Although fecal microbiota transplant has been used to prevent rec
189                                        While fecal microbiota transplant has gained increasing popula
190                                 Conventional fecal microbiota transplant is an adjuvant treatment for
191 eting of the gut microbiota (ie, by means of fecal microbiota transplant).
192 wel cleanse, a stomach-acid suppressant, and fecal microbiota transplant, and observed significant im
193 orientations of some invertons diverge after fecal microbiota transplant, potentially as a result of
194 zed therapeutic purposes, such as autologous Fecal Microbiota Transplant.
195 cacy and safety of diet-modulated autologous fecal microbiota transplantation (aFMT) for treatment of
196  We intend to review the most recent data on fecal microbiota transplantation (FMT) and critically di
197 rial to assess the safety and feasibility of fecal microbiota transplantation (FMT) and reinduction o
198 cal evaluation of the safety and efficacy of fecal microbiota transplantation (FMT) as a potential th
199                               In particular, fecal microbiota transplantation (FMT) as a treatment st
200 current Clostridium difficile infection with fecal microbiota transplantation (FMT) at a tertiary ref
201                                              Fecal microbiota transplantation (FMT) can induce remiss
202    We investigated the safety of weekly oral fecal microbiota transplantation (FMT) capsules from hea
203                                              Fecal microbiota transplantation (FMT) could be a novel
204                         We hypothesized that fecal microbiota transplantation (FMT) could be used to
205                      Through human-into-mice fecal microbiota transplantation (FMT) experiments from
206                                  The role of fecal microbiota transplantation (FMT) for Clostridium d
207 fficile infection (CDI) guidelines recommend fecal microbiota transplantation (FMT) for persons with
208 se recovery is apparent given the success of fecal microbiota transplantation (FMT) for recurrent CDI
209 e CDI, fidaxomicin for first recurrence, and fecal microbiota transplantation (FMT) for subsequent re
210 ral case series have reported the effects of fecal microbiota transplantation (FMT) for ulcerative co
211 tudies suggest that weight may be altered by fecal microbiota transplantation (FMT) from a lean donor
212                                              Fecal microbiota transplantation (FMT) from cancer patie
213                                              Fecal microbiota transplantation (FMT) has been demonstr
214                                              Fecal microbiota transplantation (FMT) has been shown to
215                                              Fecal microbiota transplantation (FMT) has been shown to
216         High-intensity, aerobically prepared fecal microbiota transplantation (FMT) has demonstrated
217         Our understanding and utilization of fecal microbiota transplantation (FMT) has jump-started
218 cerative colitis (UC) patients responsive to fecal microbiota transplantation (FMT) have reduced phag
219 round: To date, evidence for the efficacy of fecal microbiota transplantation (FMT) in recurrent Clos
220                           BACKGROUND & AIMS: Fecal microbiota transplantation (FMT) is a highly effec
221                                              Fecal microbiota transplantation (FMT) is a highly effec
222                                              Fecal microbiota transplantation (FMT) is a highly effec
223 se and replacement of the microbiota through fecal microbiota transplantation (FMT) is a promising ap
224                                              Fecal microbiota transplantation (FMT) is a promising, b
225                                              Fecal microbiota transplantation (FMT) is an alternative
226                                              Fecal microbiota transplantation (FMT) is an emerging th
227                                              Fecal microbiota transplantation (FMT) is highly effecti
228                                              Fecal microbiota transplantation (FMT) is highly effecti
229                                              Fecal microbiota transplantation (FMT) is more effective
230                                              Fecal microbiota transplantation (FMT) is recommended fo
231                                              Fecal microbiota transplantation (FMT) is used commonly
232                                              Fecal microbiota transplantation (FMT) may improve dysbi
233                      We tested the effect of fecal microbiota transplantation (FMT) on obesity, and p
234                                        While fecal microbiota transplantation (FMT) presents an attra
235 difficile infections (rCDI) are treated with fecal microbiota transplantation (FMT) provided by healt
236                           PURPOSE OF REVIEW: Fecal microbiota transplantation (FMT) re-establishes a
237 expression is regulated by the microbiota as fecal microbiota transplantation (FMT) rescues antibioti
238                                              Fecal microbiota transplantation (FMT) targeting gut mic
239 the reported promising results of open-label fecal microbiota transplantation (FMT) therapy in patien
240                    We report a case in which fecal microbiota transplantation (FMT) utilized for rela
241 rs of antibiotic-resistant microbes and that fecal microbiota transplantation (FMT) would reduce the
242                                              Fecal microbiota transplantation (FMT), a safe, effectiv
243 roaches to modifying this ecosystem, such as fecal microbiota transplantation (FMT), are being develo
244 e analyze data from two published studies of fecal microbiota transplantation (FMT), finding that hig
245 difficile infections (rCDI) are treated with fecal microbiota transplantation (FMT), using feces prov
246  of the organism has led to increased use of fecal microbiota transplantation (FMT).
247 d as antibiotics, probiotics, synbiotics, or fecal microbiota transplantation (FMT).
248 n, 15.4% vs vancomycin, 25.3%; P = .005) and fecal microbiota transplantation (response rates of 83%-
249 most current and best methods for performing fecal microbiota transplantation and summarize clinical
250 es, with recent clinical trials highlighting fecal microbiota transplantation as a strategy to enhanc
251 a diverse intestinal microbial population by fecal microbiota transplantation attenuates disease and
252 developed and higher quality data to support fecal microbiota transplantation for treating recurrent
253 udy reported higher than 80% success rate of fecal microbiota transplantation for treatment of recurr
254 ready been applied to the clinic, such as in fecal microbiota transplantation for treatment of recurr
255 el diseases (IBDs), where clinical trials of fecal microbiota transplantation have shown some efficac
256 ive therapies like probiotics, curcumin, and fecal microbiota transplantation in the management of mi
257                                              Fecal microbiota transplantation is an efficacious and i
258                                              Fecal microbiota transplantation is another emerging the
259                                              Fecal microbiota transplantation is associated with symp
260                                              Fecal microbiota transplantation is increasingly used to
261                                              Fecal microbiota transplantation led to resolution of re
262    Taken together these results suggest that fecal microbiota transplantation may be a treatment opti
263                                              Fecal microbiota transplantation may have a substantial
264 robes using nonabsorbable antibiotics and by fecal microbiota transplantation of dysbiotic cecal cont
265 responses by treatment with either a healthy fecal microbiota transplantation or defined commensal ba
266  mother-infant pairs and patients undergoing fecal microbiota transplantation to evaluate the pattern
267                  Third, we report our use of fecal microbiota transplantation to face severe Clostrid
268                                Intervention: Fecal microbiota transplantation with donor stool (heter
269 such as probiotics, prebiotics, antibiotics, fecal microbiota transplantation, and gene manipulation
270 unities for treatment of other diseases with fecal microbiota transplantation, based on findings from
271                                              Fecal microbiota transplantation, performed in one patie
272                                        After fecal microbiota transplantation, the SERT expression in
273                                              Fecal microbiota transplantation, where healthy donor st
274 iseases and findings from clinical trials of fecal microbiota transplantation.
275 commended laboratory screening practices for fecal microbiota transplantation.
276 nd could be transferred to germ-free mice by fecal microbiota transplantation.
277 ridium difficile infection as effectively as fecal microbiota transplantation.
278 or example, administration of probiotics and fecal microbiota transplantation.
279 ow that p-crAssphage can be acquired through fecal microbiota transplantation.
280 ty and reduced fitness that were restored by fecal microbiota transplantation.
281              We also discuss case reports of fecal microbiota transplantations for different disorder
282                                Donor-derived fecal microbiota treatments are efficacious in preventin
283           We observed a global alteration of fecal microbiota upon dietary AhR ligand deprivation.
284       We assessed brain activation patterns, fecal microbiota, urine metabolome profiles, serum marke
285 ygen species (ROS) in bacterial cultures and fecal microbiota using 2',7'-dichlorofluorescein diaceta
286                                              Fecal microbiota varied with diet; the concentration of
287                           The profile of the fecal microbiota was associated with colonic transit and
288                           The profile of the fecal microbiota was associated with colonic transit bef
289                                              Fecal microbiota was characterized before surgery and on
290 enters, the diversity of early pretransplant fecal microbiota was lower in patients than in healthy c
291  representation of these 14 duodenal taxa in fecal microbiota was significantly different from that i
292                                              Fecal microbiota were analyzed by V3-V4 16S ribosomal RN
293 ological, immunohistological parameters, and fecal microbiota were analyzed.
294                 Diversity and composition of fecal microbiota were determined by 16S ribosomal RNA ge
295 fects of interventions and breast-feeding on fecal microbiota were investigated.
296                Changes in the composition of fecal microbiota were not associated with liver fat or m
297                        Taxonomic profiles of fecal microbiota were obtained prospectively, and conven
298 after each of the treatments, and changes in fecal microbiota were quantified.
299 w that the abundances of Escherichia coli in fecal microbiota were significantly higher in young chil
300 lthy donors and those who received their own fecal microbiota, which may be due to limited numbers.

 
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