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1 ine (below the cut-off value of 10 mug Hb/ g feces).
2 erocytes, cause diarrhea, and be shed in the feces.
3 e evaluated on four different pools of human feces.
4 fects on the microbiota composition of human feces.
5 egg density plateau of about 1000 eggs/g of feces.
6 t correlate with a higher fungal load in the feces.
7 obotic platform for direct analysis of human feces.
8 nation of virus shedding in oropharynges and feces.
9 cteroidetes, and increased Firmicutes in the feces.
10 reater in secondary environments compared to feces.
11 g rates of pathogens originating from animal feces.
12 rstanding the molecular composition of human feces.
13 host-associated bacteria in animal or human feces.
14 ed in seawater and in gull, cat, and raccoon feces.
15 secretions, oviposition materials, and even feces.
16 HDM extracts, as well as purified bodies and feces.
17 Pseudomonadaceae and Shewanellaceae in their feces.
18 e of viable E. coli populations in livestock feces.
19 groups 1, 6, 18, and 23 well represented in feces.
20 subsequently deposited on the host with the feces.
21 discovered shared by chicken, pig and human feces.
22 significant ARG enrichment in adult chicken feces.
23 the animals but are mostly released with the feces.
24 n facility that adequately retains or treats feces.
25 IgEs to allergens present in mite bodies and feces.
26 d, as assessed by detection of SIVcpz RNA in feces.
27 spp. and Clostridiaceae in high weight hard feces.
28 d animal (avian, cattle, poultry, and swine) feces.
29 approach to extract metabolic information in feces.
30 , and Akkermansia spp. were enriched in soft feces.
31 ermine the overall metabolite composition of feces.
32 igh RNA copy numbers detectable in serum and feces.
33 ion, were measured in intestinal tissues and feces.
34 genomic and metatranscriptomic sequencing of feces.
35 ios at which nutrients are deposited through feces.
36 transfer of cholesterol from macrophages to feces.
37 for screening the metabolite composition in feces.
38 e nitrogen-to-phosphorus ratios of herbivore feces.
39 Infected badgers shed M. bovis in their feces.
40 he abundance of distinct microbial genera in feces.
41 or, subsequent to, FIT >15 mug hemoglobin/g feces.
42 versity of antimicrobial resistance genes in feces.
43 hemical signatures from animal carcasses and feces.
45 positivity thresholds <=10 mug hemoglobin/g feces, 10 to <=20 mug/g, 20 to <=30 mug/g, and >30 mug/g
48 10), and green turtles ingested more debris (feces; 15.8 +/- 33.4 g, gut; 39.8 +/- 51.2 g) than logge
49 each participant, duplicate meals (0-96 h), feces (24-120 h), and indoor/outdoor dust (<250 mum) wer
50 children who died [median (IQR): 1360 mg/kg feces (2443-535 mg/kg feces) compared with 698 mg/kg fec
51 cial debris appeared in all green turtles in feces (25/25) and gut contents (10/10), and green turtle
52 erovirus RNA was more commonly identified in feces (42 of 44 [95%]), rectal swabs (35 of 37 [95%]), a
53 s, viral RNA was detected more frequently in feces (80%) and particularly in blood (85%), and antivir
57 oth enamel and, together with vegetation and feces, analyzed for delta(26)Mg, delta(13)C, Sr/Ca, and
60 rformed RNA-Seq on exfoliated cells found in feces and compared these data to RNA-Seq from both the s
63 (ADE) was calculated from ADDM and the GE of feces and diet; apparent digestibility of fat (ADfat) wa
64 on biopsies and exfoliated colonocyte RNA in feces and fecal microbial community composition, and to
67 uency of occurrences of artificial debris in feces and gut contents collected from loggerhead turtles
69 es, Deferribacteres, and Spirochaetes in the feces and increased abundance of Pasteurella in the orop
72 These changes in bacterial abundance in the feces and oropharynx correlated with lower asthmatic res
73 between congener-specific concentrations in feces and serum were found for all BDEs except BDE-197 a
75 ples and recovery of viable virus from mouse feces and small intestine suggest that these pests may p
78 MF(lim) by controlling the Z-values of their feces and the volume reduction of the food in the gastro
79 lyzed to assess topographical homogeneity of feces and to evaluate storage duration-, temperature-, a
80 the excretion and treatment of human waste (feces and urine) in low and middle income countries (LMI
85 l cultures (e.g., intestinal contents, human feces) and reduce TMAO levels in mice fed a high-choline
86 ive result from an FIT (>15 mug hemoglobin/g feces) and subsequent colonoscopy (reference standard) w
87 feces), at a uniform threshold (15 mug Hb/g feces), and at adjusted thresholds yielding defined leve
88 ence of heavy infections (>=400 eggs/gram of feces), and total prevalence being particularly importan
90 e orally dosed compound is eliminated in the feces, and a major fraction of the absorbed compound is
91 At baseline and every 4 wk, blood, urine, feces, and anthropometric and body composition measures
94 L did not develop infection or shed virus in feces, and IgG anti-HEV antibody levels were unchanged (
98 thyl hydroxychromanol (alpha-CEHC) in urine, feces, and plasma that were catabolized from administere
100 testinal [GI]) colonization, shedding within feces, and transmission of K. pneumoniae through the fec
102 lth risks associated with exposure to animal feces; and factors influencing concentrations and sheddi
103 ors related to points of contact with animal feces; animal fecal contamination of food; cultural beha
105 mination is likely to be minimal unless bird feces are deposited close to the land-sea interface.
106 the polysaccharide compositions of food and feces are determined, further illustrating the utility o
108 study investigated the feasibility of using feces as a noninvasive matrix to estimate serum concentr
112 es, revealing coprostanol, a proxy for human feces, as the most abundant sterol (seawater: 45.1-20.3
113 rus was stable in the absence of bacteria or feces at most temperatures, M132V virus was stabilized b
114 tyrate and propionate (>=95th percentile) in feces at the age of one year had significantly less atop
117 turers' thresholds (range, 2.0-17.0 mug Hb/g feces), at a uniform threshold (15 mug Hb/g feces), and
119 (oculomotor) avoidance of disgusting images (feces) before and after an "exposure" intervention (mone
122 wn as nutrient sources through deposition of feces, but also may deposit large quantities of energy i
125 externalities; for example, safe disposal of feces by one household prevents disease transmission to
126 creased levels of 2 miRNAs and hemoglobin in feces can identify patients with AAs or CRC more accurat
127 shedding SVA in oral and nasal secretions or feces can serve as sources of infection to other suscept
129 the bacterial load of EHEC O157:H7 strain in feces, colon and caecum tissues after murine infection.
130 dian (IQR): 1360 mg/kg feces (2443-535 mg/kg feces) compared with 698 mg/kg feces (1438-244 mg/kg fec
131 serum concentrations of tetra-decaBDEs from feces concentrations and enable a noninvasive sampling m
133 T) values for bla(KPC) were higher for heavy-feces-containing than for light-feces-containing liquid-
135 e identify routes of contamination by animal feces, control measures to reduce human exposure, and pr
139 6, -207, -208, and -209 were detected in the feces creating a matched data set (feces-serum, n = 21).
140 ta diversity of the microbiome measured from feces demonstrated significant differences between The J
141 ility), did not affect the overall amount of feces deposited but led to changes in the average body s
143 sferred with Blastocystis sp. positive donor feces did not report any significant difference in bowel
144 sferred with Blastocystis sp.-positive donor feces did not report any significant differences in bowe
147 of P. aeruginosa from the bloodstream to the feces during bacteremia, a process that promotes transmi
148 ant and that this deterrence is based on the feces-emitted carboxylic acids 3-methylpentanoic acid an
150 uivalent counts of Escherichia coli in dairy feces exposed to different environmental conditions and
151 anaviruses and their detection in organs and feces followed over time by PCR, immunohistochemistry an
152 shed one strain >10(1) log10 CFU/g in their feces for 16.4 days, which persisted in the environment
153 was defined as at least 400 eggs per gram of feces for S. mansoni infection or as more than 50 eggs p
155 nce of tests in detecting M. bovis in badger feces for the Department for Environment, Food, and Rura
157 ate such effects of RT storage, we collected feces from 29 healthy infants (0-3 months) and partition
159 rom catch basins, a constructed wetland, and feces from a beef cattle feedlot were compared over a tw
162 1 copy of human REG3A transgene but were fed feces from control mice (not expressing hREG3A) as newbo
166 e assigned to groups that underwent FMT with feces from healthy donors or were given autologous fecal
168 in humans, we colonized germ-free mice with feces from healthy or cow's milk allergic (CMA) infants(
170 For humanization, we transplanted pooled feces from healthy, noncolonized human donors supplement
173 Compared with feces from wild-type mice, feces from mice with defects in TGFB signaling had incre
174 equencing of prokaryotic 16S rRNA present in feces from naive mice and those exhibiting CP-EAE or RR-
178 FAs in various biological samples, including feces from stressed rats, sera of patients with kidney d
179 e number of matching short sequence reads in feces from the 92 animals in the two clinical and the he
180 Sanitation interventions that isolate human feces from the environment may reduce transmission but h
181 d shotgun metagenomic sequencing analyses of feces from wild-type mice and mice with defects in TGFB
183 l samples from Hol(Tg/Tg) mice compared with feces from wild-type mice; fecal microbiomes of mice giv
185 Here, we unexpectedly found that transfer of feces harvested at peak disease from the experimental au
190 Extensive anaerobic subculturing of human feces identified no single commensal capable of l-carnit
192 of a parent in childhood, exposure to animal feces in infancy, birth in the dry season, or duration o
193 atter, other components in unprocessed human feces include colonocytes (~107 per gram of wet stool),
194 Differences in Pb species between diet and feces indicated that transformation of Pb species can oc
195 Gs (7762 x/Gb) was detected in adult chicken feces, indicating higher ARG contamination level than ot
196 ile acid pool (~3 g in human) is excreted in feces, indicating the large recycling capacity and high
197 Sepsis was induced by cultivated autologous feces inoculation in anesthetized, mechanically ventilat
198 Ranavirus-exposed smooth newts shed virus in feces intermittently and infection was seen in the absen
203 gallbladder can then seed the intestines and feces, leading to transmission to uninfected cage-mate m
207 to -0.09, P = 0.02), and time to passage of feces (mean difference -0.90 days, 95% CI -1.48 to -0.32
208 to -0.32, P < 0.0001) and time to passage of feces (mean difference -1.09 days, 95% CI -2.03 to -0.15
209 Escherichia coli, was retrieved from chicken feces metagenomes and was determined to carry diverse AR
211 Bacterial communities were determined from feces obtained from domestic pigs (Sus scrofa) raised un
212 Although the SARS-CoV-2 RNA was found in the feces of 3 patients and in the duodenal wall of the pati
214 lone-resistant Escherichia coli are found in feces of 8.8% of healthy women, with most bacteria belon
215 ng of a metagenomic library derived from the feces of an AB donor enabled discovery of a significantl
217 of colonic GMB communities derived from the feces of captive specimens, leaving our understanding of
219 saguini was isolated from the intestines and feces of cotton-top tamarins (CTTs) with chronic colitis
221 a miRNA-microbiome axis and suggest that the feces of diseased subjects might be enriched with miRNAs
222 significant expansion of the pathogen in the feces of I-Ab(DeltaIEC) mice compared with I-Ab(WT) mice
225 mily Erysipelotrichaceae was enriched in the feces of mice on the AhR ligand-free diet but returned t
230 ermore, O157-specific IgA levels detected in feces of the Adj-Vac group were significantly lower in N
231 lum, Elusimicrobia were more abundant in the feces of treated cattle, however, there were no differen
232 he small intestine and were shed less in the feces of wild-type mice, and such defects were rescued i
236 hs strongly preferred the extract of control feces over the fecal extract of axenic cockroaches.
238 osed to pathogens from poorly managed animal feces, particularly in communities where animals live in
245 rnover. Incubation of RV with SFB-containing feces reduced infectivity in vitro, suggesting direct ne
246 reduced food and water intake, combined with feces replete with lipid and bile acid, indicated a phen
247 h A. muciniphila after FMT with nonresponder feces restored the efficacy of PD-1 blockade in an inter
248 onic enemas with ion compositions similar to feces resulted in high local tissue levels with minimal
249 d, analysis of 24 h collections of urine and feces revealed recovery of less than 4% of the administe
252 quantify Z-values by equilibrating food and feces samples, which have been homogenized and spiked wi
253 habitation with animals, provision of animal feces scoops, controlling animal movement, creating safe
256 as-associated quinolones and rhamnolipids in feces, setting the stage for metabolome-microbiome-wide
258 esolution LC-MS analysis of bile, urine, and feces showed metabolic products derived from 4-PCB 11 su
259 f the copper pheophytins are excreted in the feces showing almost no absorption of copper-chlorophyll
261 and laundry) and blackwater (i.e., urine and feces) streams in terms of their loadings of ambient spe
262 ing ceftriaxone from the wetland compared to feces, suggesting resistance to this antibiotic may not
263 nstructed and serially filled with simulated feces tagged with four different iodine concentrations (
265 of several bacterial taxa in both blood and feces that correlate with the presence of LF, thus defin
266 where domestic animals are exposed to human feces that have been disposed in pits and open drains.
268 impacts of exposure to poorly managed animal feces transmitted via water, sanitation, and hygiene (WA
269 ed bioreactors inoculated with healthy human feces, treated with clindamycin and infected with C diff
270 pounds in urine (UHPLC-MS/MS), bile acids in feces (UHPLC-QTOF), gastrointestinal conditions (ingesti
273 ian total daily excretion of menaquinones in feces was 850 nmol/d but was highly variable (range: 64-
277 mong environmental samples, young children's feces were almost always identified as the dominant sour
284 tivity, and psychological data and blood and feces were collected at baseline and at 8 weeks and 3 mo
292 bsequently, serum samples, tonsil swabs, and feces were collected from sows (n = 22) and their piglet
294 ands, surfaces) whereas older children/adult feces were often identified as the dominant source outsi
296 ult (hemoglobin concentration of 10 mug Hb/g feces) were invited for consultation and scheduled for c
297 al RNA amplicon sequencing were performed on feces, whereas urine and fecal metabolites were analyzed
298 role in the flow of macrophage-derived UC to feces, while the plasma increase of APOB-containing lipo
299 tinguished separately from older child/adult feces with high sensitivity and specificity in water and
300 (gt3) infections were cleared from liver and feces within 8 pegIFNalpha doses in all mice and relapse