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1 en the neuropathology of aging in humans and felines.
2 transmitted, protozoan parasite that infects felines.
3 wing Toxoplasma infection is not specific to felines.
4 fically on nocturnal, solitary and dangerous felines.
5 hat the lapping mechanism is conserved among felines.
6  HIVAN in mice, rats, nonhuman primates, and felines.
7  on the brain of eight neurologically normal felines.
8 argeted marker selection utilized the recent feline 1.9x genome assembly, concentrating on regions of
9 etic analyses have led to the inference that feline A3Z3 hap V emerged approximately 60,000 years ago
10           It is particularly noteworthy that feline A3Z3 hap V is resistant to FIV Vif-mediated degra
11  Taken together, these findings suggest that feline A3Z3 hap V may have been selected for escape from
12            We also revealed that codon 65 of feline A3Z3 is a positively selected site and that A3Z3
13                           Interestingly, one feline A3Z3 variant is dominant, restrictive, and natura
14    In this study, we demonstrated that these feline A3Z3 variants suppress vif-defective FIV infectiv
15                                              Feline-adapted CWD (Fel(CWD)) was demonstrated in the br
16 ocytic cells (derived from normal marrow) in feline alpha-mannosidosis.
17  but still allowed efficient entry into both feline and canine cells without successful infection.
18 how differences in the interactions with the feline and canine TfRs that determine viral host range a
19 ped virus infection, but the introduction of feline and human cDNAs rendered them permissive.
20  the ability of FIV to bind and utilize both feline and human CXCR4 makes the feline model an attract
21 us disease in cats for applicability to both feline and human disease.
22                                          The feline and human immunodeficiency viruses (FIV and HIV)
23                                              Feline and human RFC cDNAs conferred susceptibility to T
24       Introduction of the same mutation into feline and murine infectious gammaretroviruses resulted
25 ar hypertrophy in vivo and in cultured adult feline and rat ventricular myocytes.
26 rfactant system in both NPC1 mutant mice and felines and in NPC2 mutant mice near the end of their ex
27 ina, consistent with MRI findings in rodent, feline, and baboon retinas.
28 tuted was recognized by a majority of human, feline, and canine myasthenia gravis sera.
29 s were highly conserved among human, simian, feline, and ungulate lentiviruses, which indicated that
30       In vivo, neural mapping experiments on feline animal models illustrate one mode of use for this
31 ion sites into the virus binding face of the feline apical domain reduced or eliminated both binding
32                     FIV Vif colocalized with feline APOBEC3 (fA3) proteins, targeted them for degrada
33 thermore, small interfering RNA knockdown of feline APOBEC3 genes resulted in equalization of replica
34 ability of 7 naturally occurring variants of feline APOBEC3, APOBEC3Z3 (A3Z3), to inhibit FIV replica
35                                              Felines are the only mammals that lack delta-6-desaturas
36 iminish these hallmark features in a chronic feline asthma model.
37 stered after development of chronic allergic feline asthma, MSCs failed to reduce airway inflammation
38              We therefore report the largest feline autopsy cohort to date of 32 cats ranging from 1.
39 ntrating on regions of low marker density on feline autosomes and the X chromosome, in addition to re
40 cifically detect the DNA of these viruses in feline blood and found that the domestic cat and bobcat
41 e multiple dry samples of human, canine, and feline blood for the ultimate application to forensic sp
42                 Of 101 FIV antibody-positive feline blood specimens submitted for FIV PCR diagnosis,
43 ion of the diffusivity parameters within the feline brain has not been documented.
44 ew species, we studied feline CWD (fCWD) and feline BSE (i.e., feline spongiform encephalopathy [FSE]
45                                              Feline calicivirus (FCV) and murine norovirus (MNV) are
46 solution structures of the VPg proteins from feline calicivirus (FCV) and murine norovirus (MNV), whi
47           Open reading frame 2 (ORF2) of the feline calicivirus (FCV) genome encodes a capsid precurs
48              Here, we examined the effect of feline calicivirus (FCV) infection on SG accumulation.
49 mid was engineered in which the LC region of feline calicivirus (FCV) was placed under the control of
50                                              Feline calicivirus (FCV), a member of the Vesivirus genu
51 qPCR) for feline herpesvirus type 1 (FHV-1), feline calicivirus (FCV), Mycoplasma felis, Chlamydophil
52 ause of infectious acute gastroenteritis and feline calicivirus (FCV), which causes respiratory illne
53 the attachment and infectious viral entry of feline calicivirus (FCV).
54 cule A (fJAM-A) is a functional receptor for feline calicivirus (FCV).
55  death in young cats, and virulent, systemic feline calicivirus (vs-FCV) causes a highly fatal diseas
56                          Previous studies on feline calicivirus and murine norovirus 1 (MNV1) demonst
57                            We have shown for feline calicivirus and rabbit hemorrhagic disease virus
58  NV replication were derived from studies of feline calicivirus and rabbit hemorrhagic disease virus,
59 tion cryo-electron microscopy structures for feline calicivirus both undecorated and labelled with a
60 of the canonical start/stop site in huNV and feline calicivirus but not in rabbit hemorrhagic disease
61 el neutralizing B-cell epitope, derived from feline calicivirus capsid protein, and a well characteri
62 le-stranded RNA viruses (e.g., Echovirus 12, feline calicivirus) but degraded much faster than MS2 (i
63      As has been previously demonstrated for feline calicivirus, a member of the Vesivirus genus, PSa
64          Many viruses, including the related feline calicivirus, use terminal sialic acids (SA) as re
65 in types of feline coronaviruses (FCoVs) and feline caliciviruses (FCVs), respectively, and are impor
66 hDC-SIGN) and that infection of a permissive feline cell line (Crandall-Reese feline kidney) was mark
67 rrin receptors (TfR) on the surfaces of live feline cells and to monitor how these CPV-TfR complexes
68 urface with different kinetics in canine and feline cells but, unlike transferrin, most did not recyc
69                              In FIV-infected feline cells, some intranuclear Gag was detected in the
70 FIV) replication in lymphoid and nonlymphoid feline cells.
71 tor binding, and relative in vitro growth in feline cells.
72 preading replication and passage of HIV-1 in feline cells.
73 achment, uptake, and infection in canine and feline cells.
74 e cells while they bound to the cell body of feline cells.
75 at and dog genomes, with an expansion of the feline chemosensory system for detecting pheromones at t
76 tis (CP), aggressive periodontitis (AP), and feline chronic gingivostomatitis (FCGS) are not well cha
77 rming growth factor (TGF)-beta-dependence of feline corneal keratocyte differentiation into alpha-smo
78               Anti-TGFbeta treatment reduced feline corneal myofibroblast differentiation in vitro an
79      We showed that entry of the serotype II feline coronavirus strains feline infectious peritonitis
80 f felids caused by systemic infection with a feline coronavirus.
81 rus infection are caused by certain types of feline coronaviruses (FCoVs) and feline caliciviruses (F
82 irulent strains of serotype 1 and serotype 2 feline coronaviruses.
83                  Hypercomplex cells found in feline cortex and small target motion detectors found in
84 n (GFP) in HIV-1 entry receptor-complemented feline (CrFK) cells enabled robust spreading HIV-1 repli
85 1 and hCRM1 and comparing those sequences to feline CRM1, we mapped the functional domain to HEAT (Hu
86 of transmission to a new species, we studied feline CWD (fCWD) and feline BSE (i.e., feline spongifor
87                             Upon subpassage, feline CWD was transmitted to all i.c.-inoculated cats w
88  patterns consistent with the early stage of feline CWD.
89                                Efficacy of a feline diet with an egg product ingredient containing an
90 croti infection in cats and suggest that the feline disease is a spillover from a disease maintained
91  cells permissive to other FeLV subgroups or feline endogenous retrovirus.
92 cies, including non-human primates, canines, felines, equids, ovids, suids, bovins, salmonids and mur
93 estic cats (ERV-DCs) are one of the youngest feline ERV groups in domestic cats (Felis silvestris cat
94                       The native recombinant feline erythropoietin (rfEPO) sequence was confirmed by
95                      In this in vitro study, feline erythropoietin cDNA was cloned from feline renal
96 e in vitro production of biologically active feline erythropoietin.
97 phologically divergent trajectories early in feline evolution.
98                                      Excised feline eyeballs preserved in corneal storage medium and
99                        In vitro infection of feline F81 cells with MEVB decreased cell viability and
100 s indeed blocked by FeLV-A infection, and in feline fibroblasts that naturally express feTHTR1 and no
101                                              Feline fibroblasts, T-cell lines, and primary peripheral
102                                              Feline foamy virus (FFV) is a contact-dependent retrovir
103 of rotavirus in cats and the first report of feline G6P[9], which questions the previous belief that
104  and to aid in assembly of a higher coverage feline genome sequence.
105 man genome, and a larger let-7 family in the feline genome.
106 tified viruses may have important effects on feline health and ecology.
107 , accessible species for advancing human and feline health.
108      Cardiac injury was induced in the adult feline heart by infusing isoproterenol (ISO) for 10 days
109 aMKII-CA infected NRVMs and in hypertrophied feline hearts.
110 for mammalian expression of a representative feline heavy (IGHG1a) together with a light (lambda or k
111 ced the variable and constant domains of the feline heavy chains of IgG1a (IGHG1a), IgG2 (IGHG2), and
112 um" strain Birmingham 1, a low-pathogenicity feline hemoplasma strain.
113 ted by quantitative real-time PCR (qPCR) for feline herpesvirus type 1 (FHV-1), feline calicivirus (F
114                             We established a feline HFpEF model induced by slow-progressive pressure
115                                              Feline hip dysplasia (FHD) is a debilitating condition a
116                               Sequencing the feline HMB-synthase gene revealed different mutations in
117 we report identification and analysis of the feline homologue to the human lectin DC-SIGN and show th
118 ssion of Toxoplasma gondii to the definitive feline host via predation, and this relationship has bee
119 re two different viral species with distinct feline hosts and evolutionary histories.
120 between healthy and sick cats diagnosed with Feline Hyperthyroidism (FH).
121                                              Feline hyperthyroidism is the most commonly diagnosed en
122 ailable TDCIPP exposures are associated with feline hyperthyroidism.
123 opsy results showed the animal suffered from feline hypertrophic cardiomyopathy and severe pulmonary
124 ssociated virus serotype 8 vector expressing feline IDUA from a liver-specific promoter.
125                         Here we report novel feline Ig sequences, a technique to express antigen-spec
126 GFAP-IL-1betaXAT mice were injected with the feline immunodeficiency virus (FIV) (Cre) vector in the
127                                              Feline immunodeficiency virus (FIV) and human immunodefi
128 an immunodeficiency virus type 2 (HIV-2) and feline immunodeficiency virus (FIV) but not HIV-1.
129                               Infection with feline immunodeficiency virus (FIV) causes an immunosupp
130                                              Feline immunodeficiency virus (FIV) causes progressive i
131                       We previously observed feline immunodeficiency virus (FIV) Gag accumulating at
132 omain was identified in the p2 region of the feline immunodeficiency virus (FIV) Gag protein.
133                                           In feline immunodeficiency virus (FIV) infected cats, daily
134 cells (Tregs) activated during the course of feline immunodeficiency virus (FIV) infection suppress C
135          Herein we demonstrate that in vitro feline immunodeficiency virus (FIV) infection, but not U
136                                              Feline immunodeficiency virus (FIV) infects many species
137                                              Feline immunodeficiency virus (FIV) is a lentivirus that
138                                              Feline immunodeficiency virus (FIV) is among the most co
139                                              Feline immunodeficiency virus (FIV) naturally infects mu
140                                              Feline immunodeficiency virus (FIV) OrfA is an accessory
141 ron (IFN-alpha) induced tetherin and blocked feline immunodeficiency virus (FIV) replication in lymph
142 ily conserved on both HIV type 1 (HIV-1) and feline immunodeficiency virus (FIV) reverse transcriptas
143                                              Feline immunodeficiency virus (FIV) shares with T-cell t
144 an immunodeficiency virus type 1 (HIV-1) and feline immunodeficiency virus (FIV) the least.
145 V), bovine immunodeficiency virus (BIV), and feline immunodeficiency virus (FIV) Vif appear specific
146                         Stable expression of feline immunodeficiency virus (FIV) Vif-green fluorescen
147 d from the pathogenic GL8 molecular clone of feline immunodeficiency virus (FIV), a range of viral va
148 BIV), equine infectious anemia virus (EIAV), feline immunodeficiency virus (FIV), and Rous sarcoma vi
149 cells and cell lines did not restrict HIV-1, feline immunodeficiency virus (FIV), equine infectious a
150 tes for three species groups of lentiviruses-feline immunodeficiency virus (FIV), simian immunodefici
151                     For unknown reasons, the feline immunodeficiency virus (FIV), which infects both
152 pression following a single application of a feline immunodeficiency virus (FIV)-based lentivirus vec
153  We analyzed antibody responses in sera from feline immunodeficiency virus (FIV)-infected and uninfec
154  suppresses the infectivity of vif-defective feline immunodeficiency virus (FIV).
155 es in the production of infectious HIV-1 and feline immunodeficiency virus (FIV).
156 f many viruses, including strains of HIV and feline immunodeficiency virus (FIV).
157                       An infectious chimeric feline immunodeficiency virus (FIV)/HIV strain carrying
158  transmission pathways for three subtypes of feline immunodeficiency virus (FIVPle ) in African lions
159 onprimate lentiviral genomic RNAs (HIV-1 and feline immunodeficiency virus [FIV]) vis-a-vis their Gag
160     The earliest experiments were in the cat/feline immunodeficiency virus model, followed a decade l
161  TRIMCyp transgenic cat lymphocytes resisted feline immunodeficiency virus replication.
162 l cross-species transmission of a subtype of feline immunodeficiency virus, puma lentivirus A (PLVA),
163 tranasal insulin treatment of experimentally feline immunodeficiency virus-infected animals resulted
164 ere suggest the capture and fur use of those felines in the context of highly codified and symbolical
165 roperties of offerings consisting of ceramic feline incense burners, killed juvenile llamas, and sump
166                                     Although feline infections are typically asymptomatic, infection
167 lethal systemic granulomatous disease called feline infectious peritonitis (FIP), which is one of the
168                                              Feline infectious peritonitis and virulent, systemic cal
169                                              Feline infectious peritonitis is a lethal disease of fel
170                                              Feline infectious peritonitis virus (FIPV) belongs to th
171                                              Feline infectious peritonitis virus (FIPV) is an alphaco
172                                              Feline infectious peritonitis virus (FIPV) is the leadin
173 f the serotype II feline coronavirus strains feline infectious peritonitis virus (FIPV) WSU 79-1146 a
174 sm targeting CoV main protease (M(pro)) from feline infectious peritonitis virus (FIPV), which leads
175 ndii sexual development occurs when cultured feline intestinal epithelial cells are supplemented with
176 ammal, the sexual cycle is restricted to the feline intestine.
177   In the veterinary school database, 4.5% of feline intraocular and adnexal neoplasms (234/5153) were
178                Conversely, most (74%) of the feline isolates belonged to ST4, ST6, and ST7.
179                                              Feline junctional adhesion molecule 1 (fJAM-1) was recen
180                                          The feline junctional adhesion molecule A (fJAM-A) is a func
181                                              Feline junctional adhesion molecule A (fJAM-A) mediates
182      FCV attachment and entry is mediated by feline junctional adhesion molecule A (fJAM-A), which bi
183 a soluble fragment of its cellular receptor, feline junctional adhesion molecule A.
184 ion of the capsid proteins alone in Crandell feline kidney (CrFK) cells reproduced the cleavage of th
185 nfection of human 293H cells, Crandall Renal Feline Kidney cell line and primary feline peripheral bl
186 V with a characteristic cytopathic effect in feline kidney cells.
187  assembly, but does prevent virus spread, in feline kidney cells.
188  permissive feline cell line (Crandall-Reese feline kidney) was markedly enhanced by the overexpressi
189 ity during viral spillover events.IMPORTANCE Feline leukemia virus (FeLV) can infect a variety of fel
190                                        While feline leukemia virus (FeLV) has been shown to infect fe
191 gation of FeLV infectious disease.IMPORTANCE Feline leukemia virus (FeLV) is a member of the genus Ga
192                                              Feline leukemia virus (FeLV) is a naturally transmitted
193                                              Feline leukemia virus (FeLV) is horizontally transmitted
194                                              Feline leukemia virus (FeLV) is still a major cause of m
195 7 and ERV-DC16), or can generate recombinant feline leukemia virus (FeLV).
196   The 11-aa targeting domain of the modified feline leukemia virus envelope consists of a constrained
197 is through active heme export by the group C feline leukemia virus receptor (FLVCR).
198 bon ape leukemia virus, woolly monkey virus, feline leukemia virus subgroup B, feline leukemia virus
199 ed a single-nucleotide coding variant in the feline leukemia virus subgroup C cellular receptor 1 (FL
200                                              Feline leukemia virus subgroup C cellular receptor 1a (F
201                                          The feline leukemia virus subgroup C receptor (FLVCR) is a 1
202                                          The feline leukemia virus subgroup C receptor (FLVCR) is a h
203 abrando et al. reveal that an isoform of the feline leukemia virus subgroup C receptor (FLVCR1) expor
204 ated to mRNA expression of the heme exporter feline leukemia virus subgroup C receptor 1 (beta = -0.3
205                                              Feline leukemia virus subgroup C receptor 1 (FLVCR1) is
206 key virus, feline leukemia virus subgroup B, feline leukemia virus subgroup T, and 10A1 murine leukem
207 g calicivirus, coronavirus, herpesvirus, and feline leukemia virus, did not.
208          We previously demonstrated that the feline leukemia virus, subgroup C, receptor (FLVCR) expo
209 mestic exposure to gammaretroviruses such as feline leukemia viruses (FeLVs) occurs worldwide, but th
210  to viruses that infect human cells in vitro Feline leukemia viruses (FeLVs) rank high on this list,
211 t least some conservation between murine and feline leukemia viruses is crucial for activity.
212 d Histoplasma capsulatum-infected canine and feline lungs and airway epithelial cells could serve as
213                  After cloning cDNA-encoding feline MAP4 and obtaining its deduced amino acid sequenc
214                                271 candidate feline miRNA precursors, encoding a total of 475 mature
215                    As a result, there are no feline miRNAs present in the reference miRNA databases,
216 tilize both feline and human CXCR4 makes the feline model an attractive venue for development of broa
217                                          The feline model for CHS was originally maintained for ~20 y
218                Using the naturally occurring feline model of MPS I, we tested liver-directed gene the
219 arly for cone disorders, rodent, canine, and feline models with no true macula have substantive limit
220 ith mannan considerably reduced infection of feline monocyte-derived cells expressing DC-SIGN, indica
221                              The most common feline Mycoplasma species in guigna and domestic cats we
222 m normal and pressure overload-hypertrophied feline myocardium; volume-overloaded myocardium, which h
223                TRPC3/4/6 expression in adult feline myocytes increased rested state contractions and
224            TRPC3/4/6 overexpression in adult feline myocytes induced calcineurin (Cn)-nuclear factor
225 sion of TRPC3/4/6 channels in cultured adult feline myocytes to define mechanistic aspects of these T
226 with a mutant protein containing a consensus feline myristoylation motif (MA(NOS)) and examined the i
227   This information will not only help inform feline neuroanatomy but also will serve as a reference s
228 ill serve as a reference standard for future feline neuroimaging studies.
229 exal neoplasms (234/5153) were designated as feline ocular posttraumatic sarcoma, a tumor previously
230 nsidered to be a commensal of the canine and feline oral cavities.
231 subtype of HPV76, originally isolated from a feline oral squamous cell carcinoma, was detected in 7 n
232 e oral SCC (OSCC) derived from an osteolytic feline OSCC.
233 ve naturally occurring large animal (canine, feline, ovine, caprine, and bovine) models been so essen
234  revealed two major groups related to either feline panleukopenia virus ("FPV-like") or canine parvov
235                  Canine parvovirus (CPV) and feline panleukopenia virus (FPV) are closely related par
236     Canine parvovirus (CPV) and its relative feline panleukopenia virus (FPV) bind the transferrin re
237 s a host-range variant of a virus related to feline panleukopenia virus (FPV).
238 ith canine parvovirus (CPV) or its ancestor, feline panleukopenia virus (FPV).
239 ctivity with preventive vaccine-induced anti-feline panleukopenia virus antibodies.
240 tional felinized monoclonal antibody against feline panleukopenia virus.
241 he structures of canine parvovirus (CPV) and feline parvovirus (FPV) complexed with antibody fragment
242 te routes of transmission: first, an endemic feline parvovirus (FPV) route of transmission maintained
243 rus type 2 (CPV-2) emerged as a variant of a feline parvovirus when it acquired mutations that allowe
244                     CPV, but not the related feline parvovirus, could use receptors containing a cani
245 ican lions: canine distemper virus (CDV) and feline parvovirus.
246 iables in shaping exposure risk from CDV and feline parvovirus.
247 ore parvovirus epidemiology, suggesting that feline parvoviruses are endemic in wild carnivores in th
248 he detection and characterization of diverse feline pathogen taxa.
249 address this barrier, we isolated mRNAs from feline peripheral blood mononuclear cells (PBMCs), and u
250 ll Renal Feline Kidney cell line and primary feline peripheral blood mononuclear cells, bioactive rfE
251 ete wet (n = 97) and dry (n = 80) canine and feline pet food sold in the UK was measured to assess co
252  framework for identifying genes controlling feline phenotypes of interest, and to aid in assembly of
253 ated administration of meloxicam altered the feline plasma and urine lipidome as demonstrated by mult
254  of repeated meloxicam administration on the feline plasma and urine lipidome.
255 ation of a recessively inherited early-onset feline PRA.
256          The central pathways subserving the feline pupillary light reflex were examined by defining
257 radiation hybrid panel identified SLC19A1, a feline reduced folate carrier (RFC) and potential recept
258            Pulmonary features of NPC1 mutant felines reflected the disease described in NPC1 mutant m
259 , feline erythropoietin cDNA was cloned from feline renal tissue and utilized in the construction of
260 , a significant association between clinical feline respiratory disease and influenza virus infection
261 f the genus Felis, predating cat exposure to feline retroviruses.
262 al role of Refrex-1 predates cat exposure to feline retroviruses.
263 tor, CD134, whereas cats infected with other feline RNA viruses, including calicivirus, coronavirus,
264 est; (ii) fCWD was a more efficient seed for feline rPrP than for white-tailed deer rPrP; (iii) conve
265 , FSE more efficiently converted bovine than feline rPrP; (iv) and CWD, fCWD, BSE, and FSE all conver
266                                    McDonough feline sarcoma viral (v-fms) oncogene homolog (FMS) and
267 ne homolog (FMS) and v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene homolog (KIT) are two hema
268 ent up-regulation of v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene homolog (KIT) receptor tyr
269  MET through the nonreceptor tyrosine kinase feline sarcoma-related (FER).
270           To characterize the progression of feline SMA, we assessed pathological changes in muscle a
271                                  We used the feline SNV array and whole genome sequence data to under
272 tent, if any, muscle fascicles of denervated feline soleus (SO) change length during stance of walkin
273 ree new viruses that are widespread in three feline species, indicates risk factors for infection tha
274  first GHVs identified to be native to these feline species.
275 d push-pull in the motor output stage of the feline spinal cord, a system that allows independent con
276                                              Feline spinal muscular atrophy (SMA) is a fully penetran
277 ic cats have been shown to be susceptible to feline spongiform encephalopathy (FSE), almost certainly
278 died feline CWD (fCWD) and feline BSE (i.e., feline spongiform encephalopathy [FSE]).
279 uses in human cells and proved beneficial in feline stomatitis patients.
280 sing an in vitro coculture approach to model feline T. foetus infection of the intestinal epithelium,
281 y were to determine the pathogenic effect of feline T. foetus on porcine intestinal epithelial cells,
282                      Replacement of critical feline TfR residue 221 with every amino acid had effects
283                                      Testing feline TfR variants for their binding and uptake pattern
284  reveal that the low affinity of capsids for feline TfRs limits the residence time of capsids on the
285 usive of mammalian species, such as domestic felines, that are not commonly considered intermediate h
286 sporters in mammals, THTR2, and we show that feline THTR1 (feTHTR1) and feTHTR2 both mediate thiamine
287  and characterize the cat miRNAome in normal feline tissues.
288 y used a controlled experimental paradigm in felines to examine whether relearning of motion discrimi
289 tionally, in the gut of its definitive host, felines, Toxoplasma converts into gametocytes that produ
290 rised the transcriptomic changes involved in feline TR by sequencing RNA extracted from 14 teeth (7 w
291 MP9 may be a potential therapeutic target in feline TR.
292 gue of the oncoproteins encoded by avian and feline transforming retroviruses, c-Fes has recently bee
293 hus raising the issue of potential cervid-to-feline transmission in nature.
294 Forced expression of STIM1 in cultured adult feline ventricular myocytes increased diastolic spark ra
295                            In cultured adult feline ventricular myocytes, PKA inhibition protected my
296 l rat ventricular myocytes (NRVMs) and adult feline ventricular myocytes.
297  ST5, and ST8 (zoonosis associated) and ST6 (feline) was statistically significant (P < 0.05), indica
298 hanisms to extraclassical suppression in the feline, we made simultaneous single-unit recordings from
299                 Sixteen corneas from healthy felines were obtained immediately after death.
300  Transcriptional analysis revealed that most feline X-degenerate genes have retained housekeeping fun

 
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