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4 NV replication were derived from studies of feline calicivirus and rabbit hemorrhagic disease virus,
5 further divided into the following species: Feline calicivirus and Vesicular exanthema of swine viru
6 tion cryo-electron microscopy structures for feline calicivirus both undecorated and labelled with a
7 of the canonical start/stop site in huNV and feline calicivirus but not in rabbit hemorrhagic disease
8 le-stranded RNA viruses (e.g., Echovirus 12, feline calicivirus) but degraded much faster than MS2 (i
9 el neutralizing B-cell epitope, derived from feline calicivirus capsid protein, and a well characteri
10 irus strains, MD145-12 (genus Norovirus) and feline calicivirus (FCV) (genus Vesivirus), to investiga
12 solution structures of the VPg proteins from feline calicivirus (FCV) and murine norovirus (MNV), whi
13 located at the 3' end of the genomic RNA of feline calicivirus (FCV) encodes a small (12.2-kDa) mino
14 s approach is demonstrated by the capture of feline calicivirus (FCV) from cell culture media that is
16 Here we show how longitudinal analysis of feline calicivirus (FCV) infection in an animal rescue s
21 study was to identify the active form of the feline calicivirus (FCV) RNA-dependent RNA polymerase (R
23 mid was engineered in which the LC region of feline calicivirus (FCV) was placed under the control of
26 nse was observed in CRFK cells infected with Feline Calicivirus (FCV), a virus released by cell lysis
27 LBC) epitopes in the major capsid protein of feline calicivirus (FCV), an expression library containi
28 structure with that of a related vesivirus, feline calicivirus (FCV), highlighted potentially import
29 qPCR) for feline herpesvirus type 1 (FHV-1), feline calicivirus (FCV), Mycoplasma felis, Chlamydophil
30 ause of infectious acute gastroenteritis and feline calicivirus (FCV), which causes respiratory illne
36 in types of feline coronaviruses (FCoVs) and feline caliciviruses (FCVs), respectively, and are impor
37 ed a 99.68% reduction in virus titer against Feline calicivirus in a surface time-kill test using rea
39 gs on the RdRp activity of the norovirus and feline calicivirus Pro(-)Pol enzymes were compared and f
41 death in young cats, and virulent, systemic feline calicivirus (vs-FCV) causes a highly fatal diseas
42 we have been exploiting endemic infection of feline calicivirus within five geographically distinct h