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3 nine ratio measure, and a measure of carotid-femoral pulse wave velocity (cf-PWV) and augmentation in
5 outcome, along with blood pressure, carotid-femoral pulse wave velocity (cf-PWV), lipids/lipoprotein
8 e evaluated the relationship between carotid femoral pulse wave velocity (cfPWV) and T-cell activatio
9 nts from three metro areas underwent carotid-femoral pulse wave velocity (cfPWV) assessment between 2
10 plitude, mean arterial pressure, and carotid-femoral pulse wave velocity (CFPWV) in 1480 participants
13 chial arteries (by ultrasonography), carotid-femoral pulse wave velocity (cfPWV), aortic augmentation
14 stiffness: brachial pulse pressure; carotid-femoral pulse wave velocity (CFPWV), which is related di
17 iovascular risk factors, both higher carotid-femoral pulse wave velocity (hazard ratio [HR], 1.32; 95
18 iuretic peptide were associated with carotid-femoral pulse wave velocity (men: partial correlation, 0
20 ive protein, and arterial stiffness [carotid-femoral pulse wave velocity (PWV) and carotid augmentati
21 econdary outcomes were changes in carotid to femoral pulse wave velocity (PWV) and plasma 8-isoprosta
22 Vascular stiffness was measured by carotid-femoral pulse wave velocity (PWV) and total arterial com
25 ry flow-mediated vasodilation (FMD), carotid-femoral pulse wave velocity (PWV), and aortic augmentati
28 and homocysteine was associated with carotid-femoral pulse wave velocity (r = 0.072, P = 0.036), forw
29 ity derived from arterial tonometry (carotid-femoral pulse wave velocity [CFPWV], forward wave amplit
30 -17 years and arterial stiffness (carotid to femoral pulse wave velocity [PWV]) measured at age 17 ye
32 aluated 2 aortic stiffness measures, carotid-femoral pulse wave velocity and forward pressure wave am
33 ial artery flow-mediated dilatation, carotid-femoral pulse wave velocity and post-ischaemic brachial
37 carotid artery wall echodensity and carotid-femoral pulse wave velocity demonstrated no significant
39 nge in the weight-loss group, but carotid-to-femoral pulse wave velocity tended to decrease by 0.5 m/
40 r adding systolic blood pressure and carotid-femoral pulse wave velocity to the model, forward pressu
42 ulse pressure, pulsatility index and carotid-femoral pulse wave velocity were each associated with in
43 um [CAC] score); arterial stiffness (carotid-femoral pulse wave velocity); incident hypertension, dia
45 ssure wave, reflected pressure wave, carotid-femoral pulse wave velocity, and carotid-radial pulse wa
46 ar mass, and fractional shortening), carotid-femoral pulse wave velocity, and central retinal arterio
47 sistance, total arterial compliance, carotid-femoral pulse wave velocity, and drug tolerability were
48 omography scan, such as tonometry of carotid femoral pulse wave velocity, bioelectrical impedance ana
49 evaluated carotid pressure and flow, carotid-femoral pulse wave velocity, brain magnetic resonance im
50 :599-608) present repeated measures of aorto-femoral pulse wave velocity, capacitive compliance (C1),
51 rs of subclinical CVD were assessed: carotid-femoral pulse wave velocity, carotid intima media thickn
52 troglycerin-mediated dilation (NMD), carotid-femoral pulse wave velocity, carotid-radial pulse wave v
53 stiffness and pressure pulsatility (carotid-femoral pulse wave velocity, central pulse pressure [CPP
54 sures of arterial stiffness were the carotid femoral pulse wave velocity, forward pressure wave ampli
55 hat arterial stiffness, measured via carotid-femoral pulse wave velocity, has a better predictive val
56 differences between treatment in carotid-to-femoral pulse wave velocity, high-sensitivity C-reactive
58 We analyzed the primary outcome, carotid-femoral pulse wave velocity, using a linear mixed effect
60 cranberry juice consumption reduced carotid femoral pulse wave velocity-a clinically relevant measur
65 on measures (central pulse pressure, carotid-femoral pulse-wave velocity, mean arterial pressure, for