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1 ceptor agonists (salbutamol, procaterol, and fenoterol).
2 GP-12177A as did the addition of the agonist fenoterol.
3 s like morphine, metformin, sumatriptan, and fenoterol.
4 s like metformin, sumatriptan, trospium, and fenoterol.
5 bation of bovine tracheal smooth muscle with fenoterol (10(-7) M) for 2 h increased H(1) receptor mRN
6 relative to epinephrine (100%) were 42% for fenoterol, 4.9% for albuterol, 2.5% for dobutamine, and
7 uterol (5.83 +/- 0.06 and 4.71 +/- 0.16) and fenoterol (6.33 +/- 0.07 and 5.67 +/- 0.05) were less se
8 1 (ISO-beta(1)-AR stimulation) or 0.1 microM fenoterol, a beta(2)-AR agonist (FEN-beta(2)-AR stimulat
9 onists activate both G(s) and G(i) proteins, fenoterol, a full agonist of beta(2)-adrenoceptor, selec
11 the receptor, and K(i) values determined for fenoterol analogs model much better the cAMP activity of
12 ter understand the molecular interactions of fenoterol analogs with the beta(2)-adrenergic receptor,
16 ave synthesized a series of stereoisomers of fenoterol and its derivatives and characterized their re
20 ency, with ephedrine, dobutamine, albuterol, fenoterol, and epinephrine giving 0, 7, 17, 48, and 55%
22 haled short-acting beta2-agonists albuterol, fenoterol, and terbutaline provide rapid as-needed sympt
28 ificantly to the left after 18-h exposure to fenoterol, consistent with the increase in receptor numb
30 trast to zinterol, stimulation of I(Ca,L) by fenoterol (fen-beta(2)-AR), a beta(2)-AR agonist that ac
32 lase, namely epinephrine > or = formoterol = fenoterol > terbutaline = zinterol = albuterol > salmete
39 treatment with dexamethasone did not prevent fenoterol-induced up-regulation of H(1) receptor mRNA.
40 mation, being purely entropy-driven for each fenoterol isomer, rather than a mixture of entropy and e
41 n a mixture of entropy and enthalpy when the fenoterol isomers binding was determined using [(3)H]CGP
42 effects of the beta(2)-adrenoceptor agonist, fenoterol, on the expression of H(1) receptors at the mR
43 week) administration of the beta2AR agonists fenoterol or zinterol, starting at 2 weeks after MI, red
45 hrine and the beta(2) adrenoreceptor agonist fenoterol potently inhibited LPS-induced TNF-alpha and I
46 The two strongest agonists, epinephrine and fenoterol, provoked 11-13-fold increases in the level of
47 ive activation of beta2-AR/G(s) signaling by fenoterol restores the blunted beta2-AR contractile resp
50 differential G protein selectivities of the fenoterol stereoisomers were further confirmed by photoa
52 ed with the low concentrations (0.2 and 2 nM fenoterol), these concentrations did cause significant d
53 to Leu32 caused reversal of trospium but not fenoterol uptake kinetics. Comparison of the uptake of s