ライフサイエンスコーパス: ferric

1 p-II) = 0.99 V (at pH 7, vs NHE) with D(O-H)(Ferric) = 90 kcal/mol.

2 ico observations of a change around the heme ferric active center.

3 stability in HepG2 hepatoma cells exposed to ferric ammonium citrate (FAC).

4 ain for beta-galactosidase activity (S-Gal + ferric ammonium citrate) that produces both optical and

5 assium ferricyanide/ferrocyanide and ferrous/ferric ammonium sulfate) yielded Nernstian slopes of -58

6 Iron occurs in clay minerals in both ferric and ferrous forms.

7 Incorporating both ferric and ferrous iron in their structures, these oxide

8 Both ferric and ferrous iron were found in the mucus, indicat

9 in both enzymes has His/Cys ligation in the ferric and ferrous states and the midpoint potentials (E

10 uming a sensitive balance between heme-free, ferric, and nitric oxide-sensitive ferrous sGC in cells

11 ng activity, and showed the highest reducing ferric antioxidant power (FRAP).

12 However, accumulation of ferric (bio)minerals increases competition by stimulatin

13 The presence of ferric (bio)minerals induced surface-catalytic heterogen

14 dation in the presence or initial absence of ferric (bio)minerals.

15 (III)OH(2)) couple and its associated D(O-H)(Ferric) bond strength in CYP158.

16 Formally ferric carbonyl adducts are reported in a series of thio

17 ection of iron deficiency with (intravenous) ferric carboxymaltose (FCM) affects peak oxygen consumpt

18 for hypophosphataemia following intravenous ferric carboxymaltose (FCM) in patients with IBD.

19 icantly lower following iron isomaltoside vs ferric carboxymaltose (trial A: 7.9% vs 75.0% [adjusted

20 ron deficiency, inflammation, treatment with ferric carboxymaltose and chronic kidney disease.

21 ardiovascular death for patients assigned to ferric carboxymaltose compared with placebo (369 days pe

22 death occurred in 181 (32%) patients in the ferric carboxymaltose group and 209 (38%) in the placebo

23 occurred in 250 (45%) of 559 patients in the ferric carboxymaltose group and 282 (51%) of 551 patient

24 art failure hospitalisations occurred in the ferric carboxymaltose group and 294 occurred in the plac

25 (57.2 per 100 patient-years) occurred in the ferric carboxymaltose group and 372 (72.5 per 100 patien

26 ns and cardiovascular deaths occurred in the ferric carboxymaltose group and 451 occurred in the plac

27 tween the two groups (77 [14%] of 558 in the ferric carboxymaltose group vs 78 [14%] in the placebo g

28 Intravenous ferric carboxymaltose has been shown to improve symptoms

29 was administered as a single 1000 mg dose of ferric carboxymaltose in 100 mL normal saline, and place

30 ndomly assigned (1:1) to receive intravenous ferric carboxymaltose or placebo for up to 24 weeks, dos

31 isode of acute heart failure, treatment with ferric carboxymaltose was safe and reduced the risk of h

32 n of iron isomaltoside, 1000 mg, on day 0 or ferric carboxymaltose, 750 mg, infused on days 0 and 7.

33 We aimed to evaluate the effect of ferric carboxymaltose, compared with placebo, on outcome

34 called ferric derisomaltose), compared with ferric carboxymaltose, resulted in lower incidence of hy

35 117) and headache (iron isomaltoside: 4/125; ferric carboxymaltose: 5/117).

36 No.) were nausea (iron isomaltoside: 1/125; ferric carboxymaltose: 8/117) and headache (iron isomalt

37 including 61 to iron isomaltoside and 61 to ferric carboxymaltose; 93.4% completed the trial.

38 including 62 to iron isomaltoside and 61 to ferric carboxymaltose; 95.1% completed the trial.

39 Ferric CBS (Fe(III)-CBS) can be reduced by strong chemic

40 demonstrate that 1 has a low-spin (S = 1/2) ferric center.

41 iation of FRO2 transcript levels, as well as ferric chelate reductase activity, and is causal for a p

42 s caused by an impaired ability to boost the ferric chelate reductase activity, which is an essential

43 impaired in iron-regulated transporter1 and ferric chelate reductase2 knockout mutants and was prior

44 iency by leading to low chlorophyll but high ferric-chelate reductase activity and coumarin release.

45 asein fractions, even at the lowest level of ferric chloride addition (5mM).

46 either intra-arterial thrombin injection or ferric chloride application followed by measurement of c

47 sein-iron precipitates were formed by adding ferric chloride at >/=10mM to sodium caseinate solutions

48 Up to 20mM ferric chloride could be added to sodium caseinate solut

49 ere investigated by ultrasound in a model of ferric chloride induced non-occlusive carotid artery thr

50 As adding >5mM ferric chloride to sodium caseinate solutions results in

51 By addition of ferric chloride to the reaction mixture, a selective aro

52 of NAC on larger vessels, we also performed ferric chloride-induced carotid artery thrombosis.

53 llular PAD4 on platelet-plug formation after ferric chloride-induced injury of mesenteric venules.

54 F8-/-/PN-1-/- mice than in F8-/-mice in the ferric chloride-induced mesenteric vessel injury model.

55 ndent protection from carotid occlusion in a ferric chloride-induced thrombosis model.

56 il-vein bleeding test and the carotid artery ferric chloride-induced thrombosis model.

57 in a mouse hemophilia model, when assayed as ferric chloride-induced thrombosis.

58 We found that it reduces ferric-chloride-induced experimental thrombosis in mice

59 f serious adverse events were similar in the ferric citrate (12.0%) and placebo groups (11.2%).

60 uptake systems for elemental iron (efeUOB), ferric citrate (fecCDEF), and petrobactin (fpbNOPQ) are

61 a to compare the safety and efficacy of oral ferric citrate (n=117) and placebo (n=115).

62 Significantly more patients randomized to ferric citrate achieved the primary end point (61 [52.1%

63 (18.8%) and 15 (12.9%) patients treated with ferric citrate and placebo, respectively.

64 per 1.73 m(2) 2:1 to receive a fixed dose of ferric citrate coordination complex (two tablets per mea

65 To investigate whether fixed-dose ferric citrate coordination complex favorably affects mu

66 and hospitalization, suggest that fixed-dose ferric citrate coordination complex has an excellent saf

67 The beneficial effects of fixed-dose ferric citrate coordination complex on biochemical param

68 Ferric citrate coordination complex significantly increa

69 Compared with usual care, ferric citrate coordination complex treatment resulted i

70 Of the 133 patients randomized to ferric citrate coordination complex, 31 (23%) initiated

71 ints reached statistical significance in the ferric citrate group, including the mean relative change

72 Ferric citrate hydrate (FC) is an iron-based phosphate b

73 all, in patients with NDD-CKD, we found oral ferric citrate to be a safe and efficacious treatment fo

74 (O)L(ax) ] and b) a hydroxoiron porphyrazine ferric complex [PyPzFe(III) (OH)L(ax) ], both of which i

75 Although the interaction of low-spin ferric complexes with nitric oxide has been well studied

76 secondary coordination sphere in ferrous and ferric complexes.

77 sulted in the conversion of ferritin's inert ferric core into more reactive low-oxidation-states.

78 ANES), we demonstrate that the photocycle of ferric Cyt c is entirely due to a cascade among excited

79 ample, in blocking apoptosis by reduction of ferric cytochrome c, and gentle tuning of NO concentrati

80 to oral iron, iron isomaltoside (now called ferric derisomaltose), compared with ferric carboxymalto

81 The structures of ferric enterobactin and ferric enantioenterobactin obtained in this work provide

82 coli outer membrane receptor FepA transports ferric enterobactin (FeEnt) by an energy- and TonB-depen

83 The structures of ferric enterobactin and ferric enantioenterobactin obtai

84 e since its discovery over 40 years ago, the ferric enterobactin complex has eluded crystallographic

85 cessful growth of single crystals containing ferric enterobactin using racemic crystallization, a met

86 Strikingly, heme acquisition, ferric-enterobactin transport, and pyoverdine biosynthes

87 by the rapid H2O2-mediated oxidation of the ferric enzyme to the redox intermediate compound I.

88 states very slowly returned to resting (i.e. ferric) enzyme, indicating that they represented catalas

89 using a frequently asked question approach, Ferric Fang of the University of Washington, who has bee

90 lonized benthic communities to a gradient of ferric Fe (0-15 mg/L) for 14 days to estimate the effect

91 ater (<4 mg L(-1)), reductive dissolution of ferric Fe oxides was associated with mobilization of P t

92 ed electrode to efficiently interconvert the ferric (Fe(3+)) and ferrous (Fe(2+)) forms of an immobil

93 ble to grow aerobically over a wide external ferric (Fe(3+)) iron (FeCl3) concentration range.

94 y of infected RBCs (iRBCs) is changed due to ferric (Fe(3+)) paramagnetic state in hemozoin crystalli

95 archaea have been shown to reduce iron from ferric [Fe(III)] to ferrous [Fe(II)] state, but minerals

96 enylporphyrin where the pre-equilibria among ferric, ferrous, and ferric-superoxide intermediates hav

97 The stability of heme coordination upon ferric/ferrous redox cycling is a crucial property of th

98 ase and doming upon photoexcitation, but its ferric form does not release the distal ligand, while th

99 vely flexible structure, particularly in the ferric form, such that it is able to sample a broad conf

100 ommon iron oxidation level consistent with a ferric formulation (3: 7111.5 eV, 2: 7111.5 eV; 5: 7112.

101 electron to the coproheme iron to yield the ferric harderoheme and CO2 products.

102 erimentally derived thermodynamics lead to a ferric heme hydroperoxide OO-H BDFE determination, that

103 cleophile, namely the phenyl anion, with the ferric heme nitrosyl [(OEP)Fe(NO)(5-MeIm)](+) generates

104 Here, a ferric heme peroxide complex, [(F(8))Fe(III)-(O(2)(2-))]

105 his pattern is common to a wide diversity of ferric heme proteins, raising the question of the biolog

106 ing formed via HAT reactivity of the partner ferric heme superoxide complex.

107 Et)](-) are remarkably analogous to those of ferric heme superoxide complexes.

108 erein, an electronically divergent series of ferric heme superoxo oxidants mediates the facile conver

109 ted spin states of the iron ion, causing the ferric heme to undergo doming, which we identify.

110 main (k(r) ), of dissociation of NO from the ferric heme-NO complex (k(d) ), and of oxidation of the

111 lfate and polysulfide formation, coordinates ferric hemoglobin and, in the presence of air, generated

112 y shown that at high sulfide concentrations, ferric hemoglobin oxidizes H2S to a mixture of thiosulfa

113 We have recently discovered the ability of ferric hemoglobin to oxidize sulfide to thiosulfate and

114 Ferric hexacyanoferrate, also known as Prussian blue (PB

115 hanism whereby atomic hydrogen that forms on ferric (hydr)oxide surface layers promotes As(III) reduc

116 6-lutidinium triflate, yielding the low-spin ferric hydroperoxide species, [(F(8))Fe(III)-(OOH)] (HP)

117 on of the parent oxyferrous form, displays a ferric-hydroperoxo EPR signal, in contrast to the cryore

118 -N,O-AZADO) with hydrogen atom donors yields ferric hydroxide ((Ar) L)Fe(OH)(AZAD).

119 promoted pyruvate reduction to lactate, and ferric hydroxides did not result in any reaction.

120 nd dissociation free energies (BDFEs) of two ferric hydroxo complexes, differentiated by their noncov

121 Additionally, the ferric hydroxo species are differentiated by their react

122 show here that polysulfides bind to inactive ferric IDO1 and reduce it to the oxygen-binding ferrous

123 he on-rate for hydrogen disulfide binding to ferric IDO1 was found to be >10(6) M(-1) s(-1) at pH 7 u

124 I), which selectively binds to and activates ferric IDO1.

125 onditions led to a decrease in expression of ferric import systems.

126 This design feature promotes a switch from ferric import to the more physiological relevant ferrous

127 From this ferric intermediate, hydroxylation is thermodynamically

128 metallation mechanism may be set off between ferric ion and gadoterate meglumine.

129 moderate and high purity), and the effect of ferric ion concentration and pH was studied in moderate

130 Parallel (multichip) screening of ferric ion concentration gradients (0-40 mM) and tempera

131 ly changes with the pH, temperature, and the ferric ion concentration, consistent with previous bulk-

132 solvent-derived ligand remains bound to the ferric ion in the enzyme-substrate complex.

133 cytoplasmic antioxidants and confirmed with ferric ion reducing antioxidant power (FRAP) assays.

134 Total Phenolic Content (TPC) and Ferric Ion Reducing Antioxidant Power (FRAP) of hydrolys

135 hat Cys207 and Cys216 are the ligands of the ferric ion, and His255 and His259 are the ligands of the

136 lpiperidinyl-1-oxy (TEMPO) scavenging; (iii) ferric ions (Fe(3+)) reducing power; (iv) hydrogen perox

137 It was found that ferric ions (Fe(3+)) were most responsive and effective

138 he highly selective and sensitive sensing of ferric ions (Fe(3+)).

139 nificant higher radical scavenging, reducing ferric ions and chelating activities.

140 Furthermore, the depletion of ferric ions during treatment enables monitoring of the F

141 Pyoverdins are expected to complex ferric ions naturally present in cloudwater, thus modify

142 o increase the adsorption efficiency towards ferric ions removal.

143 ting heme, iron-sulfur clusters, and ferrous/ferric ions to apoproteins remain incompletely defined.

144 The arrays developed for the detection of ferric ions, Fe(3+), using a gamma-pyrone derivative che

145 ential factors for adsorption fluctuation of ferric ions.

146 A-RssB (RssAB) directly senses environmental ferric iron (Fe(3+)) and transcriptionally modulates bio

147 directly senses and modulates environmental ferric iron (Fe(3+)) availability to determine swarming

148 evolved mechanisms to chelate and transport ferric iron (Fe(3+)) via siderophore receptor systems, a

149 [(13)C]methane, we demonstrated that soluble ferric iron (Fe(3+), as Fe-citrate) and nanoparticulate

150 Ferric iron (Fe(III)) oxyhydroxides commonly precipitate

151 reductive dissolution and transformation of ferric iron (Fe) oxides and the concomitant release of s

152 s such as phosphate (P) and silicate (Si) by ferric iron (oxyhydr)oxides (FeOx) modulates nutrient mo

153 Finally, we show that a novel, putative ferric iron ABC transporter contributes to low iron fitn

154 ltiple strategies utilized by Mtb to acquire ferric iron and heme iron.

155 can be a half a meter deep, are composed of ferric iron bound to organic polymers - the metabolic by

156 ron-limiting conditions, these high-affinity ferric iron chelators are excreted by bacteria in the so

157 gmanite cation ordering or a decrease in the ferric iron content of the lower mantle.

158 t decay via N-O bond homolysis to ferrous or ferric iron hydroxides in the presence of 1,4-cyclohexad

159 Electron transfer to ferric iron in (oxyhydr-)oxides (hereafter iron oxides)

160 A relatively small proportion of ferric iron is internalized and boosts production of iro

161 they have been consistently observed to use ferric iron minerals as an electron sink for fermentatio

162 cells, extracellular organic compounds, and ferric iron minerals.

163 nation complex (two tablets per meal, 210 mg ferric iron per tablet) or usual care for 9 months or un

164 tic sediments provided with H(2), CO(2), and ferric iron produced a chemolithoautotrophic population

165 allows directly following decreases in oxide ferric iron reducibility during the transformation of fe

166 Ferrous iron formed during microbial ferric iron reduction induces phase transformations of p

167 uent export of Mtb siderophores, followed by ferric iron scavenging and ferric-siderophore import int

168 s of STEAP3, the oxidoreductase that reduces ferric iron to the ferrous oxidation state, in the Broad

169 ses in the reactivity of the remaining oxide ferric iron toward reduction (i.e., its reducibility) ha

170 The ferric iron uptake (Fiu) transporter from Escherichia co

171 e pathogens may rely on siderophore-mediated ferric iron uptake, ferrous iron uptake, or heme uptake

172 methanogenic mesocosms with arsenic-bearing ferric iron waste from an electrocoagulation drinking wa

173 oquine resistance transport both ferrous and ferric iron, albeit with different kinetics.

174 experimental addition of haemoglobin (Hb) or ferric iron, and reduced following addition of the iron

175 identification and detection of iron (III) (ferric iron, Fe(3+)) using Nile red (NR) as a complexing

176 oxidizes ammonium to nitrite while reducing ferric iron, were conducted in the presence of PFOA or P

177 ic tunnel and displaces LeuE11 away from the ferric iron, which forces open a short tunnel to the cat

178 This allows FADH(2) to reduce the ferric iron, which forms the stable ferric-superoxide-Ty

179 assay indicated that these metabolites bind ferric iron, which suppresses their production when adde

180 spectroscopy allowed its identification as a ferric iron-nitrosyl complex.

181 ofilm formation requires large quantities of ferric iron.

182 position of organic matter co-localized with ferric iron.

183 estart the cycle, LeuE11 toggles back to the ferric iron.

184 ning 2-30 mum grains of various ferrous- and ferric-iron containing minerals, including hypersthene,

185 b) with reversibly bound O2, or paramagnetic ferric methemoglobin (metHb).

186 We hypothesize that either highly reactive ferric minerals or radical S species produced by the oxi

187 tetrakis(4-sulfonatophenyl)porphyrinate) and ferric myoglobin (metMb) to quantitatively yield [Mn(TPP

188 Ferric neuroglobin is slowly reduced by H2S and catalyze

189 formation during the titration of an acidic ferric nitrate solution with NaOH.

190 As(V) solutions, ferric nitrate, and mucilage suspensions were mixed and

191 uman fibroblasts was restored by addition of ferric nitrate.

192 rnative involves the assignment of I435 to a ferric-nitrosyl species.

193 n well studied, examples of stable high-spin ferric nitrosyls (such as those that could be expected t

194 tren co-ligand, we have prepared a high-spin ferric NO adduct ({FeNO}(6) complex) via electrochemical

195 -based and N-based nucleophiles on synthetic ferric-NO hemes.

196 Activation of both synthetic and natural ferric nontronites was observed following the introducti

197 e(III) associated with natural and synthetic ferric nontronites.

198 Hydride attack on the cationic ferric [(OEP)Fe(NO)(5-MeIm)]OTf (OEP = octaethylporphyri

199 h peroxidase and the two-electron compound I/ferric (or Fe(IV)O(Por(*))/Fe(III)OH(2)(Por)) reduction

200 of this potential as well as the compound II/ferric (or Fe(IV)O/Fe(III)OH) reduction potential in hor

201 (6)](2-), we have determined the compound II/ferric (or Fe(IV)OH/Fe(III)OH(2)) couple and its associa

202 rast, the Fe analogue undergoes a ferrous-to-ferric oxidation state conversion during this reaction.

203 xposures to Cu adsorbed to synthetic hydrous ferric oxide (Cu-HFO).

204 ) model in addition to adsorption to hydrous ferric oxide (HFO) and clay.

205 and As(V) sorption to coprecipitated hydrous ferric oxide (HFO) in the binuclear, bridging ((2)C) com

206 ion of a geothermal doublet in which hydrous ferric oxide and hydrous manganese oxide deposits had fo

207 of Fe(II) and Mn(II) by accumulated hydrous ferric oxide and hydrous manganese oxide in the well bor

208 I) coprecipitates (lepidocrocite and hydrous ferric oxide for EC-O2 and EC-H2O2, respectively), regar

209 Biofouling of water wells by hydrous ferric oxide is a widespread problem.

210 lucose by this organism in the presence of a ferric oxide mineral, hematite (Fe2O3), resulted in enha

211 Schwertmannite is a ferric oxyhydroxysulfate mineral, which is common in aci

212 Ferric P450 27C1 reduction by adrenodoxin was 3-fold fas

213 t direct metabolic patterns by the optimized ferric particle-assisted laser desorption/ionization mas

214 ed heme species in solution in the oxidized (ferric) PAS-A protein, and by mutagenesis we identify Hi

215 controversial, in the context of the role of ferric peroxide (FeO2 (-)) versus perferryl (FeO(3+), co

216 ls can react with both ferric superoxide and ferric peroxide intermediates formed during O(2) reducti

217 acetic acid, consonant with proposals for a ferric peroxide mechanism.

218 ound I mechanism, although contribution of a ferric peroxide pathway in the 17alpha,20-lyase reaction

219 , a proton transfer-independent nucleophilic ferric peroxo anion (compound 0, i.e. Fe(3) (+)O(2) (-))

220 cs of cytP450 indicate that a thiolate-bound ferric porphyrin coexists in organic solutions at room t

221 Specifically, reduction of the ferric porphyrin, [Fe(III)(TPP)](+)(,) forms the ferrous

222 native enzyme, most synthetic thiolate-bound ferric porphyrins are unstable in air unless the axial t

223 ve to be considered when biochemical data of ferric proteins are rationalized by constraints derived

224 heme for human methemoglobin, linking hemin (ferric protoporphyrin IX) disassociation and apoprotein

225 cause rice is consumed as intact grains, and ferric pyrophosphate (FePP), which is usually used for r

226 labeled ferrous sulfate (FeSO4; study 1) or ferric pyrophosphate (FePP; study 2).

227 Applying the ferric reducing ability for nanoparticle assay, we revea

228 orrelation with the spectrophotometric FRAP (Ferric Reducing Ability of Plasma) and DPPH (2,2-Dipheny

229 ical cation decolorization assay and FRAP as Ferric Reducing Ability of Plasma), and other basic chem

230 e possessed the greatest DPPH scavenging and ferric reducing activities (p<0.05), but limited ferrous

231 eased amount of hydroxyl terminal groups and ferric reducing activities.

232 on presented the higher Fe(2+) chelating and Ferric reducing activities.

233 yphenols content and antioxidant capacity of ferric reducing antioxidant potential (FRAP) of several

234 crylhydrazyl (DPPH) free radical ability and ferric reducing antioxidant potential (FRAP).

235 xidant capacity was assessed on the basis of ferric reducing antioxidant potential of each food item.

236 The antioxidant properties, measured by the ferric reducing antioxidant power (FRAP) and hydrogen pe

237 benzenothiazoline-6-sulfonic acid (ABTS(+)), ferric reducing antioxidant power (FRAP) and iron (Fe(2+

238 generated RBCF hydrolysates exhibited higher ferric reducing antioxidant power (FRAP) and oxygen radi

239 radical absorbance capacity (H-ORAC(FL)) and ferric reducing antioxidant power (FRAP) assays, exhibit

240 eu, 2,2-diphenyl-1-picrylhydrazyl (DPPH) and ferric reducing antioxidant power (FRAP) methods, respec

241 described silver nanoparticle-based (AgNP), ferric reducing antioxidant power (FRAP), 2,2-diphenyl-1

242 showed the highest antioxidant activities in ferric reducing antioxidant power (FRAP), ABTS, superoxi

243 razyl radical scavenging activity (DPPH) and ferric reducing antioxidant power (FRAP), after in vitro

244 ns (TMA), radical scavenging activity (RSA), ferric reducing antioxidant power (FRAP), and a number o

245 scavenging capacity (DPPH and TEAC) and the ferric reducing antioxidant power (FRAP).

246 PH & ABTS radicals scavenging activities and ferric reducing antioxidant power (r>0.831).

247 The ferric reducing antioxidant power was increased in the s

248 radical absorbance capacity {H-ORAC(FL)} and ferric reducing antioxidant power {FRAP}).

249 increase in radical scavenging activity and ferric reducing antioxidant power, especially in sprouts

250 e bleaching inhibition (IC(50) = 206 ug/mL), ferric reducing power (EC(50) = 35.20 ug/mL), total anti

251 es (scavenging DPPH and ABTS(+) radicals and ferric reducing power).

252 of the microbiota, exhibits an extraordinary ferric-reducing activity.

253 ng capacity of the phenolic antioxidant upon ferric-reducing antioxidant power (FRAP) and oxygen radi

254 obarbituric acid reactive substance (TBARS); ferric-reducing antioxidant power (FRAP); total oxidant

255 binding domain and does not exhibit cellular ferric reductase activity.

256 TEAP4, suggesting that STEAP1 functions as a ferric reductase in STEAP heterotrimers.

257 ormation of the unsaturated lactone; and the ferric-reductase-like enzyme RbtH, which regioselectivel

258 by the capture of the free radical DPPH and ferric reduction ability (FRAP).

259 igher total polyphenolics, anthocyanins, and ferric reduction activity power than HM (21.3% amylose).

260 ucing Iron-Regulated Transporter1 (IRT1) and Ferric Reduction Oxidase2 (FRO2) and their transcription

261 iodide, and bromide efficiently restore the ferric resting state.

262 containing alkyl Grignard reagents in simple ferric salt cross-couplings have been elucidated.

263 ive for effective cross-coupling with simple ferric salts and beta-hydrogen-containing alkyl nucleoph

264 S = 1/2 iron species in reactions of simple ferric salts with MeMgBr proposed to be an iron(I) speci

265 oxadiazolo-[4,3-a]quinoxalin-1-one-oxidized (ferric) sGC was moderate, reaching approximately 10%-15%

266 The molecular details of ferric siderophore-mediated activation of the iron impor

267 herein the CCSSD:NTSD complex forms prior to ferric-siderophore binding.

268 The ferric-siderophore complex limits local access to iron b

269 In Gram-negative bacteria, these ferric-siderophore complexes are actively taken up using

270 ores, followed by ferric iron scavenging and ferric-siderophore import into Mtb.

271 Moreover, a gene encoding a TonB-dependent ferric-siderophore receptor is adjacent to the biosynthe

272 The FD sensors monitored uptake of both ferric siderophores and hemin by the pathogens.

273 fortificants are ferrous sulfate (FeSO4) and ferric sodium EDTA (NaFeEDTA).

274 r of IDO1, which autoxidizes to the inactive ferric state during turnover.

275 tals ( approximately 2.5 nm diameter) in the ferric state.

276 a proton and shows a signal from the peroxo-ferric state.

277 ects of spin (high/low) and valence (ferrous/ferric) states on iron partitioning in the deep mantle.

278 ture of human hemoglobin containing low spin ferric sulfide, the first intermediate in heme-catalyzed

279 py indicated initial formation of a low-spin ferric sulfur-bound species followed by reduction to the

280 ted that phenols/quinols can react with both ferric superoxide and ferric peroxide intermediates form

281 bond and thus facilitates reaction with the ferric superoxide electrophile.

282 he pre-equilibria among ferric, ferrous, and ferric-superoxide intermediates have been quantified und

283 PP), which binds O(2) reversibly to form the ferric-superoxide porphyrin complex, Fe(III)(TPP)(O(2)(*

284 , allow us to propose a mechanism in which a ferric-superoxide reacts with substrate activated by dep

285 duce the ferric iron, which forms the stable ferric-superoxide-TyrB10/GlnE7 complex.

286 Nevertheless, the ferric tannates network, constituting the SAMN@TA shell,

287 e from degradation in air by stabilizing the ferric thiolate ground state in contrast to its syntheti

288 The ferric thiolate state is favored by greater enthalpy and

289 ground state compared to the five-coordinate ferric-thiolate precursor complexes.

290 e-mediated reduction of the iron center from ferric to the ferrous oxidation state.

291 For decades, the bacterial ferric uptake regulator (Fur) has been thought to respon

292 Furthermore, inactivation of the ferric uptake regulator (fur) in P(tet)-cydDC or P(tet)-

293 The ferric uptake regulator (Fur) is a global transcription

294 Deletion of the ferric uptake regulator (Fur) renders mstA cells hyperse

295 The ferric uptake regulator (Fur) senses intracellular iron

296 ron regulation of swarming works through the ferric uptake regulator protein Fur.

297 The ferric uptake regulator protein regulates expression of

298 ted genes, beyond simple iron regulation via ferric uptake regulator, have not been uncovered in this

299 The ferric-uptake regulator (Fur) is an Fe(2+)-responsive tr

300 tic clonal-complex, obtained a mutant in the ferric-uptake-regulator (Fur), and analyzed their transc