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1 displays some of the eye deficits present in fetal alcohol syndrome.
2  that may potentially modify the severity of fetal alcohol syndrome.
3 part, for the ability of EtOH to produce the fetal alcohol syndrome.
4 anol effects on L1 in the pathophysiology of fetal alcohol syndrome.
5 arning were investigated with a rat model of fetal alcohol syndrome.
6 rved in some forms of mental retardation and fetal alcohol syndrome.
7 hol-related neurodevelopmental disorders and fetal alcohol syndrome.
8 sure often produces a condition known as the fetal alcohol syndrome.
9 xecutive processing problems associated with fetal alcohol syndrome.
10 neurobehavioral deficits associated with the fetal alcohol syndrome.
11 ely contributes to the CNS manifestations of fetal alcohol syndrome.
12 region has implications for the mechanism of fetal alcohol syndrome.
13 and development observed in animal models of fetal alcohol syndrome.
14 ies, gastrointestinal diseases, cancers, and fetal alcohol syndrome.
15 ith a confirmed diagnosis of partial or full fetal alcohol syndrome.
16 ditions, including cognitive dysfunction and fetal alcohol syndrome.
17 ity and production of blood cells; and cause fetal alcohol syndrome.
18 ther there was a link between this aspect of fetal alcohol syndrome and loss of Shh.
19 g neurobehavioral and sensory dysfunction in fetal alcohol syndrome and other alcohol-related neurode
20 , which is more than that for Down syndrome, fetal alcohol syndrome, and neural tube defects combined
21 mental changes in early-onset schizophrenia, fetal alcohol syndrome, attention-deficit-hyperactivity
22                         Approximately 90% of fetal alcohol syndrome cases are accompanied by ocular a
23  of the corpus callosum are observed both in fetal alcohol syndrome (FAS) and in children with mutati
24 or MRI1 through the University of Washington Fetal Alcohol Syndrome (FAS) Follow-Up Study.
25                                              Fetal alcohol syndrome (FAS) is the leading cause of men
26  secondary effects on the embryo, as well as fetal alcohol syndrome (FAS), chromosomal abnormalities
27 sis can lead to brain defects referred to as fetal alcohol syndrome (FAS), which can include mental h
28 bid conditions occurring in individuals with fetal alcohol syndrome (FAS).
29 tion of symptoms collectively referred to as fetal alcohol syndrome (FAS).
30 mes leading to birth defects associated with fetal alcohol syndrome (FAS).
31 could contribute to cognitive dysfunction in fetal alcohol syndrome (FAS).
32 ay represent a target for ethanol related to fetal alcohol syndrome (FAS).
33  microvascular network in human controls and fetal alcohol syndrome (FAS)/partial FAS (pFAS) patients
34  may critically contribute to the genesis of fetal alcohol syndrome in fetuses, as well as to abnorma
35 eimer's disease, pediatric brain cancer, and fetal alcohol syndrome, in addition to multiinstitutiona
36 s effects on the human fetal nervous system (fetal alcohol syndrome), including components of the vis
37 ize that ethanol-induced defects observed in fetal alcohol syndrome involve ethanol inhibition of ADH
38                                              Fetal alcohol syndrome is a leading cause of mental reta
39                                              Fetal alcohol syndrome is a major cause of learning and
40                                A hallmark of fetal alcohol syndrome is microencephaly.
41 st that one of the "putative" mechanisms for fetal alcohol syndrome is the inhibition of msx2 express
42        Although microcephaly is a feature of Fetal Alcohol Syndrome, it is currently unknown whether
43         This study suggests that adults with fetal alcohol syndrome or fetal alcohol effects suffer f
44    Twenty-five subjects who met criteria for fetal alcohol syndrome or fetal alcohol effects, who wer
45 d frequency of mental illness in adults with fetal alcohol syndrome or fetal alcohol effects.
46          Neuronal loss is a key component of fetal alcohol syndrome pathophysiology.
47                                The brains of fetal alcohol syndrome patients exhibit impaired neurona
48 ted that L1 is a direct target of ethanol in fetal alcohol syndrome, since ethanol inhibits the aggre
49              In our postulated mechanism for fetal alcohol syndrome, this enzyme may well play a cruc
50 mediate the differential genes expression in fetal alcohol syndrome were unknown; microarray data fro
51       Gestational exposure to ethanol causes fetal alcohol syndrome, which is associated with cerebel