ライフサイエンスコーパス: fetal blood

1 .6/10(6) nucleated cells (69.6 +/- 10/microL fetal blood).

2 tal transfer of amino acids from maternal to fetal blood.

3 ormal, healthy adults and in placental cord (fetal) blood.

4 tissue mass, with a significant elevation in fetal blood adiponectin.

5 el of ethanol in utero (average maternal and fetal blood alcohol level of 25 mg/dl) promotes prematur

6 triads of human placenta and of maternal and fetal blood and found large subject-to-subject variabili

7 of nutrients and gases between maternal and fetal blood and is the principal site for synthesizing h

8 Monocytes from fetal blood and lung isolated from U. parvum 70 d + LPS

9 al cells (facing uninfected blood simulating fetal blood and termed "endothelial" side) are cultured

10 ns of inflammatory mediators in maternal and fetal blood, and assess clinical consequences.

11 nutrients are exchanged between maternal and fetal blood, and the smooth chorion (SC) which surrounds

12 isorganized, with thickening of the maternal-fetal blood barrier and an associated reduction in diffu

13 n-6 (IL-6), which can cross the placenta and fetal blood-brain barrier to alter brain development wit

14 D-OH was more effective in crossing the fetal blood-brain barrier, and targeting activated micro

15 d immune factors that cross the placenta and fetal blood-brain barrier.

16 NT virus transmission: the placenta and the fetal blood-brain barrier.

17 id (~10 samples tested/animal), maternal and fetal blood by culture and polymerase chain reaction.

18 uded that PTHrP is an important regulator of fetal blood calcium and placental calcium transport.

19 These results suggest that full-term fetal blood can engraft allogeneic hosts across the majo

20 decorated with immunosuppressive glycans-or fetal blood cell derived.

21 like B-1 B cells preferentially arise during fetal blood cell development.

22 ce-activated cell sorter analysis on EGFP(+) fetal blood cells revealed that surface expression of CX

23 Similar behavior was displayed when fetal blood cells were tested in vitro on immobilized re

24 cell-depleted adult bone marrow or full-term fetal blood cells, as a model of cord blood in a murine

25 al fate giving rise to a normal yolk sac and fetal blood cells.

26 ation [Ca(2+)] was significantly lower in P0 fetal blood compared with both WT and maternal blood at

27 The lower fetal blood concentrations are likely due to active effl

28 primary murine brain-derived ECs (MBECs) or fetal blood-derived ECs (FBECs).

29 immunophenotype were comparable to those of fetal blood-derived MSCs and similarly differentiated al

30 eless data acquisition system able to record fetal blood flow signals in addition to fetal blood pres

31 index ratio as an indicator of preferential fetal blood flow to the upper body parts at the expense

32 A preferential fetal blood flow to the upper body parts at the expense

33 essfully discriminated the maternal from the fetal blood flows; the two orders of magnitude differenc

34 Cardiac nerve blockade exaggerated the fetal blood gas response to haemorrhage somewhat but did

35 Fetal blood glucose and insulin were increased (P<0.01,

36 h and development, placental efficiency, and fetal blood glucose levels were low, and the fetal blood

37 e DNA-based methods has been for determining fetal blood group in pregnancies when the fetus is at ri

38 Allogeneic full-term fetal blood has poorer radioprotective capacity but grea

39 f a positive B19V-IgG or B19V-IgM finding in fetal blood increased with gestational age.

40 Fetal blood ionized calcium was significantly reduced in

41 Oxygen transport from maternal blood to fetal blood is a primary function of the placenta.

42 The normal calcium concentration in fetal blood is raised above the maternal level, an incre

43 Fetal blood lactate concentrations reached a peak at 8 h

44 mino acid transporter Slc38a4 was increased, fetal blood levels of individual amino acids were simila

45 solated and characterized in first-trimester fetal blood, liver, and bone marrow.

46 We transplanted human first-trimester fetal blood mesenchymal stem cells (MSCs) into homozygou

47 fetal blood glucose levels were low, and the fetal blood metabolome was unchanged.

48 Fetal blood MSCs could be expanded for at least 20 passa

49 Fetal blood MSCs cultured in adipogenic, osteogenic, or

50 Fetal blood MSCs supported the proliferation and differe

51 In their undifferentiated state, fetal blood MSCs were CD29(+), CD44(+), SH2(+), SH3(+),

52 cyte recruitment.(1) They report that murine fetal blood neutrophil rolling, adhesion, and extravasat

53 We show that fetal blood neutrophils acquire the ability to roll and

54 pment, but that before embryonic day (E) 15, fetal blood neutrophils display little ability to roll o

55 When 10(6) fetal blood nucleated cells (median gestational age, 10(

56 or obstetric delivery and oxygen therapy and fetal blood or blood gas or acid-base imbalance.

57 that individual PHSC from adult marrow, late fetal blood, or newborn blood each produce similar fract

58 ay of termination, samples from maternal and fetal blood, placenta, and fetal organs were collected a

59 g of the exchange properties of maternal and fetal blood pools--and thereby of placental function.

60 cord fetal blood flow signals in addition to fetal blood pressure and heart rate from free moving ewe

61 Fetal blood pressure and heart rate were monitored and f

62 nd dexamethasone induce similar increases in fetal blood pressure and similar falls in the incidence

63 A pronounced increase in fetal blood pressure occurred following both betamethaso

64 ith the phenotype (c-kit(high)Thy-1(low)) of fetal blood promastocytes at 3 wk of culture that progre

65 At a per-cell level, B cells from fetal blood recipients did not proliferate as well as th

66 Allogeneic full-term fetal blood recipients had decreased absolute numbers of

67 In addition, reconstituting T cells in fetal blood recipients had decreased mouse T-cell recept

68 Splenic T cells in allogeneic full-term fetal blood recipients proliferated poorly, were unable

69 Immunohistochemical examination of fetal blood revealed primitive pyrenocytes that were con

70 enous fetal blood saturation quantified from fetal blood samples by using a hemoximeter.

71 DNA and anti-B19V antibodies in maternal and fetal blood samples obtained from 41 pregnancies that we

72 Maternal and fetal blood samples were collected from parturients prio

73 Fetal blood samples were drawn from the fetal brachial a

74 d pressure and heart rate were monitored and fetal blood samples were drawn to measure the response t

75 V-IgG were detected in 100%, 28%, and 24% of fetal blood samples, respectively.

76 multiple organs and virus was isolated from fetal blood samples.

77 nuous cardiotocography (1.39; 1.33-1.45) and fetal blood sampling (1.30; 1.14-1.47) with admission ca

78 Serial fetal blood sampling (FBS) and intrauterine platelet tra

79 entesis, chorionic villus sampling (CVS) and fetal blood sampling are used to obtain fetal cells for

80 At the time of fetal blood sampling, all mothers were B19V-DNA positive

81 apparent, probably because of liberal use of fetal blood sampling.

82 Good agreement was found between fetal blood saturation determined by the transabdominal

83 two-layer diffusion model to deconvolve the fetal blood saturation from that of the pregnant ewe.

84 abdominal NIR method and arterial and venous fetal blood saturation quantified from fetal blood sampl

85 ucose carbohydrates and polyols are found in fetal blood, some in concentrations higher than maternal

86 vessel patterning and over 50% reduction in fetal blood space.

87 Notably, we also found that the fetal blood spaces in highland-ancestry placentas have i

88 g morphogenesis, alterations in maternal and fetal blood spaces, and failure to remodel the maternal

89 d ancestry was also associated with narrower fetal blood spaces, which could increase exchange effici

90 NKPs were also found in the fetal blood, spleen, and thymus.

91 Fetal blood stem/stromal cells engrafted in bones, diffe

92 duced in HLA-A*0201(-) mothers and enter the fetal blood system.

93 These data suggest that impaired fetal blood to the abdominal organs and smaller fetal ki

94 th a concomitant decrease in the area of the fetal blood vessel network in the labyrinthine zone, sug

95 mbryonic lethality due to the atrophy of the fetal blood vessel network in the placenta.

96 on of the placenta, due to a decrease in the fetal blood vessels, and decreased expression of the gap

97 ic retinal vasculature and the regression of fetal blood vessels, causing persistent hyperplasia of t

98 Furthermore, these fetal blood Vgamma9Vdelta2 T cells are functionally prep

99 Fetal blood Vgamma9Vdelta2 T cells are phosphoantigen re

100 Thus, our data indicate that fetal blood Vgamma9Vdelta2 T cells find their origin in

101 The presence of B19V-IgG in fetal blood was accompanied by lower B19V-DNA levels and

102 NK)/T-cell-restricted progenitor cell in the fetal blood, with a phenotype of NK1.1(+) CD117 (c-kit)+