ライフサイエンスコーパス: fetal development

1 out life (beyond guiding brain wiring during fetal development).

2 pig placentae grow heavily, on placental and fetal development.

3 lead to pregnancy complications and impaired fetal development.

4 Maternal diet and metabolism impact fetal development.

5 n the brain and increase in abundance during fetal development.

6 , presenting a heightened risk of perturbing fetal development.

7 Folate is vital for fetal development.

8 rise from abnormalities in germ cells during fetal development.

9 an early, but narrow, window of time during fetal development.

10 layers and map transcriptionally to in vivo fetal development.

11 of developmental delay (DD) originate during fetal development.

12 ncluding in the heart and lung, during human fetal development.

13 ol of gene expression is critical for normal fetal development.

14 e compensatory response that sustains normal fetal development.

15 e myelination of the peripheral axons during fetal development.

16 xposure to air pollutants is associated with fetal development.

17 t in spinal cord and vertebral growth during fetal development.

18 ticosteroids during their first trimester of fetal development.

19 nd muscle functions and important for normal fetal development.

20 gration between the 7(th) and 16(th) week of fetal development.

21 participates in beta cell programming during fetal development.

22 ously identified coexpression modules during fetal development.

23 onocytes in an undifferentiated state during fetal development.

24 expression of mouse embryonic globins during fetal development.

25 MeCP2 and interacting proteins during human fetal development.

26 e, investigated leukocyte recruitment during fetal development.

27 Cs, and some remain undifferentiated through fetal development.

28 uitment proceeds in a similar fashion during fetal development.

29 transient organ that is necessary for proper fetal development.

30 growth, revealing a role for MC1R in normal fetal development.

31 epigenetic mechanisms potentially affecting fetal development.

32 st as an underlying cause that starts during fetal development.

33 r these gene promoters epigenetically during fetal development.

34 ion from scarless to fibrotic healing during fetal development.

35 ure NK-cell progeny emerge and expand during fetal development.

36 to reconstruct exposure at specific times in fetal development.

37 utcomes across the lifespan can be traced to fetal development.

38 ity of germ cells to study especially during fetal development.

39 naling is required for eyelid closure during fetal development.

40 ing placental transport of Hcy may impact on fetal development.

41 n) is important for successful completion of fetal development.

42 ood-brain barrier permeability during normal fetal development.

43 ophoblast metabolism and function as well as fetal development.

44 e adult Eln(+/)(-) mouse are defined in late fetal development.

45 e human and mouse brain during embryonic and fetal development.

46 GACI usually occurs during fetal development.

47 ion with increases in plasma cortisol during fetal development.

48 wing to inaccessibility of germ cells during fetal development.

49 immunovascular role during placentation and fetal development.

50 rs through impacts on placental function and fetal development.

51 c-Kit(+)) that enlarged centrifugally during fetal development.

52 s a hematopoietic organ during embryonic and fetal development.

53 sterone administration in early pregnancy on fetal development.

54 ther subtler (microscopic) defects in murine fetal development.

55 ehog protein focuses on their role in normal fetal development.

56 te the effects of placental insufficiency on fetal development.

57 mechanisms to balance maternal immunity and fetal development.

58 he control of gamma to beta switching during fetal development.

59 expressed an activated phenotype throughout fetal development.

60 eyelid epithelium morphogenesis during mouse fetal development.

61 lopment of the placenta is crucial to normal fetal development.

62 bin gene coexpression and competition during fetal development.

63 Cholesterol is required for fetal development.

64 that amplifies the maternal blood supply for fetal development.

65 anscript is found in multiple tissues during fetal development.

66 specific marker of endothelial cells during fetal development.

67 , a process that is presumed to begin during fetal development.

68 r regulated IGF bioavailability during early fetal development.

69 (DETC), homes to the murine epidermis during fetal development.

70 sue-specific genes normally expressed during fetal development.

71 late homeostatic T-cell proliferation during fetal development.

72 ensatory growth of the placentae to maintain fetal development.

73 involved in neuronal differentiation during fetal development.

74 uding systematic determinations across mouse fetal development.

75 ved cellular programs operating during human fetal development.

76 the maternal reproductive tract to influence fetal development.

77 acenta that regulate maternal physiology and fetal development.

78 multinucleated cell structure essential for fetal development.

79 ial physiological functions to ensure normal fetal development.

80 the complex chemical changes associated with fetal development.

81 ration of a common foregut tube during early fetal development.

82 ions, plays a critical role in embryonic and fetal development.

83 l slowing mechanism in response to extensive fetal development.

84 t exposure to hydrocarbons can pose risks to fetal development.

85 ternal nutrition is critically important for fetal development.

86 throughout pregnancy is critical for normal fetal development.

87 in nutrients that are essential for healthy fetal development.

88 terine environment could also interfere with fetal development.

89 ered to be benign with regards to effects on fetal development.

90 ZIKV's neurotropic effects in the course of fetal development.

91 otin-transport mechanism and is critical for fetal development.

92 sm through which maternal stress may disrupt fetal development.

93 nct characteristics associated with abnormal fetal development.

94 ervous system formation during embryonic and fetal development.

95 changes required for placental formation and fetal development.

96 l environment to sperm, potentially altering fetal development.

97 derstanding of how they affect pregnancy and fetal development.

98 rnal bariatric surgery may affect subsequent fetal development.

99 sinus primordial mesenchyme in males during fetal development.

100 sed their potential to interfere with neural fetal development.

101 ts of maternal malnutrition on placental and fetal development.

102 ly regulated in the endocrine lineage during fetal development, 237 of which are transcriptional regu

103 During fetal development a subset of ILCs orchestrate the gener

104 During fetal development, a total of ten splice variants of bhm

105 It has been postulated that during human fetal development, all cells of the lung epithelium deri

106 and pathologically during critical stages of fetal development alter nervous system function and beha

107 bility of choline during critical periods of fetal development alters hippocampal development and aff

108 ues to pose a problem for pregnant women and fetal development, an incomplete understanding of placen

109 ne hippocampus presents many similarities in fetal development, anatomy, and physiology with human hi

110 its activity is particularly relevant during fetal development and adrenarche.

111 lecules and their effector mechanisms during fetal development and adult homeostasis.

112 ieved to regulate growth of the liver during fetal development and after injury in adults, because th

113 GenX alters fetal development and antibody production and elicits to

114 eight percent of such DHSs are active during fetal development and are enriched in variants associate

115 g events take place during gametogenesis and fetal development and are thought to have long-lasting c

116 iving germ cells enter meiosis at the end of fetal development and as a result, the postnatal ovary h

117 sis for our understanding of many aspects of fetal development and behaviour that remain in use in cl

118 These defects are reflected in retarded fetal development and compromised pregnancy outcome.

119 unctional in cerebral blood vessels early in fetal development and continue to play a vital role in m

120 hat low oxygen tension (hypoxia) may control fetal development and differentiation.

121 ions can be observed in the brain throughout fetal development and disappear after birth, except in c

122 number of important biologic effects during fetal development and during the aging process.

123 atus may be an additional factor influencing fetal development and effects of environmental toxins.

124 tion of biological processes can affect both fetal development and health outcomes that manifest late

125 veal an essential role for the Notch1 TAD in fetal development and identify important cell-autonomous

126 acental amino acid transport is required for fetal development and impaired transport has been associ

127 and important function for SALSA during the fetal development and in the mucosal innate immune defen

128 sion (P<0.0001) in the frontal cortex during fetal development and in the temporal-parietal and sub-c

129 in up-regulation of all genes present during fetal development and increases the cell size of neonata

130 s, exposure to FRalpha autoantibodies during fetal development and infancy could contribute to brain

131 initiation of sexual differentiation during fetal development and into postmeiotic stages.

132 DHA plays a critical role in fetal development and is linked to health endpoints in a

133 ical signaling molecule during embryonic and fetal development and is necessary for maternal health.

134 ses on the roles of sterols in embryonic and fetal development and metabolism.

135 we provide evidence that genes facilitating fetal development and nutrient transport display converg

136 cental mitochondrial function, which affects fetal development and offspring long-term health.

137 n, the effect of GWG throughout pregnancy on fetal development and other outcomes has not been extens

138 ; 2) pulmonary vascular disease accompanying fetal development and perinatal life; 3) properties of p

139 a single essential nutrient, choline, during fetal development and point to these pathways as candida

140 n most mammals, placentation is critical for fetal development and pregnancy success.

141 ts with the MAP3K1 signaling pathways during fetal development and provide strong empirical evidence

142 embryonic myosin isoform is expressed during fetal development and rapidly down-regulated after birth

143 ndirect impacts of the parasite community on fetal development and sex allocation.

144 les might influence prenatal factors such as fetal development and sex allocation.

145 for the intrauterine environment in shaping fetal development and subsequent child health and diseas

146 tus and prenatal lead exposure can influence fetal development and subsequent health.

147 ations in fetal lambs may indicate a role in fetal development and suggest that extracellular formate

148 cient erythroid URO-synthase was present for fetal development and survival.

149 These studies highlight the period of fetal development and the processes of chromatin structu

150 idely expressed in many human tissues during fetal development and throughout life.

151 receptors to a role in morphogenesis during fetal development and to a role in the metabolism of pho

152 mother for the metabolic stress presented by fetal development and to ensure appropriate nutrient all

153 expand the functional roles of oxysterols to fetal development and to the detoxification of oxidation

154 ing secretions from the placenta that affect fetal development and whether a mitochondria-targeted an

155 an extracellular matrix protein important in fetal development and wound healing, yet its antimicrobi

156 eight, and mouse stem cell aging during late fetal development and young adulthood.

157 astating consequences on pregnancy outcomes, fetal development, and maternal health.

158 fetal viral infection to pregnancy outcome, fetal development, and maternal well-being.

159 od-brain permeability early, but not late in fetal development, and pretreatment with glucocorticoids

160 are required for lymph node formation during fetal development, and recent evidence implies a role in

161 h vitamin A deficiency can be induced during fetal development, and reveals new functions for the vit

162 8(+) dT that are permissive of placental and fetal development, and reversal of this dysfunctional st

163 ained high in Bcl11a(cko/cko) embryos during fetal development, and this was further augmented in Klf

164 al obesity increases oxidative stress during fetal development, and to determine whether administrati

165 gin to endochondral bones are hypoxic during fetal development, and we demonstrate that Hif-1alpha is

166 Rather, compromised fetal development appears to establish a "meta-plastic"

167 ecursors from colonizing distal bowel during fetal development are not completely understood in many

168 euron types and enhance neurite growth after fetal development are not well understood.

169 deficiencies in the placenta that undermine fetal development are poorly understood.

170 for the maternofetal transport of Cbl during fetal development.-Arora, K., Sequeira, J.

171 ant to cancer are well known to overlap with fetal development, as reflected in reactivation of embry

172 It is likely that the stage of fetal development, as well as the state of differentiati

173 Likewise, any gene that favors embryonic/fetal development at consequent cost to the mother--by a

174 ession and adipogenic differentiation during fetal development, at least partially through reducing D

175 ever, antibodies can access the brain during fetal development before the barrier achieves full integ

176 Following differentiation during fetal development, beta cells further adapt to their pos

177 It forms during late stages of fetal development but continues into early adult life.

178 CRD-BP levels are high during fetal development but low or undetectable in normal adul

179 long bones has been studied in-depth during fetal development but not postnatally in the epiphysis.

180 is a rich source of essential nutrients for fetal development, but in contrast, it is also a well-kn

181 g mechanisms: a direct detrimental effect on fetal development by crossing the placenta and entering

182 le antiandrogenic phthalates exposure during fetal development can have greater impacts than individu

183 Programming of the immune system during fetal development can influence asthma-related risk fact

184 cal mechanism by which folate acts to affect fetal development can inform appraisal of expected benef

185 hlights how knowledge of normal processes in fetal development can provide insight into tumorigenesis

186 at the repression of HMGA2 expression during fetal development could contribute to the specific birth

187 During fetal development, cutaneous wounds heal without inflamm

188 pite normal implantation and early placental/fetal development, deletion of Bmpr2 in the uterine deci

189 Excess proliferation during fetal development disrupts distal airspace formation, me

190 d a negative effect of parasite community on fetal development, driven primarily by the nematode Mars

191 ce ABs, yet little is known about effects on fetal development during pregnancy.

192 ply of the essential nutrient choline during fetal development [embryonic day (E) 11-17] in rats caus

193 s of pregnant Etnk2(-/-) females showed that fetal development failed at the late stage of pregnancy

194 Germ cells specified during fetal development form the foundation of the mammalian g

195 ressing neurons within the brainstem late in fetal development; gender specificity derives from a tim

196 l and ventrolateral prefrontal cortex during fetal development, genes harboring damaging de novo muta

197 pression relative to histone H4 genes during fetal development has been explored.

198 Exposure to chronic hypoxia during fetal development has important effects on immediate and

199 n the Americas and its devastating impact on fetal development have prompted the World Health Organiz

200 Fetal development in a hostile gestational environment c

201 imary motor--somatosensory cortex during mid-fetal development in autism spectrum disorder and the fr

202 Lactation is necessary for both infant and fetal development in eutherians and marsupials, although

203 The bumps emerge during fetal development in macaques, indicating that they aris

204 at atypical E2Fs promote angiogenesis during fetal development in mice and zebrafish.

205 tic mechanisms during a vulnerable period of fetal development in mice.

206 ified widespread expression primarily during fetal development in myocytes and interstitial cells sug

207 ch to understand molecular events underlying fetal development in response to hypoxia.

208 epigenetic profile changes occurring during fetal development in response to in utero environment va

209 ctrum disorder and the frontal cortex during fetal development in schizophrenia.

210 in the ventricular conduction system during fetal development in sheep.

211 miRNA profiling during late fetal development in the mouse identified miR-23b cluste

212 ting, we studied gene-expression profiles in fetal development in the relevant tissues and time inter

213 During the last 3 days of fetal development in the rodent, a burst of hepatocyte p

214 exposures, including those that occur during fetal development in utero, can cause epigenetic effects

215 ed reciprocal decreases in abundance late in fetal development, in newborn and adult sheep.

216 ic signaling pathways that are active during fetal development, including Hedgehog and Hippo/Yes-asso

217 are important in driving several aspects of fetal development, including muscle fibre differentiatio

218 l functions can have a significant impact on fetal development, including the brain, outcomes that ar

219 t and human cardiac physiology and placental-fetal development indicate a need for models in precocia

220 t and human cardiac physiology and placental-fetal development indicate a need for models in precocia

221 onmental influences during the embryonic and fetal development influence the individual's susceptibil

222 is known in humans about how testosterone in fetal development influences later neural sensitivity to

223 Vitamin D exposure during fetal development influences the immune system of the ne

224 Ethanol exposure during fetal development is a leading cause of learning disabil

225 In addition, overexpression of ZAC during fetal development is believed to underlie the rare disor

226 show that HSPC engraftment of bone marrow in fetal development is dependent on the guanine-nucleotide

227 rogramming occurs when the normal pattern of fetal development is disrupted by an abnormal stimulus o

228 ected in exosomes, but their function during fetal development is poorly understood.

229 of maternal 25-hydroxyvitamin D [25(OH)D] in fetal development is uncertain, and findings of observat

230 rocarbon exposure during critical periods of fetal development is urgently needed.

231 ly highly expressed in the liver only during fetal development, is reactivated in 60% of HCC tumors a

232 idney does not ascend as it should in normal fetal development, it remains in the pelvic area and is

233 Exposure to chronic hypoxia during fetal development leads to accelerated aging of the soma

234 ing the proposal that viral infection during fetal development may play a causal role in the pathogen

235 Maternal nutrient reduction (MNR) during fetal development may predispose offspring to chronic di

236 ell S6K1 signaling, rather than IUGR, during fetal development may underlie reduced beta cell growth

237 Biological mechanisms that explain how fetal development might influence the risk of adult dise

238 he impact of maternal Delta9-THC exposure on fetal development, neonatal outcomes, and placental deve

239 During fetal development, nephrons of the metanephric kidney fo

240 During the second trimester of human fetal development, neural structures in the brain underg

241 During late stages of fetal development, non-CG methylation accumulated within

242 e mouse germ line have revealed that much of fetal development occurs normally in their absence.

243 amniotic fluid choline levels would enhance fetal development of cerebral inhibition and, as a resul

244 indicate that, in contrast to embryonic and fetal development of clones, the process of NT-ES cell d

245 ronment transplacentally prime and result in fetal development of memory T cells.

246 The fetal development of the anterior subventricular zone (S

247 The fetal development of the mammalian eyelid involves the e

248 not cause any adverse effects on pregnancy, fetal development, or offspring behavior.

249 ly nursing period are representative for the fetal development period, using serial maternal serum sa

250 arly-mid gestation may have implications for fetal development, possibly causing a long-term phenotyp

251 proliferation of cortical precursors during fetal development provides a likely environment for soma

252 , taking a lifespan perspective beginning in fetal development provides the opportunity to target abn

253 lucidating the mechanisms by which disrupted fetal development raises the risk of this disorder.

254 Choline deficiency during fetal development reduces proliferation and migration of

255 s) whereby excess maternal nutrition affects fetal development remain poorly understood.

256 Disruption of Esrra and Esrrg in mice during fetal development resulted in perinatal lethality associ

257 of glucocorticoids during specific times of fetal development results in focal and segmental glomeru

258 ues innervated by vagal motor neurons during fetal development reveal potential sites of HGF-MET inte

259 ssion, and Il7r-Cre-mediated labeling across fetal development revealed dynamic regulation of Il7r mR

260 During fetal development, several cIN subtypes derive from the

261 Hematopoietic transitions that accompany fetal development, such as erythroid globin chain switch

262 these data indicate that ARMC5 is crucial in fetal development, T-cell function and adrenal gland gro

263 teins, were expressed more abundantly during fetal development than during postnatal ages, and their

264 part, from pathogenic processes initiated in fetal development that involve sex differences in shared

265 emical changes associated with embryonic and fetal development that, when disturbed, causes congenita

266 Using 3D imaging, we find that during fetal development the villus undergoes gross remodelling

267 the peripheral lymphoid tissues early during fetal development, the adaptive immune system in the fet

268 During fetal development, the cartilaginous epiphysis of the di

269 During fetal development, the uterine environment can have effe

270 dent mechanisms whereby they could influence fetal development, these 2 nutrients also have a common

271 bolic pathways and hypoxic conditions during fetal development; this stress may contribute to increas

272 49 female) subjects from the late stages of fetal development through 9 y of age.

273 extensive series of post-mortem brains from fetal development through ageing.

274 from 111 children during the late stages of fetal development through early adolescence.

275 rth and death in the Monodelphis retina from fetal development through early postnatal life.

276 O enhances adipogenic differentiation during fetal development through inducing epigenetic changes in

277 rental disorders and offspring outcomes from fetal development to adolescence in high-income, middle-

278 ISG expression across five time points from fetal development to adulthood in wild-type and RdRP mic

279 and euploid control brains spanning from mid-fetal development to adulthood.

280 view of chromatin dynamics during mammalian fetal development to date.

281 , serves as a donor of methyl groups used in fetal development to establish the epigenetic DNA and hi

282 structed segment of the uterus and supported fetal development to term and live birth.

283 gues demonstrate a role for platelets beyond fetal development, to maintaining integrity of the adult

284 an established late gestation ovine model of fetal development under chronic hypoxic conditions, we i

285 Conditional Prdm16 deletion during fetal development using Nestin-Cre prevented the formati

286 Restricted V(D)J recombination during fetal development was postulated to limit antibody reper

287 Hearts at late fetal development were analyzed for engraftment of human

288 fter brain death, and the critical needs for fetal development were included.

289 72 hours of birth but are viable throughout fetal development when dramatic cardiovascular structura

290 Choline is critical during fetal development, when it influences stem cell prolifer

291 umber of murine B-1 progenitors peaks during fetal development whereas B-2 B cell production predomin

292 ere on inflamed yolk sac vessels during late fetal development, whereas at earlier embryonic stages (

293 nts predominantly lose CG methylation during fetal development, whereas the trend is reversed after b

294 essential for normal placental function and fetal development; whether transport of these is altered

295 of gene expression changes occurring during fetal development which are reversed in early postnatal

296 and maternal obesity (MO) negatively affects fetal development, which predisposes offspring to metabo

297 omatin in fetal tissues, potentially linking fetal development with BP regulation in later life.

298 Maternal nutrient restriction (NR) affects fetal development with long-term consequences on postnat

299 nvironmental conditions during embryonic and fetal development with risk of diseases later in life.

300 erine conditions that provoke adjustments in fetal development, with long-term consequences for stres