ライフサイエンスコーパス: fibrinogen

1 eactive protein], TNF-alpha R1, D-dimer, and fibrinogen).

2 cally and dose dependently adhered to bovine fibrinogen.

3 evels of plasminogen, alpha2-antiplasmin, or fibrinogen.

4 n to human-derived fibronectin, keratin, and fibrinogen.

5 r matrix components, such as fibronectin and fibrinogen.

6 bles such as probing depth (PD) with CRP and fibrinogen.

7 rowth at near human plasma concentrations of fibrinogen.

8 K(D) of 86 nm, ~20-fold lower than that for fibrinogen.

9 well as purified and fluorescently labelled fibrinogen.

10 gher usual levels of log-transformed CRP and fibrinogen.

11 rget epitopes in IgG1 Fc and the ACPA target fibrinogen.

12 lpha(M)beta(2) integrin-mediated adhesion to fibrinogen.

13 < .0001) minutes in clots made from purified fibrinogen.

14 integrin alphaIIbbeta3-mediated spreading on fibrinogen.

15 ides, in the absence of a T cell response to fibrinogen.

16 -fold stronger binding affinity of Abeta for fibrinogen.

17 zM sequences and binding of equine and human fibrinogens.

18 50-10,000 pg/mL), cTnI (1-10,000 pg/mL), and fibrinogen (0.1-5 mg/mL).

19 M) adhered to a fibrin matrix (0.1-0.4 mg/mL fibrinogen, 10 nM thrombin) under a variety of venous fl

20 less likely to have a source of concentrated fibrinogen (40% versus 56%, aOR 0.44, 95% CI 0.21-0.92,

21 The first case of hereditary fibrinogen Aalpha-chain amyloidosis was recognized >20 y

22 a French kindred with a novel amyloidogenic fibrinogen Aalpha-chain frameshift variant, Phe521Leufs,

23 easured levels of circulating EVTF activity, fibrinogen, activated partial prothrombin time, D-dimer,

24 easured levels of circulating EVTF activity, fibrinogen, activated partial prothrombin time, prothrom

25 hypertension-associated biomarkers, namely, fibrinogen, adiponectin, low-density lipoprotein, and 8-

26 exchange of two blood proteins, albumin and fibrinogen, adsorbing to and competitively displacing (G

27 of AAVP particles that feature resistance to fibrinogen adsorption or neutralizing antibodies and abi

28 ne, we show that UHRA does not interact with fibrinogen, affect fibrin polymerization during clot for

29 granule secretion, adhesion and spreading on fibrinogen all attenuated along with a reduction in thro

30 le to construct the structural models of the fibrinogen alpha-chain (excluding two highly flexible re

31 of novel cross-links in the alphaC region of fibrinogen alpha.

32 ed quantity among plasma proteins belongs to fibrinogen (alpha, beta and gamma chains), and this prot

33 so the possible interactions of MOF-1s with fibrinogen also studied using fluorescence spectroscopy

34 Fibrinogen, an acute phase protein, has been shown to in

35 ination of 16 genetic instruments for gamma' fibrinogen and 75 genetic instruments for total fibrinog

36 a mutations increase the interaction between fibrinogen and Abeta, which might be central to cerebrov

37 In vitro, adhesion depended on fibrinogen and coagulation factor XIII (FXIII), and supr

38 aster and showed increased spreading on both fibrinogen and collagen.

39 with markers of coagulation exacerbation as fibrinogen and D-dimers, and were increased in patients

40 ncreased adhesion of knock-in neutrophils to fibrinogen and decreased neutrophil chemotaxis to a form

41 Live cell video microscopy showed how fibrinogen and fibrin influence NETosis and neutrophil r

42 eta3-mediated interactions of platelets with fibrinogen and fibrin.

43 dicated a protective effect of higher gamma' fibrinogen and higher total fibrinogen on VTE risk.

44 Moreover, intraspinal extravasation of fibrinogen and immunoglobulins was decreased acutely at

45 imate the causal effect of total circulating fibrinogen and its isoform, gamma' fibrinogen, on risk o

46 eptide that inhibits the interaction between fibrinogen and its Mac-1 integrin receptor.

47 A core of fibrinogen and monocytes was found in 39 (93%) white-cen

48 n addition, the stronger interaction between fibrinogen and mutant Abetas led to a dramatic perturbat

49 nd sulfatides, blocking their association to fibrinogen and P-selectin, respectively.

50 flammatory functions mediated by cleavage of fibrinogen and PAR1, the trypsin-like protease thrombin

51 Maximum amplitude, a factor of fibrinogen and platelet count and function, and a measur

52 nt groups and correlated well with levels of fibrinogen and platelet count.

53 iles with additional derangements related to fibrinogen and platelet function.

54 ittle is known about the association between fibrinogen and serum amyloid A (SAA), a highly fibrillog

55 d in clinical studies due to the presence of fibrinogen and several platelet growth factors.

56 Fibrinogen and the BMP target Id2 are increased in demye

57 ith the fibrin gel formed by directly mixing fibrinogen and thrombin in solution.

58 ic roles, have a surprising association with fibrinogen and tissue remodeling.

59 Genetic instruments for gamma' fibrinogen and total fibrinogen were selected, and the i

60 we probed the molecular interactions between fibrinogen and zeolite nanoparticles using both experime

61 o alpha-granules in resting platelets, binds fibrinogen, and acts as a positive regulator of platelet

62 with a significant amelioration of ferritin, fibrinogen, and alanine aminotransferase levels.

63 -alpha, high-sensitivity C-reactive protein, fibrinogen, and albumin), and bone mineral metabolism (2

64 or blood's hemostatic components (platelets, fibrinogen, and coagulation factors).

65 ng of Abeta40 to integrin alpha(IIb)beta(3), fibrinogen, and GPVI collectively contributed to the for

66 n is dependent on the binding of M1 protein, fibrinogen, and M1-specific IgG to platelets in suscepti

67 Serum glucose, ADP fibrinogen, and mannose were among the strongest predict

68 r antagonist, TNFalpha, leptin, adiponectin, fibrinogen, and plasminogen activator inhibitor-1 were d

69 the Xps-mediated degradation of fibronectin, fibrinogen, and the cytokine interleukin-8 (IL-8).

70 bovine serum albumin antibody (anti-BSA) and fibrinogen antibody (anti-Fg) onto the pCB-coated surfac

71 results showed that both E- and D-domain of fibrinogen are bound to the EMT zeolite NPs via strong e

72 These studies establish fibrinogen as a common target for both RF and ACPAs, and

73 Using extravascular fibrinogen as a marker of vascular disruption, we found

74 multiple sclerosis patients also identified fibrinogen as a significant portion of pEV cargo.

75 al parameters, C-reactive protein (CRP), and fibrinogen as markers of inflammation.

76 sorbed onto these surfaces using lysozyme or fibrinogen as prototype common contaminants.

77 SAA also binds to fibrinogen, as determined with a fluorescent-labelled SA

78 These studies establish that the thrombin/fibrinogen axis is fundamental to host antimicrobial def

79 y the same mechanism by which FnBPB binds to fibrinogen, because FnBPB variants defective in fibrinog

80 rinogen, because FnBPB variants defective in fibrinogen binding also did not bind H3.

81 The peak and net increase of individual fibrinogen binding events correlated with the Ca(2+) tra

82 Genetic elimination of the fibrinogen binding motif to CD11b reduced neuroinflammat

83 ed by thrombin, which simultaneously induces fibrinogen binding to alphaIIbbeta3 and converts fibrino

84 n P2Y-stimulated repetitive Ca(2+) waves and fibrinogen binding to the platelet integrin alpha(IIb)be

85 ADP-evoked fibrinogen binding was initially uniform over the cell p

86 tream of P2Y(1) was essential for ADP-evoked fibrinogen binding, whereas P2Y(12) and the kinase PI3K

87 ceptor 4, activating peptide-induced soluble fibrinogen binding.

88 t each induced a rapid, discrete increase in fibrinogen binding.

89 lasma, and (iv) deletion of srr1, encoding a fibrinogen-binding adhesin, increases GBS survival in wh

90 nt a novel methodology employing nonantibody fibrinogen-binding proteins, termed Affimers, to stabili

91 ivation was observed when MKs were plated on fibrinogen but not on other matrices (fibronectin, vitro

92 tion of mechanical ventilation and admission fibrinogen, but not exposure to extracorporeal support,

93 major component of the fungal cell wall, and fibrinogen C containing domain 1 (FIBCD1) is a chitin-bi

94 , diabetes, sex, and duration of dialysis or fibrinogen, C-reactive protein, and complement C3) confi

95 plasminogen activator inhibitor-1, D-dimer, fibrinogen, C-reactive protein, sST2, galectin-3, cystat

96 cluding ESR, CBC with differential, D-dimer, fibrinogen, C3, C4, IL-6, etc.

97 Albumin and fibrinogen cause a 52.2% and 78.2% attenuation of the do

98 ction in secretion, thrombin generation, and fibrinogen cleavage.

99 Fibrinogen clustered on the surface of ionomycin-stimula

100 Fibrinogen concentrate (4 g; n = 415) or cryoprecipitate

101 Fibrinogen concentrate (FC) represents a potential thera

102 Intravenous, single-dose administration of fibrinogen concentrate (n = 60) or placebo (n = 60), tar

103 ere 16.3 (95% CI, 14.9 to 17.8) units in the fibrinogen concentrate group and 17.0 (95% CI, 15.6 to 1

104 events occurred in 26 patients (7.0%) in the fibrinogen concentrate group and 35 patients (9.6%) in t

105 To determine whether fibrinogen concentrate infusion dosed to achieve a plasm

106 fibrinogenemia after cardiopulmonary bypass, fibrinogen concentrate is noninferior to cryoprecipitate

107 Use of fibrinogen concentrate may be considered for management

108 5 minutes) were randomized to receive either fibrinogen concentrate or placebo.

109 Of 827 randomized patients, 735 (372 fibrinogen concentrate, 363 cryoprecipitate) were treate

110 brinogen replacement with cryoprecipitate or fibrinogen concentrate.

111 patients receiving L-ASP therapy, the use of fibrinogen concentrates may increase the risk of thrombo

112 Administration of fibrinogen concentrates was associated with an increased

113 impairment), physiological function (plasma fibrinogen concentration and lung function), cognitive p

114 cation in neurosurgical patients, and plasma fibrinogen concentration has been identified as a potent

115 t loads >500 nM and sensitive to the initial fibrinogen concentration.

116 astometry maximum clot firmness (FibTEM-MCF; fibrinogen contribution to clot firmness).

117 ransplant-triggered intravascular release of fibrinogen correlating with red blood cell aggregation a

118 plasmin generation and was predicted to bind fibrinogen D fragment close to tissue plasminogen activa

119 Therapeutic depletion of fibrinogen decreases BMP signaling and enhances remyelin

120 deficient mice by simultaneous imposition of fibrinogen deficiency.

121 In contrast, fibrinogen-deficient (Fib-) mice failed to clear the mic

122 receptor, indirect bilirubin, myoglobin, and fibrinogen degradation products.

123 ination with low-dose r-tPA, did not lead to fibrinogen degradation, did not cause bleeding (versus c

124 th prominent elevation of D-dimer and fibrin/fibrinogen-degradation products, whereas abnormalities i

125 Fibrinogen depletion with ancrod reduced both intrahepat

126 ing circulating histones or leucocidins; but fibrinogen depletion/consumption, as in trauma or sepsis

127 deletion in adult mice resulted in increased fibrinogen deposition in lungs and kidneys, indicating e

128 age in plasma and reduced neuroinflammation, fibrinogen deposition, and neurodegeneration in the brai

129 tive in vivo two-photon imaging showed focal fibrinogen deposits associated with loss of dendritic sp

130 Fibrinogen did not bind to or protect neutrophils stimul

131 ise temporal and spatial mechanisms by which fibrinogen-directed inflammatory events may dictate the

132 Fibrinogen effects are rescued by BMP type I receptor in

133 Additionally, fibrinogen experienced a substantial conformational chan

134 electron microscopy images, we observed only fibrinogen-exposed eosinophils undergoing this process.

135 ar CD3-positive T-lymphocyte clustering, and fibrinogen extravasation were demonstrated in the core o

136 re, we investigate the hypothesis that human fibrinogen (FBG) - an acute phase reactant - inhibits hu

137 ESR, CRP, fibrinogen, ferritin, and procalcitonin were higher in p

138 The corresponding value for functional fibrinogen (FF) TEG maximum amplitude (MA) was 19 mm.

139 aureus, has a structured A-domain that binds fibrinogen (Fg) and a disordered repeat-region that bind

140 resulting in the release of the host protein fibrinogen (Fg), which coated the bladder and implant.

141 Presurgical serum fibrinogen (FIB), carcinoembryonic antigen (CEA), and ca

142 ils after binding to cells, but formation of fibrinogen/fibrin-histone aggregates prevented cell deat

143 Water-soluble fibrinogen, following cleavage by thrombin, self-polymer

144 Upon activation, fibrinogen forms large fibrin biopolymers that coalesce

145 ity C-reactive protein, serum amyloid A, and fibrinogen from baseline to end of treatment.

146 Whereas purified fibrinogen from healthy controls displayed no post-trans

147 In vitro guanidinylation of fibrinogen from healthy subjects increased the formation

148 splayed no post-translational modifications, fibrinogen from patients on hemodialysis was glycosylate

149 95% CI, 1.03-1.23]; p = 0.011) and admission fibrinogen (g/L) (odds ratio, 0.73 [0.57-0.91]; p = 0.00

150 formation, with significant upregulation of fibrinogen gamma polypeptide, apolipoproteins A-Ib and A

151 le is the strong upregulation of albumin and fibrinogen genes, which suggest significant liver pathol

152 0.75), fibrinogen level (p = 0.67), or daily fibrinogen goal (p = 0.81).

153 he primary outcome, median blood loss in the fibrinogen group was 50 mL (interquartile range [IQR], 2

154 ematocrit >=25%, platelet count >50,000, and fibrinogen >120 mg/dL.

155 se including acute-phase reactants proteins (fibrinogen, haptoglobin and CRP), cell adhesion molecule

156 apitulates the experimental observation that fibrinogen has a higher affinity for SWCNTs than albumin

157 The results indicated that fibrinogen has a strong and thermodynamically favorable

158 The blood-clotting protein fibrinogen has been implicated in host defense following

159 Recently, we demonstrated that the fibrinogen highly contributed in the protein corona comp

160 activated enzyme catalyzes the formation of fibrinogen hydrogels through covalent intermolecular cro

161 and transendothelial migration via an ICAM1-fibrinogen-ICAM1 bridge.

162 PGI2 over activated and spread platelets on fibrinogen, identified a significant reduction in platel

163 with statistically significant enrichment of fibrinogen, IgG3, and complement components, especially

164 ne plasma samples from all participants, and fibrinogen in a subset (n = 9,380).

165 nt cross-reactivity of RF+ patient sera with fibrinogen in both western blots and ELISAs.

166 It is necessary to consider the role of fibrinogen in NETosis.

167 Together, these data suggest that fibrinogen in pEVs contributes to the perpetuation of ne

168 nd to be driven by CD8+ T cells and required fibrinogen in pEVs.

169 essed by the presence of increased levels of fibrinogen in the surrounding parenchyma and enhanced le

170 in the subendothelial matrix, by fibrin and fibrinogen in the thrombus, and by a remarkable number o

171 Since the interaction between Abeta and fibrinogen increases CAA and plays an important role in

172 ycoform of the VDKDLQSLEDILHQVENK peptide of fibrinogen increases greater than 10-fold in the HCV and

173 D(pos) patients distinctively show declining fibrinogen, increasing platelet counts, and lower white

174 rinogen and 75 genetic instruments for total fibrinogen indicated a protective effect of higher gamma

175 Thus, fibrinogen-induced spine elimination and cognitive decli

176 Fibrinogen-induced spine elimination was prevented by in

177 Fibrinogen induces phosphorylation of Smad 1/5/8 and inh

178 Here, we show that the blood protein fibrinogen induces spine elimination and promotes cognit

179 Fibrinogen inhibited neuronal differentiation in SVZ and

180 (2017) show that the blood clotting protein fibrinogen inhibits nerve repair by preventing oligodend

181 in AD, we investigated the role of the Abeta-fibrinogen interaction in HCAA pathology.

182 with a higher affinity than (GT)(6) and that fibrinogen interacts with ssDNA-SWCNTs more strongly tha

183 proteins within feedback loops, and cleaves fibrinogen into fibrin, which polymerizes into fibers to

184 yzed conversion of a soluble plasma protein, fibrinogen, into a polymeric fibrin clot, is conserved i

185 is revealed that pronounced binding of human fibrinogen is a common phenotype of human S. equi subsp.

186 Fibrinogen is a complex glycoprotein with regulatory rol

187 Fibrinogen is a key component of the coagulation cascade

188 We propose that fibrinogen is a regulator of NSPC-derived astrogenesis f

189 ledge, this is the first report to show that fibrinogen is a specific trigger for cytolytic eosinophi

190 Fibrinogen is an acute phase protein that will protect e

191 s suggest that SzM-mediated binding of human fibrinogen is an important virulence mechanism of zoonot

192 Gamma prime (gamma') fibrinogen is an isoform of fibrinogen that has anticoag

193 Furthermore, binding of human fibrinogen is associated with specific SzM types.

194 Here, we show that blood-derived fibrinogen is enriched in the SVZ niche following distan

195 he actin cytoskeleton, because surface-bound fibrinogen is highly immobile, and its motility was enha

196 the functional link between eosinophils and fibrinogen is not understood.

197 Fibrinogen is one of the key proteins that participate i

198 ncentrations of C-reactive protein (CRP) and fibrinogen, is associated with increased risk of coronar

199 (2019) show that blood-derived fibrinogen leads to dendritic spine elimination and cogn

200 of bleeding is acquired hypofibrinogenemia (fibrinogen level <1.5-2.0 g/L), for which guidelines rec

201 eeding requiring RBC transfusion (p = 0.75), fibrinogen level (p = 0.67), or daily fibrinogen goal (p

202 ncentrate infusion dosed to achieve a plasma fibrinogen level of 2.5 g/L in high-risk cardiac surgery

203 ), targeted to achieve a postinfusion plasma fibrinogen level of 2.5 g/L.

204 oderate to severe bleeding; and pretreatment fibrinogen level, 1.6 [interquartile range, 1.3-1.9] g/L

205 e, fasting glucose and insulin, RR interval, fibrinogen level, factor VII level and white blood cell

206 were FibTEM-MCF (desired range, 8-13 mm) and fibrinogen levels (desired range, 1.5-2.5 g/L).

207 D(pos) patients tended to have higher plasma fibrinogen levels and lower platelet counts.

208 in a real data example of analysis of plasma fibrinogen levels in the TOPMed program (n = 23,763), us

209 Fibrinogen levels increased from a mean (SD) of 0.91 (0.

210 as also a protective effect of higher gamma' fibrinogen levels on cardioembolic and large artery stro

211 brin(ogen) in mice, low postoperative plasma fibrinogen levels were associated with liver dysfunction

212 mildly low platelet counts, elevated plasma fibrinogen levels, and detection of both severe acute re

213 Fibrinogen levels, fibrin polymerization, platelet activ

214 ng, including the measurement of D-dimer and fibrinogen levels, is suggested.

215 n FC dose is required to achieve therapeutic fibrinogen levels.

216 rinogen production, thereby elevating plasma fibrinogen levels.

217 vated C-reactive protein (CRP), D-dimer, and fibrinogen levels; tachycardia; thrombocytosis; leukocyt

218 The fibrinogen-like (FBG) domain of FREP1 is highly conserve

219 and instead, the immunosuppressive cytokine fibrinogen-like 2 (Fgl2) was a functional ligand for Fcg

220 Here, we demonstrate that fibrinogen-like protein 1 (FGL1), a liver-secreted prote

221 of MC function, and we provide evidence for fibrinogen-like protein 2 (FGL2) as an MC mediator with

222 Here we show that fibrinogen-like protein 2 (FGL2) is highly expressed in

223 s C and S, antithrombin, factors VIII/IX/XI, fibrinogen, lipoprotein(a), homocysteine, lupus anticoag

224 work produced from the aggregates containing fibrinogen looks to be partly built around bright spots

225 n the hypoxic spinal cord and suggest that a fibrinogen-Mac-1 interaction underpins this response.

226 The peak areas of the fibrinogen marker peptides were increased by a factor of

227 Concurrently, the total surface-adsorbed fibrinogen mass is 168% greater than that of albumin.

228 Fibrinogen may be a significant contributor to AD pathog

229 Targeting fibrinogen may be an upstream therapeutic strategy to pr

230 The observed interactions with fibrinogen may be critical to host colonization and it i

231 as been suggested that the polydispersity of fibrinogen may play an important role.

232 the following proteins compared to Group II: fibrinogen (median (interquartile range) Group I: 0.09 (

233 he bloodstream, and yet avidly bind immobile fibrinogen on the surface of other platelets at the prim

234 Activated platelets display immobilized fibrinogen on their surface, thus mediating further recr

235 ort effects of genetically determined gamma' fibrinogen on VTE and ischemic stroke risk.

236 of higher gamma' fibrinogen and higher total fibrinogen on VTE risk.

237 her affinity for SWCNTs than albumin, with a fibrinogen on-rate constant 1.61-fold greater and an off

238 rculating fibrinogen and its isoform, gamma' fibrinogen, on risk of VTE and ischemic stroke subtypes

239 ty, because unlike r-tPA, it did not degrade fibrinogen or enhance experimental bleeding.

240 Targeting fibrinogen or its downstream BMP signaling pathway may h

241 (high-sensitive C-reactive protein, lipids, fibrinogen, oxidative stress, and endothelial function a

242 ching 30 mg/mL by 15 min, the model required fibrinogen penetration into the clot to be strongly diff

243 king MS, we identified the previously mapped fibrinogen peptides that are responsible for covalent D-

244 s to PAD and IgG antibodies to citrullinated fibrinogen peptides, in the absence of a T cell response

245 ailed to develop antibodies to citrullinated fibrinogen peptides.

246 rnational normalized ratio antithrombin III, fibrinogen, plasma-free hemoglobin, platelets, and decli

247 cture of the fibrins produced from those two fibrinogen preparations was determined by using x-ray sc

248 ariations between the composition of the two fibrinogen preparations were found that are much smaller

249 First, the respective compositions of both fibrinogen preparations were thoroughly determined by me

250 Two commercial fibrinogen preparations were used, a monodisperse and a

251 how that fibers made from the aggregate-free fibrinogen present a crystalline longitudinal and latera

252 Separately, fibrinogen split products induce fibrinogen production, thereby elevating plasma fibrinog

253 ntegrin receptor, alpha(M)beta(2,) abolished fibrinogen protection.

254 Mass spectrometry analysis of fibrinogen proteolytic reactions detected the presence o

255 The deficiency of fibrinogen, prothrombin, factor V (FV), FVII, FVIII, FIX

256 The postdose fibrinogen range was 1.2 to 3.3 g/L (72% within the desi

257 tor clusterin upon binding of Abeta40 to the fibrinogen receptor integrin alpha(IIb)beta(3) Here, we

258 Genetic and pharmacologic depletion of fibrinogen reduced astrocyte formation within the SVZ af

259 A genetic variation within mosquito fibrinogen related-protein 30 (FBN30) was previously ide

260 1.5-2.0 g/L), for which guidelines recommend fibrinogen replacement with cryoprecipitate or fibrinoge

261 chaemic stroke, while factor VIII and gamma' fibrinogen require further population-based studies to a

262 o GPVI, which induced the release of ATP and fibrinogen, resulting in platelet aggregation.

263 in G, glutaredoxin-1, thioredoxin, GP1b, and fibrinogen, showed a bias for oxidation, whereas annexin

264 of TG (five markers), and bovine and porcine fibrinogen (six markers each).

265 ed as catalytic microgelator were mixed with fibrinogen solution or blood containing 0.2mM H(2)O(2).

266 Two fibrinogen-specific Affimer proteins, F5 and G2, were id

267 Separately, fibrinogen split products induce fibrinogen production,

268 rted eosinophil activity against FITC-linked fibrinogen substrates.

269 is process, platelets are bridged by soluble fibrinogen that binds surface integrin receptors.

270 a prime (gamma') fibrinogen is an isoform of fibrinogen that has anticoagulant properties.

271 In the presence of LPS together with iC3b or fibrinogen, the expression levels of IL-6 and TNF-alpha

272 ains to how resting platelets ignore soluble fibrinogen, the third most abundant protein in the blood

273 ptomyces mobaraensis, and bovine and porcine fibrinogen/thrombin in restructured meat was developed u

274 g the rotational thromboelastometry variable fibrinogen thromboelastometry maximum clot firmness (Fib

275 suggest a role of CPN1 in the conversion of fibrinogen to fibrin.

276 inogen binding to alphaIIbbeta3 and converts fibrinogen to fibrin.

277 d plasma powder and PPFG using the ratios of fibrinogen to serotransferrin peptide peak areas seems t

278 ke protein SzM is crucial for the binding of fibrinogen to the bacterial surface and for survival in

279 thrombosis and bleeding disorders related to fibrinogen variants.

280 -assessed using C-reactive protein (CRP) and fibrinogen- varied across the adult age span.

281 ane of the specimen; nondiagnostic when only fibrinogen was found at the same location; and negative

282 Low fibrinogen was independently associated with chest tube

283 Fibrinogen was responsible for a second distinct mechani

284 the common pathway, increased gamma (gamma') fibrinogen was significantly associated with AIS/CES.

285 dults, who have low mean LDL-C, CRP, but not fibrinogen, was independently associated with increased

286 Fibrinogen, weight, indication for extracorporeal membra

287 L (24.0) and geometric mean (95% CI) CRP and fibrinogen were 0.90 mg/L (0.87-0.93) and 3.01 g/L (2.98

288 ronic kidney disease, lactate, ferritin, and fibrinogen were associated with myocardial injury.

289 and platelet count, endotoxin, Protein C and fibrinogen were independent predictors of a hypocoagulab

290 , while responses to thrombin or adhesion to fibrinogen were not affected.

291 ks of serum levels of C-reactive protein and fibrinogen were noticed in the first postoperative week.

292 cally relevant thrombocytopenia, and reduced fibrinogen were rare and were associated with significan

293 instruments for gamma' fibrinogen and total fibrinogen were selected, and the inverse-variance weigh

294 (NT-proBNP), cardiac troponin I (cTnI), and fibrinogen- were rapidly (5 min) analyzed from clinical

295 nt Rasa3 (H794L) show increased spreading on fibrinogen, which in contrast to wild-type platelets is

296 omarker profiles (C-reactive protein, plasma fibrinogen, white blood cell count, vitamin D, high-dens

297 No associations of fibrinogen with MCE, IS, or ICH were identified.

298 We investigated associations of CRP and fibrinogen with risks of incident major coronary events

299 Therefore, understanding the interaction of fibrinogen with zeolite nanoparticles in more details co

300 age of human kidneys induces accumulation of fibrinogen within tubular epithelium.