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1 n T cells were stimulated with TGF-beta1 and fibroblast growth factor-7.
4 miR-155 inhibition increases diabetic wound fibroblast growth factor 7 expression in diabetic wounds
6 The expression patterns of mRNAs encoding Fibroblast Growth Factor-7 (FGF-7) and its high affinity
8 ent study, we analyzed the effects of excess fibroblast growth factor-7 (FGF-7) on both the prolifera
11 As, miR-155 is significantly upregulated and fibroblast growth factor 7 (FGF7) mRNA (target of miR-15
12 with self-complementary (sc) AAV2 expressing fibroblast growth factor 7 (Fgf7) supported gland surviv
13 atinocyte growth factor (KGF), also known as fibroblast growth factor 7 (FGF7), is synthesized by ski
14 this notion, UBs cultured in the presence of fibroblast growth factor 7 (FGF7), which induces the for
16 nsforming growth factor-alpha (TGFalpha) and fibroblast growth factor-7 (FGF7) exhibit distinct expre
18 tromal fibroblasts, epidermal growth factor, fibroblast growth factor 7, fibroblast growth factor 2 a
19 atrix extracellular phosphoglycoprotein, and fibroblast growth factor-7 have been demonstrated to pla
21 lets) via the hepatic portal vein (HPV) with fibroblast growth factor 7-loaded galactosylated poly(DL
22 ation of an epithelial cytoprotective agent, fibroblast growth factor-7, maintained a stable pool of
23 Whereas dexamethasone and recombinant human fibroblast growth factor 7 promoted expansion of Foxn1(+