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1 , and absent perfusion prior to the onset of fibroproliferation.
2 e regulation of angiogenesis-mediated airway fibroproliferation.
3 on of the airway epithelium and intraluminal fibroproliferation.
4 a potential mechanism for ventilator-induced fibroproliferation.
5 ing growth factor-beta (TGFbeta), leading to fibroproliferation.
6 , current antifibrotics aim only at limiting fibroproliferation.
7 (MNC) inflammation and mesenchymal cell (MC) fibroproliferation.
8 proprotein processing, and it contributes to fibroproliferation.
9  more effective than lower doses in reducing fibroproliferation (0% in high dose versus 29% in low do
10 near complete obliteration of the lumen with fibroproliferation (96.9% occlusion, =0.001) and absent
11                                 Pathological fibroproliferation after tissue injury is harmful and ma
12 serves as a physiological restraint to limit fibroproliferation after tissue injury.
13 providing a physiological mechanism to limit fibroproliferation after tissue injury.
14 lveolar angiogenesis and fibrogenesis (i.e., fibroproliferation and deposition of extracellular matri
15 and creating a local environment that favors fibroproliferation and matrix deposition.
16 crotic cores within the plaques, and reduced fibroproliferation and neovascularization in the aortic
17  important role in the development of airway fibroproliferation and obliteration.
18 tion, microbiome contraction with epithelial fibroproliferation and profound commensal depletion with
19 nredundant role for CXCR3 in limiting tissue fibroproliferation and suggest that this effect may be m
20 ays, biochemical markers of inflammation and fibroproliferation, and infectious complications.
21 eptide III (PCP III), a biological marker of fibroproliferation, and with increased fatality rates.
22 gens, chronic inflammation, and unrestrained fibroproliferation are likely to be part of a dynamic, u
23  factor-beta 1 (TGFbeta1) expression worsens fibroproliferation following bleomycin-induced lung inju
24                         The role of CXCR3 in fibroproliferation has not been investigated.
25      CsA markedly reduced the development of fibroproliferation in allografts (19% in treated allogra
26                            Abnormal vascular fibroproliferation in CAV occurs as a result of coronary
27 immune-epithelial progenitor niche unique to fibroproliferation in respiratory PASC.
28 ents results in epithelial abnormalities and fibroproliferation in the airway lumen, changes not seen
29  use of CsA in our model system would reduce fibroproliferation in tracheal allografts.
30 yndrome (ARDS) is accompanied by progressive fibroproliferation, inability to improve lung injury sco
31                                    Exuberant fibroproliferation is a common complication after injury
32 data are consistent with the hypothesis that fibroproliferation is an early response to lung injury a
33                         We hypothesized that fibroproliferation is initiated early in ARDS, character
34 ibroblast migration is an initiating step in fibroproliferation; its involvement during acute lung in
35 mmatory/immunologic process characterized by fibroproliferation, matrix deposition, and obliteration
36 eated transgenic mice demonstrated increased fibroproliferation, myofibroblast persistence, and impai
37 yndrome (ARDS) is characterized by excessive fibroproliferation, ongoing inflammation, prolonged mech
38                                       During fibroproliferation, protein-associated extracellular ald
39        Diseases associated with pathological fibroproliferation represent a major cause of morbidity
40                   Histologic lung injury and fibroproliferation scores were positively correlated wit
41                             There was little fibroproliferation seen in either of these groups.
42 ithelial loss correlated with progression to fibroproliferation, suggesting that the epithelium plays
43 ch is to promote resolution of physiological fibroproliferation that follows injury before it becomes
44 ors, activation of cell cycle signaling, and fibroproliferation, the central events in immunopathogen
45 on and airway ischemia in the development of fibroproliferation, we used a murine orthotopic tracheal
46       Lung transplantation is complicated by fibroproliferation, which is likely mediated in part by
47 ptosis but exhibit exaggerated CHI3L1-driven fibroproliferation, which together promote HPS fibrosis.
48 cipients, is characterized histologically by fibroproliferation within small airways.