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1 l tumor, chondroblastoma, osteoblastoma, and fibrous dysplasia.
2  (111)In-pentetreotide was noted in areas of fibrous dysplasia.
3 at this pattern of expression is retained in fibrous dysplasia.
4  individuals with MAS but not in humans with fibrous dysplasia.
5 atient with axial and peripheral polyostotic fibrous dysplasia and cafe au lait spots on clinical exa
6 been found in vitro in both animal models of fibrous dysplasia and in cultured cells from individuals
7  was to assess the prevalence of polyostotic fibrous dysplasia and McCune-Albright syndrome among pat
8          To date, it remains unknown whether fibrous dysplasia and other symptoms of MAS, including n
9                       Twelve had polyostotic fibrous dysplasia, and 26 had the McCune-Albright syndro
10 perfunctioning endocrinopathies, polyostotic fibrous dysplasia, and cafe au lait pigmentation.
11 rome, and a combination of childhood injury, fibrous dysplasia, and pyarthrosis.
12 ions of MAS in a given individual, including fibrous dysplasia, are determined by the timing and loca
13 ere retrospectively screened for polyostotic fibrous dysplasia based on their preoperative abdominal
14                             Rs1 mice display fibrous dysplasia, BM aplasia, progressive loss of HSC n
15             Encasement of the optic canal in fibrous dysplasia causes narrowing of the canal, but tha
16 OC complex: progenitor cell differentiation (fibrous dysplasia), extracellular matrix production (ost
17                                              Fibrous dysplasia (FD) is a prototypical disease of G(s)
18 sative role of Wnt/beta-catenin signaling in fibrous dysplasia (FD) of bone, a disease that exhibits
19 pective review of patients with craniofacial fibrous dysplasia (FD), the clinical and radiological fi
20 MP-responsive osteogenic cells and resembled fibrous dysplasia (FD).
21 anism leading to the specific development of fibrous dysplasia in bone has not been elucidated.
22 m binding correlated with known locations of fibrous dysplasia in the periphery of individuals with M
23 ence for increased cAMP activity in areas of fibrous dysplasia in vivo.
24    Sixty-seven optic canals were affected by fibrous dysplasia; in 49 of them (73 percent) there was
25 eting surface La in a novel explant model of fibrous dysplasia inhibits excessive osteoclast formatio
26             Prkar1a(+/-) mice developed OMX, fibrous dysplasia-like lesions (FDL) and other tumors.
27 ave previously been shown to be the cause of fibrous dysplasia of bone (FD)/McCune-Albright syndrome
28 nt found in certain tumors and patients with fibrous dysplasia of bone and McCune-Albright syndrome g
29    Our data indicate the need to reinterpret fibrous dysplasia of bone as a disease of cells in the o
30                                              Fibrous dysplasia of bone frequently involves the anteri
31 entation patterns and hyperendocrinopathies, fibrous dysplasia of bone is a major finding in the McCu
32                  We studied 38 patients with fibrous dysplasia of the lesser wing of the sphenoid bon
33 be indicated on the basis of the presence of fibrous dysplasia on diagnostic images alone, since it d
34 n McCune-Albright syndrome, characterized by fibrous dysplasia, precocious puberty, and cafe au lait
35 h the de novo deposition of lesional bone in fibrous dysplasia produce a bone matrix enriched in cert
36 lting in their defective differentiation and fibrous dysplasia, we identify Galphas as a key regulato
37 al desmoid tumor, osteofibrous dysplasia, or fibrous dysplasia were examined by cytogenetic analysis