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1 neurostimulation techniques (e.g., cutaneous field stimulation).
2 ual-evoked potentials associated with visual field stimulation.
3 release of [3H]acetylcholine in response to field stimulation.
4 ains by employing weak, alternating magnetic field stimulation.
5 to cholecystokinin octapeptide or electrical field stimulation.
6 ulated with a muscarinic agonist or electric field stimulation.
7 rebound responses similar to the response to field stimulation.
8 ory thalamic neurons to peripheral receptive field stimulation.
9 nd centralized rotors by optical S1-S2 cross-field stimulation.
10 sponses to non-noxious and noxious receptive field stimulation.
11 e potential caused by extracellular electric field stimulation.
12 ted by ipsilateral rostral scratch receptive field stimulation.
13 se of [3H]NE was elicited through electrical field stimulation.
14 ncholinergic nerve stimulation by electrical field stimulation.
15 and prVEP amplitude evoked by full- and half-field stimulation.
16 es evoked by periorbital cutaneous receptive field stimulation.
17 NEP) using alternating current (ac)-magnetic field stimulation.
18 rical brain responses associated with visual field stimulation.
19 enhanced contractile response to electrical field stimulation.
20 ergic neurostimulation initiated by electric field stimulation.
21 ntractile responses to CCh and to electrical field stimulation.
22 rmal spontaneous Ca(2+) release events after field stimulation.
23 es of anesthetized mice in response to local field stimulation.
24 robiotics) and percutaneous electrical nerve field stimulation.
25 PY overflow increased 0 s and 30 s following field stimulation.
26 arterioles was assessed at 0 s and 30 s post-field stimulation.
27 ng bilateral compared with unilateral visual field stimulation.
28 stimulation and phase two started 35 s after field stimulation.
29 e of FM4-64 only during the initial phase of field stimulation.
30 cted to increasingly faster pacing rates via field stimulation.
31 , and their frequency could be elevated with field stimulation.
35 d 1000 ms) and SNA was initiated using local field stimulation (30-50 V; 1 ms at 2, 8 and 16 Hz).
36 on with elevated KCl or patterned electrical field stimulation (50 biphasic rectangular pulses of 25
37 ntitate the overflow of ADPR upon electrical field stimulation (8 Hz, 0.3 ms, 15 V, 1-2 min) of both
39 ty were evaluated using video microscopy and field stimulation after exposure to osmotic stress at 37
40 ning both a nearly normal twitch response to field stimulation and a robust positive inotropic respon
42 interactive effects of pulsatile electrical field stimulation and substrate topography on cell diffe
43 ion channels using extracellular electrical field stimulation and voltage-sensitive fluorescent prob
44 lar [Ca2+] was monitored using fura-2 during field stimulation and while membrane potential was contr
45 e cell membrane under conditions of electric field stimulation, and a counterbalancing interplay betw
46 ssed via live cell calcium imaging, electric field stimulation, and RNA sequencing of human dorsal ro
47 Delta) preparations, responses to electrical field stimulation are nearly abolished despite persisten
48 high concentration of potassium or electric field stimulation are needed to trigger the exocytosis o
49 contraction of bladder strips to electrical field stimulation, as well as activation with carbachol,
53 om wild-type (WT) mice, [Na](i) increased on field stimulation at 2 Hz from 11.1+/-1.8 mmol/L to a pl
55 left ventricular cardiomyocytes during 1 Hz field stimulation at 37 degrees C, at resting length and
56 We investigated the effects of electrical field stimulation (average frequency 2 Hz) in an irregul
58 In the normal homozygous mice, electrical field stimulation caused a biphasic response, an initial
59 auditory cortex (AC stimulation) under free field stimulation conditions using a two-tone inhibition
63 P, overflow of beta-NAD evoked by electrical field stimulation correlated with stimulation frequency
65 cholinergic responses elicited by electrical field stimulation (EFS) (8 Hz, 1 ms, 8 V for 10 s every
66 ergic (NANC) nerve stimulation by electrical field stimulation (EFS) (either short-train or continuou
67 ular transmission was achieved by electrical field stimulation (EFS) (frequency, 10 Hz; pulse voltage
68 es min(-1) using 0.1 ms pulses of electrical field stimulation (EFS) (three pulses delivered at 3-30
70 of the guinea pig vas deferens to electrical field stimulation (EFS) and norepinephrine (NE) exocytos
71 t treatment vs. neurostimulation by electric field stimulation (EFS) in bovine tracheal smooth muscle
72 xation responses were assessed by electrical field stimulation (EFS) in presence of A(2B)AR agonists,
73 S was induced by CO, NO, VIP, and electrical field stimulation (EFS) in the presence and absence of n
79 rgic stimulation by NO donors and electrical field stimulation (EFS) of intrinsic enteric inhibitory
80 ne, guanethidine and propranolol, electrical field stimulation (EFS) of the atrial preparations evoke
83 s in an organ bath in response to electrical field stimulation (EFS) under nonadrenergic, noncholiner
85 one, vasoconstrictor responses to electrical field stimulation (EFS) were not attenuated by vitamin E
86 ses, frequency-response curves to electrical field stimulation (EFS) were performed with results expr
87 resides in the combination of the electrical field stimulation (EFS) with data acquisition in spatial
88 measured in the basal state, after electric field stimulation (EFS), ganglionic stimulant dimethyl d
90 lium acts as a crucial sensor for electrical field stimulation (EFS)-enhanced osteogenic response in
93 gs of (i) caffeine-induced-, (ii) electrical field stimulation (EFS)-induced force, (iii) pCa-force,
98 n to ATP and norepinephrine (NE), electrical field stimulation (EFS, 4-16 Hz, 0.1-0.3 ms, 15 V, 60-12
99 ons were stimulated for 60 s with electrical field stimulation (EFS; vas deferens), dimethylphenylpip
100 Schematic shows brain slice, patch pipette, field stimulation electrodes and microscope objective.
104 e epilepsy were exposed to weak, DC magnetic field stimulation following computer-controlled protocol
105 eased the contractile response to electrical field stimulation for 48 hours after administration.
108 after spontaneous contraction or short-term field stimulation fusion activity increased in cardiomyo
111 tage (V(m)) of cardiac cells during electric field stimulation have a complex spatial- and time-depen
112 tylcholine release in response to electrical field stimulation in both the stomach and small intestin
113 siologically relevant patterns of electrical field stimulation in culture, including tonic stimulatio
114 spatially resolved Ca(2+) transients during field stimulation in left ventricular (LV) myocytes from
116 lateral and unilateral left and right visual field stimulation in normal and compromised systems.
117 d airway narrowing in response to electrical field stimulation in precision cut lung slices (PCLS) we
120 oth muscle hyperresponsiveness to electrical field stimulation, indicating that IgE/CD23-mediated imm
124 de of the somatic Ca2+ increase triggered by field stimulation is independent of the extracellular Ca
125 urred spontaneously and could be evoked with field stimulation: large, rapid, global Ca2+ transients
129 results are consistent with predictions from field stimulation of continuous representations of myoca
130 much has been learned about BDNF function by field stimulation of hippocampal neurons, it is not know
131 ectrical activity or responses to electrical field stimulation of intrinsic nerves in these mice.
133 ansmission have used indiscriminate electric field stimulation of neural elements within the gastric
138 the ciliary nerves in the eye, or electrical field stimulation of the cornea, evoked Ca2+ transients
140 testinal peptide (VIP) and direct electrical field stimulation of the intrinsic innervation; inhibiti
141 ing, and function was assessed by electrical field stimulation of tracheas in the presence/absence of
144 inantly observed in the contralateral visual field, stimulation of hMST also affected the ipsilateral
148 ng fluorescence technologies with electrical field stimulation or the patch-clamp technique in beatin
149 ty to neuropeptide Y (P<0.05) and electrical field stimulation (P<0.05) in mesenteric arteries was al
151 USA) delivers percutaneous electrical nerve field stimulation (PENFS) in the external ear to modulat
153 ion of skeletal muscle fibres via electrical field stimulation produced a vasodilatation (19.4 +/- 1.
154 so demonstrate that near-threshold diastolic field stimulation produces activation of deep myocardial
156 cium transient morphology at higher external field stimulation rates and augmented both calcium relea
158 ic AB levels, using repeated electromagnetic field stimulation (REMFS) in primary human brain (PHB) c
159 imetic actuation when controlled by electric field stimulation results in movement similar to that of
160 teries subjected to low-frequency electrical field stimulation revealed Ca2+ transients in perivascul
161 OFF-centre RGC responses were evoked by full-field stimulation, significantly fewer converted that re
163 ATP, and a reduced contraction to electrical field stimulation, suggesting altered P2X1 receptor func
164 (IJPs) and relaxations evoked in response to field stimulation (supramaximal voltage, 0.1 ms, single
165 mbining fluorescence imaging with electrical field stimulation, the patch-clamp method and knock-out
166 duced single action potentials by electrical field stimulation, then subjected neurons to high-pressu
167 e demonstrated the use of in situ electrical field stimulation to deactivate the adhesive property of
168 Primary cilia are sensors of electrical field stimulation to induce osteogenesis of human adipos
170 eatments include peripheral electrical nerve field stimulation to the external ear, gastric electrica
171 owed similar patterns of surface labeling by field stimulation to those shown previously by high pota
172 emonstrate that transcranial static magnetic field stimulation (tSMS) over the somatosensory parietal
174 ere, we combine transcranial static magnetic field stimulation (tSMS) with resting-state functional M
175 Maximum sarcomere shortening elicited by field stimulation was graded in the order: LVendo > LVep
176 oline (ACh) or noradrenaline (NA) release to field stimulation was measured 4 months after gene trans
177 ction potentials (S-IJPs) evoked by electric field stimulation was significantly higher in CSE-KO mou
178 cy-dependent relaxation evoked by electrical field stimulation was significantly lower in gallbladder
182 cell activity responses to somatic receptive fields stimulation, was compared between anesthetized ca
183 onophosphate elevations evoked by electrical field stimulation were diminished markedly in both nNOSD
184 C-acetylcholine (ACh) released by electrical field stimulation were measured simultaneously in strips
185 C]acetylcholine (ACh) released by electrical field stimulation were measured simultaneously in strips
186 ient amplitude increased dramatically during field stimulation when pH(i) was lowered to 6.5, consist
188 art cell contractility inducible by electric field stimulation with directionally dependent electrica
190 tment spiking neurons responding to electric field stimulation with small incremental polarization.