ライフサイエンスコーパス: fine particulate matter

1 dfire smoke, including those associated with fine particulate matter.

2 confounders, including co-pollutants such as fine particulate matter.

3 e 25th and 75th percentiles as cutpoints for fine particulate matter.

4 measurements of the chemical composition of fine particulate matter.

5 erprints for source apportionment of ambient fine particulate matter.

6 lfate measured by ion chromatography (IC) in fine particulate matter.

7 alue of statistical life and the toxicity of fine particulate matter.

8 y organic aerosol (SOA) that often dominates fine particulate matter.

9 o estimate the annual mean concentrations of fine particulate matter.

10 An average increase of 6 microg/m(3) in fine particulate matter 2 days before onset was associat

11 d Analysis Methods for Airborne Carbonaceous Fine Particulate Matter" (2003) and "Source Apportionmen

12 artile-range increase in average exposure to fine particulate matter (3.2 microg/m(3)) during pregnan

13 en maternal exposure to nitrogen dioxide and fine particulate matter (aerodynamic diameter </=2.5 mic

14 y (COWO), the role of early life exposure to fine particulate matter (aerodynamic diameter <2.5 mum;

15 Prenatal exposure to ambient PM2.5, (i.e., fine particulate matter, aerodynamic diameter </= 2.5 mu

16 Because radon, smoking, and fine particulate matter air pollutants (PM2.5) may share

17 Our goal was to evaluate fine particulate matter air pollution ([Formula: see tex

18 , unequivocal evidence of the causal role of fine particulate matter air pollution (PM(2.5), or parti

19 Exposure to ambient fine particulate matter air pollution (PM(2.5); < 2.5 mi

20 ic studies have linked long-term exposure to fine particulate matter air pollution (PM) to broad caus

21 Exposure to fine particulate matter air pollution (PM2.5) may increa

22 at further public policy efforts that reduce fine particulate matter air pollution are likely to have

23 th a causal relationship between exposure to fine particulate matter and cardiovascular morbidity and

24 associations between source contributions of fine particulate matter and emergency department visits

25 Associations between source-specific fine particulate matter and emergency department visits

26 (SOA) constitutes a substantial fraction of fine particulate matter and has important impacts on cli

27 ous compounds are a significant component of fine particulate matter and haze in national parks and w

28 e findings confirm the link between OHCA and fine particulate matter and introduce evidence of a simi

29 to estimates of annual average exposures to fine particulate matter and nitrogen dioxide at the resi

30 We examined multipollutant models (with fine particulate matter and nitrogen dioxide) and effect

31 tive models and adjustment for copollutants (fine particulate matter and nitrogen dioxide), although

32 For fine particulate matter and NO2, we used land use regres

33 ge yields annual premature deaths related to fine particulate matter and ozone (95CI: 25 000-120 000)

34 Long-term exposure to ambient fine particulate matter and renal function in older men:

35 us work has examined the association between fine particulate matter and risk of kidney disease; howe

36 tent association between increased levels of fine particulate matter and risk of primary cardiac arre

37 o describe the variation of size spectrum of fine particulate matter, and the respective contribution

38 l cardiac arrest (OHCA) are inconsistent for fine particulate matter, and, although pathophysiologica

39 (diet)/biomedical (drug) and environmental (fine particulate matter) applications of sub-mg of C sam

40 easurements were made for both ultrafine and fine particulate matter as well as black carbon inside a

41 to HEPA-filtered air or concentrated ambient fine particulate matter (CAP, 30-100 microg/m(3)) from d

42 racers of primary and secondary carbonaceous fine particulate matter collected at four midwestern Uni

43 sual Environments) network has characterized fine particulate matter composition at locations through

44 The 2003 annual average fine particulate matter concentration was not associated

45 e improve the accuracy of daily ground-level fine particulate matter concentrations (PM(2.5)) derived

46 (GWR) statistical model to represent bias of fine particulate matter concentrations (PM2.5) derived f

47 and major roads were calculated, and annual fine particulate matter concentrations, derived from a s

48 conducive to the formation of peak ozone and fine particulate matter concentrations.

49 Consequently, future fine particulate matter control efficiency will not be u

50 he association between long-term exposure to fine particulate matter, defined as particulate matter w

51 5% confidence interval (CI): 1.04, 1.39) and fine particulate matter (diameter </=2.5 mum) on lag day

52 Air pollution health studies of fine particulate matter (diameter </=2.5 mum, PM2.5) oft

53 elationships between CO and elemental carbon fine particulate matter (EC) road proximity.

54 Life cycle fine particulate matter emissions are higher for ethanol

55 Airborne fine particulate matter exhibits spatiotemporal variabil

56 Fine particulate matter exposure from both ambient air p

57 iovascular morbidity and mortality, and that fine particulate matter exposure is a modifiable cardiov

58 Conclusions: Long-term fine particulate matter exposure was associated with an

59 Fine particulate matter exposure was associated with CAC

60 nal age, race/ethnicity, education, prenatal fine particulate matter exposure, prenatal smoking expos

61 We used weekly fine-particulate-matter exposure data for 238 births in

62 and duration of exposure to fire-originated fine particulate matter (fire-PM2.5) and CHVI.

63 nergy, climate change (CC), ozone depletion, fine particulate matter formation, marine eutrophication

64 We sought to assess the role exposure to fine particulate matter ([Formula: see text]) during dif

65 The effects of exposure to fine particulate matter ([Formula: see text]) during wil

66 signed 5-y running average concentrations of fine particulate matter ([Formula: see text]), nitrogen

67 la: see text]), ozone ([Formula: see text]), fine particulate matter [Formula: see text] ([Formula: s

68 tted against estimated average daily dose of fine particulate matter from cigarette smoke along with

69 ght have received airborne virus, carried by fine particulate matter, from infected farms within the

70 udies utilizing source apportionment (SA) of fine particulate matter have shown that particles from c

71 al constraints on this critical component of fine particulate matter; however, no previous collection

72 ugh prior research has linked wildfire smoke fine particulate matter (i.e., aerodynamic diameter 2.5

73 re is consistent evidence that the levels of fine particulate matter in the air are associated with t

74 n period (OR = 1.35, 95% CI: 1.09, 1.66) and fine particulate matter in the spring (OR = 1.28, 95% CI

75 We propose that the nicotine and fine particulate matter in tobacco smoke lead to increas

76 study aimed to evaluate whether exposure to fine particulate matter interferes with lung recovery an

77 between cardiovascular disease mortality and fine particulate matter is relatively steep at low level

78 is strong evidence of an association between fine particulate matter less than 2.5 mum (PM2.5) in aer

79 Long-term exposure to fine particulate matter less than 2.5 mum in diameter (P

80 in the EU in 2019 were exposed to an annual fine particulate matter level higher than the World Heal

81 012-2016 randomized clinical trial to reduce fine particulate matter levels.

82 en long-term ambient residential exposure to fine particulate matter < 2.5 microns (PM2.5) and physic

83 re is a positive association between ambient fine particulate matter </= 2.5 mum in aerodynamic diame

84 the association between prenatal exposure to fine particulate matter </= 2.5 mum in aerodynamic diame

85 Growing evidence indicates that toxicity of fine particulate matter </= 2.5 mum in diameter (PM2.5)

86 Elevated levels of fine particulate matter <2.5 microm in aerodynamic diame

87 olar epithelial cells in culture and mice to fine particulate matter <2.5 microm in diameter (PM(2.5)

88 Previous studies have examined fine particulate matter (</= 2.5 mum; PM2.5) and preterm

89 Both short- and long-term exposures to fine particulate matter (</= 2.5 mum; PM2.5) are associa

90 Outdoor fine particulate matter (</= 2.5 mum; PM2.5) has been id

91 Given that fine particulate matter (</= 2.5 mum; PM2.5) is a mixtur

92 o satellite-based nitrogen dioxide (NO2) and fine particulate matter (</= 2.5 mum; PM2.5) predictions

93 90 days before birth for temporally resolved fine particulate matter (</= 2.5 mum; PM2.5), black carb

94 primary cardiac arrest and daily measures of fine particulate matter (</=2.5 micro m) using a case-cr

95 Laboratory studies suggest that exposure to fine particulate matter (</=2.5 mum in diameter) (PM2.5)

96 We hypothesized that exposure to ambient fine particulate matter (<2.5 mum; PM(2.5)) exaggerates

97 ad more symptoms associated with exposure to fine particulate matter measuring less than 2.5 mum in d

98 ve risk for a 13.8- micro g/m(3) increase in fine particulate matter (nephelometry: 0.54 x 10(-1) km(

99 Ambient fine particulate matter, nitrogen dioxide, and preterm b

100 d a unified modeling approach for predicting fine particulate matter, nitrogen dioxide, oxides of nit

101 Exposure to fine particulate matter, O3, and NO2 was positively asso

102 nt cohort, we found positive associations of fine particulate matter, O3, and NO2 with mortality.

103 Health risks of fine particulate matter of 2.5 microm or less in aerodyn

104 burden of disease, with ambient exposure to fine particulate matter of diameters smaller than 2.5 mu

105 he-art air pollution exposure assessments of fine particulate matter, oxides of nitrogen, and black c

106 ontextual covariates, including exposures to fine particulate matter, ozone, and nitrogen dioxide.

107 nce for detrimental effects on health is for fine particulate matter (particles <=2.5 um in diameter

108 Data on concentrations of fine particulate matter (particles that are < or = 2.5 m

109 Annual averages of fine particulate matter (particles with a mass median ae

110 Past health impact assessments of ambient fine particulate matter (particles with an aerodynamic d

111 nalyses used daily cardiovascular mortality, fine particulate matter (particles with an aerodynamic d

112 er annual levels of air pollution containing fine particulate matter (particles with an aerodynamic d

113 eses that subchronic and chronic exposure to fine particulate matter (particulate matter < or = 2.5 m

114 Exposure to ambient fine particulate matter (particulate matter <=2.5 mum in

115 diac arrhythmias and short-term exposures to fine particulate matter (particulate matter <=2.5 um aer

116 sible joint effects of cigarette smoking and fine particulate matter (particulate matter less than or

117 vestigated the short-term effects of UFP and fine particulate matter (particulate matter with an aero

118 Estimates of fine particulate matter (particulate matter with an aero

119 th effects associated with acute exposure to fine particulate matter (particulate matter with an aero

120 ationship between relatively low exposure to fine particulate matter (particulate matter with an aero

121 The impact of chronic exposure to fine particulate matter (particulate matter with an aero

122 Annual average fine particulate matter (particulate matter with diamete

123 on between short-term (or acute) exposure to fine particulate matter [particulate matter with aerodyn

124 ile exhaust contains precursors to ozone and fine particulate matter (PM </= 2.5 microm in aerodynami

125 ozone and black carbon (BC), a component of fine particulate matter (PM </= 2.5 microm in aerodynami

126 Exposure to fine particulate matter (PM with diameter </= 2.5 mum; P

127 Fine particulate matter (PM(2.5) ), a common form of air

128 organic compounds (VOCs), and reductions in fine particulate matter (PM(2.5)) affect the O(3) trends

129 Fine particulate matter (PM(2.5)) air pollution exposure

130 Fine particulate matter (PM(2.5)) air pollution has been

131 tricity generation is a large contributor to fine particulate matter (PM(2.5)) air pollution.

132 he existing scientific literature focuses on fine particulate matter (PM(2.5)) and carbon monoxide (C

133 iabetes mellitus associated with exposure to fine particulate matter (PM(2.5)) and nitrogen oxides in

134 urements of airborne metal concentrations in fine particulate matter (PM(2.5)) are important for unde

135 Annual global satellite-based estimates of fine particulate matter (PM(2.5)) are widely relied upon

136 Exposures to fine particulate matter (PM(2.5)) concentrations above t

137 Accurate data concerning historical fine particulate matter (PM(2.5)) concentrations are nee

138 This study estimated annual average ambient fine particulate matter (PM(2.5)) concentrations at 1 km

139 Average ambient fine particulate matter (PM(2.5)) concentrations have de

140 ion measures achieved an average decrease of fine particulate matter (PM(2.5)) concentrations of ~14%

141 th American decreases in population-weighted fine particulate matter (PM(2.5)) concentrations since 2

142 Exposure to fine particulate matter (PM(2.5)) during pregnancy has b

143 d health effects of greenhouse gas (GHG) and fine particulate matter (PM(2.5)) emissions from gasolin

144 ronmental health research has suggested that fine particulate matter (PM(2.5)) exposure can lead to h

145 el is used to quantify primary and secondary fine particulate matter (PM(2.5)) exposure from on-road

146 Here we project ambient fine particulate matter (PM(2.5)) exposure in India for

147 We evaluate fine particulate matter (PM(2.5)) exposure-response mode

148 arch into the global public health burden of fine particulate matter (PM(2.5)) exposures frequently r

149 Emissions of most pollutants that result in fine particulate matter (PM(2.5)) formation have been de

150 Exposure to fine particulate matter (PM(2.5)) from fuel combustion s

151 Emissions of fine particulate matter (PM(2.5)) from human activities

152 Exposure to fine particulate matter (PM(2.5)) from solid-fuel combus

153 e emerging evidence on the health impacts of fine particulate matter (PM(2.5)) from wildland fire smo

154 hough it is well documented that exposure to fine particulate matter (PM(2.5)) increases the risk of

155 bservations suggest that exposure to ambient fine particulate matter (PM(2.5)) is associated with inc

156 An important component of the fine particulate matter (PM(2.5)) is black carbon (BC) a

157 Ambient fine particulate matter (PM(2.5)) is the world's leading

158 ausing chronic health effects independent of fine particulate matter (PM(2.5)) mass is inconclusive.

159 We estimated fine particulate matter (PM(2.5)) mortality in the 250 m

160 key urban form determinants of decadal-long fine particulate matter (PM(2.5)) trends in all 626 Chin

161 Data on ambient fine particulate matter (PM(2.5)) was obtained from the

162 ution surface of the chemical composition of fine particulate matter (PM(2.5)) would offer valuable i

163 to harmful air pollutants such as ozone and fine particulate matter (PM(2.5))(4).

164 arbon, particle-bound aromatic hydrocarbons, fine particulate matter (PM(2.5)), and ultrafine particl

165 reater exposure to ubiquitous toxicants like fine particulate matter (PM(2.5)), manganese, and many p

166 red in-use emissions of pollutants including fine particulate matter (PM(2.5)), organic and elemental

167 xide (CO(2)), carbon monoxide (CO), methane, fine particulate matter (PM(2.5)), PM(2.5) elemental car

168 Air pollution, particularly fine particulate matter (PM(2.5)), poses serious health

169 Therefore, we measured outdoor fine particulate matter (PM(2.5)), species of polycyclic

170 res are increasingly a significant source of fine particulate matter (PM(2.5)), which has been linked

171 Wildland fire is a major emission source of fine particulate matter (PM(2.5)), which has serious adv

172 health-damaging pollutants such as ozone and fine particulate matter (PM(2.5)).

173 vior influence residential concentrations of fine particulate matter (PM(2.5)).

174 and often dominant component of atmospheric fine particulate matter (PM(2.5)).

175 to an increase in the atmospheric burden of fine particulate matter (PM(2.5)).

176 The impact of inhalable fine particulate matter (PM(2.5), aerodynamic diameter <

177 Fine particulate matter (PM(2.5), aerodynamic diameter <

178 xide (NO(2): 60% with 95% CI 48 to 72%), and fine particulate matter (PM(2.5): 31%; 95% CI: 17 to 45%

179 egional decreases in ozone (0.2-0.7 ppb) and fine particulate matter (PM) (0.1-0.7 mug/m(3)).

180 the search for AD risk factors; exposure to fine particulate matter (PM) air pollution increases car

181 Fine particulate matter (PM) air pollution is associated

182 temporal land use regression (LUR) model for fine particulate matter (PM) and wood-burning tracers le

183 idemiology studies rely on concentrations of fine particulate matter (PM) as a smoke tracer.

184 of magnitude higher than ambient coarse and fine particulate matter (PM) collected from downtown Tor

185 are thought to be related to exposure to the fine particulate matter (PM) component of HAP, but it is

186 Exposure to fine particulate matter (PM) during pregnancy is associa

187 Early life exposure to fine particulate matter (PM) in air is associated with i

188 anic compounds are important constituents of fine particulate matter (PM) in the troposphere.

189 Airborne fine particulate matter (PM) is responsible for the most

190 Exposures to ambient and household fine-particulate matter (PM(2.5)) together are among the

191 dependent and joint effects of heatwaves and fine particulate matter [PM < 2.5 mum in aerodynamic dia

192 We are measuring fine particulate matter [PM < 2.5 mum in aerodynamic dia

193 age), we measured 48-h personal exposures to fine particulate matter [PM [Formula: see text] in aerod

194 mine the putative adverse effects of ambient fine particulate matter (PM2.5 : PM with aerodynamic dia

195 search has identified an association between fine particulate matter (PM2.5) air pollution and lung c

196 Fine particulate matter (PM2.5) air pollution exposure h

197 Elevated ambient fine particulate matter (PM2.5) air pollution exposure h

198 Emerging evidence indicates that exposure to fine particulate matter (PM2.5) air pollution may increa

199 s with changes in nitrogen dioxide (NO2) and fine particulate matter (PM2.5) air pollution.

200 Measurements of fine particulate matter (PM2.5) and absorbance were cond

201 ions relative to baseline of 45% and 47% for fine particulate matter (PM2.5) and carbon monoxide (CO)

202 and 2013, we measured personal exposures to fine particulate matter (PM2.5) and carbon monoxide (CO)

203 ted at the patient residence using predicted fine particulate matter (PM2.5) and ground-level ozone (

204 es strongly retain atmospheric deposition of fine particulate matter (PM2.5) and metals, including Hg

205 Data on exposures to fine particulate matter (PM2.5) and ozone (O3) were obta

206 alth by increasing ambient concentrations of fine particulate matter (PM2.5) and ozone (O3).

207 tion between short-term exposures to ambient fine particulate matter (PM2.5) and ozone, and at levels

208 Vehicle emissions contribute to fine particulate matter (PM2.5) and tropospheric ozone a

209 ir pollution events and moderate exposure to fine particulate matter (PM2.5) are associated with incr

210 te phase-out scenario reduces annual average fine particulate matter (PM2.5) by an estimated 0.71 mug

211 hrough December 2011 and annual estimates of fine particulate matter (PM2.5) by census tract.

212 Exposure to ozone and fine particulate matter (PM2.5) can cause adverse health

213 48-hour personal exposure to log-transformed fine particulate matter (PM2.5) concentrations among coo

214 Short-term exposure to ambient fine particulate matter (PM2.5) concentrations has been

215 We estimated global fine particulate matter (PM2.5) concentrations using inf

216 model routinely underpredicts peak ozone and fine particulate matter (PM2.5) concentrations.

217 ity and long-term exposure to a low level of fine particulate matter (PM2.5) concentrations.

218 In highly polluted environments, fine particulate matter (PM2.5) containing redox-active

219 inorganic sulfate concentrations in ambient fine particulate matter (PM2.5) data from 2005 to 2012 a

220 ween ambient species in high time resolution fine particulate matter (PM2.5) data to form clusters th

221 gy, could increase ambient concentrations of fine particulate matter (PM2.5) due to its ammonia emiss

222 lected and analyzed within-city variation of fine particulate matter (PM2.5) elements.

223 These fuel additives also reduce fine particulate matter (PM2.5) emissions and alter the

224 nt air pollution, accounting for over 25% of fine particulate matter (PM2.5) emissions in Canada.

225 Long-term fine particulate matter (PM2.5) exposure is linked with

226 nd temporally resolved black carbon (BC) and fine particulate matter (PM2.5) exposures, residential p

227 Exposure to fine particulate matter (PM2.5) from indoor and outdoor

228 ntion advisories were designed to occur when fine particulate matter (PM2.5) from smoke, forecasted 2

229 Ambient fine particulate matter (PM2.5) has a large and well-doc

230 In adults, exposure to fine particulate matter (PM2.5) has been associated with

231 Fine particulate matter (PM2.5) has been linked to cardi

232 Reductions in ambient concentrations of fine particulate matter (PM2.5) have contributed to redu

233 ollution, up to 11 mug m(-3) for annual mean fine particulate matter (PM2.5) in northern Vietnam and

234 idence suggests that exposure to ambient air fine particulate matter (PM2.5) increases the risk of de

235 bution of DICE-Africa to annual mean surface fine particulate matter (PM2.5) is >5 mug m(-3) in popul

236 Ambient fine particulate matter (PM2.5) is a leading environment

237 Ambient fine particulate matter (PM2.5) is among the most preval

238 Exposure to atmospheric fine particulate matter (PM2.5) is associated with cardi

239 Exposure to fine particulate matter (PM2.5) is associated with cardi

240 It is unknown if ambient fine particulate matter (PM2.5) is associated with lower

241 tigating whether chronic exposure to ambient fine particulate matter (PM2.5) is associated with morta

242 Fine particulate matter (PM2.5) is thought to be respons

243 ons, can significantly impact ozone (O3) and fine particulate matter (PM2.5) pollution across the U.S

244 ls to estimate premature mortality caused by fine particulate matter (PM2.5) pollution as a result of

245 dverse health effects of chronic exposure to fine particulate matter (PM2.5) requires accurate estima

246 investigate the associations of exposure to fine particulate matter (PM2.5) with HRV as an indicator

247 summer (April-September) surface ozone (O3), fine particulate matter (PM2.5), and maximum temperature

248 Exposure to hydrocarbons, fine particulate matter (PM2.5), and other chemicals fro

249 develop land use regression (LUR) models for fine particulate matter (PM2.5), black carbon (BC), and

250 s than 1 ppb for ozone and 0.5 mug m(-3) for fine particulate matter (PM2.5), but widespread.

251 Air pollution, including fine particulate matter (PM2.5), is increasingly recogni

252 nitrogen oxides (NOx), carbon monoxide (CO), fine particulate matter (PM2.5), organic aerosol (OA), a

253 nits in z-BMI for nitrogen dioxide (NO2) and fine particulate matter (PM2.5), respectively, in PIAMA.

254 and assessed short-term exposure to ambient fine particulate matter (PM2.5), sulfates, black carbon

255 o increased inhalation exposures to PCBs and fine particulate matter (PM2.5), using disability-adjust

256 s a substantial mass fraction to atmospheric fine particulate matter (PM2.5).

257 ic health impact modeling for ozone (O3) and fine particulate matter (PM2.5).

258 h prenatal and childhood exposure to ambient fine particulate matter (PM2.5).

259 r use in a chemical mass balance (CMB) SA of fine particulate matter (PM2.5).

260 O, black carbon (BC), NO, NO2, NOx, SO2, and fine particulate matter (PM2.5)] decreased with increasi

261 l studies have provided strong evidence that fine particulate matter (PM2.5; aerodynamic diameter </=

262 g body of research suggests that exposure to fine particulate matter (PM2.5; particle size 2.5 micron

263 , the temporal pattern of the association of fine particulate matter (PM2.5; particulate matter </= 2

264 d normal chow or a high-fat diet to airborne fine particulate matters (PM2.5), and then investigated

265 ase (GBD) assessments estimated that outdoor fine-particulate matter (PM2.5) is a causal factor in ov

266 Long-term exposure to fine-particulate-matter (PM2.5) air pollution may accele

267 short-term exposure to main air pollutants (fine particulate matter [PM2.5] and ozone) and cause-spe

268 ns (particle number [PN], black carbon [BC], fine particulate matter [PM2.5], particle size).

269 from dirty-fuel-derived, outdoor, airborne, fine particulate matter pollution (PM(2.5); indicator 3.

270 Exposure to fine particulate matter pollution (PM2.5) is hazardous t

271 Ambient fine particulate matter pollution (PM2.5) is one leading

272 the same time, evidence has accumulated that fine particulate matter pollution can exacerbate asthma

273 The Salt Lake Valley experiences severe fine particulate matter pollution episodes in winter dur

274 Black carbon (BC) is fine particulate matter produced by the incomplete combu

275 Average concentrations of fine particulate matter smaller than 2.5 micrometers in

276 particles (DEP), a major component of urban fine particulate matter, suppresses antimycobacterial hu

277 and postnatal 24-hour personal exposures to fine particulate matter than the control (mean prenatal

278 100 m2) estimates of nitrogen dioxide (NO2), fine particulate matter under 2.5 mum (PM2.5), and noise

279 ion, the median 24-hour personal exposure to fine particulate matter was 23.9 mug per cubic meter in

280 crog per cubic meter in the concentration of fine particulate matter was associated with an estimated

281 Exposure to fine particulate matter was positively associated with h

282 For fine particulate matter, we observed elevated and signif

283 NO2 (a marker for traffic pollution) and fine particulate matter were also associated with mortal

284 (x) chemistry and long-distance transport of fine particulate matter with a diameter less than 2.5 um

285 annual average residential ozone (O(3)) and fine particulate matter with a diameter of 2.5 mum or le

286 ng the long-term health impacts of ozone and fine particulate matter with a diameter smaller than 2.5

287 CI, 1.101-1.147) on days of lower values of fine particulate matter with a median aerodynamic diamet

288 cal studies have linked residual oil fly ash fine particulate matter with aerodynamic diameter <2.5 m

289 personal and kitchen exposure assessments of fine particulate matter with aerodynamic diameter <= 2.5

290 ated costs; the GAINS model, which estimates fine particulate matter with aerodynamic diameter 2.5 mi

291 ntral to the pathogenesis due to exposure to fine particulate matter with aerodynamic diameter [Formu

292 y contrast, neither benzo(a)pyrene (BaP) nor fine particulate matter with aerodynamic diameter [Formu

293 s investigated whether short-term effects of fine particulate matter with an aerodynamic diameter < o

294 rom a major road, nitrogen dioxide [NO(2) ], fine particulate matter with an aerodynamic diameter 2.5

295 and nitrogen oxides) and particulate matter (fine particulate matter with an aerodynamic diameter of

296 ociated with concentrations of black carbon, fine particulate matter with an aerodynamic with diamete

297 ir quality index and PM(2.5) concentrations (fine particulate matter with diameters equal or smaller

298 3) is a key precursor species to atmospheric fine particulate matter with strong implications for reg

299 ong-term residential exposure to traffic and fine particulate matter with the degree of coronary athe

300 nt by other traffic-related pollutants: NO2, fine particulate matter (with aerodynamic diameter < or