ライフサイエンスコーパス: first instar

1 n all populations, and in salivary glands of first instars.

2 ss susceptible to Cry1Ab toxin compared with first instars.

3 d familiarity spiderlings received along the first instar and across the molt to the second instar an

4 l morphology and connectivity change between first instar and third instar larval stages using serial

5 y nontoxic to monarch first instars, whereas first instars are sensitive to Cry1Ab and Cry1Ac protein

6 Potted host plants were infested with first instar black swallowtails and placed at intervals

7 -1KO-associated paralysis and death; starved first instar DmiR-1KO larvae are essentially normal.

8 ession of pnr and Iro-C is initiated in late first instar dorsal eye margin cells.

9 between brain and ventral nerve cord of the first-instar Drosophila larva.

10 Ac reduced insect mortality by 25.5-55.6% to first instar H. virescens and M. sexta larvae, suggestin

11 meless (tim) expression are initiated at the first instar (L1) larval stage or during metamorphosis,

12 onstructions of the mushroom bodies from the first instar larva and adult Drosophila melanogaster.

13 tion, morphogenetic movements that shape the first instar larva.

14 We hypothesize that the first-instar larva (L1) of F. occidentalis mounts a resp

15 addition to adult dispersal, wind-dispersed first instar larvae also contribute to lifetime dispersa

16 ds to a dramatic delay in the growth rate of first instar larvae and ultimately death.

17 The adult males and the first instar larvae in the Mengenillidia and Stylopidia

18 The same situation exists in first instar larvae of holometabolous insects, in which

19 When first instar larvae were given access to trophic eggs, t

20 ion and the viviparous female to release the first instar larvae when the next generation of the host

21 Maternal HP1 is still detectable in first instar larvae, but disappears by third instar, sug

22 embryos from null mothers fail to hatch into first instar larvae.

23 fragmentation, and lethality at the stage of first instar larvae.

24 hibited liquid clearance from the trachea of first instar larvae.

25 hal phenotype and die as stage 17 embryos or first instar larvae.

26 y required for embryos to hatch into feeding first instar larvae.

27 s, but they die within 2 days of hatching as first instar larvae.

28 ort here the identification of six TIPs from first-instar larvae (L1), the most acquisition-efficient

29 Bioassays performed in first-instar larvae demonstrated potent activity against

30 ing proteins from Frankliniella occidentalis first-instar larvae.

31 transcriptome atlas of the entire Drosophila first instar larval brain.

32 imilarly we are able to rescue the embryonic/first instar larval lethality of an alphaPS1 null mutati

33 now identified Notch expressing HSCs in the first instar larval lymph gland.

34 ion is required in mesoderm beginning at the first instar larval stage of development.

35 e lethality occurred at the embryonic and/or first instar larval stages when raised on diet without t

36 We show that mge mutations lead to first-instar larval lethality and that Mge protein is si

37 null PSI mutation is recessive lethal at the first-instar larval stage, and lethality is fully rescue

38 First-instar mantispids use two strategies to locate spi

39 ingle-species test system was used to expose first instar, mid-instar, and late instar mayflies (Ephe

40 ehavior, by recording from photoreceptors of first instar nymphs and adult animals using the patch-cl

41 To address this hypothesis, we fed first instars of the large wolf spider Hogna antelucana

42 Molting after the normal first instar period was restored to various degrees by f

43 omatin localized gene leads to arrest at the first instar stage which can be rescued by feeding the l

44 ing Aedes aegypti, do not develop beyond the first instar when fed a nutritionally complete diet in t

45 proteins are relatively nontoxic to monarch first instars, whereas first instars are sensitive to Cr