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1 ation coverage from data for non-polio acute flaccid paralysis.
2 phalitis, and 3 had poliomyelitis-like acute flaccid paralysis.
3 s conducting surveillance for cases of acute flaccid paralysis.
4 ose IPV (fIPV) as a measure to prevent acute flaccid paralysis.
5 ated calcium channels and to an irreversible flaccid paralysis.
6 pulation immunity and prevent cases of acute flaccid paralysis.
7 esents as encephalitis, meningitis, or acute flaccid paralysis.
8 ausation or recovery from ZIKV-induced acute flaccid paralysis.
9 myelitis, a condition characterized by acute flaccid paralysis.
10 s investigated during surveillance for acute flaccid paralysis.
11 3 and early 2014 in the absence of any acute flaccid paralysis.
12 re neurological complications, such as acute flaccid paralysis.
13 sting stool samples from patients with acute flaccid paralysis.
14 naptic vesicle (SV) exocytosis, resulting in flaccid paralysis.
16 h a higher rate of reporting non-polio acute flaccid paralysis (AFP) (OR = 1.13, 95% CI 1.02-1.26 for
17 t noted an abnormal number of cases of acute flaccid paralysis (AFP) among adults, which were later c
22 n <36 mo old identified with non-polio acute flaccid paralysis (AFP) reported through polio surveilla
26 io enteroviruses (NPEVs) isolated from acute flaccid paralysis (AFP) surveillance in Shandong Provinc
27 tionnaires collected information about acute flaccid paralysis (AFP) surveillance resources, training
30 cle reviews the epidemiology of polio, acute flaccid paralysis (AFP) surveillance, and the implementa
31 ect process indicators associated with acute flaccid paralysis (AFP) surveillance, routine immunizati
33 following immunisation (AEFI); ongoing acute flaccid paralysis (AFP) surveillance; active, hospital-b
35 averaged only 57% and surveillance for acute flaccid paralysis (AFP) was suboptimal (AFP rate<1 per 1
36 1 isolates obtained from patients with acute flaccid paralysis (AFP) were compared by nucleotide sequ
42 specimens from patients reported with acute flaccid paralysis and 3171 wastewater samples were teste
43 the release of neurotransmitters that cause flaccid paralysis and are considered potential bioweapon
44 d to assess a cluster of children with acute flaccid paralysis and cranial nerve dysfunction geograph
45 ally and temporally defined cluster of acute flaccid paralysis and cranial nerve dysfunction in child
46 ne depolarization, action potential failure, flaccid paralysis and cytopathology that are characteris
48 caviruses in human diseases, including acute flaccid paralysis and diarrhea, will require further epi
49 er investigators that highlighted persistent flaccid paralysis and electrophysiological evidence of a
51 irulent variants exhibiting polio-like acute flaccid paralysis and other central nervous system manif
54 ncluding neonatal sepsis-like disease, acute flaccid paralysis, and acute hemorrhagic conjunctivitis.
55 hagic disease, encephalitis, biphasic fever, flaccid paralysis, and jaundice are typical manifestatio
58 al features of the increasing cases of acute flaccid paralysis associated with anterior myelitis note
60 children aged 0-14 years with onset of acute flaccid paralysis between Jan 1, 2001, and Dec 31, 2011.
61 aseptic meningitis, poliomyelitis-like acute flaccid paralysis, brainstem encephalitis, and other sev
62 ) is a highly potent neurotoxin that elicits flaccid paralysis by enzymatic cleavage of the exocytic
63 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting neurotransmitter-carryin
64 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting the fusion of neurotrans
65 linum toxin (BT) is a neurotoxin that causes flaccid paralysis by inhibiting the release of acetylcho
66 An analysis was conducted of 10,486 acute flaccid paralysis cases diagnosed as Guillain-Barre synd
67 ean numbers of infected and uninfected acute flaccid paralysis cases investigated in a season are der
68 like poliovirus isolates from Nigerian acute flaccid paralysis cases obtained from routine surveillan
70 nvestigated the mechanism of temporary acute flaccid paralysis caused by Zika virus infection in aged
71 nergic nerve terminals, causing a descending flaccid paralysis characteristic of the disease botulism
72 vascular shock syndrome, encephalitis, acute flaccid paralysis, congenital abnormalities and fetal de
73 tates between 2009 and 2014, using the acute flaccid paralysis database at the World Health Organizat
74 ive disease (meningitis, encephalitis, acute flaccid paralysis) develops in less than 1% but carries
76 as compared with that of children with acute flaccid paralysis due to other causes to estimate the cl
77 West Nile encephalitis; a poliomyelitis-like flaccid paralysis due to West Nile virus was recognised,
78 23 August 1991, a 2-year-old boy with acute flaccid paralysis due to wild poliovirus was detected in
79 d population groups, and surveillance (acute flaccid paralysis, enterovirus, and environmental) needs
82 documented in 27 percent of the patients and flaccid paralysis in 10 percent; in all of the latter, n
83 in 20 (35%), encephalitis in 6 (11%), acute flaccid paralysis in 4 (7%), and autonomic dysregulation
85 cribed as a bilateral, symmetric, descending flaccid paralysis in an afebrile and alert patient witho
87 dium botulinum (Clb), the causative agent of flaccid paralysis in humans that can be fatal in 5 to 10
91 here has been limited surveillance for acute flaccid paralysis in North America since the regional er
93 erosurvey was conducted among cases of acute flaccid paralysis in the 25 high-polio-incidence distric
95 d in 12 of 192 patients with non-polio acute flaccid paralysis in Tunisia and Nigeria and 0 of 96 hea
96 ingitis, and concomitant muscle weakness and flaccid paralysis may provide a clinical clue to the pre
99 ted HFMD (n = 47), meningitis (n = 8), acute flaccid paralysis (n = 1), encephalitis (n = 21), and en
100 pisodes), oculogyric crisis (four episodes), flaccid paralysis of all extremities (four episodes), tr
101 m is characterized by symmetric, descending, flaccid paralysis of motor and autonomic nerves, usually
102 lysis (hypoKPP) is characterized by episodic flaccid paralysis of muscle and acute hypokalemia during
103 sure for botulism, a fatal illness caused by flaccid paralysis of muscles due to botulinum neurotoxin
110 s of stool samples taken from cases of acute flaccid paralysis revealed the presence of mixtures of r
111 posed national surveillance system for acute flaccid paralysis should capture at a minimum the 796 GB
112 e release at neuromuscular junctions causing flaccid paralysis, specifically synaptosomal-associated
113 , the importance of maintaining strong acute flaccid paralysis surveillance even in adults, and the n
114 We conducted a retrospective review of acute flaccid paralysis surveillance in the security-compromis
115 lected from children identified by the acute flaccid paralysis surveillance program in India during 2
116 from among cases reported through the acute flaccid paralysis surveillance system between November 2
117 r understanding the sensitivity of the acute flaccid paralysis surveillance system is presented by fi
118 Health Organization requested that the acute flaccid paralysis surveillance system of Latin American
121 supplementary immunization activities, acute flaccid paralysis surveillance, and routine immunization
126 levamisole: an initial spastic paralysis; a flaccid paralysis that follows; and finally, a recovery
127 rus developed motor deficits including acute flaccid paralysis that peaked 8-10 days after viral chal
128 sm (WB) is a potentially lethal, descending, flaccid, paralysis that results when spores of Clostridi
130 e zinc proteases (serotypes A-G) which cause flaccid paralysis through the cleavage of SNARE proteins
133 nce database, in which 27,379 cases of acute flaccid paralysis were recorded between 2001 and 2007.
135 s act locally within motor neurons to elicit flaccid paralysis, while retrograde TeNT traffics to inh
136 icated a rare but distinct syndrome of acute flaccid paralysis with evidence of spinal motor neuron i
137 AE in rodents typically results in ascending flaccid paralysis with inflammation primarily targeting
138 rodents is manifested typically as ascending flaccid paralysis with inflammation targeting the spinal
139 ibe a group of patients with acute segmental flaccid paralysis with minimal or no encephalitic or sen
140 apon would cause acute symmetric, descending flaccid paralysis with prominent bulbar palsies such as
141 spital Colorado (Aurora, CO, USA) with acute flaccid paralysis with spinal-cord lesions involving mai
142 Timely investigation of children with acute flaccid paralysis, with collection of stool specimens fo
143 rome (GBS) is the most common cause of acute flaccid paralysis worldwide, and is thought to be immune
144 utants move poorly, exhibiting an incomplete flaccid paralysis, yet have normal muscle ultrastructure