ライフサイエンスコーパス: fluid resuscitation

1 inistration of normal saline solution (i.e., fluid resuscitation).

2 inistration of normal saline solution (i.e., fluid resuscitation).

3 inistration of normal saline solution (i.e., fluid resuscitation).

4 inistration of normal saline solution (i.e., fluid resuscitation).

5 icrocirculations during hemorrhage and after fluid resuscitation.

6 g severe hemorrhagic shock in the absence of fluid resuscitation.

7 roximately 40 mm Hg for 90 mins) followed by fluid resuscitation.

8 minutes after injury before any significant fluid resuscitation.

9 e has been immediate, aggressive intravenous fluid resuscitation.

10 sure 35-40 mm Hg for 90 minutes) followed by fluid resuscitation.

11 hock (blood pressure, 35 mm Hg), followed by fluid resuscitation.

12 y PulseCO and LiDCO at 10 and 120 mins after fluid resuscitation.

13 d perioperative medicine as controversial as fluid resuscitation.

14 (0.2 units/kg) or placebo during the initial fluid resuscitation.

15 eived supportive standard intensive care and fluid resuscitation.

16 65 mm Hg, both during and following adequate fluid resuscitation.

17 nary circulation in the absence of immediate fluid resuscitation.

18 stinal barrier function after hemorrhage and fluid resuscitation.

19 (two times the shed blood volume) to provide fluid resuscitation.

20 Only two of 13 deaths occurred during fluid resuscitation.

21 12 (3-68)) were administered within 5 min of fluid resuscitation.

22 nistration yielded better results than rapid fluid resuscitation.

23 tups for assessing PCLC devices intended for fluid resuscitation.

24 AKI), at any time within the first 7 days of fluid resuscitation.

25 ts with AP receiving low, moderate, and high fluid resuscitation.

26 ving hemorrhage, tourniquet application, and fluid resuscitation.

27 fine international standards for intravenous fluid resuscitation.

28 model of hemorrhagic shock in the absence of fluid resuscitation.

29 olic blood pressure less than 90 mm Hg after fluid resuscitation.

30 e level greater than 2 mmol/L after adequate fluid resuscitation.

31 be as effective and efficient as intravenous fluid resuscitation.

32 inistration of either crystalloid or colloid fluid resuscitation (1B); fluid challenge to restore mea

33 ntricular (RV) infarct (1C), the efficacy of fluid resuscitation (1C) and inotropic therapy (2C), pre

34 Interventions included fluid resuscitation (35 cases), pleural drainage (three

35 1.4 [0.53--7.9] mmol/L, p<0.0001) and after fluid resuscitation (5.5 [1.3--18.6] vs 1.3 [0.26--3.2],

36 eutic interventions included median 80-mL/kg fluid resuscitation; 65% of patients required dopamine,

37 likely to require vasopressors after initial fluid resuscitation (68.5% vs. 52.5%, p < 0.01).

38 s solution alone during the first 6 hours of fluid resuscitation after intensive care medicine (ICU)

39 Acute fluid resuscitation after trauma-hemorrhage restores but

40 Fluid resuscitation after traumatic hemorrhage has histo

41 as renal function, in a manner comparable to fluid resuscitation alone and without differences betwee

42 in a manner comparable to that achieved with fluid resuscitation alone.

43 with cecal ligation and puncture followed by fluid resuscitation, analgesia, and antibiotics.

44 ks curative options and consists of adequate fluid resuscitation, analgesics, and monitoring.

45 Early therapy of sepsis involving fluid resuscitation and antibiotic administration has be

46 After fluid resuscitation and antibiotic therapy, careful card

47 Rates of fluid resuscitation and antibiotic utilization did not d

48 ligation and puncture and were treated with fluid resuscitation and antibiotics.

49 nts, and profound shock requiring aggressive fluid resuscitation and careful hemodynamic monitoring a

50 rapy when cardiac output remains low despite fluid resuscitation and combined inotropic/vasopressor t

51 Variability in fluid resuscitation and difficulty recognizing early sep

52 Early and aggressive fluid resuscitation and early enteral nutrition are asso

53 receive after 12 hours of fecal peritonitis fluid resuscitation and either norepinephrine (group NE;

54 urfactant, glucose, insulin, hydrocortisone, fluid resuscitation and fluid removal, superior vena cav

55 istration of appropriate doses of aggressive fluid resuscitation and intravenous (IV) adrenaline in R

56 eading to hypoxemia and may be used to guide fluid resuscitation and optimize tissue oxygenation.

57 With fluid resuscitation and oxygen therapy, the patient rega

58 ssue hypoperfusion resulting from inadequate fluid resuscitation and the development of AKI after lun

59 We sought to review the evidence for rapid fluid resuscitation and to outline its clinical indicati

60 impact on the current clinical approach for fluid resuscitation and treatment of coagulopathy for tr

61 as important clinical implications regarding fluid resuscitation and treatment of coagulopathy.

62 yndrome and persistent hypotension following fluid resuscitation and vasopressor infusion.

63 n the three identified priorities related to fluid resuscitation and vasopressor therapies.

64 e in adult septic shock patients if adequate fluid resuscitation and vasopressor therapy are able to

65 Expand upon the priorities of fluid resuscitation and vasopressor therapy research pri

66 in our institution of "poorly responsive to fluid resuscitation and vasopressor therapy" being the p

67 heir "blood pressure is poorly responsive to fluid resuscitation and vasopressor therapy." Because th

68 includes hypotensive hemostasis, minimizing fluid resuscitation, and allowing the systolic blood pre

69 Large bowel resection, large-volume fluid resuscitation, and an increasing number of abdomin

70 quivalent (IE) counts, estimated blood loss, fluid resuscitation, and blood transfusions.

71 ting in significantly fewer inotropic drugs, fluid resuscitation, and mechanical ventilation requirem

72 tachycardia; and d) refractory shock despite fluid resuscitation, and vasoactive medications.

73 Conclusions: In sepsis trials, the effect of fluid resuscitation approach differed by setting, with h

74 Adequate fluid resuscitation, appropriate inotropic support, atte

75 pportive treatment consisting of high-volume fluid resuscitation (approximately 10 liters per day in

76 Intravenous and enteral fluid resuscitation are frequently used therapies in the

77 ars that pharmacologic agents in addition to fluid resuscitation are needed to restore cardiovascular

78 ation of inhalation injury and its impact on fluid resuscitation, as well as on a protective lung str

79 h differed by setting, with higher volume of fluid resuscitation associated with increased mortality

80 tion facilitated the hemodynamic response to fluid resuscitation, attenuated tissue inflammatory inju

81 Rationale: Sepsis management relies on fluid resuscitation avoiding fluid overload and its rela

82 capacity, more difficult intravenous access, fluid resuscitation based on weight with 40-60 mL kg or

83 d with % TBSA burn, inhalation injury grade, fluid resuscitation, Baux score, revised Baux score, Den

84 3) how should fluid resuscitation be individualized initially and beyo

85 eived 1-3 L of crystalloid fluid for initial fluid resuscitation before randomization.

86 Rapid fluid resuscitation benefits pediatric patients with sev

87 ic measurements, organ biomarkers, volume of fluid resuscitation, cardiac agents, and the inflammator

88 Moderate fluid resuscitation consisted of a bolus of 10 ml per ki

89 Aggressive fluid resuscitation consisted of a bolus of 20 ml per ki

90 The current strategy of fluid resuscitation could be modified according to the o

91 ere is evidence that goal-directed, moderate fluid resuscitation decreases the risk of fluid overload

92 dentified patients who received large-volume fluid resuscitation, defined as greater than 60 mL/kg ov

93 in intracranial pathologies, as small volume fluid resuscitation during spinal shock, and as maintena

94 Fluid resuscitation during the first 24 to 48 hrs after

95 351) versus 0.9% saline ( n = 357) for bolus fluid resuscitation during the first 7 days.

96 e been developed in an effort to standardize fluid resuscitation during this time.

97 Optimal sepsis management including fluid resuscitation, early antibiotic administration, an

98 critical care interventions, including rapid fluid resuscitation, early antibiotics, and patient moni

99 a dedicated study medical officer comprising fluid resuscitation, early antibiotics, and regular moni

100 re management components such as the initial fluid resuscitation, end-organ support, pain management,

101 rvention with broad-spectrum antibiotics and fluid resuscitation, even in the absence of hypotension,

102 enefit scores for restrictive versus liberal fluid resuscitation exhibited marked survival difference

103 Fluid resuscitation following hemorrhagic shock is often

104 Fluid resuscitation following severe inflammation-induce

105 ough most clinicians still generally support fluid resuscitation for multisystem blunt trauma, partic

106 ic use, lactate measurements, and aggressive fluid resuscitation for patients with suspected sepsis b

107 tion, and in 60- to 120-min intervals during fluid resuscitation for up to 300 min.

108 shock (blood pressure 35 mmHg), followed by fluid resuscitation (four times the shed blood volume in

109 aCl adenosine, lidocaine, and Mg hypotensive fluid resuscitation from the rat to the pig.

110 ents might have therapeutic potential during fluid resuscitation from trauma.

111 eatitis, post-cardiopulmonary resuscitation, fluid resuscitation > 5 L/24 hr, vasoactive or inotropic

112 s. 12% of the survivors (p<.05), and delayed fluid resuscitation (>2 hrs after burn injury), identifi

113 level greater than 2 mmol/L (18 mg/dL) after fluid resuscitation had a significantly higher mortality

114 Rapid fluid resuscitation has become standard in sepsis care,

115 Fluid resuscitation has demonstrated positive effects; h

116 Rapid fluid resuscitation has gained increased recognition sin

117 Liberal fluid resuscitation has little effect on this sequestrat

118 n remain controversial, and the best form of fluid resuscitation has yet to be identified.

119 nd efficacy of hydroxyethyl starch (HES) for fluid resuscitation have not been fully evaluated, and a

120 plications, yet the optimal type and rate of fluid resuscitation have yet to be determined.

121 eceived intravenous antibiotics and adequate fluid resuscitation, hemodynamic management according to

122 Nineteen low-value practices on imaging, fluid resuscitation, hospital/intensive care unit admiss

123 Among 4,710 patients receiving large-volume fluid resuscitation, hyperchloremic acidosis was documen

124 luid type predominantly used for the initial fluid resuscitation (i.e., >= 95% of pre-randomization f

125 urinary [TIMP-2]*[IGFBP7] following initial fluid resuscitation identify sepsis patients with differ

126 Fluid resuscitation immediately following a hemorrhagic

127 Fluid resuscitation improves clinical outcomes of burn p

128 ith two lisofylline dosing regimens added to fluid resuscitation in a shock model.

129 asma levels of IL-6, we propose that chronic fluid resuscitation in addition to acute fluid replaceme

130 O) during severe hemorrhagic shock and after fluid resuscitation in dogs.

131 ecreases in cardiac output or the effects of fluid resuscitation in dogs.

132 ized that administration of AM/AMBP-1 during fluid resuscitation in hemorrhaged animals (i.e., posttr

133 starch (HES) [corrected] is widely used for fluid resuscitation in intensive care units (ICUs), but

134 esponded to notable changes in the volume of fluid resuscitation in patients with heart failure and/o

135 ases for all randomized controlled trials on fluid resuscitation in patients with sepsis or septic sh

136 management principles, the details of early fluid resuscitation in sepsis remain contentious.

137 antly related to blood loss before and after fluid resuscitation in the 16 survivors.

138 omly assigned patients with severe sepsis to fluid resuscitation in the ICU with either 6% HES 130/0.

139 ebate about the safety and efficacy of rapid fluid resuscitation in the pediatric patient.

140 develops during abdominal surgery and after fluid resuscitation in trauma patients.

141 Hypotensive fluid resuscitation in uncontrolled hemorrhagic shock wi

142 5 +/- 5 (SEM) mm Hg for 90 mins, followed by fluid resuscitation) in male C3H/HeN mice and the animal

143 function) and required inotropic support and fluid resuscitation (including 23/29 [79%] who received

144 hibitor improved the hemodynamic response to fluid resuscitation, increased blood oxygen content, pre

145 Endotoxin administration with fluid resuscitation induced a distributive shock with a

146 ctic antibiotics, avoiding overly aggressive fluid resuscitation, initiating early feeding, avoiding

147 vasodilatory or cardiogenic shock requiring fluid resuscitation, inotropic support, and in the most

148 mmon cause of acute kidney injury (AKI), and fluid resuscitation is a major part of therapy.

149 A moderate approach to IV fluid resuscitation is associated with decreased sepsis

150 Fluid resuscitation is different in bleeding and septic

151 ical variable that plays a major role during fluid resuscitation is heart rate (HR).

152 Rapid fluid resuscitation is most commonly used for children w

153 Fluid resuscitation is the cornerstone of sepsis treatme

154 Fluid resuscitation is the recommended management of sho

155 Therefore, following the initial fluid resuscitation, it is important to identify which p

156 urring as a consequence of overly aggressive fluid resuscitation may adversely affect outcome in hemo

157 eplacement therapy, as well as goal-directed fluid resuscitation may lead to improved survival in cri

158 s and arterial catheterization, antibiotics, fluid resuscitation, mechanical ventilation, vasopressor

159 l blood lactate early (median 4 h) and after fluid resuscitation (median 12 h) in patients admitted t

160 In separate experiments, using a fluid resuscitation model we studied mitochondrial funct

161 the fluids they received during large-volume fluid resuscitation multiplied by the volume of fluids.

162 resuscitation: retransfusion of shed blood, fluid resuscitation, norepinephrine titrated to maintain

163 drated and need adequate vascular access for fluid resuscitation, nutrition, and phlebotomy for labor

164 ded by algorithms including upper limits for fluid resuscitation of extravascular lung water (<10 mL/

165 e, and prevented circulatory collapse during fluid resuscitation of hemorrhagic shock after traumatic

166 associated with statistically more rigorous fluid resuscitation of patients, greater administration

167 Recent studies show that early, aggressive fluid resuscitation of up to 60 ml/kg within 1-2 h may b

168 n = 9) or sham burn receiving anesthesia and fluid resuscitation only (n = 8) and were killed 48 hrs

169 antimicrobial administration and appropriate fluid resuscitation, optimized critical care management,

170 ged with basic critical care (stabilisation, fluid resuscitation, oxygen, and vital-organ support), b

171 sing to IAH/ACS include sepsis, large volume fluid resuscitation, polytransfusion, mechanical ventila

172 ine the relationship between a wide range of fluid resuscitation practices and sepsis mortality and t

173 analysis of within- and between-hospital IV fluid resuscitation practices showed that physician vari

174 mendations are to limit or delay intravenous fluid resuscitation preoperatively in those with uncontr

175 e evolving evidence suggests that aggressive fluid resuscitation prior to hemostasis leads to additio

176 ount of chloride received during intravenous fluid resuscitation (r = .44), with the base excess chan

177 ce endotoxemic shock or saline (control) and fluid resuscitation (R) with or without O-GlcNAc stimula

178 rocardiography and echocardiography results, fluid resuscitation, radiography results, and laboratory

179 l/L, requiring vasopressors despite adequate fluid resuscitation, regardless of shock cause.

180 Fluid resuscitation remains the cornerstone of acute bur

181 ropriate use of loperamide, and knowledge of fluid resuscitation requirements of affected patients is

182 rgan dysfunction and reduced vasopressor and fluid resuscitation requirements.

183 interest was first 24-hour blood product and fluid resuscitation requirements.

184 ualty incidents with limited availability of fluid-resuscitation resources.

185 ts with acute pancreatitis, early aggressive fluid resuscitation resulted in a higher incidence of fl

186 ted to hemorrhage and underwent a randomized fluid resuscitation scheme on separate visits 1) formula

187 trials from the past to the present include fluid resuscitation, sepsis, immune function, hypermetab

188 There were no sex differences in fluid resuscitation, shock index, coagulation, and base

189 This approach to fluid resuscitation should be abandoned.

190 Transfer patients were less likely to have fluid resuscitation started by 3 hours (54% vs 89%; p <

191 nalysis to compare liberal versus restricted fluid resuscitation strategies in trauma patients.

192 nal studies, odds for mortality with liberal fluid resuscitation strategies increased (odds ratio, 1.

193 rent evidence indicates that initial liberal fluid resuscitation strategies may be associated with hi

194 s were randomly assigned to a restrictive IV fluid resuscitation strategy (<= 60 mL/kg of IV fluid) o

195 It is unclear if a low- or high-volume IV fluid resuscitation strategy is better for patients with

196 ifiable in sepsis and respond differently to fluid resuscitation strategy.

197 psis and whether they respond differently to fluid resuscitation strategy.

198 two mechanisms may be relevant for the early fluid resuscitation strategy.Objectives: To understand t

199 tment of hemorrhagic shock in the absence of fluid resuscitation; therefore DCA may be a good candida

200 In the absence of fluid resuscitation, these changes persisted and were ac

201 dentification of septic patients, aggressive fluid resuscitation, timely antibiotic administration, a

202 and recommendations on GSP severity grading, fluid resuscitation, timing of cholecystectomy, need for

203 lowed by UHS via tail amputation and limited fluid resuscitation to maintain mean arterial pressure a

204 lterations that can be minimized by adequate fluid resuscitation to maintain tissue perfusion, early

205 to 135 mins) with hemostasis and aggressive fluid resuscitation to normalize hemodynamics; and obser

206 Acute bleeding management relies on fluid resuscitation to promote renal excretion of active

207 ible to recruit critically ill patients to a fluid resuscitation trial in U.K. EDs using 5% HAS as a

208 Without volume expansion with fluid resuscitation, trigeminal nerve stimulation signif

209 Rats were observed without fluid resuscitation until death (apnea and pulselessness

210 ide or 0.9% sodium chloride (saline) for all fluid resuscitation until ICU discharge, death, or 90 da

211 from t = 40-70 min, followed by reperfusion/fluid resuscitation until t = 300 min.

212 otal body surface area (TBSA) scald burn and fluid resuscitation using the Parkland formula.

213 fs and combinations for blood pressure (BP), fluid resuscitation, vasopressors, serum lactate level,

214 ement of early sepsis-induced hypoperfusion: fluid resuscitation volume, timing of vasopressor initia

215 ct of vasopressor dosing intensity varies by fluid resuscitation volume; and 3) determine whether the

216 injury characteristics that most influenced fluid resuscitation volumes received.

217 re assessed at 12, 24, 48, and 72 hours, and fluid resuscitation was adjusted according to the patien

218 s was easily established in all animals, and fluid resuscitation was carried out effectively through

219 After 4 hours of hypotension, fluid resuscitation was initiated with a crystalloid sol

220 As fluid resuscitation was initiated, tumor necrosis factor

221 Fluid resuscitation was performed with saline alone or i

222 After randomization, fluid resuscitation was started 2 hours after severe acu

223 d hemorrhage with an extremely low volume of fluid resuscitation was used to mimic the combat situati

224 g values for a threshold of 3.0 mmol/L after fluid resuscitation were 76%, 97%, 30, and 0.24.

225 r severe hemorrhagic shock in the absence of fluid resuscitation were analyzed.

226 ssure greater than 65 mm Hg despite adequate fluid resuscitation, were included.

227 ne output < 0.5 mL/kg/hr for > 6 hrs despite fluid resuscitation when applicable) predicts meaningful

228 subdiaphragmatic blood loss and allow for IV fluid resuscitation when intrinsic cardiac activity is s

229 pressure less than 90 mm Hg after an initial fluid resuscitation, who lacked an obvious source of hyp

230 r conventional cooling methods consisting of fluid resuscitation with 0.9% sodium chloride solution,

231 Fluid resuscitation with acadesine produced no adverse h

232 IV fluid resuscitation with balanced crystalloid or 5% HAS

233 Fluid resuscitation with commercially available crystall

234 hod within 12 hrs of diagnosis (1C); initial fluid resuscitation with crystalloid (1B) and considerat

235 Acidotic patients received fluid resuscitation with either dextran 70 or starch at

236 Patients with severe sepsis assigned to fluid resuscitation with HES 130/0.42 had an increased r

237 Numerous animal studies have suggested that fluid resuscitation with HS bolus after hemorrhagic shoc

238 Initial fluid resuscitation with lactated Ringer's solution, com

239 The data further suggest that fluid resuscitation with LR may benefit patients with se

240 Among children presenting with septic shock, fluid resuscitation with MES (balanced crystalloid) as c

241 g from normotensive to hypotensive (limited) fluid resuscitation with plasma substitutes.

242 In this prospective study, we evaluated how fluid resuscitation with PolyHSA impacts the hemodynamic

243 Additionally, we evaluated fluid resuscitation with PolyHSA in a model of polymicro

244 maintenance of vascular integrity following fluid resuscitation with PolyHSA.

245 ldly to moderately dehydrated child, enteral fluid resuscitation with the aid of an antiemetic such a

246 olloid, and electrolyte solution for limited fluid resuscitation with the smallest volume should cont

247 The findings support the notion that fluid resuscitation with unbuffered electrolyte solution

248 For fluid resuscitation within 8 hours of sepsis diagnosis:

249 septic shock requiring vasopressors despite fluid resuscitation within a maximum of 6 hours after th