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1 rosis) and enamel development (dental/enamel fluorosis).
2 vide a potential mechanism underlying dental fluorosis.
3 s associated with moderate and severe dental fluorosis.
4 rial species enriched in moderate and severe fluorosis.
5 ation of fluoride levels in water and dental fluorosis.
6 d to an increase in the occurrence of dental fluorosis.
7 uorosis; and 3.7%, mild, moderate, or severe fluorosis.
8  MMP20 plays a nominal role in dental enamel fluorosis.
9 ession is involved in the etiology of dental fluorosis.
10 ression is not solely responsible for dental fluorosis.
11 ctional Mmp20 allele are more susceptible to fluorosis.
12 understanding of the biological mechanism of fluorosis.
13  as a model organism for the study of dental fluorosis.
14 ing that ER stress may play a role in dental fluorosis.
15 sis may also play a role in the mechanism of fluorosis.
16 train is least affected, with minimal dental fluorosis.
17 sceptibility or resistance to develop dental fluorosis.
18 etic component in the pathogenesis of dental fluorosis.
19 unding areas suffer from dental and skeletal fluorosis.
20 e of ready to feed infant formula and enamel fluorosis.
21 rall, only 8% of children showed evidence of fluorosis.
22 eration in patients with moderate and severe fluorosis.
23 al caries prevention with the risk of dental fluorosis.
24 cantly associated with higher odds of dental fluorosis.
25 hift in patients with moderate/severe dental fluorosis.
26 ere associated with a greater risk of dental fluorosis.
27     Of 5,484 examined students, 71.8% had no fluorosis; 24.4%, questionable to very mild fluorosis; a
28 ed and calibrated examiners to assess dental fluorosis (a reliable and valid individual biomarker of
29 is a serious health hazard as it may lead to fluorosis, a serious bone disease.
30 been an increase in the prevalence of dental fluorosis, a side-effect of exposure to greater than opt
31                      No associations between fluorosis and any OHRQoL scales met statistical or MID t
32 fetus and young infant, and can cause dental fluorosis and cognitive deficits.
33 study were to determine the impact of enamel fluorosis and dental caries on oral health-related quali
34 osure, and prevalence and severity of dental fluorosis and dental caries.
35  association between mild-to-moderate enamel fluorosis and exposure during early childhood to fluorid
36  relationship between the severity of dental fluorosis and the oral microbiome.
37 o disturbances of bone homeostasis (skeletal fluorosis) and enamel development (dental/enamel fluoros
38 f gingival bleeding, dental fracture, dental fluorosis, and dental caries experience.
39  fluorosis; 24.4%, questionable to very mild fluorosis; and 3.7%, mild, moderate, or severe fluorosis
40 sms that underlie the pathogenesis of dental fluorosis are not known.
41 he molecular mechanisms that underlie dental fluorosis are poorly understood.
42  mechanism or mechanisms resulting in dental fluorosis are unknown.
43 of fluoride (F(-)) toxicity such as skeletal fluorosis being endemic in at least 25 countries, constr
44 ic perception of dental fluorosis, with mild fluorosis being of no aesthetic concern.
45 es) were analyzed for their association with fluorosis categories and sum of d2-3fs and D2-3MFS accor
46 rapid onset and severe development of dental fluorosis compared with that in the other strains tested
47               All students were examined for fluorosis (Dean's index) and caries experience (d2-3fs o
48 en developing incisors are most sensitive to fluorosis development.
49 und in artificially fluoridated water and in fluorosis endemic areas-is associated with memory and le
50 en in the Birth Cohort through Cohort 3, and fluorosis for children in Cohorts 1 through 5.
51  experience negatively affects OHRQoL, while fluorosis has little impact.
52  this break on the development of caries and fluorosis in children.
53 studies of intra-oral distribution of dental fluorosis in low-fluoride areas reported that teeth that
54 l study investigated risk factors for enamel fluorosis in optimally fluoridated children, born after
55 lanation for the higher occurrence of dental fluorosis in posterior teeth.
56    A weighted 87.3% exhibited some degree of fluorosis in the 2013-2014 cycle and 68.2% in the 2015-2
57 occurrence of very mild or greater levels of fluorosis in the upper anterior teeth was 7 to 10% in th
58   In the fluoridated area, the occurrence of fluorosis increased from anterior to posterior teeth.
59                                   The Dean's Fluorosis Index was used to evaluate fluorosis status fo
60                                       Dental fluorosis is a very prevalent endemic disease.
61                       The severity of dental fluorosis is also dependent upon fluoride dose and the t
62                                       Dental fluorosis is caused by chronic high-level fluoride (F(-)
63 he break had little effect on caries, dental fluorosis is sensitive to even small changes in fluoride
64 w this trend affects esthetic perceptions of fluorosis is warranted.
65     Over-intake of F(-) can result in dental fluorosis, kidney failure, or DNA damage.
66 uoride exposures were associated with dental fluorosis, logistic regression analyses were conducted.
67                                       Dental fluorosis occurs from overingestion of fluoride during t
68  association between mild-to-moderate enamel fluorosis on early forming (FRI classification I) enamel
69  association between mild-to-moderate enamel fluorosis on early forming (FRI classification I) enamel
70  formula was not found to be associated with fluorosis on either FRI classification I or II surfaces.
71  strong association between mild-to-moderate fluorosis on later forming (FRI classification II) ename
72  strong association between mild-to-moderate fluorosis on later forming (FRI classification II) ename
73 olars according to the decayed-filled index; fluorosis on the labial surfaces of the upper permanent
74 ive, but nonsignificant, association between fluorosis on these enamel surfaces and infant formula in
75 evidence in the literature on whether or how fluorosis prevalence and severity change over time after
76                                              Fluorosis prevalence for seven age cohorts whose birth y
77 sm of how excessive fluoride exposure causes fluorosis remains uncertain.
78         The mechanism of how fluoride causes fluorosis remains unknown.
79 ostasis (X-linked hypophosphataemic rickets, fluorosis, rickets and osteomalacia).
80             Fluorosis was assessed using the Fluorosis Risk Index (FRI) and Russell's criteria for di
81 Case-control status was determined using the Fluorosis Risk Index (FRI).
82 s of a clinical dental examination using the Fluorosis Risk Index (FRI).
83 k populations or settings, with no increased fluorosis risk.
84                                  Measures of fluorosis severity at the person and tooth level were ca
85                                   The dental fluorosis severity value was based on the second most af
86  tooth levels, a decline in mild to moderate fluorosis severity was observed across adolescence and y
87                                     Overall, fluorosis severity, which was initially mild to moderate
88  Dean's Fluorosis Index was used to evaluate fluorosis status for each tooth.
89                                          The fluorosis status of the subjects was determined on the b
90 strains of mice showed differences in dental fluorosis susceptibility/resistance.
91 ak, and those born 1 year after it, had less fluorosis than those aged 4-5 years at the break.
92 while a higher concentration associated with fluorosis triggered memory and learning deficits, associ
93 7 (95% CI: -2.86, 5.01) and of having dental fluorosis versus no fluorosis was 0.28 (95% CI: -3.00, 3
94 ast, excessive fluoride intake causes dental fluorosis, visually recognizable enamel defects that can
95 01) and of having dental fluorosis versus no fluorosis was 0.28 (95% CI: -3.00, 3.57).
96                        Overall prevalence of fluorosis was 44%.
97                                              Fluorosis was assessed using the Fluorosis Risk Index (F
98                                              Fluorosis was measured by the TF index, and parents or g
99 To investigate the molecular bases of dental fluorosis, we analyzed the effects of fluoride exposure
100                  Risks identified for dental fluorosis were ingestion of fluoride-containing toothpas
101 sess caries and the presence of sealants and fluorosis were performed on 2921 third-grade students in
102 dant in patients with moderate/severe dental fluorosis, while Prevotella melaninogenica and Schaalia
103 he 2 exposure measurements (%LEFW and dental fluorosis) with full-scale IQ (FSIQ) scores, controlling
104 rstanding of the public perception of dental fluorosis, with mild fluorosis being of no aesthetic con
105 and these were successful in reducing dental fluorosis without compromising caries prevention.

 
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