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1 ized lamination and an absence of cerebellar folia.
2  of Purkinje cells distributed over multiple folia.
3  found that PL projects to caudal cerebellar folia.
4 ked to variation in the number of cerebellar folia.
5 tical areas and underneath the summit of its folia.
6 orming distinct parasagittal bands in caudal folia.
7 g due to the small size and abundance of its folia.
8                        In the pars anterior (folia 1-3) of the paramedian lobule, the projection to t
9                       In the pars posterior (folia 4-6), cells targeting the two zones arose from lar
10 ng fibers that project to the uvula-nodulus (folia 8-10).
11 pontaneous discharge of SSs in contralateral folia 8-10, but blocked their modulation during vestibul
12 rded from Purkinje cells and interneurons in folia 8-10, identified by juxtacellular labeling with Ne
13 limbing fiber signaling to the contralateral folia 8-10, while leaving intact vestibular primary and
14 e cell layers with the densest AMGBi were in folia adjacent to the fourth ventricle.
15 ellum with disrupted layers in poorly formed folia and strikingly reduced granule cell production.
16 ethods to measure the geometry of cerebellar folia and to estimate the thickness of the molecular lay
17 usion of cerebral hemispheres and cerebellar folia, and aberrant migration of granule cells.
18 eometry and size of clones in long and short folia are distinct.
19  at P3 in caudal lobules and extended to all folia by P9.
20                         Cerebellar deficient folia (cdf) is a recessive mouse mutation causing ataxia
21 Mice homozygous for the cerebellar deficient folia (cdf) mutation are ataxic and have cerebellar hypo
22 eover, in engrailed 1/2 mutants with shorter folia, clone cell number and geometry are most similar t
23 at both En genes are required to ensure that folia exclusive to the vermis or hemispheres form in the
24 iously published classification of parchment folia from a copy of the Gospel of Luke, produced around
25 he first time to the level of all individual folia from multicontrast high-resolution postmortem MRI
26 rkinje cell development and the formation of folia in the cerebellum.
27  have defects in the formation of lamina and folia in the cerebral and cerebellar cortices that are c
28 ed for appropriate development of cerebellar folia layering and structure.
29 stretches of Purkinje cell bodies in various folia of cerebellum.
30 erograde tracers were microinjected into the folia of crus I of the cat cerebellum to investigate spa
31 r an area of elevated density in the ventral folia of lobules V and VI.
32 d tactile cerebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes after de
33 ellar cortical c1 and c2 zones within apical folia of the forelimb-receiving area of the rostral para
34 obule V of the anterior lobe and the rostral folia of the paramedian lobule (PML) in the posterior lo
35                                 In the lower folia of the uvula and nodulus, Purkinje cell expression
36 geometry are most similar to clones in short folia of wild-type mice.
37 atrophic cerebellar hemispheres with stunted folia, profound granule cell depletion, Bergmann gliosis
38 in structure composed of an elaborate set of folia separated by fissures of different lengths, remain
39 both xylem and phloem, making application as folia spray, soil spray, and trunk injection equally eff
40 rcm(tg) homozygotes is smaller and has fewer folia than in the wild-type, ectopic cerebellar cells ar
41 bellum consists of a highly organized set of folia that are largely generated postnatally during expa
42 length of each zone located within different folia were also shown to relate to different groups of o
43                                          The folia were shown to be mainly occupied in rostrocaudal s