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1 ding those with benign cytology, to identify follicular adenoma.
2 hyperplastic gland and to the development of follicular adenomas.
3 adenomas, whereas 2 of 15 (13%) were typical follicular adenomas.
4 igh in thyroids from Graves' patients and in follicular adenomas.
5 variant of papillary thyroid carcinomas and follicular adenomas.
6 6%) low-stage follicular carcinomas, 0 of 40 follicular adenomas, 1 of 30 Hurthle cell carcinomas, 1
7 thyroid follicular carcinomas but not in 20 follicular adenomas, 10 papillary carcinomas, or 10 mult
9 ology does not allow differentiation between follicular adenoma and carcinoma on Bethesda type IV les
11 ar thyroid carcinomas, and 26 benign tumors (follicular adenomas and hyperplastic nodules) were analy
12 nty-six % of informative benign tumors (four follicular adenomas and three Hurthle cell adenomas) and
13 thle cell carcinomas, 21 were nonfunctioning follicular adenomas, and 9 were Hurthle cell adenomas.
14 rmalities, including multinodular goiter and follicular adenomas, and are at increased risk of thyroi
16 r two thirds of the mutant females developed follicular adenomas by 10 months of age, showing that lo
19 2 differentiated thyroid tumors including 15 follicular adenomas (FA), 13 papillary thyroid cancers (
20 e the molecular signatures of normal, benign follicular adenoma (FTA), and malignant follicular carci
21 g of significantly increased risk of thyroid follicular adenoma in a screening study of children and
23 s the (131)I-associated increases in risk of follicular adenoma in the Ukrainian population and adds
24 mean, 20.5 years; median, 20 years) than did follicular adenomas (mean, 35.3 years; median, 36.5 year
25 (N-ras codon 61 CAAgln-->AAAlys), and in 3/7 follicular adenomas (N-ras codon 61 CAAgln-->CGAarg x 2,
28 en and adolescents have an increased risk of follicular adenoma, though it is smaller than the risk o
29 denomas with LOH, 3 of 4 (75%) were atypical follicular adenomas, whereas 2 of 15 (13%) were typical