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1 flock house virus, human rhinovirus-14, and foot and mouth disease virus.
2 n, livestock can be made less susceptible to foot and mouth disease virus.
3 2A self-processing peptide derived from the foot-and-mouth disease virus.
4 ere experimentally infected via aerosol with foot-and-mouth disease virus.
5 lved in genome packaging of the picornavirus foot-and-mouth disease virus.
6 xsackievirus, rhinovirus, enterovirus 71 and foot-and-mouth disease virus.
7 ctive peptide (A20FMDV2) derived from VP1 of foot-and-mouth disease virus.
8 polymerases from poliovirus, rhinovirus, and foot-and-mouth disease viruses.
9 sh-ble antibiotic resistance gene, with the foot and mouth disease virus 2A self-cleaving sequence p
10 peptide binding and explains the ability of foot-and-mouth disease virus 3C(pro) to cleave sequences
13 on mediated a salient genome segmentation of foot-and-mouth disease virus, an important animal pathog
15 nked to the gene encoding the 2A protease of foot-and-mouth disease virus and then inserted in frame
16 henotype has been documented for poliovirus, foot-and-mouth disease virus, and coxsackievirus B3 and
17 cally important members, such as poliovirus, foot-and-mouth disease virus, and endomyocarditis virus.
18 icornavirus family, including poliovirus and foot-and-mouth disease virus, are widespread pathogens o
19 AVPNLRGDLQVLAQKVART (A20FMDV2), derived from foot-and-mouth disease virus, as a potent inhibitor of a
22 uman rhinovirus, coxsackievirus, poliovirus, foot-and-mouth disease virus, enterovirus D-68, and a wi
24 n from genetic and epidemiological data in a Foot and Mouth Disease Virus (FMDV) veterinary outbreak
31 ck in secretion are induced by expression of foot-and-mouth disease virus (FMDV) 3C(pro) and that thi
34 ished data from transmission experiments for foot-and-mouth disease virus (FMDV) and African swine fe
35 s were tested in this study: one recognizing foot-and-mouth disease virus (FMDV) and another recogniz
36 IRESs) of encephalomyocarditis virus (EMCV), foot-and-mouth disease virus (FMDV) and other picornavir
38 the extent to which the genetic diversity of foot-and-mouth disease virus (FMDV) arising over the cou
40 shown that the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) blocks cap-dependent
44 use in stabilizing SAT2 vaccines.IMPORTANCE Foot-and-mouth disease virus (FMDV) causes a highly cont
48 virus strains; however, the pathogenesis of foot-and-mouth disease virus (FMDV) coinfections is larg
51 for the differential laboratory detection of foot-and-mouth disease virus (FMDV) from viruses that ca
53 hin the RNA genome of all seven serotypes of foot-and-mouth disease virus (FMDV) has been developed.
57 ocked the replication of poliovirus (PV) and foot-and-mouth disease virus (FMDV) in a variety of cell
58 ociated with clearance versus persistence of foot-and-mouth disease virus (FMDV) in micro-dissected c
63 ole of T-lymphocyte subsets in recovery from foot-and-mouth disease virus (FMDV) infection in calves
66 ecades of investigation, the manner in which foot-and-mouth disease virus (FMDV) interacts with the i
67 shown that the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) interferes with the
68 iral vectors were constructed containing the foot-and-mouth disease virus (FMDV) internal ribosome en
70 tent infection in African buffalo.IMPORTANCE Foot-and-mouth disease virus (FMDV) is a highly contagio
73 stress response.IMPORTANCE The picornavirus foot-and-mouth disease virus (FMDV) is a notorious anima
77 One of the final steps in the maturation of foot-and-mouth disease virus (FMDV) is cleavage of the V
78 We have previously shown that replication of foot-and-mouth disease virus (FMDV) is highly sensitive
82 n the initiation of immune responses against foot-and-mouth disease virus (FMDV) is poorly understood
87 domestic animals with chemically inactivated foot-and-mouth disease virus (FMDV) is widely practiced
89 dentified an aromatic hydrophobic residue in foot-and-mouth disease virus (FMDV) leader proteinase (L
93 eta interferon [IFN-alpha/beta]) can inhibit foot-and-mouth disease virus (FMDV) replication in cell
94 and II IFNs have proven effective to inhibit foot-and-mouth disease virus (FMDV) replication in swine
95 g a comparative analysis, of 103 isolates of foot-and-mouth disease virus (FMDV) representing all sev
100 ents from human rhinovirus type 2 (HRV2) and foot-and-mouth disease virus (FMDV) to control the trans
101 previously demonstrated that the ability of foot-and-mouth disease virus (FMDV) to form plaques in c
105 The development of a serological test for foot-and-mouth disease virus (FMDV) which is quick and e
108 the integrin receptors on cultured cells for foot-and-mouth disease virus (FMDV), and high-efficiency
110 ily, and for several diverse species such as foot-and-mouth disease virus (FMDV), hemagglutinin (HA)
116 " We show that the key replication enzyme of foot-and-mouth disease virus (FMDV), the RNA-dependent R
117 s of representatives of several serotypes of foot-and-mouth disease virus (FMDV), we discovered a put
119 mutants were used to map antigenic sites on foot-and-mouth disease virus (FMDV), which resulted in t
120 BP1) was documented as an essential ITAF for foot-and-mouth disease virus (FMDV), with no apparent ro
133 , we apply the method to two UK epidemics of Foot-and-Mouth Disease Virus (FMDV): the 2007 outbreak,
137 ting cross-neutralization between serotype O foot-and-mouth disease viruses (FMDVs) is critical for g
139 n a 4H junction derived from domain 3 of the foot-and-mouth disease virus internal ribosome entry sit
140 (6) and 2 x 10(7) c.f.u./ml, indicating that foot-and-mouth disease virus IRES provides high-titer bi
142 a vector with a multiple cloning site 3' to foot-and-mouth disease virus IRES, was used to construct
143 of the O(1) British field strain serotype of foot-and-mouth disease virus is a high-affinity ligand f
146 from high affinity ligands of alpha v beta6 (foot-and-mouth-disease virus, latency associated peptide
147 ar to those of core catalytic domains of the foot-and-mouth disease virus leader protease and coronav
148 liovirus (n=3 each, 18.8%); Brucella spp and foot and mouth disease virus (n=2 each, 12.5%); and vari
149 we use these methods to analyze data from a foot-and-mouth disease virus outbreak in the United King
150 aches, we investigated how highly contagious foot-and-mouth disease viruses persist in the African bu
151 the prominent G-H loop of the VP1 protein of foot-and-mouth disease virus, raised substantial levels
152 nal structure of the corresponding domain of foot-and-mouth disease virus, revealing an analogous dom
153 The larger picornavirus IRESs (those of foot-and-mouth disease virus, rhinovirus, encephalomyoca
154 Venus and a puromycin-resistant gene via the foot-and-mouth disease virus self-cleaving peptide T2A.
155 odels to predict the antigenic similarity in foot-and-mouth disease virus strains and in influenza st
156 20FMDV2 is a 20-mer peptide derived from the foot-and-mouth disease virus that exhibits nanomolar and
157 naviruses (e.g., poliovirus, rhinovirus, and foot-and-mouth disease virus), the capsid precursor prot
158 ar viruses, including all seven serotypes of foot-and-mouth disease virus, two serotypes of vesicular
159 opy of the genome-linked protein, VPg wheras foot-and-mouth disease virus uniquely encodes three copi
160 imal pathogens: classical swine fever virus; foot-and-mouth disease virus; vesicular stomatitis virus
161 s in innate responses against infection with foot-and-mouth disease virus was analyzed on consecutive