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1 plasmic membrane of the organism Oxalobacter formigenes.
2 he oxalate/formate antiporter of Oxalobacter formigenes.
3 transporting membrane protein in Oxalobacter formigenes.
4 :formate exchange transporter of Oxalobacter formigenes.
5 late/formate exchange protein of Oxalobacter formigenes.
6 rt, by the intestinal bacterium, Oxalobacter formigenes.
7 6%) of 43 CF patients were colonised with O. formigenes.
8 d, was cloned from the bacterium Oxalobacter formigenes.
9 proton-motive metabolic cycle in Oxalobacter formigenes.
10 wed even greater sensitivity in detecting O. formigenes and provided support for further division of
11    We investigated whether the absence of O. formigenes and the presence of hyperoxaluria are correla
12                                  Oxalobacter formigenes are bacteria that colonize the human gut and
13 uman anaerobic colonic bacterium Oxalobacter formigenes because of its ability to metabolize oxalate,
14 aged healthy volunteers were examined for O. formigenes by culture and DNA analysis.
15  our understanding of the epidemiology of O. formigenes carriage, and are consistent with the hypothe
16 retion in hyperoxaluric mice treated with O. formigenes CM reflects the in vivo retention of biologic
17 oxaluria type 1, rectal administration of O. formigenes CM significantly reduced (>32.5%) urinary oxa
18             Compared with control medium, O. formigenes CM significantly stimulated oxalate uptake (>
19                              Treating the O. formigenes CM with heat or pepsin completely abolished t
20 ntified significant antibiotic effects on O. formigenes colonization and urinary electrolytes and sho
21                                Intestinal O. formigenes colonization has been associated with a lower
22 s associated with the progressive loss of O. formigenes colonization in populations that have been hi
23 as no specific therapy, although Oxalobacter formigenes colonization is associated with reduced stone
24  and epidemiological studies suggest that O. formigenes colonization reduces the risk for kidney ston
25            The difficulties in sustaining O. formigenes colonization underscore the need to identify
26 ss the effects of an antibiotic course on O. formigenes colonization, urine electrolytes, and the com
27 ot differ with the presence or absence of O. formigenes colonization.
28 ent kidney stone episodes and the lack of O. formigenes colonization.
29 embling the human microbiome, with stable O. formigenes colonization; such models can define optimal
30   It was hypothesized that the absence of O. formigenes could lead to increased colonic absorption of
31     We therefore evaluated the effects of O. formigenes culture conditioned medium (CM) on apical (14
32                          We conclude that O. formigenes-derived bioactive factors stimulate oxalate t
33 trafiltration of the CM revealed that the O. formigenes-derived factors have molecular masses of 10-3
34                         The prevalence of O. formigenes, determined by stool culture, was 17% among c
35  microbes, an oxalate autotroph, Oxalobacter formigenes, dominates this function transcriptionally.
36 obligate anaerobe Oxalobacter formigenes, O. formigenes formyl coenzyme A transferase (FRC).
37                                Absence of O. formigenes from the intestinal tract of CF patients appe
38      All seven CF patients colonised with O. formigenes had normal urinary oxalate levels, but 19 (53
39 he oxalate:formate antiporter of Oxalobacter formigenes, has a lone charged residue, lysine 355 (Lys-
40 mportance of the microbiome, particularly O. formigenes in host oxalate homeostasis.
41              To confirm the importance of O. formigenes in regulating hyperoxaluria, laboratory rats
42                              Detection of O. formigenes in six of these seven patients required DNA-b
43 elop murine models to allow investigating O. formigenes in the context of its native human microbiome
44                              Detection of O. formigenes in the gastrointestinal tract has attracted a
45 les from an individual not colonized with O. formigenes, indicating unique specificity.
46                                           O. formigenes interacts with colonic epithelium and induces
47   Transplanting mice with human feces and O. formigenes introduced new microbial populations resembli
48                                  Oxalobacter formigenes is a Gram-negative, anaerobic bacterium that
49                                  Oxalobacter formigenes is a specific oxalate-degrading, anaerobic ba
50                                  Oxalobacter formigenes is an obligate anaerobe that colonizes the hu
51 se results suggest that colonization with O. formigenes is associated with a 70% reduction in the ris
52                      The role of Oxalobacter formigenes is herein discussed.
53  taken together, support the concept that O. formigenes is important in maintaining oxalate homeostas
54 he oxalate/formate antiporter of Oxalobacter formigenes, lysine 355 is within transmembrane helix no.
55 antly differed in Beta-diversity from the O. formigenes-negative samples.
56 abolism in the obligate anaerobe Oxalobacter formigenes, O. formigenes formyl coenzyme A transferase
57 e mammalian intestinal bacterium Oxalobacter formigenes ( Oxf).
58       In subjects not on antibiotics, the O. formigenes-positive samples had higher alpha-diversity a
59    Serial fecal samples were examined for O. formigenes presence and microbiota characteristics.
60 ructure differed in subjects according to O. formigenes presence.
61                                           O. formigenes prevalence was 50%.
62 e multivariable model, the odds ratio for O. formigenes remained 0.3 (95% confidence interval 0.1 to
63                  As expected, the lack of O. formigenes revealed a clear association with prophylacti
64                                 Now using O. formigenes-specific PCR, we have compared the prevalence
65 ed human donors supplemented with a human O. formigenes strain into recipient mice.
66 quences of oxc or frc were able to divide O. formigenes strains into at least two groups, consistent
67 consistent with the current separation of O. formigenes strains into groups I and II on the basis of
68 onization; such models can define optimal O. formigenes strains to facilitate clinical trials.
69 th detection as well as classification of O. formigenes strains.
70 humanized mice were stably colonized with O. formigenes through 8 weeks after gavage, whereas mice re
71 of oxalate-degrading enzymes derived from O. formigenes to determine any subsequent increased resista
72 intained in the gnotobiotic facility with O. formigenes, using either a laboratory isolate or an isol
73 s in Venezuela and the Hadza in Tanzania, O. formigenes was detected in 60-80% of the adult subjects,
74 and the CF patient with normal numbers of O. formigenes was the only one of the 43 patients who had n
75 late:formate exchange protein of Oxalobacter formigenes, was established by site-directed fluorescenc
76 he oxalate-degrading specialist, Oxalobacter formigenes, was only effective against a poor oxalate-de
77 /formate exchange transporter of Oxalobacter formigenes, was purified as a histidine-tagged variant,
78 9 (53%) of 36 patients not colonised with O. formigenes were hyperoxaluric, with the most severe hype
79 nomic DNAs of various strains of Oxalobacter formigenes were subjected to restriction endonuclease fr