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1 s with activated c-Myc after single-dose and fractionated radiation.
2 the presence of clinically relevant doses of fractionated radiation.
3      There was consensus that conventionally fractionated radiation (1.8-2 Gy daily) to a dose of 45
4 ed between arm A (the control), single daily fractionated radiation (70 Gy at 2 Gy/d); arm B, identic
5 ombination treatment of in vivo tumours with fractionated radiation and a blocking antibody to FGF2 p
6 liferation does not diminish the efficacy of fractionated radiation and suggest that disruption of ke
7 3; and arm C, a split course of single daily fractionated radiation and three cycles of concurrent in
8 FC for 12 days, and tumors were treated with fractionated radiation at a dose of 5 Gy/day to a total
9 cervical carcinomas in patients suggest that fractionated radiation can lower tumor IFP and increase
10                                          The fractionated radiation combined with ATO showed a simila
11                                 Furthermore, fractionated radiation delivered to one tumor-bearing lu
12 effects are observed in both single-dose and fractionated radiation experiments.
13 oundation for clinical testing of sequential fractionated radiation followed by sorafenib in gastroin
14 al of breast cancer cells treated with hyper-fractionated radiation (HFR), which is used clinically f
15 on would yield equivalent profiles following fractionated radiation in different tumor cell lines.
16 ced in the tumour microenvironment following fractionated radiation in murine tumours consistent with
17 diosensitive than normal cells, but only for fractionated radiation in plateau phase.
18 rate that TPZ potentiates tumor cell kill by fractionated radiation in three murine tumors (SCCVII, R
19 ase inhibitor, also enhanced the efficacy of fractionated radiation in U87 spheroids.
20 hibitors of the mevalonate pathway prevented fractionated-radiation-induced suppression of KLF2, TM,
21 odels of radioresistance through exposure to fractionated radiation is an increasingly used approach
22                                      Because fractionated radiation is routinely used during cancer r
23                                              Fractionated radiation lowered IFP by 2.5 mm Hg when the
24 mycin significantly enhanced the efficacy of fractionated radiation of established U87 xenografts in
25                 Compared to single exposure, fractionated radiation profoundly suppressed KLF2, TM, a
26 ografts grown in nude mice were exposed to a fractionated radiation protocol, which resulted in a sig
27 ithin 2 years of diagnosis, and conventional fractionated radiation remains the standard treatment.
28 agent cisplatin to conventional single daily fractionated radiation significantly improves survival,
29 the third week postinjury, with protocols of fractionated radiation similar to those for treating hum
30 of monoclonal antibodies following single or fractionated radiation that has been reported in the lit
31 breast or head and neck cancer and plans for fractionated radiation therapy (>=15 fractions) with cur
32 ly hypofractionated (H-RT) or conventionally fractionated radiation therapy (C-RT) in a national coho
33  to III NSCLC requiring a definitive dose of fractionated radiation therapy (RT) were eligible.
34            This is being termed as Spatially Fractionated Radiation Therapy (SFRT) and lattice therap
35 d to decrease acute and late xerostomia with fractionated radiation therapy alone for head and neck c
36  2 years have reported the efficacy of using fractionated radiation therapy alone.
37   Intetumumab can potentiate the efficacy of fractionated radiation therapy in human cancer xenograft
38 r model was used to study tumor responses to fractionated radiation therapy in mice with anemia induc
39 ection of radioresistant cancer cells during fractionated radiation therapy may have implications in
40 n Therapy is a preclinical form of spatially fractionated radiation therapy that utilizes synchrotron
41       The effect of DA on tumor responses to fractionated radiation therapy was observed when DA was
42                     Following craniotomy and fractionated radiation therapy with concurrent TMZ, pati
43                    We test a hypothesis that fractionated radiation therapy within a therapeutic dose
44 activation and repopulation, as occur during fractionated radiation therapy, can lead to distribution
45                           When combined with fractionated radiation therapy, DA increased the tumor g
46                           When combined with fractionated radiation therapy, intetumumab significantl
47 s an important rationale for the widely used fractionated radiation therapy.
48 tumor response during standard clinical dose-fractionated radiation therapy.
49 or-bearing mice and sensitize tumor cells to fractionated radiation therapy.
50                                We found that fractionated radiation to the thorax significantly reduc
51 c division or a fast cycle of GSCs following fractionated radiation treatment is required to yield re
52  this study was to investigate the effect of fractionated radiation treatment, given in daily doses s
53  prostate cancer cells rendered resistant to fractionated radiation treatment.
54                        In mice that received fractionated radiation, tumors were less invasive, with